Core Radiology MSK Flashcards

1
Q

H shaped vertebra are due to AVN of the endplates which is present in both Gaucher’s and Sickle cell. How can you differentiate?

A

Gaucher = hepatosplenomegaly
Sickle cell = Autosplenectomy

Gaucher = Erlenmeyer flask deformity (metaphyseal flaring)

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2
Q

Diffuse sclerosis of the bones with rugger jersey spine, what’s the diagnosis?

A

Osteopetrosis

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3
Q

What’s hereditary hyperphosphasia and how is the radiological features different from Paget’s disease?

A

Child Paget’s with trabecular and cortical thickening, bowing of the legs and osteopenia
HOWEVER there is epiphyseal sparing

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4
Q

Another name for lincon log vertebra?

A

H shaped vertebra

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5
Q

Case with marrow expansion, what’s your top differential?
How does marrow expansion look like in the
Hand
Skull
Facial bones
Long bones

A

Thalassemia

Hand: widening and squaring of the phalanx and metacarpals
Skull: hair on end
Facial bones: Obliteration of the sinuses > rodent face
Long bone: Erlenmeyer flask deformity (metaphyseal flaring)

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6
Q

What does myelofibrosis look like?

A

Demo: Old patient
Radiograph: diffusely sclerotic bones
Other findings: anemia and splenomegaly

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7
Q

What are the types of stress fractures?

A

Fatigue and Insufficiency
Insufficiency = bone is insufficient but stress is normal (bone abnormal, stress normal)
Fatigue = bone is fatigued from repetitive stress however bone is normal (bone normal, stress abnormal)

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8
Q

What is a pathological fracture?

A

Normal stress on bone weakened by underlying lesion/Pagets/Infection

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9
Q

Which sequence is best used to avoid magic angle phenomenon when visualizing the patellar tendon?

A

T2

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10
Q

What is the MRI presentation of tenosynovitis and what is a common pitfall?

A

Fluid tracking around the tendon circumferentially
Pitfall; biceps tendon with fluid tracking.

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11
Q

What are the causes of tenosynovitis?

A

Repetitive motion or surrounding inflammation (infection/inflammatory arthritis)

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12
Q

What is the MRI presentation of tendinosis and what is it’s other name?

A

Tendinosis = Myxoid degeneration
MRI: Intermediate intrasubstance signal, with normal or enlarged tendon.

NOTE: if fluid signal is seen then it’s NOT tendinosis, it’s a partial tear.

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13
Q

MRI presentation of partial tear vs complete tendon tear?

A

Both fluid signal
Partial: thinning/thickening of the tendon but incomplete disruption of the fibers.
Complete tear: retraction of the tendon

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14
Q

What is Jones fracture?

A

Fracture of the metaphyseal-diaphyseal 5th metatarsal.

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15
Q

What’s Friberg infarction and what is the age demographic?

A

Demo: young women in heels

Avascular necrosis of the second metatarsal

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16
Q

What’s the tendon involved in sesamoid fracture?

A

Flexor hallucis brevis

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17
Q

What does the Lisfranc ligament connect?

A

Medial cuniform to the second metatarsal base

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18
Q

Other name of navicular necrosis and what’s the demographic?

A

Child boy = Kohler
Adult female = Weiss

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19
Q

What’s the lover’s fracture? And what do you recommend if seen?

A

Calcaneal fracture due to high impact trauma

Associated with lumbar spine fracture, aortic/renal injury so we recommend lumbar and abdominal imaging.

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20
Q

What’s important to comment on after diagnosing a calcaneal fracture?

A

Subtalar extension because this allows us to use the Essex-Lopresti classification

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21
Q

What’s the most important talus fracture and why?

A

Talar neck fracture as it predisposes to osteonecrosis

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22
Q

What is this sign and why is it important?

A

Hawkin sign; subchondral lucent band visualized 6-8 weeks after tarsal bone fracture which indicates blood supply to the dome and no avascular necrosis (good prognosis)

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23
Q

What is this sign and what does it indicate?

A

C sign
Osseous talocalcaneal coalition

Talar beak sign (in all coalition)

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24
Q

What is this sign and what does it indicate?

A

Anteater sign
Osseous calcaneonavicular coalition

Talar beak sign (in all coalition)

