Classification of Periodontal Diseases Flashcards

1
Q

What are the 4 stages of periodontal disease?

A

1- Early/mild, <15% interproximal bone loss at worst site
2-moderate, coronal third of tooth bone loss at worst site
3-Severe, mid third of tooth bone loss at worst site
4-Very severe, apical third of tooth bone loss at worst site

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2
Q

How do you classify the extent/distribution of periodontitis?

A

Localised <30% of teeth
Generalised >30% of teeth
Molar Incisor pattern

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3
Q

How do you stage periodontal disease?

A

A- Slow, <0.5 (percentage loss over age), max bone loss is less than half the patients age
B-Moderate, 0.5-1
C- Rapid, >1.0 max bone loss is more than the patients age

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4
Q

In an assessment of current periodontitis status, what would currently stable be?

A

Bleeding on Probing <10%
PPD less than or equal to 4mm
No BoP at 4mm sites

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5
Q

In an assessment of current periodontitis status, what would currently in remission be?

A

Bleeding on probing >10%
PPD more than 4mm
No BoP at 4mm sites

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6
Q

In an assessment of current periodontitis status, what is currently unstable?

A

PPD of more than 5mm
OR
PPD more than 4 and BoP

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7
Q

What are some risk factors for periodontitis?

A

Smoking
Sub-optimally controlled diabetes
Age
Stress
Medications
OH
Genetic conditions (papillon-lefevre syndrome, down’s syndrome)
DIseases (leukaemia, HIV, agranulocytosis)

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8
Q

What order do you write a diagnostic statement for periodontitis?

A

Extent-Periodontitis- Stage- Grade- Stability- Risk Factors

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9
Q

What is a BPE?

A

Screening tool that provides a provisional diagnosis of periodontal health, gingivitis or periodontitis.

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10
Q

What effect does sub-optimally controlled diabetes have on periodontal disease?

A

Hyperglycaemia in diabetes may modulate RANKL and OPG ratio and thus contribute to alveolar bone destruction
In hyperglycaemia production of AGE increases which leads to exacerbation of inflammation

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11
Q

What effect does poorly controlled periodontal disease have on diabetes?

A

Periodontal bacteria and their products produced locally in the inflamed periodontal tissues, enter the circulation and contribute to upregulated systemic inflammation.
This leads to impaired insulin signalling and insulin resistance, thus exacerbation of diabetes

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12
Q

An increase in what levels causes exacerbation of diabetes?

A

HbA1c

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13
Q

What drugs can result in an increased deposition of connective tissue supporting a hyperproliferative epithelium?

A

Anticonvulsant: Phenytoin
Immunosuppressants: Cyclosporin
Calcium channel blockers: Nifedipine, amlodipine

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14
Q

What are the indications for surgical therapy at re-evalutation?

A

Good OH, persistent deep pockets with BOP

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15
Q

What factors influence whether a patient gets periodontal surgery or not?

A

Smoking
Compliance
Oral hygiene
Systemic diseases
Suitability of site (access, soft and hard tissue factors)
Prognosis of tooth, importance of tooth
Availability of specialist treatment
Patient preference

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16
Q

What is recession type 1?

A

Gingival recession with no loss of inter-proximal attachment
Interproximal CEJ is clinically not detectable at both mesial and distal aspects of the tooth

17
Q

What is recession type 2?

A

Gingival recession associated with loss of interproximal attachment. The amount of interproximal attachment loss (measured from the interproximal CEJ to the depth of the interproximal sulcus/pocket) is less than or equal to the buccal attachment loss

18
Q

What is recession type 3?

A

Gingival recession associated with loss of interproximal attachment
The amount of interproximal attachment loss is greater than the buccal attachment loss

19
Q

What is necrotising stomatitis?

A

When the necrosis progresses to deeper tissues beyond the mucogingival line, including the lip or cheek mucosa or the tongue etc

20
Q

What are the aims for treatment of necrotising periodontitis?

A

To arrest the disease process and tissue destruction
To control the patients general feeling of discomfort and pain that is interfering