chapter 9 Flashcards

1
Q

Characteristics of acute periodontal diseases

A

sudden onset, rapid course of progression, accompanied by pain and discomfort, may be unrelated to preexisting gingivitis or periodontitis, lesion may be localized or more widespread in mouth, may present with systemic involvement

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2
Q

Abscesses of the periodontium

A

Circumscribed (abscess is localizd to specific site) fluctuant collection of pus localizedé within gingival wall of periodontal pocket. Pus (whitish, yellow, milky exudate consisting of dead and dying neutrophils, bacteria, cellular debris and fluid leaked from blood vessels. Precise bacterial etioly of abscess = unclear, most lesions contain microflora that are predominantly gram negative and anaerobic.

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3
Q

Implications of untreated abscesses

A
  • Common dental emergency
  • Can initiate rapid periodontal destruction and lead to premature tooth loss
  • Possible link between abscesses of periodontium and systemic diseases
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4
Q

Characteristics of an abscess of the periodontium

A
  • Acute abscess: constant, localized pain
  • Chronic abscess: no pain or dull pain
  • Circumscribed
  • Possible increase in tooth mobility
  • Radiographic loss of alveolar bone not involving tooth apex
  • Tooth usually has vital pulp
  • Fever may occur (serious) not common
  • If delayed treatment, pus may drain through sinus tract
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5
Q

Causes of abscesses of the periodontium

A
  • Blockage of orifice of pocket
  • Accidentaly forcing foreign object into tissues
  • incomplete calculus removal in periodontal pocket
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6
Q

Comparaison of periodontal abscesses and pulpal abscesses

A
  • Vitalitity test results: PA(usually,vital pulp), PuA(usually, no vital pulp)
  • Radiographic appearance: PA(bone loss present and an angular deflect and/or furcation radiolycency), PuA(bone loss at tooth root apex)
  • Symptoms: PA(localized, constant pain), PuA(difficult to localize, intermittent pain
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7
Q

Classification of abscesses of the periodontium by course of the lesion

A

Acute abscess: rapid onset characterized by pain/discomfort, primarily caused by exacerbation of chronic inflammatory periodontal lesion

Chronic abscess: grows slowly and is not typically associated with pain, forms after spread of infection controlled by spontaneous drainage host response or therapy

Gingival abscess: primarily limited to gingival margin or interdental papilla without involvement of deeper structures of periodontium

Periodontal abscess: abscess of periodontium that affects deeper structures of periodontium as well as gingival tissues, usually occurs in site with preexisting periodontal disease invluding preexisting periodontal pockets, usually affects deeper structures of periodontium

Pericoronal abscess: involves tissues around crown of partially erupted tooth

Pericoronitis: soft tissue inflammation associated with abscess

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8
Q

Signs and symptoms of a pericoronal abscess

A
  • Pain at site
  • swelling of perculum
  • Possible trismus
  • Possible elevated body temperature
  • Possible lymphadenopathy
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9
Q

Management of patients with abscesses of the periodontium

A
  • Fundamental treatment steps: establishment of path of drainage for pus, thorough periodontal instrumentation of affected tooth surfaces in area of abscess, pain relief
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10
Q

Steps in treatment of a gingival or periodontal abscess

A
  • Administer local anaesthesia
  • Drain pus
  • Perform thorough periodontal instrumentation
  • Adjust occlusion, if needed
  • Prescribe antibiotics, if needed
  • Recommend warm saline rinses
  • Prescribe pain meds if needed
  • Establish follow-up appointments
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11
Q

Treatment of pericoronal abscess

A

Fundamental treatment steps: Establishment of path of drainage for pus, irrigation of undersurface of operculum, thorough periodontal instrumentation of tooth surfaces in area of abscess, relief of pain

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12
Q

Common steps in treatment of patient with pericoronal abscess

A
  • Admisister local anesthesia
  • drain pus
  • Perform thorough periodontal instrumentation
  • Irrigate under operculum
  • prescribe antibiotics, if need
  • Recommend warm saline rinses
  • Prescribe pain medications if needed
  • Evaluate for need for third molar extractions
  • Establish follow-up appointments
  • if pericoronal abscess reoccurs, reasses if surgical excision of operculum or extraction of offending tooth is warranted
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13
Q

Endodontic-Periodontal Lesions (EPL)

A
  • Formerly known as combined periodontal-endodontic lesion
  • Localized area of bacterial infection characterized by infection of pulp and periodontal tissues in same tooth
    -Can originate from either dental pulp or periodontal tissues
  • Periodontlally derived lesion: Infection enters tooth via accessory canals and/or apical foremen of root, initialtes inflammatory changes in pulp-root canal complex
  • Pulpally derived lesion: Infection escaped out of tooth of tooth, triggers secondary infection of periodontal tossues
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14
Q