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25
What are the types of tarsal coalition and where are they located? What is helpful sign?
Osseous, cartilagenous, ligamentous Located: Talocalceneal Calcaneonavicular
26
What are the lateral ankle ligaments and which is most commonly injured?
1- Anterior talofibular (most common) 2- Posterior talofibular 3- Calcaneofibular (2nd most common)
27
Which muscle connects to the base of the 5th metatarsal?
Peroneus brevis
28
Where does peroneus longus attach?
Curves under the foot and attaches to the medial cuniform
29
What makes up the medial ankle ligaments?
Deltoid complex and spring ligament complex
30
What is the deltoid complex compromised of?
1- anterior tibiotalar 2- posterior tibiotalar 3- tibiocalcaneal 4- tibionavicular
31
Contents of the tarsal tunnel anterior to posterior
Tom, Dick and Harry Posterior Tibial tendon Flexor Digitorum longus tendon Flexor Hallucis longus tendon Posterior tibial artery and nerve
32
Danis-Weber classification (Depends on the level of fibular fracture and resultant syndesmotic injury. )
Weber A; distal fibular fracture with intact syndesmosis Weber B; more proximal trnassyndesmotic fracture Weber C; high fibular fracture above the level of the syndesmosis however associated with total syndesmotic rupture and ankle mortise instability.
33
If you see a medial malleolar fracture or widening of the ankle mortise what's the next step?
Take a radiograph of the proximal fibula for associated Maisonneuve fracture
34
What is this fracture called?
Pilon fracture; comminuted vertically oriented fracture of the distal tibia likely due to axial loading.
35
What is the special name for this fracture and what does it involve?
Triplane fracture: Vertical epiphyseal fracture Horizontal physis fracture Oblique metaphyseal fracture
36
What's the special name of this fracture and what does it involve?
Tillaux fracture: Salter Harris 3 of the lateral tibial epiphysis in adolescents
37
How do you suspect achilles tendon injury on plain radiograph?
Increase soft tissue in the Kagar fat pad Notching of the soft tissue
38
What's the differential diagnosis of achilles tendon injury on radiograph?
Accessory soleus muscle; soft tissue mass in Kager fat pad Haglund disease (retrocalcaneal bursitis)
39
What is recommended in all knee dislocations and why? What's the most common knee dislocation
Anterior is most common. CT angiography is recommended for all knee dislocations because even if the distal pulses are intact there is a high risk of popliteal artery injury.
40
What is the finding?
dorsal defect of patella usually located in the superiolateral aspect
41
Which fractures are more stable, medial or lateral?
Lateral tibial plateau fractures are more stable than medial. Lateral = lemme goooo
42
If there is injury to the meniscal root what do you look for and what can it mimic?
Look for meniscal extrusion but don't confuse it for the meniscofemoral ligament
43
What is the red zone of the meniscus?
Peripheral third of the meniscus is relatively vascular called the red zone in which injuries may heal spontaneously
44
How do you differentiate meniscus degeneration from tear?
Degeneration; high signal but not fluid signal and does not extend into the articular surface. Tear; fluid signal that extends into the articular surface
45
Where are horizontal tears usually located and how does this make sense?
Posterior horn of the medial meniscus likely due to degenerative changes and less likely due to trauma. Note: Osteoarthritis is asymmetrical with predominant involvement of the medial compartment
46
What is a vertical or longitudinal tear?
Tear the follows the curve of the meniscus
47
What is the most sensitive finding for detecting a bucket handle tear?
Absent bow tie sign
48
What are the signs seen in bucket handle tear?
Double delta; fragment displaced anteriorly Double PCL; fragment displaced centrally into the intercondylar notch (most common) this only applies to the medial meniscus
49
What is this sign?
Double delta sign which indicates anterior displacement of the bucket handle tear fragment.
50
Radial tear/transverse tear
Extends from the free edge of the meniscus to the periphery occurring through the body
51
What is the sign of radial tear and what is this tear associated with?
Ghost meniscus occurring through the horn usually adjacent to the intercondylar notch. Associated with meniscal extrusion
52
What is a meniscal cyst and why is it important to be mentioned?
Extension of joint fluid through meniscal tear Importance: Can cause symptoms even after meniscus is resected and sometimes cannot be seen arthroscopically.
53
Define a discoid meniscus and how is it detected on MR?
Meniscus extending to the central tibial plateau Exact measurement: meniscus body measurement more than 1.5 cm MR: Bow tie appearance 3 slices or more (4mm thick)
54
Which meniscus is commonly involved in discoid meniscus and what is the presenting history?
Lateral meniscus Hx: Locking/clicking of the knee in an adolescent.
55
Anterior cruciate ligament attachment and insertion
Arises: femoral intercondylar notch Attaches: anterior tibial plateau lateral to the tibial spine
56
Which attachment is stronger within the ACL and what is the clinical significance?
Tibial attachment is stronger causing more tears from the femoral end.
57
ACL tear on MRI
frank discontinuity of ligament fibers or abnormal course and signal
58
ACL tears are associated with two entities, name them.
Medial meniscal and MCL tears ( O’Donoghue’s triad aka unhappy triad) Segond fracture; avlusion fracture of the lateral plateau.
59
What are the secondary findings in ACL tears?
Contusion pattern: lateral femoral condyle and posterolateral tibial plateau Buckling of the PCL
60
What is Segond fracture associated with and what's the next step?
1- ACL tear 2- Iliotibial band syndrome Next step MRI because of the previous assocations.
61
The iliotibial band inserts at
Gerdy tubercle of the tibia
62
Posterior cruciate ligament arises and inserts
Arises: femoral intercondylar notch Inserts: posterior tibial plateau.
63
PCL tear appearance on MRI
Increased laxity of the PCL with or without abnormal high signal intensity
64
Medial collateral ligament arises and attaches to Why is it not visualized by arthroscopy?
Arises: Posterior aspect of the medical femoral condyle Attaches: Medial tibial metaphysics Not visualized by arthroscopy because it's extrasynovial
65
Grades of MCL tear
Grade 1; Sprain; high signal of the soft tissue with normal signal ligament Grade 2; severe sprain/partial tear; high signal/ partial disruption of the MCL fibers. Grade 3; Complete disruption of the MCL
66
The lateral collateral ligament is compromised of many structures, 3 of which are visualized by MRI. Name them.
anterior to posterior; 1- Biceps femoris 2- LCL 3- Iliotibial band The biceps and LCL attach on the lateral fibular head. The iliotibial band attaches to the Gerdys tubercle.
67
Clinically: anterolateral knee pain in a runner. DDx
IT band syndrome; fluid surrounding the iliotibial band Lateral meniscus tear
68
The quadriceps originate from ________ except _________ it originates from _________
Femur except: rectus femoris from : AIIS
69
Define patella alta and patella baja
Alta; abnormally high patella, > 1.2 Baja; abnormally low patella, < 0.8
70
What is Jumper's knee?
Patellar tendinosis = Jumper’s knee = thickening of the patellar tendon
71
History of loss of knee extension, what's your differential diagnosis and how do you distinguish between the two?
Quadriceps tendon tear = Patella baja Patellar tendon injury = Patella alta
72
What are the findings and diagnosis?
Bone marrow contusions; lateral femoral condyle and medial patellar facet with tearing of the medial retinaculum Diagnosis: Patellar dislocation relocation
73
Define Osgood-Schlatter disease and the MRI findings.
Osteochondrosis of the tibial tubercle due to repetitive microtrauma in adolescent. MRI: 1- increase signal of distal patellar tendon 2- marrow edema in the tibial tubercle 3- edema within Hoffa’s pad
74
What are the types of cartilage injury?
surface irregularity, fissuring, delamination
75
What's the distribution of Osteochondrosis Dissecans
1- knee (lateral medial condyle) 2- ankle(posteriomedial or anteriorlateral talar dome) 3- elbow
76
What's an unstable fragment in osteochondrosis dissecans and what is the complication?
Unstable fragment, lesion not attached to bone causing secondary osteoarthritis.
77
MRI finding of unstable osteochonrosis dissecans
Curvilinear high signal intensity on fluid sensitive sequence interposed between the fragment and underlying bone.
78
If pigmented villonodular synovitis is seen outside the knee joint it is termed
Giant cell tumor of tendon sheath
79
Whats the most common site for pigmented villonodular synovitis, the radiographic and MRI findings
Knee Radiograph; knee effusion, soft tissue mass posterior to knee MRI; hemosidernin deposit; dark signal on T1 and T2, with blooming on gradient echo sequences.
80
Describe the findings and diagnosis.
Overgrowth of intracapsular synovial fatty tissue, causing lobulated and globular intra-articular fatty masses. Lipoma arborescens
81
