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Family Medicine EOR > Dermatology 5% > Flashcards

Dermatology 5% Flashcards

1
Q

lesh-colored, pink or yellow-brown lesion with a rough sandpaper feel
Occurs on sun-exposed surfaces and is a precursor to squamous cell carcinoma

A

Actinic keratosis

Treat with observation (many resolve on their own), cryosurgery, 5 FU cream, electrodesiccation or Imiquimod

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2
Q

autoimmune - attack against hair follicles. Onset usually prior to 30 years of age; men and women are equally affected. Well-documented genetic predisposition.

A

Alopecia areata

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3
Q

Scalp disorder characterized by the thinning or shedding of hair resulting from the early entry of hair in the telogen phase (the resting phase of the hair follicle)

A

Telogen effluvium

Alopecia is preceded by a psychologically or physically stressful event 6–16 weeks prior to the onset of hair loss.

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4
Q

white waxy lump or a brown scaly patch, raised pearly and rolled borders, telangiectasis, a central ulcer on sun-exposed areas, such as the face and neck

A

BCC
basal cells
Basal cells produce new skin cells as old ones die. Limiting sun exposure can help prevent these cells from becoming cancerous

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5
Q

How to treat BCC?

A

fluorouracil (FU) and imiquimod, photodynamic therapy (PDT), and surgical excision with clear margins

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6
Q

autoimmune subepidermal blistering skin disorder caused by linear deposition of autoantibodies (IgG) against hemidesmosomes in the epidermal-dermal junction

(-ve) Nikolsky sign

A

Bullous pemphigoid is less severe than pemphigus vulgaris, does not affect mucous membranes and has a negative Nikolsky sign

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7
Q

Dx of bullous pemphigoid

A

Diagnosis is made by skin biopsy with direct immunofluorescence exam shows deposition of IgG and C3 basement membrane

Treat with systemic corticosteroids

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8
Q

large number of pruritic, tense, subepidermal bullae across her upper thighs. There was no mucosal involvement and skin biopsy showed subepidermal bullae filled with eosinophils and neutrophils.

A

Bullous pemphigoides

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9
Q

treatment for bullous pemphigoides

A

High-potency topical corticosteroids (eg, clobetasol 0.05% cream) should be used for localized disease and may reduce the required dose of systemic drugs.

Prednisolone (an anti inflammatory agent) and azathioprine (an immunosuppressant) can also be used in the treatment of bullous pemphigoid.

nicotinamide is used in combination with tetracycline or erythromycin to treat bullous pemphigoid

Dapsone Is particularly effective in mucous membrane lesions.

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10
Q

Acute bacterial skin and skin structure infection of the dermis and subcutaneous tissue;
characterized by pain, erythema, warmth, and swelling. Margins are flat and not well demarcated.

Caused by Staphylococcus and Streptococcus in adults

H. influenzae or strep pneumonia in children

A

Cellulitis

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11
Q

Mild cellulitis treatment MSSA

A

Cephalexin or Dicloxacillin

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12
Q

How to treat MRSA cellulitis?

A

Trimethoprim-sulfamethoxazole (TMP-SMZ) 1 DS tab PO BID
Clindamycin 300–450 mg PO
Doxycycline 100 mg PO BID

IV Vancomycin or Linezolid

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13
Q

an indication for workup in patients with suspected cellulitis?

A

Tachypnea

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14
Q

Flesh-colored, cauliflower appearance genital warts caused by HPV types 6 and 11

A

Condyloma acuminatum (also known as genital warts or anogenital warts) refers to an epidermal manifestation attributed to the epidermotropic human papillomavirus (HPV)

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15
Q

well-demarcated erythema, erosions, vesicles

A

contact dermatitis

Allergic: Type 4 hypersensitivity
Nickel, poison ivy

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16
Q

Tx for contact dermatitis?

