Chapter 4- Motor Learning Flashcards

1
Q

Motor Learning

A

the process of the acquisition and/or modification of movement

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2
Q

Learning and Memory

A

acquisition of knowledge that leads to the retention and storage of that knowledge or ability
- learning can occur in all parts of the brain

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3
Q

Habituation

A

becoming less sensitized to a stimulus; getting used to it to the point that its effects are no longer novel

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4
Q

Short term and Long term memory

A

may not be separate categories, but may be part of a single graded memory function

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5
Q

Associative Learning

A

a person learns to predict relationships, either relationships of one stimulus to another(classical conditioning), or the relationship of one’s behavior to a consequence (operant conditioning)

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6
Q

Classical Conditioning

A

an initially weak stimulus(CS) becomes highly effective in producing a response when it becomes associate with another stronger stimulus(US)
- extension of sensitization

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7
Q

Operant Conditioning

A

we learn to predict the outcome of specific behaviors

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8
Q

Procedural learning

A

includes the learning and execution of motor and non declarative cognitive skills, especially those involving sequences. Occurs only when mvmt is performed by learners themselves through trial an error practice

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9
Q

Declarative learning

A

explicit knowledge that requires conscious process such as awareness and attention, and results in knowledge that can be expressed consciously

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10
Q

Hippocampus

A

subcortical structure that is critical for declarative learning and memory formation. Also deals with cognitive map of spatial area formation. “Memory of visiting locations”

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11
Q

Long term potentiation

A

weak input will be enhanced if it is activated in association wight the strong one

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12
Q

Motor learning

A

The efficiency of the thalamic input to the motor cortex is facilitated with repeated practice

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13
Q

Acquisition of skills

A

Performance gains during early learning strongly rely on prefrontal caudate interactions, while later learning improvements in performance involve increased activity in a subcortical circuit involving the thalamus, cerebellar nuclei, and basal ganglia as the task becomes more automatic

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14
Q

Plasticity-modulation of neural connectivity

A

Occurs at many levels:

  • brain level (glial and vascular support)
  • network level (changes in patterns of neural activation and cortical remapping)
  • intercellular level (changes between neurons at the synaptic level, including synaptic sprouting)
  • intracellular level (mitochondrial and ribosomal function)
  • biochemical level (protein conformation, enzyme mobilization)
  • genetic level (transcription, translation, and post translation modifications)
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15
Q

Restorative mechanisms

A

direct mechanisms involve the resolute of temporary changes and recovery of the injured neural tissue itself. In addition, nearby neural tissue takes over identical neural functions to the original damaged tissue

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16
Q

Compensatory mechanisms

A

reorganization that includes both function-enabling and function-diabaling plasticity. “Use it or lose it”

17
Q

Diaschisis

A

transient CNS phenomenon where nerve communication is disrupted due to injury

18
Q

Denervation Supersensitivity

A

As seen in Parkinson’s-when postsynaptic membrane of a neuron becomes hyperactive to a released transmitter substance

19
Q

Regenerative and Collateral synaptogensis

A

Recovery of function after brain damage

20
Q

Remapping following peripheral lesions

A
  • Neighboring maps expand their receptive fields to cover much of a denervated region
  • Reactivation of the cortex is due to increased responsiveness of weak inputs from neighboring areas; if denervation exceeds a certain distance, silent areas remain
21
Q

Cerebellar contributions to recovery from cortical injury

A
  • New cerebellar cortical synapses are associated with complex skill learning but not with gross motor activation in the absence of learning
  • New skill tasks must be taught to keep challenging the cerebellum
22
Q

Contributions of ipsilaterl motor pathways

A

Uncrossed pathways play an important role in recovery of function

The contralesional primary motor cortex (motor cortex in the side opposite to the affected cortex) may play a role

23
Q

Clinical Implications

A
  • Use it or lose it
  • Use it and improve it
  • Specificity
  • Repetition matters
  • Intensity matters
  • Time matters
  • Salience matters
  • Age matters
  • Transference
  • Interference