chapter 6 fluid and electrolytes Flashcards

1
Q

Equal solute concentrations, causes no fluid shifts

A

Isotonic fluid

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2
Q

lowe solute concentration

fluid shifts out

A

hypotonic

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3
Q

higher solute concentration

fluid shifts in

A

hypertonic

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4
Q

triggered by decrease blood volume and osmolarity

A

Thirst mechanism

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5
Q

Promotes reabsorption of water in the kidneys

A

Antidiuretic hormone

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6
Q

Increases reabsorption of sodium and water in the kidneys

A

Aldosterone

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7
Q

Stimulates renal vasodilation and suppresses aldosterone, increasing urinary output

A

Atrial natriuretic peptid

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8
Q

excess fluid in the interstitial space

A

edema

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9
Q

excess fluid in the intravascular space

A

hypervolemia or fluid volume excess

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10
Q

excess fluid in the intracellular space

A

water intoxication

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11
Q

manifestations as peripherial edema, periorbital edema, anasarca, cerebral edema, dyspnea, bounding pulse, tachycardia, JVD, HTN, polyuria, rapid weight gain, crackles, and bulging fontanelles

A

Fluid Excess

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12
Q

dehydration, hypovolemia or fluid volume deficit, can occur independently without electrolyte defects.
decrease in fluid level leads to increase in level of blood solutes
cell shrinkage
hypotension

A

Fluid deficit

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13
Q

positively charged electrolytes

A

cations

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14
Q

negatively charged electrolytes

A

anions

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15
Q

normal 135-145
most significant cation and prevalent electrolyte of extracellular fluid
controls serum osmolality and water balance
facilitates muscles and nerve impulses
main source is dietary intake
excreted thru the kidneys and GI tract

A

Sodium

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16
Q

excessive sodium
Hypertonic IV saline (3% saline)
causes fluid shifts

A

HYPERnatremia

17
Q

serum osmolarity decreases
anorexia, GI upset, poor skin turgor, dry mucous membranes, BP changes, pulse changes, edema, headache, lethargy, confusion, diminished deep tendon reflexes, muscle weakness, seizures, and coma

A

HYPOnatremia

18
Q
normal 98-108
mineral electrolyte
Major extracellular anion
found in gastric secretions, pancreatic juices, bile, and csf
main source is dietary intake
excreted thru the kidneys
A

chloride

19
Q

normal 3.5-5
primary intracellular cation
electrical conduction, acid-base balance, and metabolism
can’t fluctuate much without causing serious issues

A

Potassium

20
Q

normal 4-5
found in the bone and teeth
role in blood clotting, hormone secretion, receptor functions, nerve transmission, and muscular contraction
inverse relationship with phosphorus
synergistic relationship with magnesium
absorbed thru the GI tract (small intestines)

A

Calcium

21
Q

normal 2.5-4.5
found in bones, small amounts in bloodstream
role in bone and tooth mineralizaton, cellular metabolism, acid-base balance, and cell membrane formation
excreted thru the kidneys

A

phosphorous

22
Q
normal 1.8-2.5
intracellular cation
stored in bone and muscle
cardiac rhythm
excreted thru kidneys
A

magnesium

23
Q

reflects hydrogen concentrations

A

pH

24
Q

chemicals that combine with an acid or a base to change pH

A

Buffers

25
Q

most significant in the extracellualar fluid
forms from carbon dioxide reacting with water
carbonic anhydrase causes carbonic acid to separate into hydrogen and bicarbonate
carbonic anhydrase in the lungs allow for CO2 excretion and in the kidneys allows for hydrogen excretion

A

Bicarbonate-carbonic acid system

26
Q

similar to the bicarbonate-carbonic acid system
high concentrations in the intracellular fluid
act as weak acids, and some act as weak bases
primarily works in the kidneys by accepting or donating hydrogen

A

Phosphate system

27
Q

primarily occurs in the capillaries
acidity and hypoxia cause hemoglobin to release the oxygen
hemoglobin then becomes a weaker acid, taking up extra hydrogen
binding with oxygen makes hemoglobin more prone to release hydrogen
hydrogen reacts with bicarb to form carbonic acid, which is converted to carbon dioxide and released into the alveoli

A

Hemoglobin system

28
Q

most abundant buffering system
proteins can act as an acid or a base by binding to or releasing hydrogen
occurs in the intracellular and extracellular spaces
hydrogen and CO2 diffuse across the cell membrane to bind with protein inside the cell
albumin and plasma are the primary buffers in the intravascular space

A

protein system

29
Q

manages pH by altering CO2 excretion
speeding will excrete more CO2 decreasing acidity and vice versa
uses chemoreceptors
responds quickly, but is short lived

A

respiratory regulation

30
Q

alters the excretion or retention of hydrogen or bicarb
more effective because it permanently removes hydrogen
responds slowest, but lasts the longest

A

renal regulation

31
Q

results from a deficiency of bicarb or an excess of hydrogen

A

Metabolic Acidosis

32
Q

bicarb and chloride results are added together and subtracted from the sodium
normal 6-9

A

anion gap

33
Q

results from excess bicarb or deficient acid or both

A

metabolic alkalosis

34
Q

results from CO2 retention, which increases carbonic acid

A

respiratory acidosis

35
Q

results from excess exhalation of CO2, which leads to carbonic acid deficits

A

Respiratory Alkalosis

36
Q

respiratory and metabolic disorders resulting in an acidotic or alkalotic state
both the respiratory and renal systems demonstrate an imbalance of acid or base

A

mixed disorders