A

Avoid offending agent. Burrow’s solution (aluminum acetate), topical steroids, zinc oxide (diaper rash)

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17
Q

Pruritic, eczematous lesions, xerosis (dry skin), and lichenification (thickening of the skin and an increase in skin markings). Most common on flexor creases (ex. antecubital and popliteal folds)

A

Atopic dermatitis

IgE, Type 1 hypersensitivity

TX with:
Topical corticosteroids and emollients, topical calcineurin inhibitor (ie, tacrolimus or pimecrolimus)

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18
Q

Coin-shaped/disc-shaped

Treat with?

A

Nummular eczema

Tx: High- or ultra-high potency topical corticosteroids, phototherapy, systemic corticosteroids, methotrexate, cyclosporine

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19
Q

Erythematous, yellowish greasy scales, crusted lesions.

Infants- scalp (cradle cap)
Adults/adolescents- body folds

A

Seborrheic dermatitis

Scalp: antifungal shampoo

Face: low potency steroid cream

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20
Q

Treatment for seborrheic dermatitis?

A

Ketoconazole shampoo

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21
Q

What is treatment for perioral dermatitis?

A

Topical metronidazole
erythromycin

Topical steroids are contraindicated as they may cause flare of lesions

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22
Q

Acrylates and methacrylates have been significantly associated with contact allergy and allergic contact disease
T/F

A

True
acrylic nail sources and wound dressings represent emerging sources of sensitization. A separate study found that acrylates and methacrylates were significantly associated with allergic contact dermatitis.

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23
Q

Potassium hydroxide preparation or fungal culture is often indicated to exclude tinea in dermatitis of the hands and feet
T/F

A

T
Potassium hydroxide preparation and/or fungal culture to exclude tinea are often indicated for dermatitis of the hands and feet. This helps identify disorders such as tinea pedis.

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24
Q

why Patients should avoid using topical antihistamines, including topical doxepin w/ contact dermatitis?

A

because of the risk for iatrogenic allergic contact dermatitis to these agents; additionally, sedation can occur if large amounts of doxepin cream are applied.

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25
Q

Shorter courses of corticosteroids may allow poison ivy dermatitis to relapse
T/F

A

True

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26
Q

what is contraindicated as treatment for genital warts in pregnancy?

A

Podophyllum resin

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27
Q

Condylomata acuminata is caused by

A

HPV

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28
Q

Young women. Papulopustular, plaques, and scales around the mouth.

Treatment

A

Perioral dermatitis

Treatment: Topical metronidazole, avoid steroids

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29
Q

What is the correct dosage of epinephrine for the actute treatment of anaphylaxis?

A

epinephrine 0.2–0.5 mg (1:1000 [1 mg/mL] solution) IM every 5–15 min prednisone may be given to prevent recurrence.

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30
Q

a pruritic vesicular eruption comprised of clear, deep-seated vesicles without erythema erupting on the lateral aspects of fingers, the central palm, and plantar surfaces.

Tapioca pudding

A

dyshidrosis

Tapioca vesicles on hands and feet following stress or hot humid weather

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31
Q

1st line treatment for dyshidrosis

A

Topical HIGH STRENGTH steroids and cold compress

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32
Q

raised, edematous, circumscribed, hot, erythematous area, with or without vesicles or bullae frequently involving the central face or lower extremity.

A

Erysipelas

is a type of superficial cellulitis with dermal lymphatic involvement
Looks like cellulitis but it is well-demarcated and caused by group A strep (strep pyogenes)
Must rule out MRSA

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33
Q

primary etiological agent of erysipelas is

A

Beta-hemolytic streptococcus

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34
Q

MILD Erysipelas Treatment?

A

Mild can be treated with Penicillin G

Patients with allergy to penicillin can be treated with erythromycin or clindamycin

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35
Q

Moderate erysipelas Treatment>

A

Trimethoprim-sulfamethoxazole (TMP-SMX)-DS: 1–2 tablets PO BID AND penicillin VK 500 mg PO QID

or

cephalexin 500 mg PO QID

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36
Q

Severe erysipelas treatment?

