Cranial Nerves: History + Exam Flashcards

1
Q

What associated symptoms should be asked about in the SOCRATES review of a headache?

A
n&v
altered consciousness
rash
pyrexia
neck stiffness
photophobia
visual loss, blurred vision
aura, seizures
tender scalp
malaise
rhinorrhoea, lacrimation
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2
Q

What conditions might be in the differential diagnosis if symptoms are relapsing and recurring?

A

migraine
epilepsy
MS

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3
Q

What exacerbating/relieving should be asked about in the SOCRATES review of a headache?

A

exacerbating: noise, stress, bending, standing up, coughing, sneezing, blowing nose, eating, combing hair, bright/flashing lights, certain foods/drugs, dehydration
relieving: analgesia, dark environment, lying down, rest/sleep

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4
Q

What qs are important to ask in the history of an episode of altered consciousness?

A

describe in own words

onset - gradual/sudden? time of day?
what were they doing at the time? - pain, infections, hot crowded room, emotional stress, prolonged standing?
how they felt before the episode?

associated symptoms

recovery time? amnesia, aggression, crying or weakness afterwards? feeling sad/crying after episode?
previous episodes? were they like this one?
was it witnessed? what did they say, can we contact them?

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5
Q

What associated symptoms should be asked about in the history of an episode of altered consciousness?

A

dizziness, nausea, vertigo, aura, tachycardia, sweating, weakness, paraestoesia, slurred speech, headache, tongue biting/incontinence, stiffening/jerking of limbs, awareness/responsiveness during episode, eyes open or closed, groans or crying?

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6
Q

What is the most important factor to ask about any neurological symptom?

A

course

sudden in onset? how long to reach peak of symptoms? getting better or worse? static/progressive/relapsing + remitting?

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7
Q

What conditions should be specifically asked about in the PMH?

A
head/spinal trauma 
metabolic/endocrine disorders e.g. diabetes
cancer, mets?
epilepsy
HTN
AF
heart diseases
previous episodes?
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8
Q

What medications should be specifically asked about in the drug history?

A
anticonvulsants
drugs that interact with anticonvulsants/lower seizure threshold
anticoagulants/anti-platelets
analgesics
antihypertensives
antidepressants
insulin
recreational drugs
over the counter drugs
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9
Q

Whats factors are important to ask about in the social history?

A

alcohol consumption
smoking
recreational drugs
occupation
social activities/hobbies/daily living/driving
home circumstances/independence/family support/housing

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10
Q

Why is alcohol important to ask about?

A
can cause:
seizures
neuropathy
ataxia
Wernick-Korsakoffe syndrome (lack of thiamine)
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11
Q

What conditions should be specifically asked about in the family history?

A
diabetes
cerebral haemorrhage
cerebrovascular disease/stroke
ischaemic heart disease 
migraine
epilepsy
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12
Q

What 4 things need to be tested in a neurological exam?

A
  1. walking
  2. cranial nerves
  3. motor system
  4. sensory system
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13
Q

What does a Parkinson’s gait look like?

A
stooped posture, rigidity
small shuffling steps
wrists, elbows, hips and knees flexed
no arm swing
resting tremor
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14
Q

What does a hemiplegic gait look like?

A

weakness on one side > weak arm hands, drags 1 leg behind

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15
Q

What does a scissoring gait look like?

A

knees can flex and knock together

restricted stiff movement

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16
Q

What can cause a scissoring gait?

A

any condition causing spasticity

e.g. cerebral palsy

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17
Q

What does a steppage gait look like?

A

foot drops and toes drag so hips and knees flex excessively (high stepping)
foot stamps

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18
Q

What can cause a steppage gait?

A

common perineal nerve palsy

= weakness of dorsiflexion > high step to avoid falling

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19
Q

How is gait examined?

A
  1. normal heel to toe walk - look for posture, balance, stride length, arm swing
  2. tandem walk test - look for same things
  3. Romberg test - stand feet together with eyes closed, dr stands close, +ve = fall
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20
Q

What does a +ve Romberg test indicate?

A

proprioception problem

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21
Q

What is the first step in a cranial nerve examination?

A

general inspection of patient’s head and neck

look for: scars, neurofibromas, facial asymmetry, ptosis, proptosis, skew deviation of the eyes, inequality of the pupils

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22
Q

How is CN I: olfactory nerve examined?

A

ask if they have had any alteration in their sense of smell/taste > only test further if they report problems

olfactory testing bottles/easily recognised scents e.g. soap, coffee
test each nostril separately, occluding the other one with your finger

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23
Q

How is CN II: optic nerve examined?

