clinical corell PAD Flashcards
signs and symptoms of peripheral arterial disease (PAD
HPI:
Claudication
Cramping and pain in legs with walking a certain distance
Diabetics may have leg fatigue instead of pain
Rest pain
Cramping in legs with elevated, relieved with dependent position
“black” ulcers or toes
Chronic ulcers that don’t heal
Painful ulcer despite neuropathy
signs and symptoms of peripheral arterial disease (PAD
HPI:
Claudication
Cramping and pain in legs with walking a certain distance
Diabetics may have leg fatigue instead of pain
Rest pain
Cramping in legs with elevated, relieved with dependent position
“black” ulcers or toes
Chronic ulcers that don’t heal
Painful ulcer despite neuropathy
treatment options for PAD
Antiplatelet therapy Aspirin or Clopidogrel (Plavix) LDL cholesterol level of less than 100 mg/dL HgA1c of less than 7% Control of Hypertension Possibly with benefit of ACE inhibitor Tobacco cessation embelectomy angioplasty (baloon)
Risk factors PHD of PMH
Diabetes Coronary Artery Disease Hypertension Hyperlipidemia Obesity Aortic aneurism Age Race Hispanic African American Male gender
Metabolic risk factors diabeties
Hyperinsulinemia or hyperglycemia (2 hour PP after 100 gm oral dose > 200)
Hypertriglyceridemia (> 150) and low HDL (130/85)
Obesity: waist line > 40 inches in males, > 35 inches in females (waist alone will diagnose 80% of patients with metabolic syndrome)
Every increase of 1% of HgA1c, risk factor for PAD goes up 28%
PAD risk facotrs continued…PSH, SH, FH, ROS
PSH
Coronary artery stent placement or lower extremity stent placement
SH
Tobacco use
FH
Any family with history of risk factors or PAD
ROS
Same as HPI, may have someone coming in for other complaints and pick up PAD in this area
2 greatest risk factors for PAD
diabeites and smoking!!
PE derm
Skin thin, atrophic Lack of pedal hair Brittle rigid nails Cold Hemosiderin deposits May have waxy appearance Ulcers Gangrenous Granular with a lot of fibrotic tissue
Where palpate pulse during PE?
Palpate dorsalis pedis (tween 1st n second metatarsals) and posterior tibial (just behind medial maleoulus) pulses
If not palpable
may palpate popliteal and/or femoral pulses
Doppler DP, PT and perforating peroneal
Cap refill time
May be greater than 3 seconds (push on end of toe wont be able to see through the nail)
Dependent rubor
Foot looks red when dependent, pale with elevation
Doppler
Triphasic
normal
Doppler biphasic
mild to moderate PAD
Doppler monophasic
Severe PAD
ABI normal
.9-1.3
PAD with intermitent claudication
.5-.9
severe limb ischemia, rest pain n ulceration
Guidelines for Obstruction
segmental pressures
20-30mmHg difference between adjacent cuffs
30 mmHg change along leg from thigh to ankle
20mmHg or more difference between opposite leg, same level
Wagners index
Ischemic Index = leg pressure/ arm pressure Successful healing Above 0.35 in arteriosclerosis Above 0.45 in diabetes
Doppler ultrasound
Ultrasound waves travel from probe to vessels – bounce off of blood cells back to probe
Pitch – function of how fast blood cells moving – faster the cells move the higher the pitch
Loudness – function of how many blood cells the waves hit – the more cells hit the louder the sound
Doppler Ultra wave B intrep
Ventricles contract, aortic valve opens
Doppler Ultra wave c intrep
Ventricles relax, valve open, reflux in large arteries
Doppler Ultra wave d intrep
Aortic valve closes, pressure increases in large vessels
Doppler Ultra wave e intrep
Return to diastolic baseline
Normal triphasic doppler ultra sound waves
Bidirectional
Rapid upstroke/ downstroke
Flow reversal
Arterial wall rebound