A

IV and linezolid 600 mg IV/PO BID or vancomycin IV or daptomycin 4 mg/kg IV q24h.

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37
Q

Skin lesions predominantly involving the extremities (hands, feet, and mucosa). Target-like shape, raised, blanching, and lack of itchiness help characterize this rash.

A

Erythema multiforme (EM) is an acute, self-limited, and sometimes recurring skin condition that is considered to be a type IV hypersensitivity reaction

ASsociated with:
HSV

Sulfa drugs

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38
Q

The most common cause of erythema multiforme minor is

A

HSV

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39
Q

EM major (involvement of mucous membranes and systemic signs)

A

Symptomatic treatment with oral antihistamines and topical corticosteroids for mild cases; mouthwashes or topical steroid gels for oral disease.

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40
Q

EM minor (no mucous membrane involvement and no systemic signs) is usually self-limited

A

Supportive care
Early treatment with acyclovir may lessen the number and duration of cutaneous lesions for patients with coexisting or recent HSV infection.

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41
Q

The child has had a low-grade fever, headache, and sore throat for the past week. Four days ago, he suddenly developed a bright red rash on his cheeks, which during the past 2 days has spread to the trunk, arms, and legs.

DX

A

5th disease

MCC: Parvovirus-B19

Diagnosis is based primarily on clinical observations, history, and physical exam

Serology: associated with ENLARGED nuclei with peripherally displaced chromatin
PARVO B19-specific IgM antibodies and PCR

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42
Q

Erythema

Migrans

A

Lyme disease

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43
Q

Erythema MARGINATUM

A

Rheumatic fever

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44
Q

Erythema NODOSUM

A

Mono

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45
Q

Erythema

Infectiosum

A

Parvovirus-B19 ;

5th disease

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46
Q

MCC of hand foot mouth disease?

A

Coxsackie VIRUS type A

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47
Q

children< 10 years old with vesicles on pharynx, mouth, hands, feet

sores in the mouth and a rash on the hands, feet, mouth, and buttocks

A

hand foot mouth disease

The virus usually clears up on its own within 10 days
Treatment is supportive, anti-inflammatories

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48
Q

The 4 C’s - cough, coryza, conjunctivitis and cephalocaudal spread

A

Measles (Rubeola)

49
Q

Morbilliform - maculopapular, brick red rash on face beginning at hairline then progressing to palms and soles last - rash lasts 7 days

A

Measles (Rubeola)

50
Q

Koplik spots (small red spots on buccal mucosa with blue-white pale center) precedes rash by 24-48 hours.

A

Measles (Rubeola)

51
Q

Cephalocaudal spread of maculopapular rash, lymphadenopathy (posterior cervical, posterior auricular)

A

Rubella

52
Q

Cephalocaudal spread of maculopapular rash, lymphadenopathy (posterior cervical, posterior auricular)

A

Rubella

Teratogenic in 1’st trimester - congenital syndrome - deafness, cataracts, TTP, mental retardation

53
Q

How to differentiate between rubEOLA
vs
rubELLA?

A

Rubella rash =spread is much more rapid, and the rash does not darken or coalesce

54
Q

Herpesvirus 6 or 7, only childhood exanthem that starts on the trunk and spreads to the face

A

Roseola
(6th disease)

High fever 3-5days
then rose pink maculopapular
BLANCHABLE rash on
Trunk–>then face

55
Q

The lesions are erythematous papules or pustules. They are usually not painful but may burn

A

Folliculitis

56
Q

MCC of folliculitis

MCC of hot tub folliculitis

A

S.aureus

Pseudomonas

57
Q

Treatment for folliculitits?

A

1st line:
–Mupirocin ointment

—Topical benzoyl peroxide

58
Q

Severe folliculitis treatment?

