Definition Flashcards

1
Q

IV Therapy

A
Assessment – renal/cardiac function
Monitor patient status (Wt, I/O, lab studies)
Doctor’s orders - rate and solution
Patient Safety
Be aware of “high alert” drugs (KCL)
Smart pump
Computerized physician order entry
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2
Q

Flow rates via Gravity (without pump)

A

Patency of cannula
Venous spasms
Size of cannula
Blood in tubing
Present of local complications
The higher you hang a bag, the faster it will go
Patency – there should be nothing obstructing it (blood clot will cause drip rate to slow down or just blood in the tubing)
If you suspect blood in tubing, you can try and flush, but if there is a clot, DO NOT force it back into the body
Spasms – can slow flow; caused by cold temp (constricting vessels), cold temp of IVs/IV meds that are in the refrigerator

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3
Q
Peripherally Inserted 
Central Catheters (PICC)
A
Placed in the antecubital fossa
Tip resides in SVC
Low complication rates and less expensive
No BP/blood draws in this extremity 
Fluid comes out in superior vena cava and can handle more meds because it is bigger than a vessel
Nurses need to protect PICC lines
BP cuff could collapse/destroy
Do not draw blood
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4
Q

Multi-Lumen Catheter

A

Can deliver two or more solutions at same time via separate pathways
Necessary for patients who are getting multiple meds, especially blood because blood can only be given with saline
By the time they mix in the blood, all of the meds are well diluted

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5
Q

Implanted Ports

A

No part of catheter is visible
Venous system most common placement
Accessed with a needle through the skin
Helpful for kids because they will not grab at it like they would with the lines
In the SVC
Can be accessed when needed, but okay to stay there without use as well (small lump)
Can stay in for months-years
Common for chemo patients so they can administer it at home
There may be a small incision over the top of the port from where it was inserted

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6
Q

Indications for Central Venous Access Device (CVAD)

A

Medication Administration
Nutrition
Blood samples/transfusions
Conditions (renal failure, burns etc.)

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7
Q

Intraosseous Route

A

High infusion rates are possible
Pain meds may be required
Good for severely dehydrated or during cardio/resp. emergencies
Proximal humerus, proximal and distal tibia
Instills meds directly into the bone
Severe dehydration and cant get a line or in the middle of a code
Used more in ER during traumas

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8
Q

Central Line Care

A
X-Ray before starting fluids
Sterile dressing change
Observe site for S/S infection
Observe for IV systemic complications
Care depends on complication assessed
Cannot start anything until we know for sure that the tip is inserted in the correct area (SVC)
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9
Q

Complications of Central Venous Access Devices (CVADs)

A
Catheter Occlusion
Embolism
Infection
Pneumothorax
Catheter Migration
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10
Q

What is the nurse’s role in IV therapy?

A

Assessment and listen to the patients

Always investigate if there seems to be a problem

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11
Q

Local Complications: Infiltration/Extravasation

A

Infiltration/Extravasation: Skin is pulled away – fluid going into interstitial space
Edema
Pain
Cool temperature
Red
When assessing IVs, you need to look at site at least every hour (fluid build up, red, open, etc)
Compare to other area (hand v. hand, etc)
Pull the IV out if you suspect something is wrong
Make sure you think about what to do next (what med/saline was going in and why?) – what needs to be done next to keep patient safe and stable

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12
Q

Local Complications: Nerve Damage

A
Nerve damage is a big concern
Fluid compresses nerves
Tingling
Numbness in fingers (Emergent situation)
Fluid can be reversed, but nerve damage may not
Prioritize what needs attention
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13
Q

Hematomas/Ecchymosis

A

Hold pressure after removal to avoid hematomas

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14
Q

Phlebitis

A

Phlebitis is warm feeling – take catheter out of the arm to avoid more harm

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15
Q

Thrombophlebitis

A

Thrombophlebitis = clot (this warrants a call to physician, do not press on clot b/c you could dislodge it)

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16
Q

Site infection

A

Redness
Swelling
Possible fever

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17
Q

Catheter Related Infection

A

Caused by poor sterile technique/contamination
Pay attention to ABCs
BP stable
Call RR
Abrupt rise in temp, severe chills, shaking, increase HR, RR, headache

