Critical Care: Advanced Cardiac Life Support Flashcards
IV/IO dose conversion to ET
2-2.5x IV dose = ET dose
cardiac arrest
pt unale to generate aadquate cardiac ouput o support O2 demands
types of rhythms in cardiac arrestg
- v. fib (VF)
- pulseless v tach (pVT)
- asystole
- pulseless electrical activity (PEA)
when do we admin meds when treating cardiac arres
- if initially non-shockable rhythm (PEA, asystole): admin epiniphrine ASAP
- if initially shockable (VF or pVT): admin epiniphrine after the 2nd shokc and antiarrhythmics after 3rd
- epinephrine is 1 mg Q4min (2 CPR cycles) - do NOT use high dose
- antiarrhythmics: usually amio or lidocaine
general treatment for cardiac arrest
- start with CPR/O2 and attatch heart monitor/defibrillator
- 2 min cycles
- if after2 min of CPR, pt has shockable rhythm, shock and then CPR; if they do NOT have shockable rhythm, CPR + epi
- reassess in 2 min
- Whenever pt hits ROSC, swtich to post caidac arrest acre
for more specific directions on med admin, see med admi card
ROSC
treatment goal for cardiac arrest
- return of spontaneous circulation
epinephrie MOA in treatment of cardiac arrest
increase coronary and cerebral perfusion pressure -> enhance orang pefusion
- can help achieve ROSC faster but doesn’t necessarily mean better neuro utcomes
antiarrhythmic agents in treatment of cardiac arrrest
- amio
- lidocaine
- Mg - ONLY if pt has torsadews
antiarrhythmics MOA in treatment of cardiac arrest
potnetially normalzie abnormlaly depolarizing and conducting myocardial cell
amio considerations in cardiac arrest
- do NOT bolus if pt has pulse
- DO bolus if pt is pulseless
- follow admin with 10-20 mL of NS to help get it into circulatoin
- diluent can cause hypotension and bradycardia, consider giving with vasopressor to reduce
- ADR: QT prolongatio risk
remeber that it’s not even used until after 3rd shock
when to consider lidocaine in treatment of cariac arres
- ONLY after 3 shocks
- if amio unavailable
- torsades (dt lower QT prolognation risk than amio)
reversible causes of cardiac arrest: Hs
- hypovolemia
- hypoxia
- hydrogen ion (acidosis) - do NOT treat with bicarb
- hyperkalemia
- hypothermia
- hypoglyceia
reversible causes of cardiac arrest: Ts
- tension pneumothorax
- tamonade, cardiac
- toxins (opioids, local anesthetics, TCAs)
- thrombosis (pulmonary or ardiac)
How to handle hyperkalemia induced cardiac arrest
- treat cardiac arresy (duh)
- treat hyperkalemia
1. give Ca - stabilzes myocardial membrane
2. give temporary measure (bicarb or insulin w/dextrose)
3. once ROSC, consider long term measuers: dialyses, kayexelate, diuresis
what treatment decreases mortality in cardiac arrest
- high qualtiy compressions and appropriate defib
- limited to no med efficacy