MRSA:

A

PO ABX: DICLO-xacillin + CEPHALExin

MRSA tx TMP-SMX,
Clinda
or
Doxy

59
Q

Chronic follicular occlusive disease manifested as recurrent inflammatory nodules, abscesses, sinus tracts, and complex scar formation

A

Hidradenitis suppurativa

Pea- to marble-sized nodules under the skin that can be painful and tend to enlarge and drain pus.

They usually occur where skin rubs together, such as in the armpits, groin, and buttocks.

60
Q

Hidradenitis suppurativa

Treatment of

A

Intralesional triamcinolone is 1’st line treatment

Oral and topical antibiotics, hygiene, warm soaks, and sometimes surgery can help manage symptoms

61
Q

A highly contagious skin infection that causes red sores on the face

The main symptom is red sores that form around the nose and mouth. The sores rupture, ooze for a few days, then form a yellow-brown crust
“honey-colored” and weeping

A

Impetigo

62
Q

MCC impetigo?

Treatment

Complication for impetigo?

A

S.aureus

Treatment is topical mupirocin, dicloxacillin, cephalexin for more severe illness

Complications: poststreptococcal glomerulonephritis

63
Q

“grains of sands” on a red base on the buccal mucosa opposite the second molars.

Complication of measles x3

A

Rubeolla Koplik spot

Measles

  • Encephalitis
  • Bronchopneumonia
  • Otitis media
64
Q

First sign of measles observed?x2

A

High fever >104F + Koplik spots

65
Q

the quickest method of confirming acute measles?

A

capture immunoglobulin M (IgM) antibody assay

66
Q

supplementations has been associated with reductions in morbidity and mortality in patients with measles?

A

Vitamin A

67
Q

Purple, red, or brown skin blotches are a common sign.

lesions to grow in the skin, lymph nodes, internal organs, and mucous membranes lining the mouth, nose, and throat. It is associated with human herpesvirus 8 and is an AIDS-defining cancer

A

Kaposi sarcoma

Treatment may include radiation or chemotherapy. Rarely, surgery may be needed

68
Q

Pruritic scalp, body or groin. Nits are observed as small white specs on the hair shaft
Body (corporis); Pubic (pubis)

A

LICE

Treat with permethrin 1%

For resistant cases consider oral ivermectin

Screen for other STIs in patients with pubic lice - abstain from sexual contact until the infestation clears

69
Q

purplish, itchy, flat-topped bumps. On mucous membranes, such as in the mouth, it forms lacy white patches, sometimes with painful sores

Clinically characterized by 5Ps purple, papule, polygonal, pruritus, planar

Wickham striae: whitish lines visible in the papules of lichen planus and other dermatoses

A

Lichen planus (LP)is a chronic papulosquamous inflammatory dermatosis of unknown etiology, probably autoimmune in origin

–Treatment: Topical steroids

70
Q

a chronic dermatitis resulting from chronic, repeated rubbing or scratching of the skin. Skin becomes thickened with accentuated lines (“lichenification”). The constant scratching causes thick, leathery, brownish skin

Treatment?

A

Lichen simplex chronicus

Treatment: Break the itch-scratch cycle (anti-histamines, occlusive dressing)

71
Q

benign fatty tumors, generally slow-growing, and usually harmless

just under the skin and move easily when pressure is applied. They commonly occur in the neck, shoulders, back, abdomen, arms, and thighs
If the presenting lesion is fast-growing, suspect another diagnosis

A

Lipomas

Treatment generally isn’t necessary, but if the lipoma is bothersome, painful, or growing, surgical excision or liposuction may be needed

72
Q

Painless:
solitary, soft, well defined, mucin-filled lesions

Characteristically, they have a smooth surface and a small opening to the surface of the skin, known as a punctum

A

The term epidermal inclusion cyst refers specifically to an epidermoid cyst that is the result of the implantation of epidermal elements in the dermis

Treatment includes close observation of the tumor in asymptomatic cases and surgical management, if necessary

73
Q

Usually a pigmented lesion with an irregular border, irregular surface, or irregular coloration

Asymmetrical, unevenly pigmented patch/plaque with a nodule and an irregular border

A

Melanoma occurs when the pigment-producing cells that give color to the skin (melanocytes) become cancerous

74
Q

What is prognosis of melanoma strongly associated with?