Where is a Baker cyst located and what is an alternative diagnosis.
Located between the semimembranosus tendon and medial head of the gastrocnemius muscle. (Baker’s M&M) DDx popliteal aneurysm.
82
Tennis leg
Tear of the plantaris tendon or medial head of the gastrocnemius
83
The center of the acetabulum that is not covered by cartilage is called
Pulvinar
84
Findings and diagnosis
PD; FAT SAT Structure mostly the plantaris tendon. Note the abnormal signal within the medial head of gastrocnemius Dx: Ruptured plantaris - tennis leg
85
Which muscles attach to the greater trochanter?
Gluteus medius, minimus Obturator internus and externus Piriformis
86
Which muscle attaches to Lesser trochanter? Note: Fracture in the lesser trochanter is always pathological until proven otherwise.
Ilipsoas
87
What is an easy way to establish whether or not there is a fracture to the anterior or posterior column of the acetabulum?
Iliopectinal and ilioischial lines disruption
88
Which acetabular column is commonly fractured?
Posterior
89
Name the intracapsular and extracapsular femoral fractures and why is it important
Femoral head and neck fractures = intracapsular, complications include femoral head osteonecrosis Intertrochanteric and subtrochanteric = extracapsular, direct injury, rarely cause osteonecrosis.
90
Types of femoral neck fracture
Subcapital (most common) Transcervical Basicervical
91
Technical factors to adjust when patient with metallic prosthesis undergoes MRI
1- Decreasing the field strength 2- Fast spin echo sequences instead of gre 3- Increasing the receiver bandwidth, which will increase the noise, so we need to increase the number of acquisitions. 4- Artifacts can be directed in the superior-inferiro plane so the region 5- Decrease voxel size by decreasing slice thickness and increasing matrix size.
92
Indications of hip MRI
Occult traumatic fracture since radiographs are insensitive for detection of nondisplaced femoral neck fracture. Occult stress fracture Stress fractures; either fatigue or insufficiency fractures Radiographs insensitive for detection of hip stress fractures, if present looks like a band sclerosis.
93
Classic location for femoral stress fractures
Inferomedial femoral neck
94
Less commonly superior femoral head fractures can mimic avascular necrosis, how to differentiate on MRI
AVN smooth and concave to articular surface, insufficiency fracture irregular low intensity fracture line convex to the articular surface.
95
If there is a stress fracture in an atypical location, what can be a clue during history taking
Bisphosphanate use
96
Arterial supply to the femoral neck
Medial femoral circumflex artery
97
Non traumatic causes of AVN
1- Red cell abnormalities 2- Abnormality of marrow packing (Gaucher) secondary effects of meds (steroids) alcohol immunosuppression which tend to be bilateral. Diving (Cassion's disease)
98
Most common site for AVN?
Proximal femur and proximal humerus
99
Ficat staging for AVN
Stage 1; normal radiograph with signs of AVN on MRI Stage 2; cystic and sclerotic changes Stage 3; loss of normal spherical shape due to collapse of the subchondral bone. Subchondral lucent line = cresent sign. Stage 4; Flattening of the femoral head and secondary osteoarthritis. Tx joint replacement.
100
What is this sign?
Subchondral lucent line = cresent sign which indicates avascular necrosis. Unlike Hawkin's sign in the talar dome, it's a lucent line within the talar dome if present decreases the likelihood of developing AVN.
101
Describe the findings and what is it pathognomonic for?
Geographic subchondral lesion outlined by a serpentine low signal rim on T1 and on T2, two intensities are seen Double line sign Diagnosis: AVN
102
Transient bone marrow edema/ transient osteoporosis of the hip MRI finding and demographic Note: It's a diganosis of exclusion
Demo: severe hip pain, young, middle aged adult, men, pregnancy MRI: T1, diffuse low signal, high signal on T2 with signal extending from the femoral head into the neck.
103
Hip labral injury Most common cause Predisposing factor Best diagnostic study Most common location
MCC: chronic repetitive trauma rather than acute Predisposing factor: Femoracetbaular morphological changes i.e DDH and femoral acetabular impingement Best diagnostic study: MRI arthrogram Most common location: anteriosuperior
104
Femoroacetabular impingement types How do we make the diagnosis?
Cam Pincer- type Mixed You need clinical symptoms for diagnosis (controversial topic)
105
Cam impingement Causes Demographic Treatment
Many causes examples 1- Legg-calve-perth 2- DDH, slipped capital femoral epiphysis Demo; young athletic males Treatment: osteoplasty
106
Name the deformity and diagnosis
Pistol grip deformity Dx: Cam impingement
107
Chance fracture/ Seat belt fracture definition
Flexion distraction injury, horizonal splitting of the vertebra beginning posteriorly in the spinous process/lamina
108
What are the measurements for craniocervical dissociation and ligamentous laxity
More than 12 mm BD interval; craniocervical dissociation More than 2.5 mm AD interval adults and more than 5 in pediatrics ; ligamentous laxity
109
Define a Jefferson's fracture Which view on xray is helpful and what can you appreciate?
symmetrical fractures of the anterior and posterior arches of c1 Due to axial loading to the vertex Open mouth- odontoid view = lateral masses are displaced laterally.
110
What are the 3 types of odontoid injury? How does this fracture come about?
1 = tip of the dens 2= base of the dens (unstable) 3 = base of the dens extending into the vertebral body (stable/unstable) Flexion injury
111
Hangman’s fracture definition What's characteristic on lateral radiograph?
Hyperextension causing traumatic spondylolysis of C2 = bilateral fractures through the pedicles of C2 Characteristic radiograph finding: disruption of the spinolaminar line.
112
Burst fracture
Compression fracture w/ retropulsion
113
Flexion teardrop
Avulsed bony fragment from anterior inferior aspect (anterior longitudinal ligament avulsion) with posterior displacement of the vertebrae into the spinal cord
114
What's the most severe cervical spine injury and why
flexion tear drop due to it's complication; Anterior cord syndrome = complete paralysis and loss of pain and temp, dorsal column is intact
115
Extension teardrop fracture
It's a stable fracture usually at C2-C3 with an anterior inferior avulsion fragment No subluxation Spinolaminar line not affected.
116
Clay Shoveler’s fracture
Displaced avulsion fracture of the spinous process Lower cervical spine
117
Bilateral interfacetal dislocation (locked facet)
1. Hyperflexion injury 2. Complete disruption of all spinal ligaments 3. Anterior dislocation of the affected vertebrae Sign: naked facet sign (axial) Perched facet: less severe variation (subluxation but not displacement)
118
Is a unilateral facet dislocation stable or unstable?
stable
119
Grisel syndrome
Non-traumatic rotatory subluxation C1-c2 caused by inflammatory mass i.e pharyngitis or retropharyngeal abscess.
120
Most common glenohumeral dislocation Best seen on
Anterior GH dislocation where it's displaced anterior inferiorly. Best seen on axillary view
121
Define Hill sachs and Bankart
Hill sachs = compression fracture of posterolateral aspect of humeral head Bankart = injury to the anterior-inferior rim of glenoid usually cartilaginous
122
What are the two signs seen in posterior shoulder dislocation
Light bulb sign = fixed internal rotation Trough sign = compression fracture of the antero- medial humral head (reverse hill sach)
123
What happens in inferior shoulder dislocation?
1. Rotator cuff injury 2. Greater tuberosity fracture 3. Injury to the axillary n + a
124
The subacromial and subdeltoid bursae normally communicate with each other and not the with the glenohumeral joint. IF you inject contrast into the glenohumeral joint and it extends into the subacromial/subdeltoid bursae, what does that indicate?
Complete tear
125
Type 3 and 4 acromion and clinical significance
Type 3; hooked undersurface Type 4; convex undersurface Significance; lead to rotator cuff tears
126
Os acromial
Persistent accessory ossification center of the acromion in patinets above 25 y MRI findings with edema in this area suggests that it's the cause of the patient's shoulder pain.
127
Rotator cuff tendon insertion
Greater tuberosity; supraspinatus, infraspinatus, teres minor Lesser tuberosity; subscapularis
128
Tendinosis/Tendiopathy MRI finding and differential diagnosis
(Mxyoid degeneration of tendon) MRI: diffuse/focal thickening with intermediate signal intensity on T1/T2 DDx: Magic angle artifact, may stimulate tendinosis/ partial tear, look for the T2
129
What is the footprint of the shoulder?
Site of attachment of tendons at the greater tuberosity
130
Most commonly injured rotator cuff tendons from most to least
Most commonly injured; supraspinatus, next infrapspinatus, least affected teres minor
131
Partial thickness tear MRI finding
MRI: abnormal signal intensity of the muscle/tendon but not extending through it’s entire thickness AND fluid signal.
132
Types of partial tear in the rotator cuff tendons? and what's the most common type
