CONCEPT- EPIDIOMOLOGY Flashcards

1
Q

Brings back the architecture and function

A

Regeneration

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2
Q

Scar tissue formation, tooth will be mobile

A

Repair

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3
Q

Treatment for periodontitis and the junctional epithelium will be longer.

A

Root planing

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4
Q

A concept that is all gingivitis becomes periodontitis. Aka CONTINOUS DISEASE PROCESS

A

Classic model

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5
Q

Acc to russel, loe and lindhe, how many mm normally loss of attachment

A

0.1mm per yr

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6
Q

Not all gingivitis progress to periodontits, there individuals that are not progress periodontitis even though without therapy

A

Moskow 1978

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7
Q

What are the 6 sites usually done by probing by Goodson Et Al?

A

Distobuccal
Midbuccal
Mesiobuccal
Mesiolingual
Midlingual
Distolingual

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8
Q

It founds out that only 5% of 1115 sites showed attachment loss. Not all site exhibit attachment loss.

A

Goodson et al 1982

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9
Q

Only 2.8% of 3414 sites with significant attachment loss

A

Haffajee et al 1983

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10
Q

Who are the researchers contradicts the classic model

A

Goodson et al
Haffajee
Socransky
Lindhe

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11
Q

Reason why dispute the classical model

A

Sites with periodontal destruction are brought under control by unknown mechanism

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12
Q

Destruction is based on random time not on site. Aka episodic burst model

A

Random burst model

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13
Q

Destruction will burst at diff time and diff sites

A

Asynchronous multiple burst model

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14
Q

Some sites are inactive, but we should do root planing to prevent future burst

A

Treatment implication

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15
Q

Microbial composition of healthy gingiva

A

More gram + bacteria

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16
Q

What are the microbes mostly gram +

A

Streptococci and actinomyces

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17
Q

Composition associated with gingivitis

A

More gram - bacteria

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18
Q

Composition associated with periodontits

A

Several bacteria, usually gram - bacteria and increase the total microbial load

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19
Q

Give 3 criteria of periodontal pathogen

A

Be eliminated or decreased in sites
Demonstrate a host response
Demonstrate virulence

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20
Q

What are the 3 most common pathogen

A

Porphyromonas gingivalis

Actinobacillus actinomycetemcomitans or AGGRETIBACTER ACTINOMYCETEMCOMITANS

Bacteriodes forsythus or TANNERALLA FORSYTHIA

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21
Q

Mechanisms of periodontal pathogens

A

Colonize up to sub gingival area
Multiply
Can kill leukocytes

22
Q

Ability of btwo genetically distinct bacteria to recognize and adhere to one another

A

Coaggregation

23
Q

It damage the tissue cells

A

Direct pathway

24
Q

Induce the host to produce substances

A

Indirect pathway

25
Q

It breaks down the sugar

A

Saccharolytic

26
Q

It needs CO2

A

Capnophilic

27
Q

It kills the leukocytes such as WBC, macrophage

A

Leokotoxin

28
Q

It induce the bone resorption in actinobacillus actinomycetemcomitans

A

GRO-EL

29
Q

Produces susbtances to destroy the host tissue

A

Lipopolysaccharides

30
Q

Inhibits the s. Sanguis

A

Bacteriocin

31
Q

What is the drug of choice in aggregatibacter actinomycetemcomitans

A

Metrodinazole and tetratcycline

32
Q

Can’t break down carbohydrates for energy

A

Asaccharolytic

33
Q

It reduces the inflammation

A

Gingipains

34
Q

Cell to cell communication in immune response

A

Cytokines

35
Q

Another name for bacteriodes forsythus

A

Tannarella forsythia

36
Q

This microbe is elevated on the ANUG periodontitis or acute necrotic ulcerative gingivitis

A

Prevotella intermedia

37
Q

It induces cytokine,elastase and oxygen radical release from leukocytes

A

Fusobacterium nucleatum

38
Q

Capable of stimulating human gingival fibroblast to produce IL-6 and IL-8

A

CAMPYLOBACTER RECTUS

39
Q

It induce bone resorption in campylobacter rectus

A

Interleukin 6

40
Q

Has a distinct specificity for neutrophils

A

Inteleukin 8

41
Q

Recognized as pathogen

A

Eikenella corrodens

42
Q

Higher numbers at sites of periodontal destruction

A

Peptostreptococcus micros

43
Q

With tumbling motility and a tuft of flagella

A

Selemonas species

44
Q

Has limited evidence to periodontitis

A

Eubacterium species

45
Q

Highly motile microorganism

A

Spirochetes

46
Q

Study of distribution of a disease or a physiologic condition in human population

A

Epidemiology

47
Q

Aim of epidemiology

A

Prevention of disease
Maintenance of health

48
Q

3 uses of epidemiology

A

It can identify and measure the importance of health problems

It is essential for disease surveillance and control

Key instrument of in the formulation of health policies

49
Q

Characterize disease occurence and describes the phenomenon

A

Descriptive studies

50
Q

Simply observes and uses cross sectional studies and cohort studies

A

Observational studies

51
Q

Development of new hypothesis and uses the clinical trials and community trials

A

Experimental studies or intervention

52
Q

Used mainly for patient education to reinforce the patient’s oral hygiene

A

Disclosing solution