Dental Anomalies 2 Flashcards
anomalies of shape
dens evaginatus, dens invaginatus
anomalies of structure
enamel and dentin
anomalies of eruption
ectopic eruption, ankylosis, natal/neonatal teeth
dens evaginatus
evagination of the inner enamel epithelium
dens evaginatus anterior teeth
extra cusp in cingulum region
talon shape
may extend to the incisal edge
dens evaginatus posterior teeth
extra cusp in the central groove
most common in premolars
dens evaginatus clinical considerations
occlusal interference
plaque trap
pulp exposure
dens invaginatus
invagination of the inner enamel epithelium
dens in dente
location: max incisor or canines most common
dens invaginatus clinical findings
deep lingual pit
deep/long lingual groove
enamel and dentin in the invagination may be defective or absent
explorer stick in pit/groove
dens invaginatus clinical considerations
plaque
direct communication between pulp and oral cavity
anomalies enamel
amelogenesis imperfecta : genetic
enamel hypoplasia : systemic, localized
amelogenesis imperfecta main types
hypoplastic (most common)
hypomaturation
hypocalcification
ALL teeth affected
hypoplastic AI
predominantly AD
decreased enamel quantity/normal quality
radiographically: decreased enamel thickness
clinically: teeth small, open contacts, temperature sensitivity
anterior open bite
hypomaturation AI
normal quantity of enamel
lack of enamel maturation
low mineral content
porous mottled surface
enamel easily fractured
decreased enamel radiodensity
hypocalcification AI
normal quantity of enamel
poorly calcified
enamel easily fractured: incisal edges
acquired developmental enamel defects
an enamel deficiency/defect encountered during enamel formation
systemic causes, local causes
acquired developmental enamel defects systemic causes
fever
low birth weight
vitamin deficiency
chromosomal defects
neurological defects
allergies/asthma
radiation
no known cause
molar-incisor hypomineralization
first permanent molars/incisors
may be no determined cause
often secondary to fever/infection
mild/moderate/severe
temperature sensitive
post-eruptive breakdown/caries
acquired developmental enamel defects local causes
trauma
infection
anomalies dentin
dentinogenesis imperfecta: inherited/genetic
regional odontodysplasia: not inherited
dentinogenesis imperfecta
inherited
all teeth in both dentitions affected
more common than AI
dentinogenesis imperfecta types
Shields Type I, Shields Type II
Shields Type I
with osteogenesis imperfecta
brittle bones
bowing of the limbs
bi-temporal bossing
blue sclera
primary teeth more severely affected
Shields Type II
autosomal dominant
hereditary opalescent dentin
primary and permanent teeth affected equally
dentinogenesis Imperfecta Type II clinical findings
amber translucency
enamel fractures from dentin
severe attrition
abscess in absence of caries
dentinogenesis imperfecta type II radiographic findings
bulbous crowns
slender roots
pulp chamber obliteration: abnormal, continuous proliferation of dentin matrix
pulp canals - small, ribbon-like
root fractures
stainless steel crowns
regional odontodysplasia clinical findings
delayed eruption or lack of eruption
defective enamel and dentin formation
erupted teeth are dysmorphic
frequently abscess soon after eruption
regional odontodysplasia radiographic findings
ghost teeth
decreases radiodensity
thin layer of enamel/dentin
large pulp chambers
regional odontodysplasia tx
extraction, prosthetic replacement
anomalies of eruption
ectopic eruption: max 1st perm molar, mand perm incisors
anyklosis: primary molars
natal and neonatal teeth
ectopic eruption maxillary perm 1st molars
prevalence: 3-4%
mesioangular eruption path
primary second molar: distal root resorption, dental/periodontal abscess (uncommon)
ectopic eruption mand perm incisors
central or lateral: excessive lingual eruption position
prevalence 10%
ectopic eruption mandibular permanent incisors etiology and tx
etiology: abnormal eruption path, transitional crowding, arch length deficiency
tx: mobile - observe or extract, not mobile -extract
tongue will usually move into the arch
ankylosis etiology and prevalence
etiology: fusion of cementum with alveolar bone at anytime during eruption… does not have to be whole tooth
overall prevalence: 1.3-38.5%, most common - primary molars
50% of patients - more than 1 ankylosed tooth
ankylosis clinical findings
below plane of occlusion
lack of physiologic mobility
ankylosis radiographic findings
lack of lamina dura, PDL space
more common with congenitally missing permanent successor
natal and neonatal teeth
rare
location: mand central incisors
normal/hypoplastic/dysmorphic
must diagnose radiographically
most are not supernumerary
riga fede
lingual tongue ulceration
smooth incisal edge
composite resin
extract
natal and neonatal teeth indications for extraction
excessive mobility: aspiration risk
feeding difficulties
supernumerary