derm Flashcards
age of onset:
- erythema toxicum
- neonatal acne
- milia
- erythema toxicum = within first 72h
- neonatal acne = ~3-6weeks
- milia = 6 weeks
erythema toxicum vs transient neonatal pustular melanosis
erythema toxicum:
- first 72h
- papules > pustules
- significant surrounding erythema
- spare palms and soles
- no PIH
pustular melanosis:
- often at birth
- pustules and macules, no surrounding erythema
- palms and soles, leave PIH
infantile vs neonatal acne
neonatal:
- 3-6 weeks onset
- from oil glands with yeast
- ketoconazole if need to treat
- no PIH
infantile:
- 3-4mo onset
- from hyperplasia of sebaceous glands secondary to androgenic stimulation, so more common in boys
- treat as can cause PIH e.g. benzyl peroxide or retinoid
maculopapular mastocytosis
reddish brown spots that are chronic, and redden up when you touch them
cradle cap vs scalp eczema
cradle cap 1 week to 4 mo, greasy waxy flakes. no need to treat, but can use moisturiser/soft toothbrush to get it off.
scalp eczema > 4mo, treat
nappy rash vs candidiasis
nappy rash spares the folds
What is acrodermatitis enteropathica?
from zinc deficiency, present after weaning off breastmilk. Classic triad:
1) Peri-acral and periorificial dermatitis - sharply demarcated
2) Diarrhoea
3) Alopecia
accessory tragus =
goldenhar
ectodermic dysplasias have problems where?
hair, teeth, skin, nails, and sweating
haemangioma - natural history
most not present at birth
grow 6-9mo
>1y - spontaneous involution, 10% per year
beard haemangioma, think =
airway risk
large facial haemangioma, think =
PHACES
Segmental haemangioma overlying lumbosacral spine =
tethered spinal cord
> 5 haemangiomas, think =
visceral, esp hepatic/GI
most common side effects of oral propranolol
diarrhoea (most common)
hypoglycaemia!! educate - all symptoms masked except sweating!
hypo/brady 2h post dose
migraine
bronchochonstriction
hyperkalaemia
PHACE syndrome + beta-blocker?
Risk of stroke given often have cervical stenosis and arteriopathy (coarctation) as part of syndrome – hypotension from BB resulting in ischemia
key complication of Kaposiform haemangioendothelioma (KHE)
Kasabach-Merritt phenomenon
Sturge-Weber syndrome vs Klippel-Trenaunay syndrome vs Parkes-Weber syndrome
Sturge-Weber: capillary malformation over V1/V2
Klippel-Trenaunay: over limbs, soft tissue/bone hemihypertrophy
Parkes-Weber: same as KT but have AV shunts
haemangioma vs port wine stain
port wine stain:
- present at birth
- grow with overall growth, do not regress
- flatter and darker, does not blanch with pressure
- doesn’t usually pass the midline