Signs and symptoms of EPLs

A
  • Deep pocketing that extends close to apex
  • Negative or altered response to pulp sensitivity tests
  • Bone resorption in apical or fungal regions of tooth
  • Spontaneous pain
  • Pain upon palpation or percussion
  • Purulent suppuration
  • Tooth mobility
  • Presence of sinus tract
  • gingival color alterations
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15
Q

Necrotizing periodontal disease (NPD)

A

Distinct characteristics: interdental tissue necrosis, intense gingival pain, spontaneous gingival bleeding

Secondary clinical characteristics: Fetid breath, pseudomembrane formation, systemic involvement

  • Noncommunicable, destructive, inflammatory diseases
  • Limited to interdental and marginal gingiva
  • Historically called Vincent’s infection (no longer valid)
  • Distinctive characteristic is tissue necrosis of gingiva
  • Progression follows predictable course: interproximal gingival necrosis, causes punched out appearance, spreads to affect marginal gingiva
  • Most often found in mandibular anterior region
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16
Q

Etiology of necrotizing gingivitis

A
  • Late 19th cent, vincent identifies spirochètes and fusiforme deep in tissues
  • Mid 20th cen Listgarden confirmed spirochetal invasion deep with tissues of NG lesions
  • More recent testing revealed presence of diverse array of microorganisms deep within NG lesions
  • Still debate wether invasion of specific bacteria species occurs before or after onset of NG
  • Inflammatory response major contributing factor to development of NG
  • HIV - positive individuals at higher risk of NG (may be indications of HIV infection)
17
Q

Prevalence of NG

A
  • Can affect subjects of any age, most common between ages of 20 - 30
  • Lower prevalence in developed countries compared to developing countries
    -95% of cases in north america occur in caucasians
18
Q

Characteristics of NG

A
  • necrosis and ulcers in interdental papilla
  • Gingival bleeding
  • Pain
  • Pseudomembrane formation
  • Halitosis
  • Lymphadenopathy
  • Fever
19
Q

Treatment for NPD

A

Management should focus on:
- Reducing patient pain/discomfort
- arresting destructive progression of disease
- restoring form and function of involved periodontal tissues
- Preserving stability of periodontium following initial management

20
Q

Stage 1: management of the Acute stage NPD

A

First day of treatment
- Remove pseudomembrane and soft and mineralized deposits and provide self-care regimen

Second day of treatment
- Initiate subgingival periodontal instrumentation and provide further instruction in self care to control systemic predisposing factors

Third visit
- Complete subgingival instrumentation, evaluate patient for resolution of symptoms, further counsel patient on predisposing factors and home care

Sever cases many require use of systemic antibiotics

21
Q

Stages 2 & 3: management of acute stage of NPD

A

Stage 2: control preexisting conditions
- Comprehensive clinical assessment to identify and manage underlying periodontal disease

Stage 3: corrective surgical management
- May be required for some patient to reestablish natural contours of gingiva

22
Q

Stage 4: maintenance phase (management of acute stage of NPD)

A
  • Assess periodontal status
  • reinforce self-care
  • Control predisposing factors
    -Perform any nessecary periodontal instrumentation
    Factor in patient compliance levels
23
Q

Necrtotizing periodontitis (NP)

A
  • Same clinical features as necrotizing gingivitis but tissue necrosis spreads to underlying periodontal attachment apparatus
  • Can lead to bone loss and clinical attachment loss
24
Q

Typical treatment of Necrotizing periodontitis

A
  • Similar to necrotizing periodontitis treatment, but may be more complex due to extensive, irreversible tissue destruction
  • Referral to periodontist recommended
  • Close collaboration with patient’s medical practitioner also warranted
25
Q

Necrotizing stomatitis (NS)

A
  • Extension of NG or NP where necrosis progresses to deeper tissues beyond mucogingival line
  • Symtoms similar to those of NG and NP
  • Rare but most extensive and invasive form of NPD
  • Refer immediately to oral pathologist, oral maxillo-facial surgeon and physician
26
Q

Primary Herpetic Gingivo stomatitis

A
  • Caused by herded simplex virus (HSV)
  • unnoticeable ins one patients, but severe symptoms in others
  • Contagious
    -Several stagesL prodrome, macule, papule, vesicle, ulcer, scab, healed area with redness, complete healing
  • Vesicle and ulcer stages most important from dental management standpoint
  • HSV- 1 spread by direct contact (can also spread from one part of body to another)
  • Most common in children and young adults but can occur at any age
  • Recurrence can be triggered by many factors: stress, trauma, sunlight, fever
27
Q

Clinical signs of Primary herpetic gingivostomatitis

A
  • Oral pain with difficulty in eating, drinking
  • Swollen and/or bleeding gingival tissue
  • Vesicles and ulceration of gingival tissue, lips, tongue, and/or palate, with ulcerations surrounded by red halo
  • Elevated body temperature
    -Malaise
  • Swollen lymph nodes
28
Q
A