CVS Flashcards

1
Q

GUidelines for Primary PCI when managing STEMI

A
  1. within 12 hours from starting symptoms AND
  2. within 90 min from medical contact at capable facility
  3. within 120 min at a non-capable facility
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2
Q

Treatment on hypertension in Pregnancy

A

First line: betablockers (labetalol) , calcium channel blockers (nifidipine) , hydralazine, methyldopa
Second line: clonidine, thiazides,

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3
Q

Treatment of HTN in nonpregnant

A

First line: thiazide, Ca channel blockers, ACEI/ARB

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4
Q

characteristics of hypertrophic cardiomyopathy

A
  1. asymmetric LV wall thickening favoring septum
    2.reduced LV cavity size
    3.Left atrial enlargement
  2. ECG showing prominent Q waves and Twave inversion in 2 leads
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4
Q

Initial management of STEMI

A

oxygen
Aspirin (chewed)
clopidogrel
nitroglycerin
betablocker(meto/ateno)
anticoagulation (heparin)

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4
Q

Vasospastic Angina
-pathophysiology
-risk factors
-clinical presentation
-diagnosis
-treatment

A
  1. hyperactivity of coronary smooth muscles (similar to reynaud)
    2.smoking, age <50
  2. recurrent chest pain at rest or during sleep resolves in 15 min
  3. ECG (ambulatory)will show st elevation transient
    Cath will not show coronary artery disease
    5.Ca channel blocker is preventative
    nitroglycerin abortive
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4
Q

Management of Primary Mitral Regurg

A

-Surgery if LVEF 30-60%
consider surgery if success if likely in : –>asymptomatic >60%
>symptomatic <30%

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4
Q

Characteristics of Athletes Heart

A
  1. Eccentric LV hypertrophy with enlargement of LV mcavity size and slight increase in wall thickness
  2. enlarged RV cavity size
    3.increase stroke volume (no change in EF)
  3. abscence of Left atrial enlargement
  4. sinus bradycardia
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5
Q

Pre-op evaluation of valvular heart disease

A

-If mild: can go ahead with their noncardiac surgery
-moderate/severe?
if symptomatic: repair valve first
if asymptomatic and pt is undergoing intermediate or high risk surgery then assess the valve for EF and pulm htn if present repair first

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6
Q

chronic aortic regurgitation etiologies

A

-congenital bicuspid aortic valve
-Post inflammatory(rheumatic heart disease or endocarditis)
-aortic root dilatation (Marfan/syphillis)

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7
Q

chronic aortic regurgitation clinical findings

A

-decrescendo diastolic murmur
-widened pulse pressure
-water hammer pulsation
-abrupt carotid distension and collapse (pistol shot femoral pulses)

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8
Q

what are the cardiovascular effects of hyperthyroidism

A
  1. increased rate:tachycardia/palpitations, afib
    2.increased contractility: increased EF and CO, increased myocardial oxygen demand, increase pulmonary artery pressure
    3.peripheral vasodilation: decreased SVR
    4.decreased diastolic pressure, increased systolic
    5.increased pulse pressure
  2. high output heart failure
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9
Q
A
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