Division; bursal-surface, articular surface, or intrasubstance MC type: articular
133
40 % of supraspinatus tears are associated with
infraspinatus tears Note: Infraspinatus is rarely torn in isolation
134
Chronic full-thickness rotator cuff tear due to RA has a classical appearance
Humeral head migrates superiorly and may articulate with acromion
135
Fatty degeneration of the muscle usually occurs within _____ weeks of injury of the tendon
4 weeks!!!
136
Adhesive capsulitis/ Frozen shoulder
Thickening and contraction of the glenohumeral joint capsule. Clinical diagnosis however MRI; thickening of the joint capsule and synovium > 4mm assessed at the level of the axillary pouch.
137
Where do the glenohumeral ligaments attach?
Superior and middle = lesser tuberosity to the supraglenoid tubercle of the scapula Inferior = inferior glenoid labrum to the anatomic neck of the humerus
138
What is the rotator interval is a triangular region Where is it located and what is it's purpose?
Purpose: allows rotational motion around the coracoid process Located between the supraspinatus and subscapularis tendons
139
What does the rotator interval contain?
1. Coracohumeral ligament (CHL) 2. Long head of the biceps tendon (LHBT) 3. Superior glenohumeral ligament (SGHL)
140
Where do the glenohumeral ligaments arise from
Labrum
141
Most important component of the capsulolabral complex for maintaining stability in abduction and external rotation
Inferior glenohumeral ligament (IGHL)
142
Where does the biceps tendon attach?
Anterosuperior glenoid rim along with the SGHL and MGHL
143
The biceps tendon sheath connects with ______ and it's importance is
The sheath communicates with the glenohumeral joint and thus fluid in the tendon sheath may be normal.
144
The biceps pulley is formed by
1. Superior glenohumeral ligament 2. Coracohumeral ligament 3. Distal fibers of the subscapularis tendon.
145
Bicep tendon tears are associated with
Supraspinatus tear oR Risk of impingement by the supraspinatus
146
Biceps tendon subluxation vs dislocation
subluxation = still contact with the groove dislocation = no contact with the groove
147
Biceps subluxation is associated with ______________ Biceps dislocation is associated with ______________
Subluxation = transverse ligament ear Dislocation = subscapularis tendon injury
148
Most shoulder instability lesions occur at this location __________________ and are associated with
Anterior-inferior aspect of the glenohumeral joint and associated with anterior band of the ligament.
149
Define bankart lesion and it's differential
Injury to the anteroinferior labrum due to anterior glenohumeral dislocation with stripping of the scapular periosteum Labrum might migrate superiorly and appear as balled up mass-like object producing glenoid labrum ovoid mass (GLOM sign). DDx dislocated biceps tendon, air bubble (MR arthrogram was performed)
150
What is an osseous bankart and it's complications?
Fracture of the anterior-inferior glenoid rim Complication; predisposition to recurrent dislocation due to glenoid insufficiency
151
Define Hill-Sach fracture and how to detect it on MRI
Impaction fracture of the posterolateral humeral head caused by anterior dislocation 5mm slices, normal humeral head should appear round in the 3 consecutive slices Subtle hill sach; bone marrow edema without of fracture superiorly.
152
What is anterior labro-ligamentous periosteal sleeve avulsion (ALPSA) Variant of Bankart
Variant of Bankart Also representing anterior-inferior labral injury. How to differentiate? Scapular periosteum is intact
153
What is Perthes lesion and how is it visualized?
Avulsion of the anterior-inferior labrum Labrum remains attached to scapular periosteum Very difficult to visualize MR arthrography with ABER is often necessary for diagnosis
154
What makes the borders of the quadrilateral space and what is it's significance?
posterior aspect of axilla, humerus laterally, long head triceps medullary, teres minro superior, and teres major inferiorly. Significance: axillary nerve and posterior humeral circumflex artery
155
What is seen in axillary nerve injury
Axilary nerve supplies both the deltoid and the teres minor, however if there is injury only the teres minor will atrophy
156
Define a paralabral cyst. Where is it seen? What is it's complication?
Very specific finding for labral tear even when labral signal is not altered. Usually seen in soft tissue adjacent to the labrum but may extend into the bone. May cause nerve entrapment.
157
Suprascapular nerve entrapment Location: Nerve supply: Caused by
Location: Suprascapular notch Proximal Innervation; supra and infraspinatus. Caused by: paralabral cyst
158
Elbow dislocation is 1. Associated with 2. Most common type 3. Next step
Ulnar fractures Most common: Posterior dislocation of the radius and ulna with respect to the humerus Next step: Obtain full forearm radiographs because the fracture can be distal.
159
What is the sail sign and what is considered more specific?
Sail sign; elevation of anterior fat pad only and less specific Elevation of the posterior fat pad is nearly diagnostic
160
Most common elbow fracture in pediatrics and adults
Pediatrics: Supracondylar fracture Adults: Radial head fracture
161
If fat pad sign is seen and no fractures what's the next step?
Recommend additional views or CT scan
162
Essex- Lopresti fracture-dislocation
Radial head fracture and tearing of the interosseous membrane with ulnar dislocation at the distal radioulnar joint.
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Monteggia fracture -dislocation
Ulnar fracture and radial dislocation at the elbow
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Galeazzi fracture-dislocation
Fracture of distal third of radius with ulnar dislocation at the distal radioulnar joint.
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Colles fracture
Distal radius frature with dorsal angulation Usually intra-articular FOOSH
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Hutchinson (Chauffeur’s) fracture
Distal radius fracture of lateral aspect extending into the radial styloid and radiocarpal joint.
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Perilunate vs lunate dislocation
Perilunate = capitate dislocation Lunate = lunate dislocated volarly, but capitate is aligned with the radius
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Scapholunate ligament injury Definition Cause Complication
Capitate migrates proximally to fill the gap between scaphoid and lunate Caused: FOOSH Complication: scapholunate advanced collapse wrist (this can also be caused due to other things)
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What's the best view for Triquetral fracture
Best seen on lateral radiograph as avulsion fragment dorsal to the triquetrum
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Keinbock disease
Avascular necrosis of the lunate Associated w/ negative ulnar variant
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Mallet finger
Distal phalanx, may have avulsion fragment of the distal phalanx, DIP joint flexion.
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Boutonniere deformity Defintion Association
Medial slip (entrapment of the tendon), fixed flexion of the PIP Associated w/ RA, may be post traumatic
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Gamekeeper’s thumb (skier’s thumb)
Injury to the ulnar collateral ligament at the base of the thumb; proximal phalanx. Thumb is forced abduction. May have avulsion fragment of the proximal phalanx.
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What is this sign and what does it indicate?
Terry Thomas sign, increased distance of the scapholunate interval indicating scapholunate ligamentous injury
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Bennett fracture
Intra-articular fracture of the base of the thumb metacarpal
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Rolando fracture
Comminuted bennett fracture
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Boxer’s fracture
Metacarpal neck fracture, likely 5th
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Volar plate fracture
Avulsion fracture of the volar aspect of the proximal phalanx.
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What is the hallmark of arthritis and how is this depicted on radiographs?
Hallmark: Cartilage destruction Radiographs: Joint space narrowing
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What are the types of arthritis?
1. Degenerative 2. Inflammatory 3. Crystal 4. Hematological 5. Metabolic
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Osteoarthritis/osteoarthrosis/degenerative joint disease KEYWORDS
1. Osteophytes 2. Subchondral cysts 3. Asymmetrical joint space narrowing 4. Subchondral sclerosis Note: No erosions
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DDx for extensive subchondral cystic changes seen in osteoarhtirits
Calcium pyrophosphate dihydrate crystal deposition disease.
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Osteoarthritis distribution in the following: Hand Foot Knee Spine Hip Sacroiliac joint
