Cranial nerves Flashcards

1
Q

What makes up a cranial nerve exam

A

General inspection
Assessment of each cranial nerve

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2
Q

General inspection

A

Speech abnormalities: may indicate glossopharyngeal or vagus nerve pathology.

Facial asymmetry: suggestive of facial nerve palsy.

Eyelid abnormalities: ptosis may indicate oculomotor nerve pathology.

Pupillary abnormalities: mydriasis occurs in oculomotor nerve palsy.

Strabismus: may indicate oculomotor, trochlear or abducens nerve palsy.

Limbs: pay attention to the patient’s arms and legs as they enter the room and take a seat noting any abnormalities (e.g. spasticity, weakness, wasting, tremor, fasciculation) which may suggest the presence of a neurological syndrome).

Look for objects or equipment on or around the patient that may provide useful insights into their medical history and current clinical status:

Walking aids: gait issues are associated with a wide range of neurological pathology including Parkinson’s disease, stroke, cerebellar disease and myasthenia gravis.

Hearing aids: often worn by patients with vestibulocochlear nerve issues (e.g. Meniere’s disease).

Visual aids: the use of visual prisms or occluders may indicate underlying strabismus.

Prescriptions: prescribing charts or personal prescriptions can provide useful information about the patient’s recent medications.

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3
Q

Cranial nerves

A

1- Olfactory (S)
2- Optic nerve (S)
3- Oculomotor (M) PARASYMPATHETIC
4- Trochlea (M)
5- Trigeminal (B)
6- Abducens (M)
7- Facial (B) PARASYMPATHETIC
8- Vestibulocochlear (S)
9- Glossopharngeal (B) PARASYMPATHETIC
10- Vagus (B) PARASYMPATHETIC
11- Accessory (M)
12- Hypoglossal (M)

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4
Q

Olfactory nerve (CN1) Foramen and fissure (F&F)

A

Foramen/Fissure – Cribriform plate
Function – sense of smell
Sensory

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5
Q

CN1 test and outcomes

A

Ask Pt: How they noticed any changes in their sense of smell? If no ask about taste to confirm, more of a recognisable loss.
Smell test - use different odours (Citrus, mint or coffee). Unilateral test – cover 1 nostril and patient closes their eyes)

Outcomes –
Anosmia – Complete loss of smell due to olfactory nerve damage (Genetics, Parkinsons, fracture of the Cribriform plate, SOL.)
Parosmia – Distorted sense of smell (bacterial or viral infections)
Hyperosmia – overwhelming sensitivity to smells (migraine, genetics, Epilepsy)

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6
Q

Optic nerve (CN2) F&F

A

Foramen/Fissure – Optic foramen
Function – Sight
Sensory

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7
Q

CN2 Tets

A

Visual acuity - Snellen wall chart (Unilateral test). Patient covers one eye and reads down the chart until the 6th line if possible.

Visual fields test - Practioner covers opposite eye to patient. Starting with the right eye (patient) using your left arm take it into the periphery and flex your first digit. Pt should be staring at a fixed point (i.e your nose). Following that, with the same eye, use you right arm, perform the same process. Then switch eyes.

Pupillary reflexes - Patient will cover the mid point in their face creating a divide between the eyes. Shine the light in one eye and observe bi-lateral constriction. Progress then to ‘swinging light test’. Patient will need to focus on a point in front of them, not directly the light.

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8
Q

CN2 outcomes

A

Visual acuity - If the patient reads the 6/6 line but gets two letters incorrect, you would record as 6/6 (-2). Could be due to age related muscular degeneration or optic neuritis

Visual fields - Information from the L optic nerve from the L medial field (Nasal field) towards the chiasm but remains on the same side before heading to the visual cortex via the L geniculate body. Information from the L optic nerve from the R lateral field (temporal field) heads towards the chiasm and desiccates and heads towards the L geniculate body. Due to the nature of the anatomy, results can be confusing.

Pupillary reflex - Direct response suggests the optic nerve is receiving information and the oculomotor nerve is constricting the pupil. Consensual response but no direct response suggests that the optic nerve is receiving the signal but the oculomotor is failing to constrict the pupil. Absent pupillary reflex suggest the optic nerve is not recognising the stimulus. Relative afferent pupillary defect, optic nerves is damaged, both pupils will constrict less when light is shone into the affected eye compared to the healthy eye (large retinal detachment or optic neuritis)

If a defect is found – refer to an optician.

