Control system - peripheral 2 Flashcards

1
Q

receptors

A
  • Muscle
  • Tendon
  • Joint
  • Ligament
  • Skin
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2
Q

Muscle spindles

A

DETECT CHANGE IN MUSCLE LENGTH
Sensitive to length and velocity of lengthening
• Contractile element – intrafusal muscle fibres control sensitivity of muscle spindle
• Controlled by gamma-motoneurons
• Coactivated with alpha-motoneurons
• Important for perception of movement
• Stimulation of single afferent does not give conscious perception

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3
Q

Golgi tendon organs

A

• Proprioceptive - changes in muscle tension
Inhibitory input to alpha-motoneurons
• Not just involved in strong forces
• Each attached to small group of muscle fibres – sensitive to small forces
• Important feedback

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4
Q

Joint afferents

A

• Majority fire at end-range
• Some receptors fire over certain
ranges of motion
• Stimulation modifies muscle activity

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5
Q

ligaments

A

ALSO HAVE SENSORY FUNCTION
mechanical properties > joint stabaility
sensory properties >The γ-muscle spindle system > Movement and position sense or control of muscle stiffness and coordination > joint stability

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6
Q

ligament reflexes

A

Protective reflexes are too slow

State of changeable (continuously regulated) muscle stiffness at thetime of displacement/ trauma

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7
Q

Skin receptors

A

• Tactile receptors
• Important for perception of joint motion – stimulation can induce illusion of joint motion
• Relationship between firing of afferents and motor units in finger muscles
* rub on skin can stimulate muscle

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8
Q

Loss of feedback

A
  • Direct trauma to mechanoreceptors
  • Subfailure trauma to ligaments or joint capsule – compromised sensitivity
  • Abnormal feedback – Poor proprioception (almost all injuries have some form of proprioception loss)
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9
Q

What are the consequences of impaired proprioception?

A

injurt > mechanoreceptors (corrupted transductor signals > controlls (corrupted command) > Incoordinated muscle activity which leads to corrupted feedback, Increased Trauma, load, Inflammation and pain

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10
Q

Loss of feedback

A

– impaired proprioception
– Impaired control
– Potential factor in perpetuation of painand injury or recurrence
– Exercise must be specific to retrain control

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11
Q

Effusion in facet joint

A

• Reduced response to disc stimulation

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12
Q

Loss of sensory function of ligaments

• Summary

A

– Impaired contribution of mechanoreceptors to muscle control
– Exercise must be specific to retrain control

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13
Q

Reflex inhibition

A
  • Reduction in alpha motoneurone excitability in response to altered afferent input from injury
  • Can occur in absence of pain
  • Near-linear relationship between effusion volume and inhibition (Isles et al 1990)
  • What receptor – Ruffini ending in joint capsule?
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14
Q

Are all muscles affected equally?

A

*Pinching of the joint capsule leads to inhibition of knee extensors & facilitation of knee flexors
• Isolated wasting of the quadriceps with hamstring sparing in knee joint injuries
• Not uniform = greater change in slow muscles (i.e. more type I muscle fibres) leads to more functional implications

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15
Q

Atrophy of paraspinal muscles is common in low back pain

A

• Chronic low back pain
– Atrophy of paraspinal muscles – ↓ cross-sectional area - L4/5
– Signs of denervation

• Acute low back pain
– ↓ cross-sectional area at level of pain, ipsilateral to symptoms

  • Involves changes at multiple levels of the motor system: May be competitive
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16
Q

Reflex inhibition

• Summary

A

– Appears to be anatomically specific
– Particularly affects muscles that are important for joint control (extensors and antigravity muscles)
– Likely to have functional implications
– Exercise – needs to be specific
* about precision and specificity

17
Q

Muscle spindles & pain

• Summary

A

– Diffuse reflex effects – Increased activity

– Exercise must be specific – retrain patterns

18
Q

Central Neuro-immune interactions Spinal cord

A

Repetitive loading leads tp Neuropeptides & inflammatory cytokines in spinal cord

19
Q

summary

A
  • Peripheral elements of the control system are profoundly affected by pain, injury and pathology
  • Requires specific attention in rehabilitation
20
Q

Prevention and rehabilitation

A

Muscles that are inhibited will not be retrained automatically
• Retraining very important after injury

21
Q

• Activation of inhibited muscles.

A

– EMS
– EMG biofeedback
– Voluntary activation of muscles involved
– Isometric strengthening programs + EMG biofeedback
– Ultrasound feedback

22
Q

Prevention and rehabilitation

• Facilitation techniques

A
–  Manual contact
–  Verbal commands
–  Stretch facilitation
–  Traction
–  Approximation
–  Gentle resistance
23
Q

Prevention and rehabilitation

• Train sensory function

A
–  Repositioning
•  Return to target position (neutral) 
–  Acuity
•  Joint motion 
•  Vibration
–  Function
• Balance boards, etc