cleft exam 4 Flashcards

1
Q

VPI: anatomical defects

PLUG WIT

A

(1) Anterior palatal levator insertion
(2) Absence or weakness of uvular muscle
(3) Glossopalatine muscle problems
(4) Webbing of pharyngopalatine muscle
(5) Tongue impairments

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2
Q

VPI: Palatopharyngeal disproportion ADATA

A

(1) Abnormal basocranial angle
(2) Large anterior-posterior dimensions of pharynx
(3) Reduced horizontal aspect of velum
(4) Tonsils and adenoids

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3
Q

VPI: interference with motion AGMP

A

(1) Too anterior glossopalatine muscle insertion
(2) Abnormal pharyngopalatine muscle insertion
(3) Maxillary advancement surgery

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4
Q

VPI: structural factors VLM ACB PNS RAP

A

vii) Velar length and mobility: 85% moving
ix) Angulation of cranial base- tilted inferiorly causes greater distance between velum and pharyngeal wall
x) Position of nasal spine -posterior part of posterior bone where velum attaches, can direct velum upward or downward
xi) Role of adenoid pad- Enlarged may impede closure

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5
Q

Bankson and Byrne

A

(1) S/z
(2) Speech difficulty is not due to distorted or missing dentition
(3) Mixture of other problems
(a) Coordination of tongue
(b) Maxillary arch too narrow

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6
Q

Starr SMArT move

A

(1) Significant malocclusions is the problem
(2) Can cause artic difficulty
(3) Difficult for tongue to move where it needs to

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7
Q

Counihan

A

(1) Maxillary arch too narrow, short

(2) maxillary arch problems→ artic problems

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8
Q

Powers

A

good artic can occur despite anomalies

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9
Q

McCutcheon

A

even something as small as reduced ruggae produced errors

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10
Q

neuromotor patterns

A

learned in first two years, very hard to correct negative compensatory behaviors once they are learned– INTERVENE EARLY

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11
Q

compensatory speech/language

A

substitutions

i) Glottal stops and pharyngeal fricatives
iii) Voice disorders
(1) Hyponasality- pharyngeal flap, obturator bulb too big
(2) Harshness/ hoarseness/ vocal roughness
(3) Intensity
(4) Hyper-hypofunction cycle
(5) aspirate

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12
Q

obligatory speech/language

A

triad: hypernasality, nasal emission, weak pressure consonants

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13
Q

articulation etiologies LOAN

A

learning problems
occlusal (not dentition)
auditory ability
nasal emission

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14
Q

pseduo/prognathis

A

pseudo- maxilla retruded

prognathis- mandible protruded

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15
Q

craniofacial team

A

i. Pediatrician- crucial role- big picture, assesses and manages health aspects
ii. plastic/ recon surgeon- closes lip/palate, secondary procedures for VPI, soft tissue reconstructions of face
iii. oral max surgeon- maxilla mandible
iv. orthodontist- teeth, jaw
v. pediatric dentist- dental health of child, assists in recommendations, works with makers of obturators etc
vi. Otolaryngologist- expertise in ears- PE tubes
vii. geneticist- family history analysis, clefts, recurrence
viii. clinical psychologist- helps family cope with difficulty, counseling
ix. Social worker- counsels family and individuals, patient and family advocacy, investigates services available and obtains services
x. **Pediatric nurse practitioner- very important
xi. **
nurse- very important, preps patient
xii. Audiologist- does hearing testing, impact of HL on development, speech
xiii. coordinator- often SLP

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16
Q

resonance testing Ct TOM

A

oral manometer
tongue anchor
counting 60-70-90

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17
Q

nasal emission SACS

A

grimace (ala pinching)
clouding- mirror
scape-scope
stethoscope/airplane headphones

18
Q

phonation

A

aspirate, hyper-hypofunction

19
Q

speech: functional factors BEFOR W

A

reward, bonding, feeding/ breathing, overprotection, wait until surgery, impact on childs ego