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18
Q

Circulatory Overload

A

Patient got too much fluids and body couldn’t handle it
Increase head of bed
Give O2
Turn to KVO rate (keep vein open) – slow down so your don’t do more harm to patient
If bad, call RR
If you catch it early, call physician – they will likely order Lasix to get rid of fluid excess quicker
Increase BP, distended neck veins, Shortness of breath

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19
Q

Pneumothorax

A

Generally occurs when they are putting a central line in
Intervention with chest tube
Decreased or absent breath sounds, respiratory distress, distended unilateral chest

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20
Q

Air embolism

A

BP is low unlike circulatory overload where it increases
Occurs when you change a central line – if you don’t cap it correctly
Prime line incorrectly
Intervene by positioning in left lateral modified Trendelenburg (head slightly down, feet slightly up, left side) – traps air in right atrium and it gets pulled out
Chest pain, SOB, decrease BP, increase HR, cyanosis, anxiety, confusion

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21
Q

IV Push

A

Always flush before and after to make sure all meds are in patient
We don’t want to mix meds if there is some left over from previous push
Push slowly
Not all drugs can be pushed
Faster reaction from patients
If they do have a reaction, the nurse will be there to intervene quickly
Disadvantage: cant take back and cant slow down, adverse reaction could be more severe and quicker

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22
Q

IV Piggyback

A

Assess patients and teach them meds
Goes into primary line with drip rate (over longer course of time)
Make sure it is diluted in the correct type of solution***
Advantage: Don’t have to be in the room the whole time, less risk of adverse rxn because it is diluted, if there is a rxn majority of med will still be in bag so we can stop it
Disadvantage: Not in the room the whole time to see reaction when it occurs

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23
Q

PCA Pumps

A

Enables the patient to self administer medication on prn. basis
Programmed according to medical orders (dosage, time intervals between doses, and lock-out intervals)
Can record the amount of medications received and the number of requests by the patient

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24
Q

Perfusion

A

refers to the flow of blood through arteries and capillaries, delivering nutrients and oxygen to cells.

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25
Q

Central Perfusion

A

Force of blood movement generated by cardiac output
Requires adequate cardiac function, blood pressure, and blood volume
Cardiac output (CO) = Stroke volume × heart rate
Central perfusion is cardiac output – need blood volume to maintain perfusion
If the heart is not pumping, you will see signs all over the body

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26
Q

Peripheral Tissue or Local Perfusion

A

Volume of blood that flows to target tissue
Pressure generated from each myocardial contraction supplies blood to peripheral vascular system
Valve in each vein keeps blood flowing in direction toward heart
Local perfusion or tissue perfusion is a local effect (ex. Blood not flowing to feet)
Local manifestations
Most of the effects stay local, although it could create a chain reaction and become systemic

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27
Q

Impaired Tissue (Local) Perfusion

A

Impairment of tissue perfusion is associated with loss of vessel patency or permeability, or inadequate central perfusion
Results in impaired blood flow to the affected body tissue (localized effect)
Leads to ischemia and, ultimately, cell death if uncorrected

28
Q

Atherosclerosis

A

Begins when waxy cholesterol (atheromas) becomes deposited on the intima of the major arteries
Atheromas interfere with the absorption of nutrients by the endothelial cells that compose the vessel lining and obstruct blood flow
Build up of plaque in the artery
Lumen narrows and interferes with blood flow
Blood flow does not get into vessel itself and can cause destruction
is fatty substances within the walls of the arteries

29
Q

Arteriosclerosis

A

a thickening and loss of elasticity and hardening of the arterial walls

30
Q

Impaired Central Perfusion

A

Impairment of central perfusion occurs when cardiac output is inadequate.
Reduced cardiac output results in a reduction of oxygenated blood reaching the body tissues (systemic effect).
If severe, associated with shock
If untreated, leads to ischemia, cell injury, and cell death
Centers around cardiac output
You may see shock (major organ damage), stroke (not enough O2 to brain, kidney failure (not enough blood to kidney), etc.

