CV Integration Lab Flashcards
How does venous return change during inspiration?
- increases return to RV
- decrease in return to LV
- these changes lead to altered murmur intensity
- inspiration increases intensity of right sided murmurs
How do insp/exp affect S3 and S4?
- increase with insp
- decrease with exp
How does opening snap in TS change with expiration and inspiration? MS?
- TS - diminishes with exp; increases with insp
- MS - increases with exp; decreases with insp
When might you hear a loud S1? soft? variable intensity?
- Hyperdynamic (fever, exercise), MS, Atrial myxoma (rare)
- Low CO (rest, heart failure), Tachycardia, Severe mitral reflux (caused by destruction of valve)
- Atrial fibrillation, Complete heart block
If you hear P2 audible at apex, what should be on your ddx?
- significant pulmonary HTN
- ASD
Define the following:
- Cardiogenic Shock
- Distributive Shock
- Hypovolemic Shock
- Shock that is caused by failure of the heart as a pump to deliver adequate perfusion to peripheral tissues
- Shock that is caused by failure of the arterial circulatory bed to maintain adequate perfusing pressure to peripheral tissues
- Shock that is caused by significant reduction in circulating vascular volume, resulting in reduced perfusing pressure to peripheral tissues
What are causes of a split S2?
What influences Stroke Volume?
- LV preload/afterload
- Myocardial contractility
nothing to add - just figured i should put this in here because of the questions he put by it
If you see a pt with unexplained sinus tachycardia and hypotension, you should think of …
shock
* If you see a patient with shock who has bradycardia or a lower-than-expected heart rate, consider ways to increase the heart rate
What two things should you measure in pt with suspected shock to help with prognostic info/therapy guidance?
- arterial blood gases
- lactate levels
The dx of pericarditis requires at least 2 of what criteria?
- typical chest pain
- pericardial friction rub
- widespread ST segment elevation
- new or worsening pericardial effusion.
If you suspect pericarditis, what is the most important next step?
- assess its hemodynamic impact, both clinically and echocardiographically, looking for evidence of cardiac tamponade, which is a medical emergency.
*Clinical features of cardiac tamponade include dyspnea, hypotension, tachycardia, jugular venous distention, and pulsus paradoxus of >10 mmHg
If a pt has sharp pleuritic chest pain that improves when they sit up and lean forward, you should think …
acute pericarditis
*dyspnea, palpitations, diaphoresis, + sx of underlying cause may also be present
*characteristic finding is pericardial friction rub
Where is the best place to hear a pericardial friction rub?
scratchy sound best heard with diaphragm over left sternal border