Colorectal Flashcards
What are haemorrhoids and where are they located?
Haemorrhoidal tissue is part of the normal anatomy which contributes to anal continence. They are mucosal cushions found in the left lateral, right posterior and right anterior portions of the anal canal – 3, 7 and 11 o’clock positions, respectively.
Haemorrhoids exist once they become enlarged, congested, and symptomatic.
How are haemorrhoids classified by their location?
Can be internal or external
External – originate below the dentate line, prone to thrombosis and are painful
Internal – originate above the dentate line and generally are not painful
How are haemorrhoids classified by their features?
Grade I – do not prolapse out of the anal canal
Grade II – prolapse on defecation but reduce spontaneously
Grade III – can be manually reduced
Grade IV – cannot be reduced
What clinical features do haemorrhoids present with?
Painless rectal bleeding seen on toilet paper and dripping into pan
May be history of straining and altered bowel habit
Importantly blood not mixed in with the stool
Pruritic
Pain – usually no significant unless piles are thrombosed
Soiling may occur as a result of 3rd or 4th degree piles
How should suspected haemorrhoids be investigated?
All patients presenting with anal bleeding should have a DRE and procto-sigmoidoscopy as a bare minimum
Routine bloods
How can haemorrhoids be managed?
Conservative
• Soften stool by increasing dietary fibre and water intake
• Topical local anaesthetic and steroid creams may be useful
Outpatient
• Rubber band ligation
• Injection sclerotherapy
Surgery (reserved for large symptomatic haemorrhoids not responding to the above)
• Haemorrhoidectomy
• Doppler guided haemorrhoidal artery ligation operation (HALO procedure)
• Stapled haemorrhoidopexy – may cause urgency as a side effect
Residual skin tags may be removed by surgical means if necessary, after haemorrhoid treatment.
How do acutely thrombosed haemorrhoids present
How are they managed?
Typically present with significant pain and a purplish, oedematous tender subcutaneous perianal mass. If presenting within 72 hours, then referral for excision should be considered. Otherwise manage conservatively with stool softeners, ice packs, topical GTN or diltiazem to reduce sphincter spasms and analgesia, symptoms should settle within 5-7 days.
What are anal fissures?
Longitudinal or elliptical tears of the squamous lining of the distal anal canal.
What are the risk factors for anal fissures?
Constipation
Inflammatory bowel disease
Sexually transmitted infections – especially HIV, syphilis and herpes
What are the clinical features of anal fissures?
Painful bright red rectal bleeding
90% occur on the posterior midline (if elsewhere consider underlying cause e.g. Crohn’s)
Chronic fissure triad – ulcer, sentinel pule and enlarged anal papillae
How should anal fissures be investigated?
All patients presenting with anal bleeding should have a DRE and procto-sigmoidoscopy as a bare minimum
Routine bloods
How are acute anal fissures managed?
Dietary advice – high fibres and high fluid intake
Bulk forming laxatives – if not tolerated then lactulose
Lubricants such as petroleum jelly prior to defecation
Topical anaesthetics
Analgesia
How are chronic anal fissures managed?
Continue management as for acute anal fissures
Topical glyceryl trinitrate first line for chronic anal fissures or diltiazem cream
If not effective after 8 weeks, then referral to secondary care for sphincterotomy or botulinum toxin.
What are anorectal abscess?
Definition – pus filled space adjacent to the anus. Very common in Crohn’s disease.
Where can anorectal abscess be located?
- Perianal (most common)
- Ischiorectal – found between the obturator internus muscles and external anal sphincter
- Supralevator – infection tracks superiorly from the peri-sphincteric area to above the levator ani
- Intersphincteric – rare and found between internal and external anal sphincters
- Pelvirectal – horseshoe abscess found in the potential space between the coccyx and anal canal and can form as a complication of other anorectal abscess.
What organisms usually cause anorectal abscess?
E. Coli
Staphylococcus Aureus and MRSA
What underlying conditions can lead to anorectal abscess?
Inflammatory bowel disorders (especially Crohn’s)
Diabetes mellitus – effect on wound healing
Underlying malignancy
What are the clinical features of anorectal abscess?
Dull aching pain, worse when sitting down or just before defecation
Pain relieved after defecation
Feeling of hardened tissue in the anal region
Pus-like discharge from the anus
How should suspected anorectal abscess be investigated?
Good history and examination
To investigate the cause – colonoscopy and blood tests e.g. cultures and inflammatory markers
MRI and transperineal USS (gold standard) but these are rarely used
Must rule out haemorrhoids
How should anorectal abscess be managed?
Surgical incision and drainage under local anaesthetic. Wound is usually packed or left open but not stitched healing in around 3-4 weeks
Antibiotics only used if systemic sign of infection
What is a volvulus?
Torsion of the colon around its mesenteric axis resulting in compromised blood flow and closed loop obstruction.
Where are the most common locations for volvuli to occur?
Sigmoid is the most common location accounting for 80% of colonic volvuli, Caecal volvuli make up the other 20% and this usually occurs in people where developmental issues has resulted in their caecum not being retroperitoneal.
What are the risk factors for sigmoid volvulus?
Older patients
Chronic constipation
Chagas disease
Neurological conditions e.g. Parkinson’s disease and DMD
Psychiatric conditions such as schizophrenia