clinical approach to chest pain Flashcards

1
Q

what is the most common cause of non-emergent chest pain

A
musculoskeletal pain (36%)
while cardiac is only 16%
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2
Q

what are the 4 killer chest pains

A

Acute Coronary Syndromes
Pulmonary Embolism
Aortic Dissection
Tension Pneumothorax (least common)

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3
Q

what are common causes of cardiac chest pain

A
Angina
Myocardial infarction
Aortic valve disease
Hypertrophic or congestive cardiomyopathy
Aortic dissection
Pericarditis
Mitral valve prolapse
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4
Q

list areas that angina present

A

most common: the substernal area

next most common: jaw, epigastrium and inner aspect of the left arm

3rd most common: the neck, right shoulder and inner right arm

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5
Q

Physical Signs in Acute CAD

A

Pallor
Sweating
Anxiety
Tachycardia
Rise in blood pressure (all of the above are due to sympathetic stimulation due to pain)
S4 gallop
Mitral regurgitation murmur (w/ inferior wall ischemia or infarct)
Paradoxically split S2 (LBBB)
Pulsus alternans (indicate impending LV failure and cardiogenic shock)

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6
Q

Acute Coronary Syndrome

A

Unstable Angina
NSTEMI (non ST-seg elevation MI)
STEMI (ST-segment elevation MI)

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7
Q

method of how coronary thrombosis is caused

A
  • plaque equilibrates between vulnerable and non-vulnerable plaques.
  • when it is vulnerable physical or mental stress can trigger rupture.
  • rupture triggers intracoronary thrombosis which can be occlusive if coagulability or vasoconstriction increase.
  • this causes symptoms of angina, unstable angina, myocardial infarction, or even sudden death depending on the size.
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8
Q

in an acute MI can an EKG be normal?

A

yes - about 10% of the time it will be, especially with an occlusion of the circumflex artery

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9
Q

signs of a MI on an EKG

A

elevated ST segments, inverted T waves, the development of Q waves within 12 hours.

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10
Q

what other syndromes can look like an MI on an EKG

A

Pericarditis, J-Point elevation, W-P-W Syndrome

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11
Q

how can you tell the difference between a NSTEMI and unstable angina

A

EKG will look the same so you need to look at the biomarkers

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12
Q

general rule for ST elevation vs depression

A

ST depression = ischemia,

ST elevation = infarction.

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13
Q

reciprocal changes on an EKG

A

is an electrical phenomena - go away when an ST segment elevation goes away

If the ST depression goes away in 24 hours then it is attributed to electrical reciprocity due to the myocardial infarction. If it stays, it may indicate ischemia.

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14
Q

Troponin I & T

A

Specific for cardiac injury
Most sensitive when you take the diagnostic window out to 24 hours
It is not an earlier marker!
The majority of Troponins are not elevated in the first few hours
It remains elevated for many days!
When elevated is a marker for increased risk with non-ST elevation Acute Coronary Syndrome

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15
Q

Stable angina

A

Angina that occurs at a predictable amount of energy expenditure or emotion

goes away with rest or nitroglycerin

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16
Q

true or false pain occurs when ischemia begins

A

False: pain does not occur until 30 seconds after the occlusion. This is after there have been EKG changes present

17
Q

grades of Angina Pectoris

A

I- only w/ strenuous activity
II- slight limitations on normal activities (fast walking/stairs)
III- limitations to ordinary activity- occurs when walking 2 blocks or 1 flight of stairs)
IV- inability to carry on physical activity w/o pain, may even ocurr at rest

18
Q

when should a stress test be used

A
  • if the history is indeterminate for cardiac chest pain vs. musculoskeletal pain

may also be used:
Estimating progress and severity of disease
Evaluation of therapy
Screening for latent coronary disease (not common)
Evaluation of arrhythmias

19
Q

Bruce protocol

A

5 stages of a stress test increasing speed and incline to increase the HR and BP of the patient and monitor symptoms, signs and the ECG to look for coronary insufficiency.

20
Q

normal stress ECG

A

upsloping ST-segment

21
Q

abnormal stress ECG changes

A

The ST position 2 mm from the end of the QRS (or J point) is observed. 1mm or greater depression with a horizontal or downward slope is (+) for ischemia.

22
Q

you can increase the sensitivity of a stress test by

A

adding imaging - either echo or nuclear

23
Q

Pericarditis – EKG

A

The EKG in pericarditis may show diffuse (every lead) ST elevation of lumen to possibly 2mm. This, if present, is diagnostic. Not all pericarditis has a positive ECG. Myocardial infarction ST elevation is usually greater or localized. (For example: ST elevation is 23F leads only).

also fever and chest pain is better when sitting up

24
Q

3 major symptoms of aortic stenosis

A

CHF – 2 year survival
Syncope – 3 year
Angina – 5 year

25
Q

GI causes of chest pain

A

GERD
diffuse esophageal spasm (present like angina and is relieved by nitro)
cholecystitis and cholelithiasis

26
Q

pulmonary causes of chest pain

A

pulmonary hypertension
pneumothorax
pulmonary embolism

27
Q

neuromuscular causes of chest pain

A

Herpes zoster (shingles)
Cervical arthritis
Chest wall pain and tenderness

28
Q

describe the pain associated with an aortic dissection

A

Cataclysmic onset - (sudden and severe)
Most severe at onset, “tearing”, “stabbing”
Tendency to migrate (may tell you were the tear is)
Anterior thorax (proximal); interscapular (distal)

29
Q

signs of an aortic dissection

A
Pulse deficit (proximal), aortic regurgitation (proximal)
Neurological deficits (proximal) – CVA, paraparesis, peripheral neuropathy, vasovagal
(EKG is often normal)
30
Q

who is more likely to have an aortic dissection

A

Men 2x, 50 – 60 year old, proximal 2x

Predisposing factors:
HTN, pregnancy, congenital (bicuspid aortic valve, coarctation, Marfan, Ehlers-Danlos)

31
Q

sign of a pulmonary embolism

A

positive D-dimer test

32
Q

Pneumothorax

A

Air in the pleural space
Spontaneous
Secondary
Tension pneumothorax can be quickly fatal