Chapter 3 Flashcards

1
Q

Why are normal, everyday cardiac rhythms called normal sinus rhythm?

A

Because each beat originates with depol of SA

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2
Q

What rate does heart normally beat?

A

60-100 bpm

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3
Q

Define arrythmia.

A

Any disturbance in rate, regularity, site of origin, conduction of electrical impulse

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4
Q

What are several characteristic sx of arrhythmias?

A
  1. Palpitations (awareness of one’s own heartbeat - accel/decel/regular/irregular)
  2. Syncope/light-headedness (due to decreased CO)
  3. Angina (chest pain, due to increased O2 demands of myocardium –> can precipitate CHF)
  4. Sudden death
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5
Q

What arrhythmogenic factors should be considered for pts w arrythmias?

A
  • HIS DEBS
  • Hypoxia
    • Myocardium deprived of O2 = irritable
    • Pulm disorders = major precipitants of cardiac arrythmias
  • Ischemia + irritability
    • Myocardial infarctions + angina w or w/o cell death = major precipitants
    • Myocarditis (inflamm of heart muscle, often caused by routine viral infections) can also induce arrhythmia
  • Sympathetic stim
    • Can be from hyperthyroidism, CHF, nervousness, exercise
  • Drugs, even antiarrhythmic drugs (e.g. quinidine)
  • Electrolyte disturbances
    • Hypokalemia
    • Ca + Mg imbalances
  • Bradycardia (incl. bradytachycardia aka sick sinus syndrome)
  • Stretch (englargement + hypertropy of atria/ventricles, CHF, cardiomyopathies, valvular disease)
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6
Q

(T/F) 12-lead EKG is sufficient to view heart rhythm if arrhythmia is suspected.

A

F, run rhythm strip (long tracing of single or mult leads)

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7
Q

What is the diff b/w Holter and event monitors?

A
  • Holter or ambulatory monitor
    • Worn for 24-48h
    • Monitor can employ 1-2 leads (1 precordial and 1 limb lead)
    • Rhythm stored for later analysis of arrhythmias
    • Best for infrequent occurance
  • Event monitor
    • Worn over course of several months
    • Initiated by pt during palpitations
    • Records only 3-5min of rhythm strip
    • Best for even less frequent occurances
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8
Q

How to det HR from EKG?

A
  1. Find R wave that falls on heavy lines
  2. Count number of boxes until next R wave
  3. Determine rate:
    • 1 lg square: 300bpm
    • 2 lg squares: 150bpm
    • 3 lg squares: 100bpm
    • 4 lg squares: 75bpm
    • 5 lg squares: 60bpm
    • 6 lg squares: 50bpm
    • Can divide 300 by number of lg squares or 1500 by number of sm squares

*Can also count number of cycles within two 3s intervals and mult by 10

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9
Q

What are the 5 basic types of arrhythmias?

A
  1. Arrhythmias of sinus origin - follows usual conduction pathways (too fast/slow/irregular)
  2. Ectopic rhythms - electrical activity originates elsewhere than SA node
  3. Reentrant arrhythmias - electrical activity trapped within electrical racetrack, can occur anywhere within heart
  4. Conduction blocks - electrical activity originates in sinus node, follows usual pathways but encounters unexpected blocks and delays
  5. Preexcitation syndromes - electrical activity follows accessory conduction pathway other than normal –> electrical shortcut/ short circuit
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10
Q

Describe sinus tachycardia and sinus bradycardia.

A
  • Depol originates in SA node
  • Above 100bpm or below 60bpm
  • Can be normal or pathologic as with sig heart disease
    • CHF, severe lung disease, hyperthyroidism in elderly –> tachy
    • Early stages of acute MI, enhanced vagal tone –> brady
    • Brady can lead to fainting
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11
Q

Define sinus arrhythmia.

A
  • Slightly irregular but in all else appears NSR = normal phenomenon
  • Variation in HR accompanies inspir + expir
    • Inspir –> accel HR
    • Expir –> decreases HR
  • Diminishes w age, obesity, in pt w long-standing HTN, DM pt w autonomic neuropathy
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12
Q

What is the difference between sinus arrest, asystole, and escape beats?

A
  • Sinus arrest –> SA node stops firing
  • Prolonged arrest = asystole
  • Escape beats –> all cells have pacemaker ability –> other pacemakers spring into action
    • SA node: usually has overdrive suppresion of other foci
    • Atrial automaticity focus: 60-80bpm
    • Junctional automaticity focus: 40-60bpm (most common; AV node typically won’t take over if SA node fails)
    • Ventricular automaticity focus: 30-45bpm (any of purkinje fibers)
    • What can happen at any of these foci is reentrant rhythm (e.g. Afib
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13
Q

What is a junctional escape rhythm?

A
  • Depolarization occuring at/near AV node
  • Usual pattern of atrial depol doesn’t occur –> no normal P wave but may see retorgrade P wave
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14
Q

What is retrograde P wave?

A
  • Depol of atrium moving backwards from normal direction
  • Axis of P wave reversed 180 degrees –> inverted P wave
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15
Q

(T/F) It is impossible to determine whether prolonged sinus pause is due to sinus arrest or sinus exit block.

A

T, but sometimes can be distinguished

  • Sinus arrest: resumption of sinus electrical activity occurs at random time
  • Sinus exit block: resumption in some integer multiple of normal cycle (fired regularly but silently)
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16
Q

What is sinus exit block?

A

Failure of sinus depol to be transmitted out of node into atria

17
Q

What are two major causes of nonsinus arrhythmias?

A

Ectopic and reentrant rhythms

18
Q

What are ectopic rhythms?

A
  • Disorder of impulse formation: abn rhythms arising elsewhere from SA node
  • Single + isolated or sustained
  • Cause:
    • Enhanced automaticity of non-SA node, overdrives normal sinus mechanism
      • Often due to digitalis toxicity and beta adrenergic stim from inhalaer therapies used to treat asthma + COPD
        *
19
Q

What are reentrant rhythms?

A
  • Disorder of impulse transmission: Depol arriving at two adjacent regions of myocardium and one wave transmits more slowly than the other (e.g. from ischemic disease/fibrosis/diff degrees of ANS input) –> wave of depol pushes through slower pathway –> revoling circuit with current sending waves of depol in all directions
    • Normally: depol arriving at two adjacent regions of myocardium, conducting current at same rate –> depol continues normally