A

Prognosis of melanoma is most strongly associated with the depth of the lesion, based on the Clark Classification System of Microstaging

75
Q

Clark Classification System of Microstaging

Level I to V

A

I–> confined to epidermis

II–> papillary dermis

III: papilary reticular interface

IV: reticular dermis

V: subQ fat

76
Q

Treatment of melanoma?

A

Treatment may involve Mohs surgery, radiation, medications, or in some cases chemotherapy

77
Q

Skin condition due to ↑ in estrogen during pregnancy or from sun exposure

Appear as dark, irregular, well-demarcated macules/patches,

A

Melasma

Also known as chloasma = “mask of pregnancy”

In women, melasma often fades on its own after pregnancy or after an affected woman goes off birth control pills

78
Q

Melasma treatment?

A

Treat with sunscreen and topical hydroquinone (bleaching agent)

79
Q

caused by the poxvirus, pearly papules with central umbilication

A

Molluscum contagiosum
MCC-> POXVIRUS

Treatment: The bumps usually disappear on their own

May be removed with IMIQUIMOD (Aldara)

curettage, cryotherapy, or acid or exfoliative peel - tretinoin,

80
Q

WORKUP for ONYCHOMYCOSIS

Treatment?

A

Fungal culture +
KOH prep

Terbinafine x6weeks for fingernails

12 for toenails

81
Q

What to monitor with antifungal regimens like terbinafine?

A

LFTs

82
Q

Superficial inflammation of the lateral and posterior folds of skin surrounding the fingernail or toenail

Caused by candida if chronic

and

staph aureus if acute

A

Paronychia

83
Q

Paronychia
Treatment:

Without abscess formation

A

topical antibiotics and warm water or antiseptic soaks (eg, chlorhexidine, povidone-iodine)

84
Q

When is paronchyia needs Emperic oral ABX?

A

in more severe cases: An antistaphylococcal agent such as dicloxacillin (250 mg four times daily) or cephalexin (500 mg three to four times daily) is appropriate first-line therapy

85
Q

Paronychia with abscess

A

I & D

tetanus booster prn

86
Q

An abnormal skin growth located at the tailbone that contains hair and skin
Will usually present as a teenager with pain, discomfort and swelling above the anus or near the tailbone that comes and goes
Often includes drainage of pus or blood

A

Pilonidal disease

Treatment involves drainage and surgical removal of the cyst - look for sinus tract

87
Q

Herald patch: Large oval plaque with central clearing and scaly border. 1st sign

A

pityriasis rosea

Pruritic erythematous plaque with central scale in Christmas tree pattern on the trunk

Langer’s lines (cleavage lines) in a Christmas tree-like pattern.

88
Q

The rash consists of oval scaly lesions that line up along skin folds (Langer lines); it often resembles a “CHRISTMAS TREE” distribution

Pityriasis rosea is self-limiting and usually lasts for 3-8 weeks and disappears spontaneously

A

The cause is unknown but is thought to be viral. It is thought to be caused by herpesvirus 7

89
Q

How to treat pityriasis rosea?

