CIEDs Flashcards

1
Q

CT and MRI can both interfere with ICD function

A

true

CTs can cause oversensing

MRIs not sufficient evidence. Manufacturers may list conditional circumstances when to perform. Most centers will not do MRI

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2
Q

imaging can help identify the device when it is not known

A

true

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3
Q

Indications for CIED

A
bradycardia
Sinus node disease
long QT
HCM
Dilated cardiomyopathy -  3chambers (RA and both ventricles)
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4
Q

Subcutaneous ICD

A

larger than transvenous models
no antitachy functions or sustained antibrady functions
higher defib threshold

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5
Q

CIEDs respond to low-amplitude electrical signals nd can be altered by extraneous signals such as:

A
electrocautery
RFI ablation
MRIs
Radiation therapy
lithotripsy
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6
Q

Unipolar leads

A

more easily effected since the + and - ends are further from each other

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7
Q

Bipolar leads

A

most modern PM

better shielding of lead ends reduces risk of interference

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8
Q

PM code

1st letter

A

denotes cardiac chamber being paced
Atrium
Ventricle
Dual

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9
Q

PM code

2nd letter

A
cardia chamber for electrical activity being sensed 
A
V
D
0 - none
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10
Q

PM code

3rd letter

A
Response to sensed signal
0-none
I - inhibition
T - triggering
D - dual inhibition and triggering
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11
Q

PM code

4th letter

A

Rate response features

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12
Q

PM code

5th letter

A

denotes chambers in which multisite pacing is delivered

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13
Q

Most common PM settings

A

AAI
VVI
DDD

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14
Q

Ansynchronous pacing

A
simplest form
Modes are:
AOO
VOO
DOO 
meaning aria are paced with no sensing and no response since there is no sensing)

It can compete with pt’s intrinsic rate and depletes the battery life faster

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15
Q

Single chamber pacing

A

can be atrial or ventricular

A-pacing requires the AV node and bundle of HIS to be intact (AAI).

Those with AV node disease can have a single chamber ventricular pm (VVI). senses the R wave, and if present, the PM discharge in inhibited

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16
Q

PM syndrome

A

can occur with VVI from loss of AV synchrony
symptoms are syncope, PNS, orthopnea, hypotension, pulmonary edema

changing settings to DDD can relieve this

17
Q

DDD

A

more common methods when the SA or AV node is diseased

physologica pacing since it retains AV synchrony

there is a lead in the RA and one in the RV

If the atria is sensed, then the atrial pacer is nin habited

If the ventricle is sensed, the nteh ventricles is inhibited

This mode allows the SA node ot increase rate with exercise

18
Q

DDI

A

there is atrial and ventricular sensing but only responds t]wit inhibition

only used when there are tachy dysrhythmias

19
Q

Rate adaptive PM

A

can be used in pts who don’t have appropriate HR responses during exercise

BB, CCB or sick sinus common causes

20
Q

ICDs

A

include a pacing and shocking function to manage brady or tachy arrhythmias

Senses the R-R interval
-short intervals interpreted as tachy

RV lead to sense ventricular depolarization -
if Vfib sensed, a second waveform analysis occurs
-10 to 15 sec delay
-pt may lose conciousness

21
Q

ICD modes

1st letter

A
chamber shocked
O-none
A
V
D
22
Q

ICD modes

2nd letter

A
Anti-tachy pacing chamber
O
A
V
D
23
Q

ICD modes

3rd letter

A

Tachycardia detection mechanism
E - electrogram
H - hemodynamic

24
Q

ICD modes

4th letter

A
Anti-bradycardia pacing chamber
O
A
V
D
25
Q

CRT

A

used for CHF pts caused from asynchrony and conduction blocks

Using 3 pacing leads in the RA, RV, coronary sinus, the device can be programmed to resynchronize the heart

indications are EF <35%, QRS prolongation, NYHA 3 or 4

26
Q

ICDS/surgery

A

Timely interrogation pre-operatively (ASA recommends 3 months prior, others say 12 months)
Re-programming preferred vs magnet
Pad placed so current heads away from device
bi-polar cautery
current 5 cm from pacer leads
Post-op interrogation
*anti-tachycardic functions and rate response functions are usually suspended just before surgery if any EMI is possible

27
Q

Pacer insertion

A

under conscious sedation or MAC
routine monitoring
pt supine with head turned away from operative side
anti-arrhythmic drugs available

28
Q

complications from pacer insertion

A

pnemo/hemo thorax
air embolism
*tension pneumo suspected if pt suddenly deteriorates in PEA
cannulation of subclavian artery

29
Q

Factors that can increase depolarization threshold of PM

A
hyperkalemia
acidosis
antidysrhythmic meds
hypoxia
hypoglycemia
local anesthetics
MI
hypothermia
30
Q

Factors that can decrease depolarization threshold of PM

A
hypokalemia
increased catecholamine levels
sympathomimetic drugs
anticholinergics
glucocorticoids
stress/anxiety
hyperthyroidism
hypermetabolic state
31
Q

Magnets and surgery

A

Magnet response is not standardized

not intended to treat PM emergencies or prevent EMI effects
incorporated to allow for assessment of battery life and pacing threshold

Placing a magnet over a generator may produce no change at all bc not al PM switch to asynchronous mode. Some may only go into asynchronous temporarily

Those that do - have HR btwn 85-100

Some will have loss of pacing or transition into continuous pacing

Need to know what happens when you put a magnet on the device

32
Q

Pts with CIED will have one of the following:

A

sustained or intermittent brady or tachy arrhythmia

heart failure

33
Q

Asynchronous mode is used why?

A

to prevent inappropriate oversensing

34
Q

Harmonic scalpels

A

US

used w/o concern for EMI

35
Q

Temporary PM

A

common after cardiac surgery
Transvenous pacing is the most reliable temporary PM means

PACs sometimes have a pacing function

Transcutaneous is quickest emergent pacing modality -

36
Q

transcutaneous pacing

A

V3 and posteriorly at inferior scapula. Captures the RV then activates the LV.

Maintained at 5-10 mA above capture threshold as tolerated