COMP 9 and OSCE Flashcards
When treating the hip flexion dysfunction with ME:
a. what is normal hip flexion ROM?
b. In the supine technique, in which direction is the patient’s activating force?
C. In the supine technique, what is the patient’s position? doc’s position?
D. In the supine technique, Which ASIS does the doc stabilize ipsilateral or contralateral?
E. In the prone technique, what is the position of the ipsilateral knee?
F. In the prone technique, which direction is the patient’s activating force?
G. In the prone technique, what landmark and on which side does the doc stabilize with the cephaled hand?
A. Knees extended: 90. Knees bent: 120-135
B. Up towards the ceiling
C. Patient at the edge of the table with affected side hanging off the table. Doc at the side of dysfunction
D. Contralateral
E. Flexed to 90
F. Towards the table
G. Ipsilateral ischial tuberosity
When treating hip extension dysfunction with ME:
A. What is the patient’s position? Doc’s position?
B. Which ASIS does the doc stabilize?
C. What is the patient’s activating force?
A. Patient supine, doc at the side of dysfunction
B. Contralateral ASIS
C. Down toward the foot of the table
When treating the Hip internal rotation somatic dysfunction with ME:
A. what is the normal ROM of internal hip rotation?
B. What is the patient’s position? What is the doc’s position?
C. What is the patient’s activating force?
A. Normal ROM for internal hip rotation is 30-40
B. Patient supine. Doc at the side of dysfunction
C. Internal rotation.
When treating the hip external rotation somatic dysfunction with ME:
A. what is the normal ROM of external rotation?
B. What is the patient’s position and doc’s position?
C. What is the patient’s activating force?
A. Normal ROM for external rotation is 40-60
When treating the Hip adduction/abduction somatic dysfunction with ME:
A. what is the normal ROM for hip adduction? Hip abduction/
B. What is the patient’s position? Doc’s position?
C. In treating adduction dysfunction, what is the patient’s activating force?
D. In treating abduction dysfunction, what is the patient’s activating force?
A. Hip adduction: 20-30. Abduction: 45-50
B. Patient supine, doc at the foot of the table
C. adduction
D. Abduction
When treating knee flexion/extension somatic dysfunction with ME:
A. what is the normal ROM for knee flexion and how is it tested?
B. What is the normal ROM for knee extension and how is it tested?
C. In treating knee flexion somatic dysfunction, what is the patient’s position and doc’s position?
D. In treating knee flexion somatic dysfunction, what is the patient’s activating force?
E. In treating knee extension somatic dysfunction, what is the patient and doc’s position? In what direction is the patient’s activating force?
A. 145-150, tested while pt is prone
B. 0, tested while pt is supine
C. Pt supine, doc at the side of dysfunction
D. Patient’s activating force is towards knee flexion
E. pt prone. doc at the side of dysfunction. Pt’s activating force is towards extension
When treating Posterior and anterior fibular head somatic dysfunction:
A. How is fibular head dysfunction diagnosed?
B. What is a posterior fibular head somatic dysfunction accompanied by?
C. what is an anterior fibular head accompanied by?
D. When treating, what is the patient’s position? Doc’s position?
E. When treating posterior fibular head dysfunction, how do you set up the patient? what is the patient’s activating force?
F. when treating anterior fibular head dysfunction, how do you set up the patient? what is the patient’s activating force?
A. Pt supine with knee flexed to 45 and gliding the fibular head anterior and posterior
B. Foot inversion, adduction, plantarflexion, tibia internal rotation.
C. Foot eversion, abduction, dorsiflexion and tibial external rotation
D. Patient is supine, with knee flexed to 90, doc at the side of dysfunction.
E. Cephaled hand holds fibular head with thumb and index finger. Caudad hand brings the foot everted, dorsiflexed, abducted, and tibia externally rotated. Patient’s activating force is opposite of that (invert, plantarflex, adduct, and tibia internally rotate)
F. Cephalad hand holds fibular head with thumb and index finger. Caudad hand brings the foot to inversion, plantarflexion, adduction, and internally rotate tibia. Patient’s activating force is the opposite of that.
When treating posterior fibular head somatic dysfunction using HVLA:
A. What is the patient’s position? Doc’s position?
B. How do you set up the patient? How is the restrictive barrier engaged?
C. How and In what direction is the HVLA?
A. Patient supine with ipsilateral knee flexed to 90. Doc at the side of the lateral on the affected side
B. Doc’s index MCP of cephalad hand on the posterior aspect of fibular head and the other hand contacts the foot/ankle to evert, abduct, dorsiflex, and externally rotate tibia to engage the restrictive barrier.
C. HVLA thrust is applied by hyperflexion of the knee and an anterior thrust on the posterior fibular head from the cephalad hand.
When treating anterior fibular head somatic dysfunction using HVLA:
A. What is the patient’s position? Doc’s position?
B. How do you set up the patient? How is the restrictive barrier engaged?
C. How and In what direction is the HVLA?
A. Patient is supine with ipsilateral hip and knee extended (leg flat on table)
B. Thenar eminence of cephalad hand over the anterolateral aspect of fibular head and the other hand contacts the foot/ankle to invert, adduct, plantarflex, and internally rotate tibia to engage the barrier
C. HVLA thrust is applied by a posterior thrust on the anterior fibular head from the cephalad hand
When treating knee (tibial) internal/external rotation somatic dysfunction using MFR:
A. what is the normal ROM of tibia internal and external rotation?
B. To treat, what is the patient’s position, doc’s position? how is restrictive barrier assessed?
C. To treat an internal rotation dysfunction using direct MFR, how do you set up the patient and force is applied? External rotation dysfunction?
D. To treat an internal rotation dysfunction using indirect MFR, how do you set up the patient and how is the force applied? External rotation dysfunctioN?
A. 10 for both
B. Patient supine with knee flexed to 90, doc at the side of dysfunction. Encompass tibial plateau with both hands, with thumbs on anterior aspect of tibia and the fingers on posterior aspect. Apply a slight distraction force and rotate internally and externally noting ease and restriction of motion
C. For direct, force is towards restriction of motion. For internal dysfunction: Rotate the tibial plateau externally and is maintained until tissue relaxation stops. For external dysfunction: rotate tibial plateau internally and maintain force until tissue relaxation stops
D. Force is towards ease of motion. For internal dysfunction: rotate tibial plateau internally and maintain force until tissue relaxation. For external dysfunction, rotate tibial plateau externally and maintain force until tissue relaxation.
when treating fibular head anterior/posterior somatic dysfunction using MFR:
A. what is the patient’s position? Doc’s position?
B. How do you set up the patient for the treatment?
C. How is direct and indirect MFR performed for anterior dysfunction?
D. How is direct and indirect MFR performed for posterior dysfunction?
A. patient supine with knee flexed to 45. Doc at the side of dysfunctions
B. Hold the fibular head between thumb and index finger with cephalad hand and caudad hand stabiizes distal tib/fib
C. For anterior fibular head dysfunction: Direct: apply posterior force on the fibular head and maintain until tissue creep. For indirect take it to it’s ease of motion so bring to anterior and hold until tissue creep.
D. for posterior fibular head dysfunction; Direct: apply anterior force on fibular head and maintain force until tissue creep. Indirect: apply posterior force on fibular head and maintain force until tissue creep.
When evaluating motion of tibia on femur, posterior glide is accompanied with_, and anterior glide is accompanied with _.
Flexion
Extension
