Chapter 33 - Burns and Frostbite Flashcards
First degree burns
involve epidermis only
no blistering
no \tissue death
second degree burns
penetrate dermal layer
most painful
regenerative dermal compnents are in the hair follicles and sweat glands - no skin grafting required
third degree burns
leave no tissue capable of spontaneous regeneration
all dermal layers involved
nerve endings gone - not painful
fourth degree burns
require flaps
bone and tendon involvement
Protein rich edema fluid after burns promotes what?
scarring, contracture, stiffness
HO in burns
most commonly about the elbow between the medial epicondyle and the olecranon
1-3% of burn patients
escharotomy incisions should be placed where?
midaxial along the elbow and digits
fasciotomy is more commonly required in what type of burns?
electrical
burned hands tend to fall quickly into what posture?
intrinsic minus - extended MCP, Flexed PIP, DIP
how to prevent intrinsuc minus contracture?
splint in intrinsic plus - mcp flexion pip, dip extension
Web space contractures - cause and how to treat
fibrosis of the CMC capsule, adductor pollicis, first dorsal interosseus musculature
z plasty, skin grafting, intermetacarpal pinning
how to treat intrinsic minus clawing deformity
release of the contracted MCP capsule, palmar IP capsule, subcu tissue and skin
if extensor mechanism unable to be mobilized, PIP arthrodesis
how to treat elbow contractures
simple contractures - z-plasty, more complex - scar excision plus skin grafting or flap coverage
electrical burns cause what mechanism of damage?
electrical energy yis converted to heat in deeper tissues - results in vessel thrombosis and coagulation necrosis
cardiac complications in electrical burns
with AC currents (most common) - ventricular arrhythmias
other physiologic complications of electrical burns
myoglobinuria - keep UOP at 2ml/kg/hr
Acid burns pathophys
cause cellular dehydration, cell membrane destruction, liquefaction necrosis
alkali burn pathology
less common than acid burns
penetrate deeper than acid burns via cellular dehydration
cause saponification of adipose tissues
specific neutralization agents for chemical burns
hydrofluoric acid -> fluoride binding agents (calcium gluconate)
phenol -> not water soluble so requireds polyethylene glycol or glycerol
white phosphorous -> copper sulfate in solution
what temperatures cause frostbite
sensory nerve dysfunction starts at 10C
ice crystals form -6- -15C - cellular injury begins here with intracellular dehydration
local inflammation, coagulation, resultant microvascular thrombosis and tissue necrosis after thawing
management of frostbite
warm IV fluids
rapid rewarming with 40-42C water bath
TPA administration within 24 hours decreases rate of amputation
pediatric effects of frostbite
young children - premature physeal closure 2/2 chondrocyte injury
older children - (>10yo) short digits, excess skin, joint laxity, degen joint changes
late complications of frostbite in adults
cold intolerance, hyperhidrosis, trophic changes, raynaud phenomenon