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9
Q

Oculomotor nerve (CN3)

A

Foramen/Fissure - Superior orbital fissure
Function – The oculomotor nerve supplies all extraocular muscles except the superior oblique (CN4) and the lateral rectus (CN6)
Motor
 PARASYMPATHETIC – Pupillary reflex
 Elevation of the upper eyelid

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10
Q

CN3 tests and outcomes

A

H-Test
Light pupillary reflex - Ask the patient to focus on a target approximately half a metre away whilst you shine a pen torch towards both eyes.Inspect the corneal reflex on each eye: If the ocular alignment is normal, the light reflex will be positioned centrally and symmetrically in each pupil. Deflection of the corneal light reflex in one eye suggests a misalignment.

Outcomes –
 Ptosis (Innervation to the levator palpebrae superioris) - (Horner’s syndrome, Myasthenia gravis, muscle damage)
 Down and out (Divergent squint)
 Pupil dilation
 Eye movements restricted

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11
Q

Trochlea Nerve (CN4) F&F

A

F Foramen/Fissure - Superior orbital fissure
Function - Innervates the superior oblique (ability to look down).
Motor

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12
Q

CN4 Test and outcomes

A

H-Test

Outcomes - Vertical diplopia - when looking inferiorly, due to loss of the superior oblique’s action of pulling the eye downwards
 Diplopia common when going downstairs or reading when palsied.

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13
Q

Trigeminal nerve (CN5) F&F

A

Foramen/Fissure:
Ophthalmic (Superior orbital fissure)
Maxillary (Foramen Rotundum)
Mandibular (Foramen Ovale)

Both sensory and motor
Function –
O - Sensory to scalp, forehead, nose and upper eyelid.
Mx – Sensory to palate, upper jaw, upper teeth, lower eyelid and nasal cavity
Ma – Sensory to lower lip, lower teeth, anterior 2/3 of tongue, lateral cheek, temporal region. Motor to muscles of mastication, digastric, mylohyoid and the middle ear.

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14
Q

CN5 tests

A

Sharp and soft sensory test. (Pin & cotton wool) V1,V2,V3) - should be performed in all 3 regions. To ensure the patient understands what is normal, place the cotton wool/pin prick on their sternum or hand for reference. Patient closes eyes and states “sharp” or “soft”. Need to be able to visualise the three separations of the trigeminal nerve:

Mastication test (V3) - Ask the patient to chew. Encourage them to move from left to right or in a circular motion. Palpate the temporalis, masseter and pterygoids.

Jaw reflex - Place your left index finger on their chin and striking it with a tendon hammer. This should cause slight protrusion of the jaw. Brisk jaw jerk could be sign of bilateral UMN lesion
Corneal reflex

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15
Q

CN5 outcomes

A

 Loss of sensation
 Loss of distinction between sharp and soft
 Weakness or inability to masticate
 increased reflex (mouth closing)
 Loss of blinking reflex
 Trigeminal neuralgia - sudden, severe facial pain. It’s often described as a sharp shooting pain or like having an electric shock in the jaw, teeth or gums.
 Often associated with MS.

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16
Q

Abducens nerve (CN6) F&F

A

Foramen/Fissure - Superior orbital fissure
Function - Innervates the lateral rectus. (ability to look laterally.
Motor

17
Q

CN6 tests and outcomes

A

H-Test

Outcomes –
Medial gaze – If the lateral rectus fails, the medial rectus will pull the eye inwards.
Inability to look laterally
Strabismus – where eyes point in different directions. (Eye alignment). Not to be confused with Amblyopia (lazy eye)

18
Q

Facial nerve (CN7) F&F

A

Foramen/Fissure - Internal auditory meatus/Stylomastoid foramen

Both sensory and motor - PARASYMPATHETIC
Function – Sensory taste from the anterior 2/3 of the tongue. Sensation (Part) of external ear and palate. Motor to muscles of facial expression. Innervation of Stapedius. Parasympathetic innervation of salivary glands (sub-mandibular and sub-lingual), lacrimal gland.

19
Q

CN7 test and outcomes

A

Ask the patient about their taste, hearing and saliva, tears.
Facial expression test- Ask the patient to perform a series of facial expressions (smile, pursed lips) eyebrow raise). Utilise resistance. Resist patient opening eyes. Blowing out cheeks.