20
Q

wells: strengthen. CHYBESS

A

yawning, yawn-phonate, chewing, puffing, swallowing, blowing, sucking, ejecting

21
Q

wells: excursion SIBS

A

stimulate velum and pharyngeal wall, speech bulb, stop a sneeze, quick inhalation

22
Q

wells: airstream

A

spitball, blow pen

23
Q

shelton no:BARS

A

sensory/somasthetic feedback doesnt not improve VPC; process of using speech appliances and reduction is not helpful; biofeedback does not help generalization

24
Q

kuehn CPR MANE

A

CPAP provides resistance against velum to strengthen; motor activities may not be as effective bc there are fewer nerve endings as you go deeper into oral cavity- no motor memory

25
Q

first meeting MDQ

A

2-3 mos post surgery or after lip has healed, model attachment and vocal play, describe/compare palatal structures, describe surgeries, embryology (blame), field Qs about childs intelligence

26
Q

second meeting LLN

A

7-12 months old, demonstrate language stimulation (vocal play continues, pitch changes, varied parts of speech, early sounds etc REDUCE GESTURE), explain naturalistic learning, teach developmental norms

27
Q

third meeting

A

6-8 weeks after palatal surgery (by 20 mos old), add more sounds (k/g, plosives), relaxation to avoid grimacing/tension, simple sentences, teach developmental order of sounds

28
Q

language risk factors

A

external: negative or non-reactions of caregivers,
lack of adaptive interactions, pity anxiety
internal: hearing, neurological, cognitive

29
Q

newborn risks

A

mood set by CF team; lack of mutual comforting, bonding, communication; parents must work to counteract early exposure to pain

30
Q

infant/toddler risks

A

parents must be intimately involved, positive mood

31
Q

identification/intervention risks

A

early intervention a must! knowledgeable transdisciplinary team necessary

32
Q

language stimulation training

A

motivation of child (bonding, naturalistic teaching), ID/use positive compensatory behaviors (lower lingual placement), reduce parents stress by teaching about child’s language, involve parents, additional management (referrals: ET, neuro, devt, physical)

33
Q

informal-structured situations

A
  1. Expansion of fragments
  2. Repetition of correct production
  3. Parallel talk
34
Q

reactions to child utterances

A

try to understand errors, reinforce correct productions, repeat incorrect productions correctly (model) but keep in mind the developmental progression, extinguish negative compensatory behaviors

35
Q

early parent training

A
nurse- feeding, hygienic technique
coach parents (acknowledging childs progress, strengths, weakness)
36
Q

palatal training: indirect

A
  1. No touching structures
  2. No ejecting blowing etc
  3. Emphasis on artic: k, g, ng hoping will effect VP closure
  4. Mixed results- improve artic, but no change in VP closure
37
Q

palatal training: direct

A
  1. Manipulation- move velum
  2. Stroking massaging velum and ph wall
  3. Wells activities: Blow, swallow, ejecting
  4. Speech appliances and reduction (25%)
38
Q

palatal lift

A

i. fits into hard palate vault
ii. may be connected to dentition,
iii. almost touches pharyngeal wall
iv. velum sits on posterior part of appliance (bulb)
v. require less movement for velum to touch ph wall
vi. reduction: if velum strengthens, you can shave off some bulb to see if velum can close

39
Q

palatal stimulating device

A

i. k g, non nasals, sibilants
ii. velum moves up against bulb
iii. isometric exercises to strengthen velum
iv. reduction- of bulb to see if velum can still be pressed against smaller bulb- strengthened

40
Q

palatal stimulating during non-speech acts

A

a. Blow suck move palate
b. Massengill: Swallowing best for VP closure
c. Peterson: Touch stroke seemed to improve VPC but no carryover

41
Q

biofeedback

A

a. Client observes movement of own velum with nasoendoscopy
b. Shelton: Does not carryover, become automatic
c. Shprintzen: DOES help with VP closure
PROMISING