Course 2 Flashcards
pertinent positives
Specific symptoms that raise the physician’s suspicion for a particular disease.
Pertinent Negatives
Specific symptoms that are not present which cause the physician to doubt certain diagnoses.
How do physicians rule out certain diseases?
They will order a specific Objective study that can diagnose it or rule it out. Also the physical exam can rule out some diseases.
What DDx can be drawn from chest pain (the subjective complaint)?
PE, Musculor-skeletal chest pain, MI.
Risk Factors
What puts the patient at risk
Chief Complaint (CC)
Typical major sx. Want to know Timing (constant, intermittent, waxing and waning), quality (dull, sharp, pressure, cramping), what makes it better or worse.
Assoc Sx
pertinent positives; sx that raise the physician’s suspicion for the disease.
Pert. Neg
pertinent negatives; important sx that are not present.
Assoc MEd
medications related to the disease.
PE
Common physical exam findings associated with the disease.
Diagnosed by
how the disease is ruled out or diagnosed
CAD Etiology
Narrowing of the coronary arteries limits blood supply to the heart muscle causing angina.
Angina
Chest pain specifically due to heart-muscle ischemia. Sx of CAD
CAD CC
CP or chest pressure. worse with exertion, improved by rest and NTG.
CAD Assoc Med
Aspirin (ASA) 324mg PO
Nitroglycerin (NTG) 0.4mg SL
CAD diagnosed by
Cardiac catheterization ( not in the ED)
A patient has CAD if they…
Have a PMHx of Angina, MI, CABG, Cardiac stents, angioplasty.
Any patient complaining of CP should..
Receive ASA PO unless given PTA.
MI etiology
acute blockage of the coronary arteries results in ischemia and infarct of the heart muscle.
CAD catch phrase
chest pain with physical exertion.
MI Catch phrase
Chest pressure with diaphoresis, N/V, and SOB
MI Risk Factors
CAD, HTN, HLD, DM, smoker, FHx of CAD <55
MI CC
Chest pain or chest pressure
MI study
EKG (STEMI) or elevated Troponin (non-STEMI)
MI Assoc Med
ASA NTG B-blocker Thrombolytic (Heparin)
B-blocker
Reduces blood pressure by blocking epinephrine. Makes beat more slowly and less force.
CHF Etiology
The heart becomes enlarged, inefficient, and congested with excess fluid
CHF Catch phrase
SOB with pedal edema and orthopnea
CHF CC
SOB, orthopnea, PND, DOE
orthopnea
SOB worsen when lying down
PND
Paroxysmal Nocturnal Dyspnea - SOB at night
CHF PE
Rales, JVD in nec, Pitting pedal edema
CHF assoc Med
diuretics (Lasix, Furosemide) - unrinate extra fluid.
CHF study
CXR or elevated BNP
A FIb CC
Palpitations (Fast, Pounding, Irregular)
AFib risk factors
Paroxysmal A Fib, Chronic A Fib
AFib PE
Irregularly irregular rhythm, Tachycardia
AFib study
EKG
AFib Assoc Med
Coumadin (Warfarin): Blood thinner, prevents blood clots in atria. Digoxin: Slows down heart rate
pericarditis
Inflammation of the sac surrounding
the heart causing CP
Pleurisy
Inflammation of the sac surrounding
the lungs causing pleuritic CP
Costochondritis
Irritation of the ribs causing CP
worsened by pressing on the sternum
Chest Wall Pain
Irritation of the chest wall causing pain
with palpation of the chest
Pleural Effusion
Fluid collecting around the lungs causing
SOB or CP
AFib
electrical problem, irregular HR
MI
Heart attack
CHF
fluid traffic jam
CAD
Risk for MI
PE ET
A blood clot becomes lodged in the pulmonary artery and blocks blood flow to the lungs
PE catch phrase
Pleuritic chest pain with tachycardia and hypoxia
PE risk factors
Known DVT, PMHx of DVT or PE, FHx, Recent surgery, Cancer, A-Fib, Immobility, Pregnancy, BCP, Smoking
PE CC
SOB or Pleuritic chest pain (CP worse with deep breaths)
PE study
CTA Chest (CT Chest w/ IV contrast) or VQ scan. D-dimer aids in detecting clots, but cannot diagnose a PE.
PNA ET
Infiltrate (bacterial infection) and inflammation inside the lung
PNA Risk Factors
Elderly, Bedridden, Recent chest injury, Recent surgery
PNA CC
SOB or Productive cough
PNA Assoc Sx
Cough with sputum, Fever, Chest pain
PNA Assoc Med
Rocephin and Zithromax (antibiotics)
PNA PE
Rhonchi
PNA study
CXR
Pneumothorax
PTX
PTX Et
Collapsed lung due to trauma or a spontaneous small rupture of the lung
PTX CC
SOB and one-sided chest pain
Sudden onset. Often trauma patients.
