CHF Flashcards
epidemiology of CHF
effects 10% of men and 8% of women over 60yo
incidence on the rise, especially diagnosis 3mil visits/yr
5yr mortality after diagnosis is 50%
etiology of CHF
- CAD & HTN are most common
- cardiomyopathies - alcoholic, viral myocarditis, idiopathic, infiltrative, post partum
- valvular dz
- cardiotoxins - chemo, anabolic steroids
- arrhythmias, diabetes, and congenital heart disorders
- dz promotion - renal dz, NSAIDS, CCBs
s/s systolic left sided failure
fatigue
crackles/rales - small clicking, bubbling or rattling sounds in lungs
DOE
orthopnea
PND
Cheyne-Stokes respiration
diminished pulse pressure, cyanosis, JVD, S3 gallop, tachycardia, murmurs
what is cheyne-stokes respirations
apnea = hyperventilation = apnea
caused by diminshed sensitivity of resp center to arterial PCO2
what is diastolic (right sided) failure
disruption in diastolic filling or contractility of RV secondary to MI, obstructive outflow process (anatomical vs inc p ulm capillary wedge pressures resulting from L sided failure)
what is left sided failure CHF
LV remodeling secondary to MI, HTN, or obstructive outflow process
s/s of diastolic (right sided) failure
- peripheral pitting edema - may be on back or buttocks if sedentary
- GI tract congestion - decreased appetite, hepatomegaly,ascites
- JVD, prolonged L parasternal impulse with RV hypertrophy
DDX for CHF - cardiovascular
acute coronary syndrome valvular/septal rupture aortic dissection arrhythmia critical aortic stenosis endocarditis/myocarditis HTN crisis pericardial tamponade
DDX for CHF - pulm
COPD PE multilobar pneumo ARDS asthma
ddx for chf - other
acute renal failure iatrogenic volume overload post transfusion overload sepsis med non compliance new meds
w/u for CHF
(results are related to progression of dz)
- labs: CBC, CMP, UA, A1C, lipid panel, TSH, BNP, troponin
- EKG: LV hypertrophy, MI/prior MI (Q waves), arrhythmia/tachycardia
- ECHO: assess for LV/RV hypertrophy, wall motion abnormalitites, ejection fraction (>50% normal), valvular abnormalities***essential for DX, Tx, management
- CXR (see other flash card for findings)
what is BNP
vasodilatory protein released by stretched myocardium in response to increased ventricular filling pressures. attempts to offset the effects of neurohormonal activation by promoting diuresis, vasodilation and suppressing the renin-angiotensin system.
***>500 is diagnostic
definition of CHF
cardiac pumping capacity that is inadequate to meet the body’s metabolic demands
CHF tx
- treat reversible causes - HTN, DM, CAD, arrhythmias, structural
- diuretics
pharacology tx for CHF - diuretics
- diuretics: most effective agents for chronic symptomatic relief –thiazides for mild CHF
- HCTZ
- chlorthalidone
- loop diuretics - used for mod-severe HF - K+ sparing - hypo K, dehydration, hypouricemia due to diuretics (used in combo with thiazide or loop