Outcomes -
 Hypoguesia (reduced taste ability) ageusia (loss of taste)
 Loss of hearing noted (tested in conjunction with CN8) (Ramsey Hunt syndrome - Herpes Zoster effects the facial nerve)
 Facial expression test -Facial palsy (Bell’s palsy or stroke)

20
Q

How to distinguish between an UMNL & LMNL in facial paralysis

A

Pt wont be able to control the frontalis and move the eyebrow up if its a LMNL e.g. Bells palsy.
But they are able to in the case of an UMNL e.g. stroke.
Bell’s palsy the same side as palsy stroke opposite

21
Q

CN7 Patho

A

Damage to innervation can arise from upper and lower motor neuron lesions

Facial nerve palsy caused by a lower motor neuron lesion presents with weakness of all ipsilateral muscles of facial expression, due to the loss of innervation to all muscles on the affected side. The most common cause of lower motor neuron facial palsy is Bell’s palsy.

Facial nerve palsy caused by an upper motor neuron lesion also presents with unilateral facial muscle weakness, however, the upper facial muscles are partially spared because of bilateral cortical representation (resulting in forehead/frontalis function being somewhat maintained). The most common cause of upper motor neuron facial palsy is stroke.

22
Q

Vestibulocohlear CN8 F&F

A

Foramen/Fissure - Internal auditory meatus
Function - Sense of hearing and balance
Sensory

23
Q

CN8 test

A

Ask the patient about their hearing

Numbers repetition test

Weber’s – 512hz tuning fork on the forehead. Can you hear the noise? Should be the same in both ears. If they hear the noise louder in one ear that means: Conductive deafness in ear perceiving the louder sound OR Sensory neural deafness in the other ear.

Rinne’s test – 512 hz tuning fork onto the mastoid process (bone conduction). By the ear (air conduction). Ask the patient if they can hear it. When diminished, bring away from the mastoid process and place outside of the external auditory meatus and ask the patient if they can hear it, air conduction is stronger than bone conduction (normal) confusingly referred to as a “Rinne’s positive” result (air conduction)

Rinne’s and Weber’s are always tested alongside one another

24
Q

CN8 outcomes

A

Numbers test – Air conduction loss
Weber’s - If they hear the noise louder in one ear that means conductive deafness in ear perceiving the louder sound OR sensory neural deafness in the other ear.
Rinne’s – Bone conduction or air conduction loss.

25
Q

Glossopharyngeal & Vagus nerve (CN9 & 10) F&F

A

Foramen/Fissure - Jugular faramen (CN9 & CN10)

Both sensory and motor + PARASYMPATHETIC
Function –
* CN9 - provides motor, parasympathetic and sensory information to your mouth and throat. Among its many functions, the nerve helps raise part of your throat, enabling swallowing.
* CN10 vagus nerve is responsible for the regulation of internal organ functions, such as digestion, heart rate, and respiratory rate, as well as vasomotor activity, and certain reflex actions, such as coughing, sneezing, swallowing, and vomiting-

26
Q

CN9/10 tests

A

Ask patient to cough – can they cough properly from the back of the throat? Is there a Bovin cough.
 Recognise if their voice sounds hoarse.
 Swallowing test - Ask patient to drink a sip of water and observe the ability of the pharynx. Afterwards, has their voice been affected by the water? If so, the glottis may not be functioning.

Test for uvula deviation (“Ah”) Ask the patient to say “ahh“: Inspect the palate and uvula which should elevate symmetrically, with the uvula remaining in the midline. A vagus nerve lesion will cause asymmetrical elevation of the palate and uvula deviation away from the lesion.

Gag reflex. Very uncomfortable and unpleasant. This should not be performed unless there is an overriding reason to do so.

27
Q

CN9/10 Outcomes

A

 Uvula deviation away from the palsied side.
 Weakness in the patient’s voice
 Loss of gag reflex
 Dysphagia

28
Q

Accessory nerve (CN11)

A

Foramen/Fissure - Foramen Magnum/Jugular foramen

Function - Motor to pharynx, trapezius and sternocleidomastoid (SCM).
Motor

29
Q

CN11 Test and outcomes

A

SCM test (Patient rotates their head against practitioner resistance).
Trapezius test (Patient shrugs shoulders against practitioner resistance)

Outcomes –
 Weakness
 Muscle wasting

30
Q

Hypoglossal nerve (CN12) F&F

A

Foramen/Fissure - Hypoglossal canal
Function - Motor to the tongue and throat muscles

31
Q

CN12 tests and outcomes

A

Resisted tongue movement
Tongue protrusion

Outcomes -
Tongue deviation - In hypoglossal palsy, the tongue will deviate towards the palsied side.
Tongue weakness
Motor