31
Q

Cardiac Output (CO)

A

CO = Stroke Volume X Heart Rate
Stroke Volume = amount of blood ejected from the left ventricle with each heartbeat
Heart Rate = Beats/minute
If you change one of these factors, you change CO
When blood volume drops, HR increases
Hypoxia = increased HR
If you increase the heart rate, you increase CO

32
Q

Stroke Volume

A

Influenced by Preload

& Afterload

33
Q

Preload

A

refers to amount of blood in left ventricle at the end of diastole
The greater the preload the greater the “stretch” or contractility of the heart muscle resulting in a greater stroke volume (Starling’s Law)

34
Q

Starlings Law

A

when I stretch the fibers of the ventricle, it will cause a greater output
If you overwork the muscles, they will die
ex rubberband

35
Q

Afterload

A

reflects the amount of resistance the ventricles have to contract against (also know as systemic vascular resistance – SVR)
influenced by diameter of blood vessels and blood volume
An increase in afterload results in a decrease in stroke volume
resistance; what the heart has to pump against to get the blood out
When we decrease the afterload, we decrease how hard the heart has to work

36
Q

Perfusion: Modifiable Risk Factors

A

risk factor is one in which an individual may exercise control by changing a lifestyle or personal habit
Weight
Cholesterol
Blood sugar

37
Q

Perfusion: Non-Modifiable Risk Factors

A

risk factor is a consequence of genetics over which an individual has no control
Age
Gender
Family history

38
Q

Perfusion: Baseline History

A

family history, smoking, diet (cholesterol and sodium), exercise, occupation, sleeping (sleep apnea), stress, history of hypertension, medications (specifically BP meds, cholesterol, asthma, diuretics, blood thinners, OTCs (psudofed increases BP and HR, potassium supplements, calcium supplements like Tums))

39
Q

Perfusion: Focused History

A

Chest pain
Edema – excessive fluid is a sign of heart failure
Dizziness – blood flow to brain is perfusion issue
Extremities are local perfusions
Pain
Dyspnea
Edema
Dizziness (altered mental status)
Extremity changes (temp, skin, color etc.)

40
Q

Common Diagnostic Tests

A
Doppler Studies
Ankle-brachial index  (ABI)
Laboratory tests
Creatine kinase, natriuretic peptides, troponin, homocysteine, C-reactive protein, serum lipids, platelets, prothrombin time (PT), partial thromboplastin time (PTT), international normalized ratio (INR)
Electrocardiogram (ECG) 
Cardiac stress tests
Exercise or pharmacological test
Radiographic studies
Chest x-ray, ultrasound, arteriogram, 
Cardiac Catheterization
41
Q

Biomarkers

A
Creatine Kinase (CK)
BB – brain
MM – skeletal muscle
MB – cardiac muscles
CK-MB
Rise & fall over 2-4 days
Onset about 3-6 hrs.
Peak within 10-24 hrs.
42
Q

Troponin

A

complex of three regulatory proteins integral for muscle contraction
Troponin I used for myocardial damage
Released after myocardial injury
Levels are usually so low, cannot be detected
The higher the troponin, the greater the damage
Start to rise within hours after infarct (most within 6 hours after attack)
If levels are normal within 12 hours - infarction unlikely
Levels detectable for 10-14 days
Levels drawn upon arrival to ER and repeated twice within a 12-16 hour window
This test does things CK-MB did not do
Shows results within an hour or so – patients already release Troponin when they are having a heart attack
Troponin can last up to 2 weeks – if a patient comes in days later, there is no CK-MB, but there will be Troponin
Troponin correlates with injury – the more severe a heart attack, the more Troponin will be there
Physicians will do a second test if the first comes back inconclusive

43
Q

hs-CRP (high sensitivity C-reactive Protein)

A

Reflective of endothelial inflammation
Treatment to lower levels include aspirin, smoking cessation, exercise lipid lowering agents, Omega-3,
Patients with rheumatoid arthritis – looks at inflammation

44
Q

Homocysteine

A

Amino acid result breakdown of dietary protein
When levels are elevated, blood clotting may increase and the vascular endothelium may be damaged
Treat with folic acid/B vitamin complex (supplements/diet)

45
Q

Serum lipids

A

-Triglycerides (<150 mg/dl)
- HDL (high density) (40-60 mg/dl)
- LDL (low density) (<100)
- TC (total cholesterol) (<200)
(Risk factors = smoking, low HDL, family history of premature CHD and age)
HDL is good, LDL is bad
It is okay if HDL is high because it is cardio protective – may negate another risk factor
If you are diabetic, LDL needs to be less than 70

46
Q

Electrocardiogram (EKG)

