chaptera 29, 30, 34, 35, 36, 37, 38, 39, 58, 59, 60, 62, 65 Flashcards

to leatn these subjects for the final

1
Q

Measures to prevent relapse of pneumonia?

A

Health promotion:
There are many nursing interventions to help prevent the occurrence of pneumonia, as well as the morbidity associated with it. Teaching a client to practise good health habits, such as proper diet and hygiene, adequate rest, and regular exercise, can help the client maintain the natural resistance to infecting organisms. If possible, exposure to URIs should be avoided. If a URI occurs, it should be treated promptly with supportive measures (e.g., rest, fluids). If symptoms persist for more than 7 days, the person should obtain medical care. The individual at risk for pneumonia should be encouraged to obtain both influenza and pneumococcal vaccines.

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2
Q

Assessment of client with COPD? and cardiopulmonary complication?

A

Diagnosis of COPD is based on a history of exposure to risk factors and the presence of airflow limitation that is not fully reversible, with or without the presence of symptoms. Patients who have chronic cough and sputum production with a history of exposure to risk factors should be tested for airflow limitation, even if they do not have dyspnoea. For the diagnosis and assessment of COPD, spirometry is the gold standard as it is the most reproducible, standardized, and objective way of measuring airflow limitation. FEV1/FVC < 70% and a postbronchodilator FEV1 < 80% predicted confirms the presence of airflow limitation that is not fully reversible. Health care workers involved in the diagnosis and management of patients with COPD should have access to spirometry. Measurement of arterial blood gas tensions should be considered in all patients with FEV1 < 40% predicted or clinical signs suggestive of respiratory failure or right heart failure.

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3
Q

Assessment for pleural effusion?

A

Pleural effusion: is a collection of fluid in the pleural space. It is not a disease but rather a sign of a serious disease. Pleural effusion is frequently classified as transudative or exudative accord- ing to whether the protein content of the effusion is low or high, respectively. The type of pleural effusion can be determined from a sam- ple of pleural fluid obtained via thoracentesis (a procedure to remove fluid from the pleural space). Exudates have a high pro- tein content and the fluid is generally dark yellow or amber. Transudates have a low protien contecnt or contain no protien, and the fluid is clear or pale yellow. The fluid can also be analyzed for RBC or WBC malignent cells, bacteria and glucose.

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4
Q

Assessment for Pleurisy?

A

Pleurisy (pleuritis) is an inflammation of the pleura. The most common causes are pneumonia, TB, chest trauma, pulmonary infarctions, and neoplasms. The inflammation usually subsides with adequate treatment of the primary disease. Pleurisy can be classified as fibrinous (dry) with fibrinous deposits on the pleural surface or serofibrinous (wet) with increased production of pleural fluid that may result in pleural effusion. The pain of pleurisy is typically abrupt and sharp in onset and is aggravated by inspiration. The client’s breathing is shallow and rapid to avoid unnecessary movement of the pleura and chest wall. A pleural friction rub may occur, which is the sound over areas where inflamed visceral pleura and parietal pleura rub over one another during inspiration. This sound is usually loudest at peak inspiration but can be heard during exhalation as well. Treatment of pleurisy is aimed at treating the underlying dis- ease and providing pain relief. Taking analgesics and lying on or splinting the affected side may provide some relief. The client should be taught to splint the rib cage when coughing. Intercostal nerve blocks may be done if the pain is severe.

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5
Q

Assessment of client with and cardiopulmonary complication?