A

The disease is self-limiting: topical or systemic steroids and antihistamines are often used to relieve itching. Asymptomatic lesions do not require treatment

90
Q

erythema of localized area, usually non-blanching over the bony surface

A

Stage I pressure ulcer aggressive preventive measures, thin-film dressings for protection

91
Q

—partial loss of dermal layer, resulting in pink ulceration

–full dermal loss often exposing subcutaneous tissue and fat

A

Stage 2
occlusive dressing to maintain healing, transparent films, hydrocolloids
Stage 3

92
Q

full-thickness ulceration exposing bone, tendon, or muscle. Osteomyelitis may be present

A

Stage 4 Debridement of necrotic tissue. Exudative ulcers will benefit from absorptive dressings such as calcium alginates, foams, hydrofibers. Dry ulcers require occlusive dressing to maintain moisture, including hydrocolloids, and hydrogels.

93
Q

a well-demarcated, erythematous plaque with silvery scaling. Patients may also present with no rash and only joint symptoms - pain in both hands and nail changes such as pitting and onycholysis

A

Psoriasis is an immune-mediated disease that causes raised, red, scaly patches to appear on the skin

94
Q

What is Auspitz sign?

and
Kobner’s phenomenon associated with?

A

Psoriasis Auspitz sign (bleeds when the scale is picked), Koebner’s phenomenon (minor trauma causes new lesion)
Psoriasis Vulgaris: most common. Noted on extensor surfaces
Guttate Psoriasis: children, after URI. small lesions
Inverse Psoriasis: intertriginous areas.
Pustular Psoriasis: contains pustules

95
Q

How to treat psoriasis?

Mild

Severe

A

Topical steroids

Vitamin D analogs

when severe, methotrexate, oral retinoids, immunomodulatory agents (biologics), or immunosuppressants.

96
Q

How to Dx psoriasis?

A

Diagnosis is based on appearance and distribution of lesions.

Biopsy is confirmatory and will be consistent with Plaque psoriasis

Elevated: ESR + Serum uric acid levels

97
Q

women aged 30-50,

facial erythema, telangiectasias, papules, may cause rhinophyma.

Triggers include heat, alcohol, spicy foods

A

Rosacea
Differentiate from acne by lack of comedones (blackheads)

treat with topical metronidazole

98
Q

Pruritic papules. S-shaped or linear burrows on the skin. Often located in web spaces of hands, wrists, waist with severe itching (worse at night)

what is the common site?

A

SCABIES

WRist is common site

99
Q

How to treat scabies?

A

Treat with topical permethrin 5%, all clothing bedding, towels washed and dried using heat and have no contact with body for at least 72 hours

Oral ivermectin

100
Q

waxy “stuck-on” appearance.Most common benign skin tumor seen in fair-skinned elderly patients with prolonged sun exposure
Brown, black, or tan growth with waxy, “stuck on” appearance, commonly referred to as barnacles of old age

A

Seborrheic keratosis is not premalignant and needs no treatment unless the lesions are irritated, itchy, or cosmetically bothersome

101
Q

Necrotic wound - Local tissue reaction causes local burning at the site for 3-4 hours → blanched area (due to vasoconstriction) → central necrosis erythematous margin around an ischemic center “red halo” → 24-7 hours after hemorrhagic bullae that undergoes Eschar formation → necrosis

A

Brown violin on the abdomen

Brown Recluse: = necrotic

102
Q

How to treat brown recluse bite?

A

Treatment:

For brown spider bites, use wound care, local symptomatic measures, and sometimes delayed excision

103
Q

Neurologic manifestations - May not see much at bite site:

–toxic reaction: nausea, vomiting, HA, fever, syncope, and convulsions

A

Black Widow:

Red hourglass on the abdomen

104
Q

How to treat black widow bite?

