CN Flashcards
Unilateral cn III, IV, VI
Cavernous sinus lesion
Combined unilateral cn IX, X, XI
Jugular foramen syndrome
Combined bilateral cn X, XI, XII
If lower motor neuron Bulbar palsy
If upper motor neuron. Pseudobulbar palsy
Olfactory nerve
CN I
Observe external nose
Observe internal nose
Check smell by texting each side separately from its different scent
Optic nerve
CN II Observation of eyes Test visual acuity...snellen Test visual field (peripheral) aka confrontation Pupillary light reflex Look in eye
Oculomotor, Trochlear, abducens
CN III, IV, VI Confrontation....50, 60, 70, 90* Corneal light reflex...look for reflexion 6 Cardinal planes of gaze Accommodation Convergence with finger to nose
Trigeminal
CN V Corneal blink reflex Facial sensation Sharp/light touch Motor: open mouth, clench teeth, palpate muscles Test jaw jerk General sensation ant. 2/3 tongue
Facial
CN VII Inspect facial drop asymmetry Facial expression muscles: look up, wrinkle forehead Shut eyes tightly Smile..compare nasolabial grooves Taste ant. 2/3 tongue
Vestibulocochlear
CN VIII Observe external ear Observe internal ear Webers test: lateralization Rinnes test: air bone conduction
Accessory
CN XI
Examine SCM and trapezius atrophy
Shrug shoulders turn head against resistance
Glossopharyngeal, vagus
CN. IX, X Hoarse or nasal? Says AH...asymmetrical soft palate movement Gag reflex (sensory IX motor X) Kuh la mi
Hypoglossal
XII
Listen to articulation
Inspect tongue for wasting , fasciculations
Protrude tongue: unilateral deviates to affected side
Tongue to cheek
Bell’s palsy
Lesion is peripheral to the geniculate ganglion Lower motor neuron Spares forehead Cn VII No loss of taste
Affected taste
CN VII
Sensory
Peripheral lesions in cn VII must be PROXIMAL to the stylomastoid foramen to affect taste
Unilateral cn V, VII, VIII
Cerebellopontine angle lesion
Primary neurons
Olfactory
Unmyelinated processes of the ciliated receptors in the upper part of the nasal mucosa are gathered into about 20 branches which pas through the crib reform plate of the ethmoid bone to the olfactory bulb.
Secondary neurons
Olfactory
Myelinated processes of the bipolar cells of the bulb form olfactory tract and terminate in the primary olfactory cortex
Tertiary neurons
Olfactory
Neurons extend from the primary olfactory cortex to the entorhial cortex, lateral preoptic area, amygdaloid, and medial forebrain bundle
Retrobulbar neuritis
Involves the optic nerve or tract, the most common cause is multiple sclerosis
Optic or bulbar neuritis
Includes various forms of retinitis
Papilledema.
Choked disc
A commonly seen symptom of increased intracranial pressure due to brain tumors, abscesses, hemorrhage, hypertension
Optic atrophy
Associated with decreased visual acuity and a change in the color of the optic disc to light pink, white, gray
Primary optic atrophy
Caused by processes that involve the optic nerve and do not produce papilledema
Secondary optic atrophy
Is a sequel of papilledema
Primary simple optic atrophy
May be due to neuritis, glaucoma, or increased intracranial pressure
Fosters Kennedy syndrome
May be caused by tumors at the base of the frontal lobe and is characterized by ipsilateral blindness and anosmia (with atrophy of the optic and olfactory nerve ) and contralateral papilledema
Amaurotic familial Idiocy
Aka Tay Sachs disease
Cerebromacular degeneration with severe mental deficiency occurring in Jew families and is associated with blindness, optic atrophy, and dark cherry red spot in place of the macula lutea
Argyll Robertson pupil
Reacts only to accommodation. It has neither a direct or indirect reaction to light. It was once considered pathognomonic of tabes dorsalis, but it has now been found to occur as a diabetic complication
Holmes aide syndrome
Characterized by a tonic pupillary reaction and the absence of one or more tendon reflexes. The pupil is said to be “myotonic” with a very slow, almost imperceptible contraction to light and in near vision, a slower dilation upon removal of the stimuli. Ppl with this syndrome have an abnormal sensitivity to weak solution, instilled into the conjunctival sac is demonstrable in affected eyes; tonic pupils constrict, whereas pupils of normal eyes remain unaffected
Holmes adie syndrome
Aka adies pupil/tonic pupil
Is a pupil that reacts to,light very slowly, remains constricted longer, then dilates slowly. This occurs most often I. Young women and is considered benign