A

complications of CODP: Cor Pulmonale.
cor pulmonale is hypertrophy of the right side of the heart, with or without heart failure, resulting from pulmonary hypertension. In COPD, pulmonary hypertension is caused primarily by constriction of the pulmonary vessels in response to alveolar hypoxia, with acidosis further potentiating the vasoconstriction. Chronic alveolar hypoxia causes pulmonary arteriolar muscle hypertrophy. Chronic hypoxia also stimulates erythropoiesis, which causes polycythemia and increases the viscosity of the blood. Cor pulmonale is a late mani- festation of COPD with a poor prognosis; approximately 40% of clients with severe COPD have cor pulmonale. when pulmonary heypetension develops, the pressures on the right side of the heat must increse to push blood into the lungs. Eventually, right sides heat faliure develops. Heart sound changed include accentuation of the pulmonic component of the second heart sound, right sided ventricular diastolic S3 gallop and early systolic ejection clicking along the left sternal border. Overt manifestations of right sides heart faliure may develop, which include jugular venous distension hepatomegaly with right upper quadrant tenderness, ascites, epigastric distress, peripheral edema, and weight gain. Diuretics are generally used, but serum creatinine and blood urea nitrogen (BUN) must be monitored since diuretics can cause volume depletion. Electrolytes must be monitored, since hypokalemia can predispose to dysrhythmias.

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6
Q

TB skin test?

A

The body’s immune response can be emonstrated by hypersensitivity to a tuberculin skin test. A positive reaction occurs 2 to 12 weeks after the initial infection, cor- responding to the time needed to mount an immune response. A positive reaction indicated the persence of a turbculosis infection, but it does not show whether the infection is latent or active. Two step testing is recommended for initial screening of health care workers who will be gettig regularly retested in the future, and for those who have decreased reasopnse to allergens.

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7
Q

DVT (Deep Vein thrombosis) invervetions?

A

Nursing care for the client with venous thrombosis is directed toward the prevention of embolus formation and the reduction of inflammation. While the client is receiving anticoagulation therapy, the nurse should closely observe for any indication of bleeding, including epistaxis and bleeding gingivae. Urine should be assessed for gross or microscopic hematuria. A smoky appear- ance to the urine is sometimes noted if blood is present. A speci- men should be checked daily for hematuria. Particular attention should be paid to the protection of skin areas that may be trauma- tized. Surgical incisions should be closely observed for evidence of bleeding. Stools should be tested to determine the presence of occult blood from the gastrointestinal tract. Mental status changes, especially in the older client, should be assessed as a possible indi- cation of cerebral bleeding. IM injections should not be given.

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8
Q

Assessment of medication effectiveness for client with asthma?

A

less exaserbations of asthma becuase of continuess drug therapy

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9
Q

Measures to prevent relapse of pneumonia

A

-be sure to Finnish all prescribed antibiotics
-teach client to cough productively
-practise good health habits, such as proper
diet and hygiene, adequate rest, and regular exercise
-exposure to URIs should be avoided
-should be encouraged to
obtain both influenza and pneumococcal vaccines

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10
Q

Assessment of client with COPD and cor pulmonale complication

A

listening to heart sounds: Heart sound changes include accentuation of the pulmonic component of the second heart sound, right-sided ventricular diastolic S3 gallop, and early systolic ejection click along the left sternal border. which include distended neck veins (jugular venous distension), hepatomegaly with right upper quadrant tenderness, ascites, epigastric distress, peripheral edema, and weight gain.

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11
Q

Assessment for pleurisy and pleural effusion

A

Common clinical manifestations of pleural effusion are progressive dyspnea and decreased movement of the chest wall on the
affected side. There may be pleuritic pain from the underlying disease. Physical examination of the chest will indicate dullness to percussion and absent or decreased breath sounds over the affected area. The chest radiograph will indicate an abnormality if the effusion is greater than 250 mL. Manifestations of empyema
include the manifestations of pleural effusion, as well as fever, night sweats, cough, and weight loss. A thoracentesis reveals an
exudate containing thick, purulent material.