A

PQRST wave forms to assess cardiac function

Deviations from normal sinus rhythm can indicate heart abnormalities

47
Q

Holter Monitoring

A

Recording of ECG rhythm for 24-48 hours to assess arrhythmias
Normal patient activity during recording
Recorder stores information
Information analyzed
Patient needs to keep diary of activities/any symptoms
No bath or shower during monitoring
Skin irritation may develop from electrodes
Can pick up potassium imbalances
The patient will keep a diary while wearing EKG electrodes so that if an abnormality occurs or if the patient feels something, they will know what they are doing when it occurs
Steps
Eat
Sleep
Rest vs. active

48
Q

Echocardiogram

A

Uses ultrasound waves
Can measure heart structures and size, blood flow and ejection fraction
Ejection fraction (EF) = percentage of blood volume that is ejected during systole
Normal EF is > 55%
Provides information about the left ventricle’s function
The ability of a person’s heart to maintain cardiac output
If it falls below 50%, that could be an early sign of heart failure
Patients needs to lay on their side

49
Q

Transesophageal Echocardiography (TEE)

A

Provides more precise information about the heart
Flexible endoscope with an ultrasound transducer
Used in inpatient and outpatient
Contraindicated in patient with esophageal disorders
Require conscious sedation
Behind the heart
Put tube down esophagus and see the heart from the back side
If there is an issue that is not getting resolved, this may be an option
Patient is under monitored sedation
Gag reflux has to be numbed – don’t let the patient go until the gag reflux comes back (ABCs)
NPO for at least 4 hours prior

50
Q

Exercise Stress Test

A

Involves walking on treadmill
ECG & BP recording included before during and after exercise
Patient instructed to report any chest pain, SOB etc. immediately during procedure
Wear comfortable shoes
Light meal or NPO 2 hours prior
No caffeine
Avoid smoking prior to the test
Some medications may be held
Chest pain is a lack of O2 to the heart
Most of the time, heart attack is already ruled out at this point, but they need to figure out what is wrong
No caffeine or smoking before this

51
Q

Stress Echo

A

Same as exercise stress, but Includes echo ultrasound before and immediately after walking
Pt. needs to get back on table as soon as exercises ceases so post exercise images can be obtained
Outpatient test – they can drive after
Get a baseline with EKG
Put patient on table and do an echo
As long as the patient seems okay to do so, they will slowly start treadmill walking
Cardiologist will slowly increase rate of treadmill to put some stress on heart
If the patient has chest pain, SOB, etc, the test is stopped immediately – if this happens, it is a failed test and they will go for further testing
After testing, they have 30 seconds to get back on the table to redo the echo

52
Q

Nuclear Stress Test

A

Studies involve injection of radioactive isotopes
Uptake is measured by a scan
Provides info regarding contractility, perfusion and cell injury
Patient lays still on back with arms extended overhead for 20 minutes; newer scans have seats
Involves exercise on treadmill or medication to simulate activity for patients with ambulation issues
Scans are repeated before & after exercise for comparison of images
Sometimes the patient is on the treadmill, sometimes there will be a die injection
This test is used more commonly on elderly patients to simulate walking on the treadmill
Concern is allergy to die

53
Q

Cardiac Catheterization

A

Right-sided: vein Left-sided: artery
Views heart, chambers, O2 status and chamber pressures
Involves insertion of a catheter & injection of contrast media & possible placement of stent
Coronary angiogram – dye injected into coronary arteries to evaluate condition/blood flow
Test to view chambers
Can see how blocked coronary arteries are
With kids, we are looking at holes in the heart and other congenital issues
Cath is threaded into heart and inserted with dye

54
Q

Primary Prevention

A

Smoking and nicotine cessation
Diet
Exercise
Weight control
Monitor BP, Cholesterol levels, Blood glucose
They need to STOP smoking all together (not just cut down)
Prevents further destruction of the area

55
Q

Secondary Prevention (screening)

A

Identify High Risk patients and screen
Blood pressure screen
Lipid Screening

56
Q

Altered Peripheral (Tissue)Perfusion

A

Arterial Disorders
Issue: poor perfusion and oxygenation
Atherosclerosis is the leading cause in majority of cases
Causes narrowing of the lumen, obstruction by thrombosis, plaque ulceration, aneurysm, rupture
Local perfusion
Arterial – carries oxygenated blood away from the heart (blood isn’t getting to where it needs)
Legs and arms
Big component is plaque build up

