Chapter 20 Flashcards

Vision problems

1
Q

What is a cataract

A

age-related (senile cataracts) or other (ex. DM, trauma, smoking) opacity in the lens; maybe one or both eyes (vision may be worse in one than the other)

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2
Q

how is a cataract formed

A

Altered metabolic processes in lens cause h2o accumulation and changes in lens fiber structure -> altered transparency -> vision changes.

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3
Q

What does a cataract look like

A

A totally opaque lens creates the appearance of a totally white pupil.

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4
Q

Describe how cataracts affect one’s vision.

three

A
  1. Graduel decreased vision,
  2. abnormal color perception,
  3. and glare (due to light scatter caused by lens opacities) which can be worse at night.
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5
Q

Cite the 2 ways which cataracts can be detected.

A

Ophthalmoscope: Directly observes opacity
Slit lamp examination: Microscopic examination

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6
Q

what gets implanted when a cataract is removed

A

Most pts have an Intraocular Lens (IOL)

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7
Q

what drops (2) are used after cataract surgery

A

Post Op meds include antibiotic drops to prevent infection and corticosteroid drops to decrease inflammatory response. Drops are gradually reduced in frequency and then stopped as eye heals

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8
Q

Describe activities to be avoided after cataract removal. (4)

A

Bending, stooping, coughing, lifting (activities that increase IOP)

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9
Q

Identify what vision is affected by age related macular degeneration.

A

Central vision

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10
Q

Review the difference between dry and wet AMD; cite which is more severe. P 369

A
  1. Dry (nonexudative): atrophy of macular cells
    *More common
    *Slow, progressive, painless loss of vision
  2. Wet (exudative):
    *More severe; abnormal blood vessels develop in or near macula
    *Rapid onset of vision loss; AMD-related blindness
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11
Q

Describe the changes in vision caused by AMD. (4)

A

Acute vision loss; blurred or darkened vision, scotomas (blind spots) and metamorphopsia (visual distortion)

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12
Q

Cite the location and purpose of injections to help control AMD.

A

Medications injected every 4–6 weeks into the vitreous cavity to stop new vessel formation and slow vision loss. One of the few tx options for WET AMD

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13
Q

Describe dietary supplements that may help decrease the progression of AMD.

A

*Vit C, E, beta-carotene, and zinc are the main ones. Lutein/zeaxanthin supplements may help some pts.
*Teach pt to eat dark green, leafy veggies containing lutein (eg. kale, spinach) and fatty fish at least twice a week

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14
Q

Describe glaucoma and its effect on vision. (what and effects)

A

*What: group of disorders characterized by increased IOP and its consequences and optic nerve atrophy
*Visual effects: peripheral visual field loss leading to permanent blindness.

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15
Q

Describe how glaucoma damages the optic nerve (2)

A

*Primary open-angle glaucoma (POAG): outflow of aqueous decreased; drainage channels clogged -> optic nerve damage.
*As glaucoma progresses, optic disc cupping may be one of the first signs of POAG. The optic disc becomes wider, deeper, and paler (light grey or white).

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16
Q

Identify the long term effect of untreated glaucoma.

A

Blindness

17
Q

Review the 2 types of glaucoma and which one is more common.

A
  1. Primary open-angle glaucoma (POAG): outflow of aqueous decreased; drainage channels clogged -> optic nerve damage.
    *Initially asymptomatic
    *Gradual loss of peripheral vision
    *Late or untreated: tunnel vision
  2. Angle-closure glaucoma (ACG): Reduced outflow from angle closure
18
Q

Cite what type of vision is lost due to primary open angle glaucoma (POAG).

A

Gradual loss of peripheral vision

19
Q

Describe the symptoms associated with acute angle-closure glaucoma (AACG) (four)

A

*Severe, sudden pain in or around eye
*N/V
*Colored halos, blurred vision, ocular redness

20
Q

Cite the goals of glaucoma therapy. (3)

A
  1. No progression of visual impairment
  2. Comply with therapy
  3. No post op complications
21
Q
A