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12
Q

tb skin test

A
  • The PPD test is used to determine if someone has developed an immune response to the bacterium that causes tuberculosis
  • The standard recommended tuberculin test is the Mantoux test, which is administered by injecting a 0.1 mL volume containing 5 TU (tuberculin units) PPD into the top layers of skin of the forearm.
  • Skin tests should be read 48-72 hours after the injection.
  • The basis of the reading of the skin test is the presence or absence and the amount of induration (localized swelling).
  • a negative test does not always mean that a person is free of tuberculosis. People who have been infected with TB may not have a positive skin test (known as a false negative result) if their immune function is compromised by chronic medical conditions, cancer chemotherapy, or AIDS.
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13
Q

DVT

A
  • A DVT is a blood clot that forms in the deep veins of the legs most commonly, but can also occur in the veins of the upper extremities. That’s because the blood clot, which usually forms in a calf or thigh deep veins, can partially or completely block blood flow back to the heart and cause damage to the one-way valves in the veins.
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14
Q

DVT interventions

A

Encourage REST, elevate the extremities above the level of the heart (DO NOT PUT THINGS UNDER KNEES)- Moist compress to prevent embolus from dislodging- (DO NOT MASSAGE EXTREMITY) - Use SCD’s and antiembolism stalking- promote blood flow - Administer Meds (comp-bleeding) - LOW MOLECULAR WT HEPARIN, WARFARIN, ANALGESICS -Thrombolytic therepy- must be initiated within 5 days

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15
Q

Assessment of medication effectiveness for client with asthma

A

Asthma severity levels can change for better or worse over the course of the client’s life. This is particularly true for children with asthma, since asthma severity often decreases with age. Good asthma control correlates with minimal to no symptoms, the abil¬ ity to sleep through the night, and participation in sports, exercise, and strenuous activity. Once asthma control has been maintained for several months, an attempt should be made to reduce medica¬ tion while maintaining acceptable asthma control.
Signs of severe or poorly controlled asthma include a history of a previous near-fatal asthma episode (loss of consciousness, need for intubation); recent hospitalization or emergency department visit for asthma; nighttime symptoms; limitations in daily activities; and the need for inhaled p2-agonists several times each day or night.

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16
Q

Pursed breathing

A

Pursed lip breathing is one of the simplest ways to control shortness of breath. It provides a quick and easy way to slow your pace of breathing, making each breath more effective.

  • Improves ventilation
  • Releases trapped air in the lungs
  • Keeps the airways open longer and decreases the work of breathing
  • Prolongs exhalation to slow the breathing rate
  • Improves breathing patterns by moving old air out of the lungs and allowing for new air to enter the lungs
  • Relieves shortness of breath
  • Causes general relaxation
17
Q

CF interventions

A

(1) promote clearance of secretions, (2) control infection in the lungs, and (3) provide adequate nutrition. Management of pulmonary problems in CF is directed at relieving airway obstruction and controlling infection. Drainage of thick bronchial mucus is assisted by aerosol and nebulized treatments of medications used to liquefy mucus and to facilitate coughing.
1. Give medications as ordered. Administer pancreatic enzymes with meals and snacks.
2.Perform chest physiotherapy, including postural drainage and chest percussion several times a day as ordered.
Administer oxygen therapy as ordered.
3.Provide a well-balanced, high calorie, high protein diet for the patient.
4.Make sure the patient receives plenty of fluids to prevent dehydration.
5.Provide exercise and activity periods for the patient to promote health.
6.Provide the young child with play periods, and enlist the help of physical therapy department.
7.Provide emotional support to the parents of children with cystic fibrosis.
8.Be flexible with care and visiting hours during hospitalization to allow the child to continue school.
9.Include the family in all phases of the child’s care.

18
Q

HTN risk factors(hypertension)

A
  • Age
  • Alcohol & cigarette smoking
  • Diabetes mellitus
  • Elevated serum lipids
  • Excess dietary sodium
  • Gender & Family History
  • Obesity
  • Ethnicity
  • Sedentary life style
  • Socioeconomic status
  • Stress
19
Q

Health teaching regarding HTN

A

-Maintain a healthy weight.
-Be physically active.
-Follow a healthy eating plan, which includes foods
lower in salt and sodium.
-If you drink alcoholic beverages, do so in moderation. 1-3
-If you have high blood pressure and are prescribed
medication, take it as directed.

20
Q

Stages of HTN

A

Prehypertension

This hypertension stage is defined as a systolic blood pressure between 120 and 139 or a diastolic pressure between 80 and 89. It is meant to identify adults who are at high risk for developing hypertension. If you have prehypertension, your doctor will advise you to begin lifestyle modifications to bring your blood pressure down to the normal range. Blood pressure medication isn’t usually recommended unless the patient has diabetes or kidney disease and recommended lifestyle changes are not working.