Hand: 1st CMC, PIPs, DIPS Foot; 1st Metatarsal phalangeal joint, talonavicular joint. Knee; Asymmetric involvement of the medical tibiofemoral compartment, severe osteoarthritis can involve all the 3 compartments. Spine; facet joint, atlantoaxial, uncovertebral disc, costovertebral and sacroiliac (synovial joints) Hip; superolateral migration is classic or inferiomedial Sacroiliac joint; only the inferior portion of the sacroiliac is synovial.
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Vacuum phenomenon is pathognomonic for but don't confuse with
Vacuum phenomenon = pathognomonic for degenerative but don’t mistake for Kummel disease = gas in the vertebral body due to compress fracture representing osteonecrosis.
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Complications of osteoarthritis within the spine.
1. Spinal stenosis 2. Neural foramina narrowing 3. Degenerative spondylolisthesis
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What's a differential for osteoarthritis in the spine?
Diffuse idiopathic skeletal hyperostosis = DISH Bridging anterior osteophytes and ossification of the posterior longitudinal ligaments. DISH = CT, not MRI.
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Erosive arthritis Demo Location Distribution Features
Demo: Elderly Females Location: Only hands Distribution: Like osteoarhtirits Features: Like rheumatoid
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What is this sign and what does it indicate?
Gull wing sign; erosive arthritis
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What is the first location where rheumatoid arthritis hit?
Feet first
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What are the features for rheumatoid arthritis?
1. Marginal erosions which occur first in the bare area 2. Soft tissue swelling 3. Symmetric joint space narrowing 4. Periarticular osteopenia 5. Joint subluxation/Ankylosing *complication*
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Rheumatoid involvement of the Hand Feet Hip Knee Spine Shoulder
Hand; symmetric, MCPs, PIPs, carpals Feet: MTPs Hip; Causes concentric cartilage loss so causes axial migration, if severe it’s called protrusio acetabuli Knee; All 3 compartments are affected Spine; involved in more advanced disease, mostly the cervical spine, features; subluxation, osteopenia, erosions of odontoid, end plates, spinous process and facet joints, no bone production Shoulder; erosions in the lateral head of the humerus, and “penciling” of the distal clavicle causing chronic rotator cuff tears = high riding humerus.
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What are the features of rheumatoid arthritis in the knee
Erosions are not prominent, however there is symmetrical joint space narrowing and possibly secondary OA especially if the patient is young.
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There are two complications of RA: Joint subluxation and ankylosing. Give examples of joint subluxation
1) Boutonniere deformity; PIP flexion, DIP hyperextension 2) Swan neck deformity; PIP hyperextension and DIP flexion 3) Ulnar subluxation at the MCPs
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What are the four spondyloarthropathies?
1. Ankylosing spondylitis 2. Psoriatic arthritis 3. Reactive arthritis (Reiter’s) 4. Inflammatory bowel disease associated arthropathy
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What is the hallmark for spondyloarthropathies, it's association and hos it can be differentiated from OA
Association:- HLA B27 positive Hallmark; - Sacroilitis, first involving the ilias aspects of the joint making it distinctive.
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Ankylosing spondylitis Demo Features Location
Demographic; young male associated with lung fibrosis, aortitis Features; widening, erosions, sclerosis Location: Stats in the SI joint and moves up the spine
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What are the 5 possible appearances of ankylosing spondylitis in the spine
(1) Romanus lesion = erosion of the anterior superior or anterior inferior edges of the vertebral endplates caused by enthesis sclerosis = shiny corner. (2) Bamboo spine = syndesmoses (3) Dagger spine = fusion of the spinous process (4) Squaring (5) Andersson lesion = pseudoarthrosis in a completely ankylosing spine after fracture
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Psoriatic arthritis hallmark
diffuse soft tissue swelling of the digit = sausage digit
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Psoriatic arthritis features
1. Preserved mineralization 2. Various patterns
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Psoriatic arthritis
1) SI joint; Bilateral, asymmetric involvement of the SI joint 2) Hand; PREDOMINANT, pencil in cup erosions = DIPS usually 3) Hand rare presentation; Main en lorgnette; opera- glass hand; telescoping of the digits seen in the severe pattern arthritis mutilans 4) Foot; Plantar calcaneal spur with periosteal reaction 5) Spine; coarse bony bridging indistinguishable from reactive arthropathy.
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Reactive arthropathy pt history
cervitis, diarrhea, urethritis
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Difference between reactive and psoaritic arhtirits
Psoriatic = predominant hand involvement Reactive = predominate feet involvement
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Features of reactive arthropathy
1. Soft tissue swelling 2. Joint space narrowing 3. Aggressive marginal erosions 4. Juxta articular osteopenia however bone mineralization is preserved in the later stage.
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Psoaritic features in the foot are similar to
rheumatoid arthritis, however calcaneal spur formation like reactive arthritis
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Reactive arthritis involvement
1. Foot; calcaneus is common site of involvement; fluffy periosteal reaction, secondary Achilles tendinitis. 2. Hands; MTPs, DIPs, PIPs (same distribution of RA) 3. Spine; coarse bony bridging indistinguishable from psoraitic arthropathy.
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Features of SLE and differential diagnosis
Reducible subluxation of the MCPs and PIPs more apparent with Norgaard (ball catcher) or oblique views. Ddx RA
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Scleroderma
Atrophy of the distal soft tissue Acroosteolysis; resorption of the distal portion of the distal phalanges is characteristic especially with calcification.
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DDx for acrosteolysis
1. Collagen vascular disease 2. Neuropathy 3. Thermal injury 4. Hyperparathyroidism 5. Polyvinyl chloride exposure
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Hydroxyappetite deposition disease other name
Calcific tendinitis
210
Why is HADD asymmptomatic?
Periarticular tissue (tendons) not directly into the joint thus why it’s commonly asymptomatic
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Which tendon is commonly affected in calcific tendinitis?
Supraspinatus tendon is most commonly affected
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Milwaukee shoulder
Leads to rapid destruction of the rotator cuff and glenohumeral joint.
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When can HADD mimic a prevertebral abscess?
Prevertebral longus coli muscle = neck pain, odynophagia, fever, prevertebral effusion that may mimic prevertebral abscess
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Calcium pyrophosphate dihydrate deposition disease Demo Associated with
Demo: Pt's usually 50 years and older Associated: Hemochromatosis Hyperparathyroidism Hypophosphatasisa
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CPPD (chondrocalcinosis) Wrist Knee Hand
Wrist; chondrocalcinosis affects the triangular fibrocartilage complex with advanced disease causing scapholunate advanced collapse = proximal migration of the capitate between the dissociated schaphoid and lunate. This is not specific, it can be seen in trauma or RA. Knee; patellofemoral compartment affected first, prominent subchondral cysts. Hands; hook like/drooping osteophytes from the radial aspect of the metacarpal heads isolated to the 2nd and 3rd metacarpals.
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DDx of drooping osteophytes
Hemochromatosis = all metacarpal heads CPPD (chondrocalcinosis) = raidal aspect of the 2nd and 3rd metacarpals.
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Gout Hx Hallmark
Hx; renal insufficiency, chemotherapy tx, Lesh Nyhan in young patients Hallmark; Sharply marginated erosions with overhanding margins associated with soft tissue gouty tophi preserved joint space and bone mineralization.
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Hemochromatosis hand involvement
MCPs, hook like osteophytes, with involvement of ALL the MCPs
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Acromegaly joint changes
Excess growth hormone => enlargement of cartilage  degenerates.
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What is this sign and what is the diagnosis?
Shoulder pad sign; Atrophic muscles, bulky soft tissue nodules due to protein deposition in bones, soft tissue and joints. AMYLOID deposition