PTX PE
Absent breath sounds unilaterally
PTX study
CXR
COPD Et
Long-term damage to the lung’s alveoli (emphysema) along with inflammation and mucous production (chronic bronchitis)
COPD Risk factors
smoking
COPD CC
SOB
COPD PE
Decreased breath sounds, Wheezes, Rales
COPD Assoc Meds
Home O2 (Document how much O2 they use at baseline)
COPD study
CXR hx smoking
Reactive Airway Disease (RAD) Et
(asthma) Constricting of the airway due to inflammation and muscular contraction of the bronchioles, known as a “bronchospasm”
RAD CC
SOB/Wheezing
Improved by nebulizer “breathing treatments” (bronchodilators)
RAD PE
Wheezes (Inspiratory or Expiratory)
RAD Assoc med
Inhalers, Nebulizers, Corticosteroids
RAD study
clinically
I-CVA Et
Blockage of the arteries supplying blood to the brain resulting in permanent brain damage
I-CVA CC
Unilateral focal neurological deficits: One-sided weakness/numbness or changes in speech/vision
I-CVA Risk Factors
HTN, HLD, DM, hx TIA/CVA, Smoking, FHx CVA, AFIB
I-CVA PE
Neurological deficits: hemiparesis, unilateral paresthesias, aphasia, visual field deficits
I-CVA study
Clinically, Potentially normal CT Head
H-CVA ET
Traumatic or spontaneous rupture of blood vessels in the head leads to bleeding in the brain
H-CVA CC
Headache Sudden Onset (Thunderclap, Worst of life)
H-CVA Assoc Sx
Changes in Speech, Vision, Sensation (numbness), or Motor strength (weakness), AMS, Seizure, Headache
H-CVA PE
Unilateral neurological deficits
H-CVA study
CT head or LP
TIA ET
Vascular changes temporarily deprive a part of the brain of oxygen (Symptoms usually last less than 1 hour)
TIA CC
Transient focal neurological deficit
Changes in Speech, Vision, Strength, or Sensation
TIA Study
clinically
Meningitis Et
Inflammation and infection of the meninges; the sac surrounding the brain and spinal cord
Bacterial vs. viral
Meningitis CC
Headache and neck pain.
Meningitis Assoc Sx
fever, neck pain, neck stiffness, AMS
Meningitis PE
Meningismus, Nuchal rigidity
Meningitus study
LP
Spinal cord injury Et
Injury to the spinal cord may create weakness or numbness in the extremities past the site of the injury
Spinal cord injury cc
Neck pain or Back pain, Bilateral extremity weakness
Spinal cord injury PE
Midline bony tenderness, deformities, or step-offs, Bilateral extremity weakness, Numbness, Decreased rectal tone
Spinal cord injury study
CT Cervical Spine (Neck)
CT Thoracic Spine (Upper back)
CT Lumbar Spine (Lower back)
Seizure (SZ) Et
Abnormal electrical activity in the brain leading to abnormal physical manifestations. Often caused by epilepsy, ETOH withdrawals, or febrile seizure in pediatric pts
SZ CC
Seizure activity, Syncope
SZ assoc Sx
Injuries (tongue bite), Confusion, Headache,
Incontinence (urinary or fecal)
SZ PE
Somnolent, Confused (Post-Ictal)
SZ Med
Dilantin, Tegretol, Keppra, Depakote, Neurontin
Bells Palsy Et
Inflammation or viral infection of the facial nerve causes one-sided weakness of the entire face
Bells Palsy CC
Facial Droop
Sudden Onset
Bells Palsy Assoc Sx
Jaw or ear pain, Increased tear flow of one eye
Bells Palsy Pert Neg
No extremity weakness, No changes in speech or vision.
Bell Palsy PE
Unilateral weakness of the upper and lower face
Bell Palsy Study
clinical
HA Et
Various causes including hypertensive headaches (from high blood pressure), recurrent diagnosed migraines, Sinusitis, etc.
HA CC
Headache (gradual onset)
Pressure, Throbbing
HA Pert Neg
No fever; No neck stiffness; No numbness/weakness; No changes in speech or vision
AMS Et
Multiple causes: most common are hypoglycemia, infection, intoxication, and neurological
AMS Risk Factors
Diabetic, Elderly, Demented, EtOH use, Drug use
AMS CC
Confusion, Decreased responsiveness, Unresponsive
AMS study
case dependent