Lifestyle changes may include losing weight, eating a low-fat, low-sodium diet, exercising daily, limiting alcoholic intake, and not smoking.

Hypertension Stage 1

If your systolic blood pressure is between 140 and 159 or your diastolic pressure is between 90 and 99, you are considered to be in hypertension stage 1. Your doctor will recommend the same lifestyle changes mentioned above, but you will also probably need to take medication. The JNC 7 report recommends that the first medication to use is a thiazide-type diuretic. A diuretic is a medication that lowers blood pressure by helping your body get rid of extra fluid and sodium. Diuretics are usually very effective, have few side effects, and are inexpensive.

Hypertension Stage 2

If your systolic pressure is 160 or higher or your diastolic pressure is 100 or higher, you have hypertension stage 2. People at this stage usually must modify lifestyle habits and take a diuretic and another type of antihypertensive drug (maybe a third type if necessary). More than two-thirds of hypertensive patients require two or more different medications.

Other factors will determine your treatment. African Americans, who have a higher risk of developing hypertension-related complications, may require more aggressive treatment. If you have an underlying condition, such as heart disease, diabetes, or kidney disease, your doctor will factor that into the treatment choice.

Because none of the stages of hypertension has symptoms, everyone, even children, should get their blood pressure checked regularly.

21
Q

grades of hypertention

A

Optimal <90

22
Q

Etiology og hypertensive crisis

A

Hypertensive crisis occurs most commonly in clients with a history of hypertension who have failed to comply with their prescribed medications or who have been undermedicated. In this setting, rising BP is thought to trigger endothelial damage and the
release of vasoconstrictor substances. A vicious cycle of BP elevation ensues leading to life-threatening damage to target organs.
Hypertensive crisis related to cocaine or crack use is becoming a more frequent problem. Other drugs such as amphetamines,
phencyclidine (PCP), and lysergic acid diethylamide (LSD) may also precipitate hypertensive crisis that may be complicated by
drug-induced seizures, stroke, MI, or encephalopathy. Hypertensive crisis is classified by the degree of organ damage and the rapidity with which the BP must be lowered.

23
Q

Assessing adverse affects of HTN medications

A

24
Q

Nutrition health teaching for CAD

A

Eating a heart healthy diet is very important to prevent future complications of heart disease. Six strategies to reduce coronary artery disease include:
Eat more vegetables, fruits, whole grains, and legumes Choose fat calories wisely
Eat a variety, and just the right amount of protein foods Limit dietary cholesterol Use complex carbohydrates for energy, and limit the intake of simple carbohydrates Place less emphasis on sodium and increase your intake of potassium, magnesium and calcium

25
Q

Characteristic of AMI (acute myocardial infarction)

A

-Pain. Severe, immobilizing chest pain not relieved by rest, position
change, or nitrate administration is the hallmark of an MI. Persistent and unlike any other pain, it is usually described as heaviness, pressure, tightness, burning, constriction, or a crushing
sensation. Common locations are substernal, retrosternal, or epigastric areas. The pain may radiate to the neck, the jaw, and the arms or to the back
-On physical examination, the client’s skin may
be ashen, clammy, and cool to touch.
-BP and HR may be elevated initially.
Later, the BP may drop because of decreased cardiac output (CO). If severe enough, this may result in decreased renal perfusion and
urine output. Crackles may be noted in the lungs, persisting for several hours to several days, suggesting left ventricular dysfunction. Jugular venous distension, hepatic engorgement, and peripheral edema may indicate right ventricular dysfunction.
-Nausea and Vomiting. The client may be nauseated and vomit. Nausea and vomiting can result from reflex stimulation of the vomiting centre by the severe pain. These symptoms can also result from vasovagal reflexes initiated from the area of the infarcted myocardium -Fever. The temperature may increase within the first 24 hours up to 38°C and occasionally as high as 39°C. The temperature elevation may last for as long as 1 week. This increase in temperature is a systemic manifestation of the inflammatory process caused by myocardial cell death