A

For black widow spider bites treatment includes wound care, local symptomatic measures, sometimes parenteral opioids, benzodiazepines.
Treat with anti-venom available for elderly and kids

105
Q

serious hypersensitivity complex that affects the skin and the mucous membranes.

begins with a prodrome of flu-like symptoms, followed by a painful red or purplish rash that spreads and blisters. Layers of skin peel away in sheets (+) Nikolsky’s sign (pushing blister causes further separation from dermis)

A

SJS
Stevens-Johnson syndrome (SJS) is a milder form of toxic epidermal necrolysis (TEN) with LESS THAN 10% of body surface area detachment

Treatment: Treat the underlying cause and supportive (burn unit)

106
Q

A highly contagious, fungal infection of the skin or scalp. KOH - long, branching fungal hyphae with septations

A

Tinea infections

107
Q

-papules pustules, around hair follicles

–Athlete’s Foot: pruritic scaly eruptions between toes. Trichophyton rubrum is the most common dermatophyte causing athlete’s foot

A

Tinea Barbae
Treatment: Oral antifungal therapy is necessary - two- to four-week course of griseofulvin microsize (500 mg per day) or oral terbinafine

Tenia PEDIS
Topical antifungals - azoles (1% clotrimazole, 2% ketoconazole),

108
Q

–“Jock Itch” diffusely red rash in the groin or on the scrotum

-(ringworm): usually seen in younger children or in young adolescents with close physical contact with others (i.e. wrestlers)

A

Tinea Cruris:
Topical antifungals - azoles (1% clotrimazole, 2% ketoconazole)

Tinea corporis:
Topical azole antifungals (1% clotrimazole, 2% ketoconazole) or 1% terbinafine cream

109
Q

Most common fungal infection in the pediatric population. Occurs mainly in prepubescent children (between ages 3 and 7 years). Asymptomatic carriers are common and contribute to spread

A

Tinea capitis:

Systemic therapy warranted to penetrate hair shaft
Oral griseofulvin (Drug of Choice):
110
Q

caused by Malassezia furfur, a yeast found on the skin of humans. Lesions consist of hypo or hyperpigmented macules THAT DO NOT TAN

A 20-year-old male with no significant past medical history presents complaining of patchy tanning. He states that he has been out in the sun without a shirt several times. Areas on his chest and back just don’t tan, and he is becoming self-conscious

A

Tinea versicolor: short hyphae and clusters of spores (“spaghetti and meatballs”)
Treatment: selenium sulfide 2.5% applied to affected skin for 10 minutes.
Wash off thoroughly. Apply daily for 7–10 days. Monthly applications may help prevent recurrences

Candidiasis: budding yeast, pseudohyphae

111
Q

A rare, life-threatening skin condition that is usually caused by a reaction to drugs

A

TEN is > 30% of body

in toxic epidermal necrolysis older patients

Confirm the diagnosis by biopsy

112
Q

how to treat SJS/TEN?

A

treat SJS/TEN in a burn unit and with intensive supportive care

Consult ophthalmology if the eyes are affected

Cyclosporine and possibly plasma exchange for severe cases

113
Q

blanchable, pruritic, raised, red, or skin-colored papules, wheels or plaques on the skin’s surface
(+) Darier’s sign:

A

Urticaria Angioedema: painless, deeper form of urticaria affecting the lips, tongue, eyelids hand and genital

If anaphylaxis give epinephrine: 0.3–0.5 mg

114
Q

localized urticaria appearing where the skin is rubbed (histamine release)

A

(+) Darier’s sign:

115
Q

a rare, lifelong hereditary disorder characterized by chronic infection with HPV

A

Epidermodysplasia verruciformis

The most ugly warts on bilateral feet picture

116
Q

Loss of skin color can affect any part of the body, including the mouth, hair, and eyes. It may be more noticeable in people with darker skin

A

Vitiligo is caused by autoimmune destruction of melanocytes causing these pigment-producing cells to die or stop functioning

117
Q

Wood’s light exam: a “milk-white” fluorescence over the lesion

A

Vitiligo Sharply demarcated ivory white patches

118
Q

How to treat vitiligo?

A

Treat with sunscreen, cover-up, corticosteroids, tacrolimus (an immunosuppressive drug), and vitamin D

Treatment may improve the appearance of the skin but doesn’t cure the disease

Family Medicine EOR (4 decks)

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