Chapter 33 - Burns and Frostbite Flashcards

1
Q

First degree burns

A

involve epidermis only

no blistering

no \tissue death

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2
Q

second degree burns

A

penetrate dermal layer

most painful

regenerative dermal compnents are in the hair follicles and sweat glands - no skin grafting required

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3
Q

third degree burns

A

leave no tissue capable of spontaneous regeneration

all dermal layers involved

nerve endings gone - not painful

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4
Q

fourth degree burns

A

require flaps

bone and tendon involvement

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5
Q

Protein rich edema fluid after burns promotes what?

A

scarring, contracture, stiffness

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6
Q

HO in burns

A

most commonly about the elbow between the medial epicondyle and the olecranon
1-3% of burn patients

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7
Q

escharotomy incisions should be placed where?

A

midaxial along the elbow and digits

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8
Q

fasciotomy is more commonly required in what type of burns?

A

electrical

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9
Q

burned hands tend to fall quickly into what posture?

A

intrinsic minus - extended MCP, Flexed PIP, DIP

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10
Q

how to prevent intrinsuc minus contracture?

A

splint in intrinsic plus - mcp flexion pip, dip extension

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11
Q

Web space contractures - cause and how to treat

A

fibrosis of the CMC capsule, adductor pollicis, first dorsal interosseus musculature

z plasty, skin grafting, intermetacarpal pinning

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12
Q

how to treat intrinsic minus clawing deformity

A

release of the contracted MCP capsule, palmar IP capsule, subcu tissue and skin

if extensor mechanism unable to be mobilized, PIP arthrodesis

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13
Q

how to treat elbow contractures

A

simple contractures - z-plasty, more complex - scar excision plus skin grafting or flap coverage

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14
Q

electrical burns cause what mechanism of damage?

A

electrical energy yis converted to heat in deeper tissues - results in vessel thrombosis and coagulation necrosis

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15
Q

cardiac complications in electrical burns

A

with AC currents (most common) - ventricular arrhythmias

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16
Q

other physiologic complications of electrical burns

A

myoglobinuria - keep UOP at 2ml/kg/hr

17
Q

Acid burns pathophys

A

cause cellular dehydration, cell membrane destruction, liquefaction necrosis

18
Q

alkali burn pathology

A

less common than acid burns

penetrate deeper than acid burns via cellular dehydration

cause saponification of adipose tissues

19
Q

specific neutralization agents for chemical burns

A

hydrofluoric acid -> fluoride binding agents (calcium gluconate)

phenol -> not water soluble so requireds polyethylene glycol or glycerol

white phosphorous -> copper sulfate in solution

20
Q

what temperatures cause frostbite

A

sensory nerve dysfunction starts at 10C

ice crystals form -6- -15C - cellular injury begins here with intracellular dehydration

local inflammation, coagulation, resultant microvascular thrombosis and tissue necrosis after thawing

21
Q

management of frostbite

A

warm IV fluids

rapid rewarming with 40-42C water bath

TPA administration within 24 hours decreases rate of amputation

22
Q

pediatric effects of frostbite

A

young children - premature physeal closure 2/2 chondrocyte injury

older children - (>10yo) short digits, excess skin, joint laxity, degen joint changes

23
Q

late complications of frostbite in adults

A

cold intolerance, hyperhidrosis, trophic changes, raynaud phenomenon