Class 5: Mental Health Flashcards
Positive symptoms of pyschosis
-Delusions, hallucinations, disorganized thinking and behaviors
-Poor social functioning
-Sudden onset improved with antipsychotic medication
Negative symptoms
-Absence of thought and behavior patterns contributing to inappropriate social functioning
-Slow onset that worsens
Delusions
-aThought content, fixed false beliefs
-Cannot be corrected by reasoning or evidence
Examples of delusions
-Grandiose, persecutory, somatic & nihilistic
-GPSN
Nihilistic
Belief that they’re dead
Persecutory
Being watched
Somatic
aBodily function
Disorganized thinking
Illogical speech, impaired reasoning, loose associations
Abnormal motor behaviour
-Agitation, aggression (perceived or toward others), catatonic excitement, echopraxia, hypervigilance, mutism, rigid (could be r/t medications), waxy flexibility
Echopraxia
Imitating others behaviors
Catatonic excitement
Hyperactivity of purposeless activities and abnormal movements such as grimacing and posturing
Waxy flexibility
Posture held in an unusual position
Brief pyschotic disorder criteria rules
-2 or more of the following, presenting for 1 month. At least one of these needs to be: Delusions, hallucinations or disorganized speech
-Episode lasts at least 1 day but less than 1 month with a return to a normal level of functioning
Brief psychotic disorder criteria manifestations
Catatonic behaviour, delusions, disorganized speech and behavior, hallucinations
Brief psychotic disorder etiology
Can result from changes in physical status, major life events, drug use, and environmental changes
Prevalence of brief psychotic disorders
Onset in the mid 30s, can occur at any age, twice as common in females, more common in developing countries
Delusional disorder criteria rules
-1 or more delusions over 1 month
-Delusion criterion for schizophrenia has not been met, if hallucinations are present they are not prominent or r/t delusion
-Function is not markedly impaired
Subtypes of delusional disorder
-Jealous, somatic, erotomania (sexual), & grandiose
-JSEG
Delusional disorder prevalence
-Rarely exists on its own, comorbidities include: Mood disorders, OCD, and personality disorders (specifically paranoid, schizoid, avoidant)
-Can begin in adolescence
Delusional disorder may go..
Undiagnosed because behavior is not noticeably abnormal
If delusions are…
Somatic, individuals risk unnecessary medical investigations and legal interventions
Neurobiological etiology of delusional disorder
-Asymmetrical temporal lobes, possible neuro-degenerative component
-Sensory alterations in the nervous system associated with cortical changes
-Perceptions linked with an interpretation that has deep emotional significance but no verifiable basis
SDOH + delusional disorder etiology
Early life experiences
Schizophrenia criteria rules
-2 or more of the following over a 1 month period. Must include delusions, hallucinations, or disorganized speech
-Lasts >1 month >6 months
Schizophrenia manifestations
Delusions, disorganized speech and behavior, negative symptoms, and hallucinations
Phase I (acute) assessment of schizophrenia
-Onset of illness, loss of function abilities
-Florid, disruptive symptoms (positive or negative)
Phase II (stabilization) assessment of schizophrenia
-Period of wellness
-Move from acute care to community-based services, still require supervision
Phase III (maintenance) assessment of schizophrenia
Progress in not a linear
Prevalence of schizophrenia
-Late teens/ early adolescents
-Becomes chronic in 80% of individuals who are diagnosed, poor functioning before onset of disease
-More common in males, except in later onset
Early onset of schizophrenia
Early onset ( 18-25 yrs.): Increased brain abnormality & increased apathy
Later onset of schizophrenia
Later onset (25-35 yrs.): More likely to be female, less structural brain abnormality
Schizophrenia genetic etiology
-Heredity, polygenic disease, irregularities on chromosomes 13 and 15
-Rate increases by 10 x if you have a first degree relative with schizophrenia
Schizophrenia neurobiological etiology
-Neurotransmitters; dopamine, and serotonin, glutamate
-Dopamine & serotonin; based on the use of use of conventional antipsychotics
-Glutamate; involved in neural communication, memories, learning, regulation, CNS maturation
-GSD
Schizophrenia brain structure abnormalities + etiology
-Differences in structure of the ventricles
-Lower brain volume, increased CSF, decreased blood flow to the frontal lobe
Schizophrenia comorbidity + etiology
Obesity & diabetes
Schizophrenia etiology + prenatal risk factors
Viral infections, poor nutrition, hypoxia, toxins, birth complications, age of conception
Schizophrenia etiology + psychological stressors
-Prolonged increased stress results in aHypothalamus development
-Recreational drugs increase dopamine
-Trauma
-PRT
Schizophrenia etiology + SDOH
Adverse living conditions, migration
Across the lifespan considerations + children & adolescents
-Look for patterns
-Consider if the difficulties are r/t developmental tasks (i.e. exploration with drugs, alcohol, and sex; seeking autonomy)
Across the lifespan considerations + older adults
Presence of additional factors may increase patients’ stress and potential for secondary concerns
Delusional disorder general appearance + engagement
-No functional impairment, may become more distracted as it progresses
Delusional disorder general appearance + grooming & dress
Disrupted self-care
Delusional disorder affect
Anxiety with progression of illness
Delusional disorder thought content + delusions
Fixed false beliefs
Delusional disorder thought content + suicide, homicide, self-harm, depressive & anxious cognitions
Assess impulsivity to act in relation to the delusion