220
What is this sign and what is the diagnosis?
Shoulder pad sign; Atrophic muscles, bulky soft tissue nodules due to protein deposition in bones, soft tissue and joints. AMYLOID deposition
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Hemochromatosis involvement
Knee; Recurrent hemarthrosis = synovial hypertrophy and hyperemia which causes epiphyseal enlargement and early fusion. Characteristic appearance; widening of the intercondylar notch and squaring of the patella Elbow; Enlarged radial head and widened trochlear notch
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Pseudotumor of hemophilia
Benign lesion caused by recurrent intraosseous or subperiosteal bleeding leading to bony scalloping and pressure erosion
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DDx of hemochromatosis on imaging
Juvenile idiopathic arthritis may have similar findings especially in the knee and elbow because it causes hyperemia and same pathophys
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Juvenile idiopathic arthritis Demo Variant
Demo: Children less than 16 Variant: Still disease is a variant of JIA featuring febrile illness, rash, pericariditis.
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Hallmark of Juvenile idiopathic arthritis
abnormal bone length/morphology due to hyperemia.
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Involvement in JIA
Knee; 1. widening of the intercondylar notch 2. metaphyseal flaring (Erlenmeyer flask deformity) 3. uniform joint space narrowing ddx hemophilia 4. epiphyseal overgrowth and enlargement; “ballooning” 5. premature fusion of the growth plate which may cause brachydactyly (short digit). Hand; premature closure of growth plates causing brachydactyly. Hips; 1. symmetrical cartilage space narrowing 2. protrusion deformity (RA) 3. gracile appearance of the femoral shaft. Spine; ankylosing (AS) /(also in the wrist)
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DDx to JIA in the spine
DDx Klippel-Feil syndrome
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Neuropathic arthropathy/ Charcot joint defintion
Destructive arthropathy due to lack of sensation causing fragmentation of bone and cartilage.
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Hypertrophic charcot joint features
1. destruction 2. dislocation/subluxation 3. debris 4. disorganization 5. normal mineralization
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Atrophic charcot joint what should you consider
Shoulder involvement 1. humeral head resorption with sharp surgical-like margin Syringomyelia shoulder be suspected as the cause and can be confirmed by cervical spine MRI.
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Sarcoidosis
Lace-like lytic lesions in the middle/distal phalanges. Bone manifestations are rare.
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Differential for subluxation
Nonreducible think RA Reducible think SLE Another point: the presence and absence of erosions
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How to assess the bone mineral density and what does it help in?
Cortical thickness of the second metacarpal shaft which should be at least 1/3 of the total width of the metacarpal shaft. Helps distinguish inflammatory from non-inflammatory arthritis.
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Osteophytosis DDx
1. OA 2. CPPD 3. Hemochromotosis
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Arthritiswith periosteal reaction
Psoriatic arthritis Reactive arthritis, Less likely RA
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Bone ankylosing
Wrist and spine= Juvenile idiopathic arthropathy Advanced rheumatoid arthritis Ankylosing of the DIPs is a typical finding in psoriatic arthritis.
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Chondrocalcinosis
1. CPPD 2. Hyperparathyroidism 3. Hemochromatosis
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Calcification of the tendons
1. HADD 2. CPPD
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Soft tissue calcifications + arthritis
1. Gouty tophi 2. Scleroderma 3. Dermato/polymyositis 4. SLE
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Cartilage spaces Preserved Asymmetric Symmetric Increased
Preserved; gout Asymmetric; osteoarthritis and grout (region of gouty tophi and erosion) Symmetric; inflammatory arthropathy Increased cartilage space; acromegaly
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Wrist Diffuse
1. Inflammatory arthropathy 2. Post traumatic OA
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Erosion types and pathology
Variable; RA Pencil in cup; psoriatic Gullwing; erosive osteoarthritis Overhanding margin; gout
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Soft tissue swelling
1. Symmetric swelling; RA 2. Asymmetric swelling; OA Heberden and Bourchard 3. Swelling of entire digit; in hand think psoriatic arthritis/reactive arthropathy vs foot think reactive arthropathy 4. Lymph bumpy; gouty tophi, sarcoidosis, amyloid
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Types of periosteal reaction and in ascending order the least to most aggressive
Nonaggressive; solid Aggressive; lamellated/ onion skin i.e Very aggressive; sunburst/ hair on end Very very aggressive; codman triangle
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Types of bone matrix
Fluffy cloud like; osteoid Ring and arc/popcorn like; chondroid Ground glass; fibrous i.e fibrous dysplasia
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Fallen fragment
Fallen fragment is seen in a simple (unicameral bone cyst) with pathological fracture
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Tumoral calcinosis Hx
Painless lumps that may cause compression of the adjacent structures, excudate chalky fluid invloving large joints No erosions or there is no erosion or osseous destruction.
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Aggressive lytic lesion Above 40 Below 20
above 40 = metastasis or multiple myleoma under 20 = eosinophilic granuloma, infection, Ewing sarcoma, osteosarcoma
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Differentiating Ewing vs osteosarcoma
Ewing below 10 Osteosarcoma above 10 with more soft tissue component
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Benign incidental bone forming lesions
1. Enostosis (bone island), if multiple with kleoids osteopoikilosis 2. Osteopathia striata 3. Osteoma; cortex or skull/frontal ethmoid sinus assocition: Gardner syndrome; multiple intestinal polyps
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What are the 3 benign osseous lesions
1. Melorheostosis 2. Osteoid osteoma 3. Osteoblastoma
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What is this appearance called and what is associated with?
Candle-wax appearance ; thickened and irregular cortex Associated with Melorheostosis;
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How to differentiate a large bone island from osteoblastic mets/osteosarcoma/osteoid osteoma
Bone san is normal
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Melorheostosis on bone scan
Intense uptake
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Osteoid osteoma Presentation Features Bone scan Treatment
Presentation:night pain relieved by aspirin in a young adult. Diaphysis of long bones, leg bones commonly affected Features: Osteoid surrounded by reactive sclerosis Bone scan; double density sign Treatment; radiofrequency ablation
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Why is osteoid osteoma difficult to see on MRI?
Edema obscures the lesion
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Osteoblastoma Hx Mimics Location Complication
Hx: young adults with pain not relieved with pain killers DDx: Same as osteoid osteoma but > 2cm Location; spine mostly but can occur in the leg bones too! Complication: Secondary aneurysmal bone cyst especially in the spinal location
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Secondary Osteosarcoma
Pagets Previous radiation therapy
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How to differentiate telangiectatic osteosarcoma from aneurysmal bone cyst
MRI nodular component
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Primary Osteosarcoma Presentation Features Most common types Complications
Presentation; young adult, knee pain, lesion within the metaphysis Featuers; osteoid, aggressive periosteal reaction(codman/sunburst), soft tissue mass Conventional intramedullary (75%) Complication: Lung mets with calcificaiton
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Benign cartilage forming lesions
1. Osteochondromatosis/synovial chondromatosis 2. Enchondroma 3. Osteochrondroma 4. Chondroblastoma 5. Chondromyxoid fibroma
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Enchondroma Hx: Features: DDx; Complication; Multiple;
Hx: patients are old Features: long bones, medullary, metaphysis with ring arc calcification DDx; bone infarction, and chondrosarcoma, MRI is required to differentiate between infarction and enchondroma, enchondroma = high on T2 Complication; fracture, malignant transformation Multiple; ollier and maffuci (+pheliboliths), these syndromes make the risk for malignant transformation much higher.