A
  1. visual acuity
    - snellen chart if available
    - if not: cover one eye, ask them to state how many fingers you’re holding up/different font sizes from a newspaper
    - wear glasses if they usually do
    - if no glasses: pinhole test > won’t correct acuity in people with an optic nerve issue
  2. visual fields
    - sit 1m opposite pt with eyes at same level
    - cover one of own eyes, pt covers eye opposite
    - ask pt to stare into your other eye
    - bring object/finger from point outside visual field diagonally into visual field
    - ask them to signal when they first see it
    - repeat in all 4 quadrants for each eye
  3. visual inattention/visual extinction
    - hold hands up in periphery of pts vision
    - move tip of index finger up and down on one side at a time, then on both sides together
    - ask pt to report which side is moving
    - visual inattention/extinc = can detect if one is moving individually, ignores one side when both move at same time
  4. direct and consensual light reflexes
    - pt looks straight ahead
    - bring light in from side so pt doesn’t focus on it
    - shine light into eye and look for constriction of that pupil (direct light reflex) and the contralateral one (consensual light reflex) = both constrict
  • check relative afferent pupillary defect
  • swing flashlight between eyes > should remain constricted
  • if one dilates = RAPD (problem in ipsilateral optic nerve)
  1. accommodation
    - pt focuses on a distant point, then your finger, held - 30cm in front of their nose
    - normal = constriction of both pupils
  2. ophthalmoscopy, testing colour vision, assessing size of blind spot
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24
Q

On fundoscopy, what might a lost red reflex indicate?

