Clinical management of dental anomalies Flashcards
Hypoplastic form of amelogenesis imperfecta with gross calculus deposits
Calculus as too sensitive to brush - practically no enamel formation
Management with full composite crown coverage and preformed metal crowns
Genetic anomalies of dentine
Dentinogenesis imperfecta - loss of enamel with brown/ yellow opalescent dentine - which wears rapidly
-DSPP mutation affecting non-collagenous proteins; variation in severity of presentation
Radicular dentine dysplasia - radicular pathology; aetiology unknown
DI with OI (syndromic) - collagenous proteins affected
Dentinogenesis imperfecta epidemiology (3)
Prevalence 1:6000
Usually autosomal dominant inheritance
Primary and Secondary dentitions – but primary always more severely affected
Dentinogenesis imperfecta clinical features (2)
Enamel usually fractures off early due to lack of dentine support. Soft exposed dentine then quickly wears
Dentinogenesis imperfecta histological features (1)
Irregularly formed and poorly mineralised dentine
Dentinogenesis imperfecta radiographic features (3)
Bulbous crowns, short thin blunt roots, obliteration of root canal
Clinical considerations of treating A and DI (4)
Masking of underlying discolouration
Reduced shear bond strength of resin composites
Orthodontic management
-would rather use removable appliances due to bonding problems
Life-long maintenance of restorative interventions
Tooth morphology anomalies (4)
Dens in dente / dens invaginatus
Dens evaginatus + talon cusp
Double teeth (Fusion, Germination)
Microdontia - peg/conical lateral incisors
Principles of management: tooth morphology anomalies (3)
Early diagnosis of dens-in-dente - as subsequent RCT is difficult, aim to fissure seal / occlude communicating channels or caries-prone sites
Judicious grinding of talon cusps + duraphat applications after root formation is complete
Management of double teeth - very complex, seek specialist opinion (orthodontic assessment) management is dependant on pulp morphology
Talon cusps - facts and figures (6)
Prevalence: uncommon, increased in some racial groups (7.7% North Indian population; 0.17% American population; 0.06% Mexican population)
Increased in males (2:1 male:female)
More common in maxillary teeth
3 times more common in permanent dentition than primary dentition
The maxillary lateral incisor is the most frequently affected tooth
Labial surface of teeth may also (uncommonly) be affected
IF PT HAS TALON CUSP - LOOK OUT FOR OTHER DENTAL ANOMALIES AS WELL
Talon cusps - aetiology (5)
Multifactorial (polygenetic and environmental factors involved) disturbance of tooth formation
Familial associations
Increased prevalence in patients with consanguineous parents
Increased prevalence in some syndromes (e.g Ellis-van Creveld; Rubinstein-Taybi; Sturge-Weber)
May be associated with other dental anomalies (e.g supernumerary teeth; dens invaginatus; microdent lateral incisors; shovel-shaped incisors; bifid cingulum; enlarged cusps of Carabelli)
Talon cusps - clinical problems (5)
Occlusal interference which may lead to: displaced teeth; TMJ dysfunction; acute apical periodontitis
Caries
Poor aesthetics
Tongue irritation
Attrition or fracture of cusp with pulpal exposure and ensuing pulp necrosis
Is there pulpal tissue in the talon cusp? (4)
You can not be sure!
Histological studies have shown some talon cusps do contain pulpal extensions and others do not
Radiographs are of little diagnostic help
Large talon cusps that project away from the tooth surface are the MOST likely to contain pulpal tissue
Talon cusp tx: caries prevention (2)
Early use of fissure sealant or composite to occlude caries-prone fissures between talon cusp and tooth surfaces
Give appropriate oral hygiene instruction
Talon cusp tx: cusp reduction (5)
Be aware of possibility of pulp exposure
Objective is to undertake GRADUAL cusp reduction with reparative dentine formation (and pulpal recession)
-you can do it all at once if pt wants but try to wait until the root is mature in case you expose! Put Dycal on
Literature suggests variable time intervals: 4 weeks, 6 weeks, 8-weeks, 4 months (go for 3 months)
1-1.5 mm of tooth tissue reduction recommended each visit
Placement of F varnish after each visit to desensitise exposed dentine