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What's special about enchondroma in the hand?
Lytic with no matrix
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Osteochondroma Presentation Features Complication Multiple
Presentation; palpable mass, stops growing at skeletal maturity Features; Pedunculating bony growth continuity of the cortex grows from the metaphysis and away from the epiphysis Complication; malignant transformation to chondrosarcoma which can be determined if the patient is complaining of pain, MRI; cartilage thickness >2cm suggests malignant transformation Multiple osteochondroma = multiple hereditary exostoses
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Chondroblastoma Features What's special about it's condroid matrix? How is it seen on MRI Treatment
Features: Located eccentrically, epiphysis, skeletally immature patient, knee/humerus (Calcified chondroid matrix might not be seen on radiograph however identified on CT) MRI: Unqiue chondroid lesion; low/intermediate on T2 because most chondroid are T2 hyperintense Tx; curettage, cryosurgery, radiofrequency ablation
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Chondromyxoid fibroma Location Features Chondroid matrix
Eccentric tibia/femoral metaphysis knee Featurs: sclerosis margin Rarely demonstrates chondroid matrix
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Secondary Chondrosarcoma
1. Enchondroma (more common maffuci/ollier) 2. Pagets 3. Osteochondroma (more common osteochondromatosis)
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Features of chondrosarcoma
Medullary Expansile Ring and arc chondroid matrix Thickening and endosteal scalloping Soft tissue mass
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Nonossifying fibroma/fibrous cortical defect/fibroxanthoma Demo Location+ features Name discrepancy Where does it come from
Young pt Location + features: Eccentric, no sclerosis, medullary, diaphysis/metaphysis Nonossifying fibroma if the lesion is more than 2 cm/symptomatic Arise from the periosteum
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Fibrous dysplasia WHAT'S SPECIAL ABOUT IT BEING POLYOSTOTIC Features
Demo: Congenital, non-neoplasic condition of children and young adults Types: When polyostotic it tends to be unilateral Features: medullary, diaphysis, central and meta-diaphyseal causing bowing deformity (extreme varus is called shepherd’s crook)
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Fibrous dysplasia Rib/ long bones Skull base + DDx Pelvis Appearance Associated syndromes (2) Complications
In the ribs/long bones; indistinct/ground glass Pelvis; cystic Skull base; expansile and highly unusal on MRI DDx Pagets but age can help differentiate McCune-albirght syndrome; 1. polyostotic fibrous dysplasia 2. Precious puberty 3. Café au lait spots Mazabraud Syndrome; Fibrous dysplasia with intramuscular myxoma Complication; fracture
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Hemangioma; when can it cause neruological compromise?
Can be associated with a soft tissue mass causing neurologic compromise. Look for phleboliths/ soft tissue calcification if it’s not in the bone and it’s within the soft tissue
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Angiosarcoma commonly presents with
Lung metastasis (Pleural disease)
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What are the hematopoietic bone lesions
1. Giant cell tumor (osteoclastoma) 2. Esoinophilic granuloma (Langerhans cell histiocytosis) 3. Ewing sarcoma 4. Multiple myeloma 5. Lymphoma
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Giant cell tumor (osteoclastoma) Demo Features Tx
Demo: Skeletally mature patient between 20-40 Features: Epiphyseal lesion, eccentric, end of long bones, arises from metaphysis but crosses the physis and into the epiphysis. Tx; curettage or mid resection
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Langerhan cell histiocytosis Demo Skull Mandible Spine Long Bones
Demo: Children (5-10)  Skull; lytic lesion with a “beveled edge” aka hole within hole appearance  Maxilla/Mandible; floating tooth  Spine; vertebra plana (flattening)  Long bones; destructive radiolucent lesion with aggressive lamellated periosteal reaction which may look like lymphoma or ewing sarcoma
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Ewing sarcoma Presentation Features
 Presentation; children/ young adults, male predominance, pain, fever thus DDx include osteomyelitis which is hard to differentiate from Ewing  Features: permeative bone destruction aggressive periosteal reaction (lamellated) soft tissue mass (but less than osteosarcoma)
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Ddx for Ewing sarcoma
LCH Metastatic neuroblastoma Osteomyelitis
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Lamignant Hematopotien
280
Most common primary bone tumor in patients over 40 AND why does it not involve the spine pedicles
Multiple myeloma Comes from the bone marrow thus doesn’t involve the spine parts without marrow like pedicles
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Features and DDx of Multiple myeloma
 Features: Multiple lytic lesions with the most severe; diffuse myelomatosis with endosteal scalloping  DDX; metastasis help differentiate by bone scan which is negative for multiple myeloma
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Lymphoma Demo Features Sign
 Demo: adults over 40  Features: Aggressive lytic lesion with associated soft tissue mass  Sign: Ivory vertebra
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Lipoma (Benign) Location Features MRI features
Location: Calcaneus/subtrochanteric Features: central/ring-like calcification is present Note: Most lipomas contain some non-adipose tissue which does not suggest malignancy
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What features can suggest liposarcoma
Large size >10 cm Thick septations Nodular soft tissue
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Chordoma Arises from Location Features
 Malignant lesion from notochord remnant arising from the midline of the axial skeleton Location: spheno-occipital region, body of C2 or sacrococcygeal location  Features: Highly destructive, irregular scalloped borders, might have calcifications due to necrosis
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Lesions of unknown cellular origin
1. Simple bone cyst/unicameral bone cyst 2. Aneurysmal bone cyst 3. Adamantinoma
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Simple bone cyst  Demo:  Features:  Complication;  MRI:  Treatment;
 Demo: Local disruption of bone growth in children and young adults  Features: The lesion is hollow/ fluid filled, medullary lesion, proximal diaphysis of humerus/femur, no periosteal reaction which differentiates it from an aneurysmal bone cyst as long as there is no fracture.  Complication; highly associated with pathological fracture, fallen fragment sign which is pathognomonic  MRI: fluid-fluid levels  Treatment; intralesional injection with methylprednisolone which promotes bone growth
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DDx for MRI features of fluid-fluid levels
DDx 1. Simple bone cyst 2. Aneurysmal bone cyst 3. Giant cell tumor 4. Telangiectatic osteosarcoma
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Aneurysmal bone cyst Demo: Location Features: MRI
 Demo: Adolescents and children  Features: Expansile, multicystic lesion  Location: Medulla or cortex unlike simple bone cyst  MRI; fluid fluid levels
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Important consideration in aneurysmal bone cyst
Blood filled sinusoids and solid fibrous elements secondary to a pre-existing tumor; thus if you have a high suspicion and the pathology report is ABC then you’re not going to be happy about it.
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Adamintinoma
 Low grade tumor occurring in the anterior tibia, arising from the cortex  Features: Soap bubble appearance  DDx fibrous dysplasia
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Osseous metastasis (3 locations where to raise the suspicion)
Pedicle or posterior vertebral body suggests metastasis Fracture of the lesser trochanter raise concern for pathologic fracture. Solitary sternal lesion in a patient with breast cancer is highly predictive of mets
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Benign tumor mimics
1. Myositis ossificans/ heterotopic bone formation 2. Brown tumor 3. Osteomyelitis
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Myositis ossificans/ heterotopic bone formation
 Demo: Underlying history of trauma, even though sometimes trauma may not be remembered or minor.  Features: Peripheral osteoid mass abutting the anterior humeral cortex, usually elbow and thigh because they’re more prone to trauma. Appearance evolves over period of weeks to months  DDx parosteal osteosarcoma how to differentiate; parosteal osteosarcoma more heavily calcified centrally, while myositis ossificans is more calcified peripherally. (Zoning)
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Brown tumor
