critical care Flashcards

1
Q

what is critical care

A

organ system support - single vs multiple

  • level 2 (HDU) - single organ support
  • level 3 (ICU) - multiple organ support

initial assessment - ABCDE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what are the 2 types of respiratory failure

A

1 - oxygenation failure
2 - oxygenation and ventilation failure (failing to clear CO2 aswell)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

management of type 1 resp failure - ward based therapies

A

increase oxygen delivery

  • nasal cannula (2-4L/min)
  • Hudson mask (4-10L/min)
  • non rebreather mask (15L/min)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

management of resp failure - ICU

A

high flow nasal cannula (warms and humidifies air, means you can deliver more /min) - up to 70L/min
NIV - non-invasive ventilator

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what are the advantages of delivering large amounts of oxygen in type 1 resp failure

A

bulk flow of gas and pressure effect - contributes to improving their work of breathing and opening up areas
high flow rate - doesn’t matter how fast/deep patient is breathing, they will still be receiving the set amount of oxygen on the machine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

when are high flow nasal cannulas used

A

type 1 resp failure
patients who are struggling a lot
hypoxic and needing more support than is available on the ward

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

how does NIV work

A

tight fitting mask
machine supplies pressure to support patient’s breathing

helps keep things open and reduce the work of breathing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

m

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

main use of NIV

A

treatment of type 2 resp failure
predominantly COPD patients - CO2 retention

used as a means of avoiding intubation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what method can be used to manage severe resp failure

A

intubation and ventilation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

how does intubation work

A

tube enters trachea
tube is cuffed and forms a seal - pressure of air applied is going straight into lungs and not escaping; protects airway from secretions etc

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what are the advantages of being able to generate high pressures in severe resp failure

A

lungs require lots of pressure to open them back up again (more than NIV or patient can provide)

allows muscles time to rest and recover

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what can be done by the ventilator

A

can alter amount of oxygen going in - precise %
closed system between lungs and ventilator - nothing done with room air

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what are the 2 main outcomes following ventilation

A

infection/resp problem improves and tube is removed

patient requires prolonged ventilation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

disadvantages of being intubated

A

can’t swallow
can’t eat/drink - NG tube for nutrition and drugs
can’t talk
pressure ulcers
dental hygiene issues

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

what is an alternative to prolonged ventilation

A

tracheostomy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

advantages of tracheostomy

A

avoids need for sedation
can speak and eat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

define shock

A

acute circulatory failure w/ inadequate or inappropriately distributed tissue perfusion resulting in cellular hypoxia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

what are the 5 types of shock

A

distributive (septic)
hypovolaemic
anaphylactic
neurogenic
cardiogenic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

what happens in septic shock

A

abnormally dilated blood vessels
blood’s going to the wrong place
heart is still able to pump blood but fluid is in the wrong place

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

what can cause hypovolaemic shock

A

prolonged dehydration
blood loss

not enough blood to go around
body prioritises where the blood will go and other tissues suffer

22
Q

what happens in anaphylactic shock

A

abnormally dilated blood vessels - blood going to the wrong areas
degree of cardiac stunning - pump problem

23
Q

what is neurogenic shock

A

trauma to spinal cord
disruption of symp NS
abnormal vasodilation within peripheral tissues
blood in the wrong place

24
Q

what is cardiogenic shock

A

heart isn’t able to get blood out properly and therefore oxygen into the tissues

25
how do we calculate cardiac output and stroke volume
``` CO = HR X SV SV = preload/contractility/afterload ```
26
managing cardiovascular failure
better monitoring and access
27
how to improve access in CV failure
arterial line: introducer needle, guidewire, cannula - go into artery (radial, brachial, femoral), introduce guidewire into needle, 25g cannula over guidewire central line - sits in IJV, can also be in SC vein or femoral
28
benefits of arterial lines
better reading of BP - beat by beat reading, useful for arrythmias or unstable repeated blood sampling - ABG etc
29
advantages of central lines
able to give different concentrated and potent drugs directly into the central circulation that would cause damage if given peripherally multiple lumens - variety of different amounts of drug through one port aseptic and can stay in 7-10 days - longer duration of access access to central venous circulation - samples and pressure
30
drugs to support CV failure
vasopressors - metaraminol, noradrenaline inotropes - adrenaline, dobutamine
31
what do vasopressors do
case vasoconstriction improve preload by reducing venous volume and returning blood to the heart increase afterload by causing arterial contraction and increasing the pressure the heart is able to generate
32
how does metaraminol work
**alpha 1 agonist** causes vasoconstriction small amount of beta activity - positive contractility in the heart can be given peripherally
33
how does noradrenaline work
predominantly alpha agonsit little bit of beta activity far more potent than metaraminol - give through central line shorter half life than metaraminol therefore more titratable
34
when are vasopressors used
mostly for pts w/ septic shock
35
when are inotropes used
pump dysfunction and contractility issues
36
how does adrenaline work
both alpha and beta agonism alpha - vasoconstriction more beta effect than vasopressors - increased HR and contractility
37
when is adrenaline used
2nd line agent in patients who are really struggling and need additional support
38
when is dobutamine used and how does it work
pure beta agonist - increase HR and contractility used when contractility support is needed but vasoconstriction isn't an issue used as supportive therapy in pts w/ heart failure as a bridge to recovery
39
decisions when using fluids
colloids vs crystalloids maintenance or resuscitation how much - 30ml/kg limit in an acute setting before referring to senior/critical care
40
what is a colloid
less commonly used now fluid w/ large osmotically active molecules within it artificial molecules can cause renal issues, risk of renal failure 2y to colloid use
41
what is a crystalloid fluid
fluids with small molecules in e.g. plasmolyte, Hartmans, 0.9% saline some of the fluid stays in the intravascular tree but lots leaks into the tissues and cells still used for resuscitation
42
what is a problem with 0.9% saline
massive chlorine load - metabolic acidosis if used too much serum chloride should be between 92-99mmol/L 0.9% saline has 154mmol/L of chlorine
43
when are resuscitation and maintenance fluids used
maintenance - someone who can't eat/drink, prevent dehydration resuscitation - intravascularly deplete, markers of developing shock
44
normal regime of fluid administration
250-500mlns at a time use lower end in patients at risk of overload e.g. chronic heart failure etc higher end for otherwise healthy person
45
signs that a fluid challenge is working
HR come down BP gets better urine output does the patient look and feel better
46
what are the 4 types of neurological failure
metabolic trauma infection stroke
47
what is metabolic neurological failure
people who have come in with another disease process and have a reduced conscious level e.g. severe sepsis, inflammation, acute renal failure and uraemia, electrolyte disturbances
48
what is traumatic neurological failure
e.g. traumatic head injuries reduced conscious level that may or may not improve quickly
49
what causes infectious neurological failure
CNS infections meningitis, encephalitis
50
why is neurological failure seen in critical care
problems with the patients ability to breathe, ventilate and protect the airway
51
what can happen with traumatic brain injuries and swelling
brain swelling if left unchecked can cause severe catastrophic neurological damage up to and including coning can be fatal
52
management of traumatic head injuries to prevent swelling
ventilate the patient appropriately - reduce CO2 level to reduce intracranial pressure give patient enough oxygen to reduce intracranial pressure prevent fluid overload but give enough to have decent blood pressure going into the head