57
Q

Altered Peripheral Perfusion Manifestations

A

Intermittent Claudication
Thin, shiny, taut skin
Loss of hair on the lower legs
Diminished or absent pulses
Skin changes related to color or temperature
“Dependent rubor”
“Elevation pallor”
Ulcers (usually on toes or near toes, +pain, neurologic deficits present)
Pain at rest
Occurs in the foot or toes
Aggravated by limb elevation
The pt will experience intermittent claudication (pain with walking that is relieved with rest)
When the pain is occurring at rest, there is a severity increase (red flag)
Changes in skin because there is a lack of nutrients to the area
Diminished you can live with, but absent is severe
Ulcers tend to be near or on toes because of lack of nutrients and O2 to area – arterial disorder
Pain deficits or lack of sensation

58
Q

Carotid Artery Disease

A

Risk of stroke
Manifestations: depending on size of obstruction
Diagnostic: Carotid duplex ultrasound, MRI, CT (with contrast)

59
Q

Carotid Endarterectomy (CEA)

A

Opening the carotid artery and removing the obstructing plaque
Nursing Care: Monitor vital signs, neuro status, pulses, bleeding

60
Q

Peripheral arterial bypass

A

Operation with autogenous vein to carry blood around the lesion or occlusion

Nursing care
Important to assess for occlusion (pulses, temp cap. refill, etc.)
Report of pain (type of pain)
Bedrest for 18-24 hours
Check blood pressure
Notify surgeon immediately of abnormal findings

61
Q

Raynaud’s Phenomenon

A

Extreme sensitivity to cold aggravated by nicotine, emotion, chilling, limited to hands and feet
Treatment and education related to preventing vasoconstriction
Stress
Smoking
Painful
Goal is to treat patient how to prevent this - (ex if they go into freezer to get something, wear warm gloves)
Give them something to reverse the constrictions (vasodilator) – be careful because this decreases BP though (MONITOR)

62
Q

Thromboangiitis Obliterans or Buerger’s Disease

A

Non-athrosclerotic, recurrent inflammatory vaso-occlusive disorder resulting in microscopic occlusion (thrombi) of distal vessels of upper and lower extremities
Caused by history of tobacco/marijuana use
Directly related to smoking
With the increased use of marijuana, there is also an increase in bureger’s
Autoimmune response
Lost arms/legs
Causes inflammation process and occludes vessels and clots
Pt needs to stop smoking – this will slow down inflammation and prevent further worsening

63
Q

Virchow’s triad

A

Venous stasis
Damage of endothelium
Hypercoagulability of blood

Pain, warmth, redness, and edema

64
Q

Venous thrombosis

A

formation of a thrombus in association with inflammation of the vein
Superficial vein thrombosis (SVT)
Formation of thrombus in superficial vein
Generally benign disorder
Deep vein thrombosis (DVT)
Formation of thrombosis in deep vein
Most commonly iliac and femoral veins involved

65
Q

Venous thromboembolism (VTE)

A

Spectrum of pathology from DVT to PE
Watch for signs of pulmonary embolus
If they think the pt has a clot, they will immediately be put on heparin

66
Q

Venous Insufficiency Disorders

A

Results of prolonged venous hypertension that stretches the vein and damages the valves
Difficulty eliminating waste
Builds up in the tissues resulting in stasis, ulcers, edema, cellulitis, brownish “brawny” appearance
Prominence of superficial veins
Ulcers
Usually ankle area, +pulses, no claudication, no neurologic deficits
Walking around helps move fluid and get it back to the heart
Deoxygenated blood and waste moving back to heart
This is a problems of the waste products are not going away
There is pain and sensation in the area – very painful
Location is key – tells you if it is arterial or venous
Who is at risk –
Diabetics
Smokers
Standing in one place
Heart disease
Immobile
Hypertension
Obesity
Hormone therapy patients
Tests ordered –
Ultra sound/doppler to see circulation leaving the area
Blood test (INR)
Cholesterol levels
Dye to find what they are looking for

67
Q

Venous Insufficiency Disorders: Care & Patient Teaching

A
Avoid sitting/standing for long periods
Avoid trauma to limbs
Elevate legs above level of  heart to reduce edema
Compression therapy 
Proper nutrition 
Exercise 
Collaborate with Wound Care specialist
Varicose veins 
VNUS procedure
Laser
Adhesive (FDA approved in 2015)