 Features: Lytic lesion with a patient with hyperparathyroidism, doesn’t have any specific imaging features but may be difficult to differentiate from giant cell tumor both radiologically and pathologically.
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Brown tumor Fingers Spine
 Fingers: Osteopenia, subperiosteal bone resorption especially 2nd and 3rd middle phalanges and acromial ends of the clavicles.  Spine: If secondary to renal failure, renal osteodystrophy; rugger jersey spine.
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Osteitis Periostitis Sequestrum Involcrum Cloaca Sinus tract Brodie abscess
Osteitis =inflammation of the cortex Periostitis = inflammation of the periosteum Sequestrum = necrotic bone separated from viable bone by granulation tissue, it needs surgery because it’s a nidus for recurrent infection Involucrum = living bone surrounding necrotic bone Cloaca = opening in the involcrum Sinus tract = opening from the infection to the skin surface Brodie abscess = subcutaneous osteomyelitis; central lucency and peripheral sclerosis ddx osteoid osteoma.
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Osteomyelitis location in children and why?
Metaphsis because of sluggish flow.
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Difference between hematogenous and direct inoculation osteomyelitis
Hematogenous = inside out and vise versa because of the bridging vessels through the physis.
300
Location of osteomyelitis in infants up to 12 months
Metaphysis, epiphysis and joint
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Periostitis is more prominent in children why?
In adults, the periosteum is more adherently attached to the cortex.
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Hematogenous osteomyelitis in adults is common in what location
Spine
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Chronic drainage sinus predisposes to
squamous cell carcinoma
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Osteomyelitis differential in peds
Ewing sarcoma
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Neuropathic joint vs diabetic foot osteomyelitis
Neuropathic foot : 1. Midfoot 2. Polyarticular involvement 3. Absence of soft tissue infection 4. Absence of sinus tract 5. Bony cortex intact vs diabetic foot which is almost always associated with cutaneous ulcer and sinus tract to the bone with the cortex involved often disrupted
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When is biopsy warranted in osteomyelitis?
When it's due to contiguous spread Usually polymicrobial unlike hematogenous spread with biopsy warranted to ensure proper treatment.
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Bow to differentiate chronic from acute chronic osteomyelitis?
active/non-active osteomyelitis but serial radiographs can show development of periosteal reaction
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 Most common cause of hematogenous spread  Sickle cell  Puncture wound through sneaker  Tuberculosis
 Most common cause of hematogenous spread; Staph  Sickle cell; Salmonella  Puncture wound through sneaker; Pseudomonas  Tuberculosis; Spine
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How to differentiate osteomyelitis from cellulitis on Tc99m MDP
Positive in all 3 phases Cellulitis is positive on flow and blow pool and negative in delayed
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When is WBC scan correlation with Tc99m MDP study for osteomyelitis not ideal
When the suspected osteomyelitis is within the spine. This is because the WBCs dont get t o the spine to begin with.
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Best MRI sequence for osteomyelitis whats the role of contrast administration
Most important: T1 Gadolinium 1. fluid collections 2. nonenhancing necrotic bone (sequestrum)
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Spetic arhtirtis  Septic arthritis: gold standard  Imaging and clinical hallmark  Cause in children;  Complication  IV drug abuser osteomyelitis
 Septic arthritis: gold standard is joint aspiration  Imaging and clinical hallmark; joint effusion  Children; think hip by contiguous extension from the proximal femoral metaphyseal osteomyelitis. (((Proximal femoral metaphysis is within the hip joint capsule)))  Complication; if not treated it leads to ankylosing.  Osteomyelitis of the sacroiliac and acromioclavicular joints = think IV drug abuser
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Osteoporosis T score and Z score
 T score <-2.5  Z score = age matched women not for osteoporosis
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What is Wembergers sign and Pelkin line and pelkin fracture
315
Osteomalacia Specific sign Common sites Complication
Looser zone (pseudofracture/milkman line) is highly specific, it’s a stress fracture that’s filled with unmineralized osteoid thus appearing as a lucency through the cortex. Common sites; 1. medial proximal femurs 2. distal scapula 3. pubic bones Complication; insufficiency fracture.
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Findings in acromegaly
Head; 1. enlargement of the frontal sinus 2. thickening of the cranial bones 3. enlarged jaw Hands; 1. beak-like osteophytes of the metacarpal heads 2. spade-like overgrowths of the distal phalanges 3. Initially joint spaces are widening but may become narrowed later in the disease due to secondary osteoarthritis. Feet; increased heel pad thickness greater than 24mm
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Hallmark of hyperparthyroidism
1. Diffuse bony demineralization 2. Subperiosteal and subligamentous bone resorption
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Findings of hyperparathyrodisim in Skull Hands Clavicle Knee Teeth Anywehre Everywhere Complications
 Skull; salt and pepper appearance due to trabecular resorption  Hands; subperiosteal resorption of the radial aspect of 2nd and 3rd middle phalanx  Clavicle; subperiosteal resorption of the distal end of the clavicle  Knee; subperiosteal bone resorption medial proximal tibial metaphysis  Teeth; loss of lamina surrounding the tooth socket  Anywhere; brown tumors  Everywhere; osteopenia  Complications: insufficiency fracture and increased propensity for ligaments/tendon rupture.
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Renal osteodystorphy Findings of two other systemic diseases, what are they Two findings
 Two things; abnormal vitamin D (osteomalacia) and secondary hyperparathyroidism from prolonged renal failure. 1. Rugger jersey spine; sclerosis of the vertebral body endplates 2. Soft-tissue/vascular calcifications are often present
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Hypothyrodism findings (usually children)
1. Bullet shaped vertebral bodies 2. Wormian bones in skull. 3. Slipped capital femoral epiphysis
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Hypoparathyroidism
Metastatic deposition of calcium i.e 1. subcutaneous tissues 2. basal ganglia
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Pseudohypoparathyroidism and pseudo pseudo hypoparathyroidism what is this Clinical features Classic findings
 Psuedohypo = parathyroid hormone receptor problem  Pseudo pseudo hypo = PTH normal, PTH receptor normal but the phenotype is indistinguishable form pseudo  Clinically; obese, round facies, short, brachydactyly  Classic findings; short metacarpal either 4th or 5th digit.
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Skeletal feature of hyperthyroidism
Accelerated bone maturation
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Pagets disease Demo Skull Vertebra Pelvic Long bones Complications
Demo: Older adults, rare in patients under 40 Skull; phase 1; sharply marginated geographic lytic region called osteoporosis circumscripta phase 2; cotton wool skull Vertebral bodies; 1. picture frame vertebral body/ivory vertebrae Pelvis; 1. asymmetric coarsened trabecular thickening 2. thickening of the iliopectineal and ilioischal lines 3. possible acetabular protrusion. Long bones phase 1; proximal articular end into the diaphysis with sharply marginated border; blade of grass/flame shaped margin progression through phases; causes bowing, coxa vara deformity Complications; 1. pathological fracture 2. malignant degeneration (osteosarcoma) 3. secondary giant cell tumor 4. secondary osteoarthritis
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Hereditary hypophosphatasia (Juvenile pagets), what makees it diffrent then adult pagest?
Epiphyseal sparing
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Osteopetrosis what is this what does it cause what appearance does it give in the spine
 Deficiency of enzyme carbonic anhydrase = inability of osteoclasts to resorb bone  Diffuse marked sclerosis of skeleton leading to brittle bone with multiple fractures  Vertebral bodies = sandwich/rugger jersey appearance.
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Osteopetrosis what is this what does it cause what appearance does it give in the spine
 Deficiency of enzyme carbonic anhydrase = inability of osteoclasts to resorb bone  Diffuse marked sclerosis of skeleton leading to brittle bone with multiple fractures  Vertebral bodies = sandwich/rugger jersey appearance.