26
Q

PCI (percutaneous coronary intervention)

A

An intervention to treat coronary artery disease in which a catheter equipped with an inflatable balloon tip is inserted into a narrowed
coronary artery and the balloon is inflated; common as elective procedure and also used in emergent situations

27
Q

Purposes and indications for PCI (percutaneous coronary intervention)

A

The goal is to open the affected artery within 90 minutes of arrival at the ED. The advantages of PCI are that (1) it provides an alternative to
surgical intervention; (2) it is performed with local anaesthesia; (3) the client is ambulatory 24 hours after the procedure; (4) the length of hospital stay is approximately 1 to 3 days compared with the 4- to 6-day stay necessary with CABG surgery, thus reducing hospital costs; and (5) there is rapid return to work (approximately 5 to 7 days after PCI) instead of a 2- to 8-week convalescence after CABG.

28
Q

complications of PCI

A

The most serious complication of PCI is dissection of the newly dilated coronary artery. If the damage is extensive, the coronary artery could rupture, causing cardiac tamponade, ischemia
and infarction, decreased CO, and possible death. There is also danger from infarction should the lesion be calcified and a portion of the plaque dislodge and occlude the vessel distal to the
catheter. Coronary spasm from the mechanical irritation of the catheter or the balloon can occur as well as chemical irritation from the contrast medium injection used to visualize the artery.

29
Q

Indications of an MI

A

Patients with typical myocardial Infarction may have prodromal symptoms of fatigue, chest discomfort, or malaise in the days preceding the event; alternatively, typical STEMI may occur suddenly, without warning.
Myocardial infarction occurs most often in the early morning hours, perhaps partly because of the increase in catecholamine-induced platelet aggregation and increased serum concentrations of plasminogen activator inhibitor-1 (PAI-1) that occur after awakening. In general, the onset is not directly associated with severe exertion. Instead, it is concomitant with exertion. The immediate risk of myocardial infarction increases 6-fold on average and by as much as 30-fold in sedentary people.

30
Q

How is ACS diagnosed

A

When myocardial ischemia is prolonged and not immediately reversible, acute coronary syndrome (ACS) develops; this syndrome encompasses the spectrum of unstable angina (UA),
non-ST-segment-elevation myocardial infarction (NSTEMI), and ST-segment-elevation myocardial infarction
-In the emergency setting, electrocardiography (ECG) is the most important diagnostic test for angina. ECG changes that may be seenduring anginal episodes include the following:
Transient ST-segment elevations, Dynamic T-wave changes: Inversions, normalizations, or hyperacute changes.
ST depressions: These may be junctional, downsloping, or horizontal Laboratory studies that may be helpful include the following:
Creatine kinase isoenzyme MB (CK-MB) levels, Cardiac troponin levels, Myoglobin levels, Complete blood count, Basic metabolic panel
Diagnostic imaging modalities that may be useful include the following:, Chest radiography
Echocardiography, Myocardial perfusion imaging
Cardiac angiography, Computed tomography, including CT coronary angiography and CT coronary artery calcium scoring

31
Q

Complications of MI

A

Complications of MI include arrhythmic, mechanical, and inflammatory (early pericarditis and post-MI syndrome) sequelae, as well as left ventricular mural thrombus (LVMT). In addition to these broad categories, right ventricular (RV) infarction and cardiogenic shock are other possible complications of acute MI

32
Q

Cardiac Markers

A

Certain proteins, called serum cardiac markers, are released into the blood in large quantities
from necrotic heart muscle after an MI. These markers, specifically serum cardiac enzymes and troponin, are important in the diagnosis
of MI. When cardiac cells die, their intracellular enzymes are released into circulation. The increase in serum cardiac markers that occurs after cellular death can indicate whether cardiac damage is present and the approximate extent of the damage. Creatine kinase (CK) and troponin are typically measured to diagnose an
MI. (Figure 35-12 indicates the peak level and duration of these markers in the presence of MI.)