Chapter 58: Stroke management Flashcards
Non-modifiable risk factors
Age, gender, ethnicity/race, family history
Modifiable risk factors
HTN (#1 CAUSE!!!), heart disease, smoking, diabetes, alcohol, obesity, sleep apnea, lack of exercise, poor diet, drug abuse. Most c/b atherosclerosis–> thrombus and embolus formation
Transient episode of neurologic dysfunction c/b focal brain, spinal cord, or retinal ischemia, but without acute infarction of the brain. Sx typically last less than 1 hour. Teach the pt to seek tx for any stroke sxs, since there is no way to predict if it will resolve or if it is in fact the development of a stroke. May be d/t micro emboli that temporarily block blood flow. Warning sign of progressive cerebrovascular disease.
Transient ischemic attack.
Results from inadequate blood flow to the brain from partial or complete occlusion of an artery. Nearly 80% of strokes.
Ischemic stroke
Occurs from injury to a blood vessel wall and formation of a blood clot. The lumen of the blood vessel becomes narrowed and, if it becomes occluded, infarction occurs. Thrombosis develops readily where atherosclerotic plaques have already narrowed blood vessels.Account for about 60% of stroked. 2/3 are associated with HTN, DM, or both. Usually have had some TIAs.
S/S: no decrease in LOC, sx progress for 72 hours
Thrombotic stroke
Occurs when an embolus lodges in and occludes a cerebral artery, resulting in infarction and edema of the area supplied by the involved vessel. Second most common cause of a stroke. Severe clinical sx that occur suddenly. Warning signs less common than with thrombotic. Often occurs rapidly, giving little time to accommodate by developing collateral circulation. Pt usually remains conscious although they may have a headache. Prognosis r/t the amt of brain tissue deprived of its blood supply. Usually have a cardiac hx. Recurrence is common.
S/S: sudden onset of sx, usually remain conscious
Embolic stroke
Results from bleeding into the brain tissue itself (intracerebral or intraparenchymal) or into the subarachnoid space or ventricles.
Hemorrhagic stroke
Bleeding within the brain c/b a rupture of a vessel. Prognosis is poor. HTN is the most common cause. Sudden onset of sx with progression over minutes to hours because of ongoing bleeding. Initially, pts experience a severe headache with N/V. Weakness of one side, slurred speech, deviation of the eyes. Progression- hemiplegia, fixed and dilated pupils, abnormal body posturing, coma.
S/S: Neurological deficits, headache, nausea, vomiting, decreased LOC, and HTN.
Intracerebral hemorrhage
Intracranial bleeding into the cerebrospinal fluid-filled space between the arachnoid and pia mater membranes on the surface of the brain. C/b rupture of a cerebral aneurysm. Other causes can be trauma and illicit drug (cocaine) abuse. Pt may have warning sx if the ballooning artery applies pressure to brain tissue, or minor warning sx may result from leaking of an aneurysm before major rupture. Viewed as the “silent killer,” since individuals do not have warning signs of an aneurysm until rupture has occurred. Loss of consciousness may or may not occur. Pt’s LOC may range from alert to comatose. Neurological deficit, nausea, vomiting, seizures, and stiff neck.
Subarachnoid hemorrhage
FAST for strokes
Facial droop, uneven smile
Arm numbness, arm weakness
Slurred speech, difficulty speaking or understanding
Time- call 911 and get to the hospital immediately
Sx: slow, cautious; depressed; anxious; slow speech; right paralysis; impaired right/left discrimination; aware of deficits; impaired comprehension r/t language, math.
Left sided stroke
Sx: impulsive; denies problems; talks fast; safety risks; left paralysis; left side neglect.
Short attention span; impaired judgement; impaired time concepts
Right sided stroke
Motor function and stroke
Motor deficits are the most obvious effects of a stroke. Loss of skilled voluntary movement (akinesia), impairment of integration of movements, alterations in muscle tone, and alterations in reflexes. The initial hyporeflexia (depressed reflexes) progresses to hyperreflexia for most. A lesion on one side of the brain affects motor function on the opposite side of the body (contralateral).. The arms and legs of the affected side may be weakened or paralyzed to different degrees depending on which part of and to what extent the cerebral circulation was compromised. An initial period of flaccidity may last from days to several weeks and is r/t nerve damage. Spasticity of muscles, which follows the flaccid stage, is r/t interruption of upper motor neuron influence.
Communication and stroke
Left hemisphere is dominant for language skills in right-handed persons and in most left-handed persons. Pt may experience aphasia (receptive- loss of comprehension; expressive- inability to produce language; global- total inability to communicate). Aphasia often occurs when a stroke damages the dominant hemisphere of the brain.
Dysphagia refers to impaired ability to communicate. Patterns of aphasia may differ, since the stoke affects different portions of the brain. Nonfluent aphasia- minimal speech activity with slow speech that requires obvious effort. Fluent aphasia- speech is present but contains little meaningful communication.
Dysarthia- a disturbance in the muscular control of speech. Does not affect the meaning of communication or the comprehension of language, but it does affect the mechanics of speech.
Affect and stroke
May have difficulty controlling their emotions. emotional responses may be exaggerated of unpredictable. Depression and feelings associated with changes in body image and loss of function can make this worse. Pts may also be frustrated by mobility and communication problems.
Intellectual function and stroke
Memory and judgement may be impaired as a result of stroke. These impairments can occur with strokes affecting either side of the brain. A left-brain stroke is more likely to result in memory problems r/t language. Pts with left-brain stroke often are cautious in making judgements. PT with a right-brain stroke tends to be impulsive and move quickly.
Spatial-perceptual alterations
Pts with right-sided stroke are more likely to have problems with spatial-perceptual orientation. Can occur in left too. Damage to parietal lobe-incorrect perception of self and illness (may deny illnesses or their own body parts). Pt neglects all input from the affected side (erroneous perception of self in space. May be worsened by homonymous hemianopsia- blindness occurs in the same half of the visual fields of both eyes. Difficulty with spatial orientation, such as judging distances). Agnosia- the inability to recognize an object by sight, touch, or hearing. Apraxia- inability to carry out learned sequential movements on command.
Pts may or may not be aware of their spatial-perceptual alterations