A

cataracts

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25
A lesion in the left optic nerve will cause what kind of visual field defect?
left eye vision loss (ipsilateral)
26
A lesion in the optic chiasm will cause what kind of visual field defect?
bitemporal hemianopia | outer fields of both eyes
27
A lesion in the right optic tracts will cause what kind of visual field defect?
left homonymous hemianopia (contralateral) | left field of both eyes lost
28
A lesion in the left optic radiations will cause what kind of visual field defect?
right homonymous hemianopia with macular sparing (contralateral) (right field of both eyes lost with circle in middle spared)
29
What are the 3 symptoms of Horner's syndrome?
ptosis (drooping of eyelid) miosis (constriction of pupil) anhydrosis = on affected side
30
What happens to the eye in 3rd nerve palsy?
ipsilateral: eyeball deviated out + down (superior rectus palsy) mydriasis (pupils dilated) ptosis > eye looks closed
31
What happens to the eye in 4th nerve palsy?
ipsilateral eye elevates (superior oblique palsy) | contralateral head tilt to lessen diplopia
32
If both eyelids are closed, what condition is it more likely to be?
something affecting the muscles | myasthenia?
33
How are CN III: oculomotor, IV: trochlear and VI: abducens nerves examined?
- ask pt to keep head still and follow finger with their eyes (midway between you and pt) - trace a large H shape and central I shape - take pts eyes to limit of their gaze as you do - ask if they experience diplopia at any point - observe for dysconjugate eye movements, nystagmus
34
How is CN V: trigeminal nerve examined?
1. sensation in skin - demonstrate stimulus (cotton wool) on sternum withers open, check they can feel it - pt closes eyes, check they can feel it on each dermatome of CN V - can they feel it, is there a difference in the sensation between the 2 sides of the body? 2. motor component - clench teeth + palpate contraction of masseter and temporalis muscles - pt to open mouth against resistance: look for jaw deviation > towards side of paralysed pterygoid 3. jaw jerk - ask pt to half open mouth - put index finger vertically over midline of lower jaw - tap on index finger with tendon hammer - sudden brisk closure > UMN lesion above pons 4. corneal reflex - not usually done - touch eye with cotton wool, normal = involuntary blinking
35
What does CN V innervate?
sensory: facial + corneal sensation motor: jaw closure, mastication
36
What are the 3 branches of CN V called?
ophthalmic maxillary mandibular
37
On opening against resistance, the pt's jaw deviates to the right. What is this a sign of?
right paralysed pterygoid | = CN V palsy
38
Pt presents with a brisk jaw jerk. What might this indicate?
UMN lesion above pons
39
How is CN VII: facial nerve examined?
look for asymmetry/appearance of face, especially forehead, obicularis oculi muscles 1. screw up eye, don't let them be opened by you 2. puff out cheeks, maintain on your resistance 3. raise eyebrows > wrinkles indicate intact nerve supply, no elevation on side of UMN lesion 4. purse lips 5. show their teeth, count teeth on each side, weak side = more covered
40
What kind of UMN supply do muscles of the upper face have compared to muscles of the lower face? What affect does this have on the impact of a lesion?
``` upper face (frontal, obicularis oculi): bilateral UMN supply, can be spared in UMN lesions e.g. stroke = forehead sparing on side of lesion always ``` lower face: unilateral UMN supply, weakened on the side of the UMN lesion = BELL'S PALSY
41
Pt presents with unilateral loss of taste and hyperacusis on the same side. What does this indicate?
LMN lesion
42
What can trigger trigeminal neuralgia?
cold, touch, shivering, shaving, eating, drinking
43
What is Bell's palsy?
lower VII lesion, all facial muscles of affected side are weak can be due to a UMN or LMN lesion
44
A pt presents with facial drop/asymmetry. Is the lesion in the brain or the nerve itself?
if in the brain > UMN lesion e.g. stroke | if in the nerve itself > LMN lesion
45
How does Bell's palsy due to a UMN lesion present?
forehead sparing > can close eye, wrinkles seen affects the CONTRALATERAL side facial muscle weakness in lower half of face due to bilateral UMN innervation
46
How does Bell's palsy due to a LMN lesion present?
all facial muscles affected, no forehead wrinkles, facial droop can't close eye, see whites of eye affects IPSILATERAL SIDE
47
How is CN VIII: vestibulocochlear nerve examined?
1. cover opposite ear with hand, whisper a number to the pt, ask them to repeat it abnormality suspected? 2. Rinnes and Weber's tests to determine if it's a sensory or conductive defect 3. dizziness = common symptom - vestibule ocular reflex/Hall pike and Unterberg could be done - vor: pt focus on your nose, jerk head fast to L/R for them, their eyes should stay focussed on your nose - peripheral problem = eyes move in same direction as jerk then do a corrective jerk/nystagmus
48
How are CN IX: glossopharyngeal and CN X: vagus nerves examined?
1. ask pt to open their mouth wide, is the uvula in the midline at rest? 2. ask pt to say 'aah', note asymmetry of movement > uvula will deviate away from the CN IX/X palsy 3. does pt have any difficult swallowing? (gag reflex doesn't need to be performed routinely) 4. observe drinking a sip of water > problem if they cough/choke 5. ask pt to cough > bovine (non-explosive, weak) cough = vagal palsy 6. note any hoarseness of the voice
49
A weak bovine cough indicates a lesion in which CN?
X | due to inability to close glottis
50
Pt presents with uvula deviation to the left. Which nerve has been affected?
right vagus pulling muscles so the working muscles pull it to their side
51
How is CN XI: accessory nerve examined?
1. test trapezius: shrug shoulders against resistance > look at back for asymmetry/scapula winging 2. test sternocleidomastoid: turn head against resistance, palpate during
52
What does CN XI innervate?
trapezius + sternocleidomastoid
53
Pt presents with winging of the scapula. Why might this happen?
weakness of trapezius due to VIth nerve palsy
54
How is CN XII: hypoglossal nerve examined?
1. ask pt to open mouth and observe tongue for fasciculation 2. protrude tongue, any deviation? > deviation occurs towards the side of a lesion 3. ask pt to push tongue into their cheek against the resistance of your finger, assess power
55
What signs are seen in CN XII palsy?
ipsilateral atrophy/wasting | deviation towards side of lesion
56
What is Rinne's test? What are the findings in conductive hearing loss compared to sensorineural hearing loss?
- tuning fork held on mastoid until sound is no longer heard - then held near external acoustic meatus, sound should continue to be heard as air > bone conduction conductive hearing loss: bone conduction > air conduction, sound not heard at the EAM sensorineural hearing loss: air + bone conduction decreased by similar amount
57
What is Weber's test? What are the findings in conductive hearing loss compared to sensorineural hearing loss?
- tuning fork held against forehead in midline - vibrations normally perceived equally in both ears conductive hearing loss: sound louder in abnormal ear sensorineural hearing loss: sound louder in normal ear sensitivity increased by pt blocking external ear canals with fingers if +ve > do rinne's test
58
How is meningism tested for? What is it's pathognomonic feature? What can it indicate?
1. ask about neck pain > whilst lying, touch chin to chest 2. you support head with hands, move back and forth > check for stiffness and pain (incl abdominal pain) = won't affect SIDE TO SIDE movement (must be MSK) indicates meningitis, SAH
59
How does an UMN lesion present?
everything INCREASES part of the motor system in the brain + spinal cord increased spastic tone pyramidal pattern of weakness increased reflexes
60
What 2 types of increased muscle tone are there?
1. spasticity - first increase then decrease in tone - typical for UMN lesions 2. rigidity - increased tone over entire radius of joint movement - present in parkinson's
61
How does an LMN lesion present?
everything DECREASES in the PNS > horn, roots, plexus decreases muscle mass (atrophy + fasciculations - check tongue) decreased tone peripheral pattern of weakness decreased reflexes