DERMATOLOGY: NCLEX REVIEW I Flashcards

1
Q

CELLULITIS

  1. S/S
  2. caused by
A
  • red
  • warm
  • tender
  • painful
  • swollen
  • skin glossy appearance
  • fever maybe present

Streptococcus pyogenes or staphylooccus

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2
Q

Strep pyogenes also causes

A
  • strep throat
  • rheumatic fever
  • necrotizing fascitis and glomerulonephrits
    *
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3
Q

Staph aureus cause

A

meningitis, osteomyelitis

sepsis

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4
Q

Pseudomos aeruginosa causes

A
  • UTI
  • atypical pneumonia
  • infections in burns
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5
Q

Wet to dry dressing aids in

Promotes healing by __?__ intention

A

debridement of exudate and necrotic tissue

Secondary Intention

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6
Q

Primary Intention Wound Healing

Healing is outside to in

A
  • sutures
  • glue
  • staples
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7
Q

Secondary Intention means

A

wound heals from the inside-out

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8
Q

Tertiary Intention

A

wound cleaned and left open for several days for observation

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9
Q

Hydrocolloids Dressings fxn

A

minimize discomfort and prevent contamination of microbes

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10
Q

BURNS

Prioritize (4)

A
  1. Airway
  2. Restoring hemodynamic stability
  3. Initiating fluid rescuscitation
  4. Infection prevention throughout treatment
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11
Q

“Hemodynamic instability”

http://www.virtualmedstudent.com/links/cardiovascular/hemodynamically_unstable_patient.html

A

First off, what is “hemodynamic instability”? Hemodynamics is the study of blood movement; when this movement is compromised you get hemodynamic instability. If left untreated, it will cause multi-organ failure and death.

In essence, you can think of hemodynamic instability as the collapse of the cardiovascular system. This collapse is manifested clinically by a significant drop in blood pressure.

There are numerous causes of hemodynamic instability, and the beauty is that you do not need to “know” all of them to manage a patient who is acutely experiencing cardiovascular collapse. A few basic tenets of physiology, when examined in the right order, will help you manage a patient who is unstable.

The tenets are as follows:

(1) Preload (volume status)
(2) Afterload (systemic vascular resistance)
(3) Heart rate
(4) Heart rhythm
(5) Contractility

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12
Q

After a burn injury

Increase/Decrease

  • Hematocrit
  • Heart Rate
  • K levels
  • Lactic Acid
A
  • Hematocrit: increase due to blood concentration
  • Heart Rate: increase due to hypovolemia associated with third spacing
  • K levels: Increase due to cellular destruction
  • Lactic Acid: increase due to impaired tissue perfusion
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13
Q
  • Contact dermatitis rash is caused by__?__
  • Treatment (2)
A
  • contact with an irritant
  • Cool wet dressing OTC oral antihistamine
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14
Q

Stage I Pressure Ulcer

An erythematous lesion that loses all redness when pressed is termed “blanchable.”

Blanchable lesions are due to vascular dilatation. Non-blanchable, erythematous lesions are due to the presence of red blood cells outside of blood vessels (extravasation).

A
  • Skin intact
  • red
  • non blanching
  • warm
  • Painful
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15
Q

Stage II Pressure Ulcers

A
  • Skin is not intact
  • loss of the dermis occurs
  • pink/red
  • open wound shallow
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16
Q

Stage III Pressure Ulcers

A
  • Full thickness skin loss
  • extends into the dermis and subq tissue
  • slough and tunneling may be present
17
Q

Stage IV Pressure Ulcer

A
  • Full thickness skin loss
  • exposed bone, tendon, or muscle
  • slough or escar
  • tunneling