Chapter 19: Endocrine and Hematologic Emergencies Flashcards
Alpha cells
Produce glucagon (stimulates breakdown of glycogen).
Beta cells
Produce insulin
Normal blood glucose
80-120
Hyperglycemia
- Gradual onset
2. AMS, lethargy, severe thirst, visual or sensory deficits, partial paralysis or muscle weakness, seizures.
Hypoglycemia
- Rapid onset
- Normal to shallow or rapid respirations, pale or moist skin, diaphoresis, dizziness or headache, rapid pulse, AMS, seizure, weakness on one side of the body.
Polyuria
Frequent urination. Occurs in DM1 patients as their kidneys are overwhelmed by too much glucose, and it comes out in urine.
Polydipsia
Increase in fluid consumption, frequent urination causes dehydration.
Polyphagia
Increased food intake.
Diabetes Mellitus Type 1
- Autoimmune disorder where the immune system produces antibodies against pancreatic beta cells, and the pancreas can’t produce insulin.
- Many patients experience polyuria, polydipsia, polyphagia, and have pumps to measure blood glucose for them.
Ketones
Acid wastes produced as the body burns fat. Kidneys can’t maintain acid base balance and patients breathe rapidly to reduce acid level.
Diabetic Keto Acidosis
Occurs when DM 1 goes untreated and fat metabolism and ketone production continues. Indicated by abdominal pain, body aches, nausea, vomiting, and AMS.
Diabetes Mellitus Type 2
- Cellular resistance to insulin. Obesity creates predisposition.
- Can be improved with diet and exercise or medications.
- Indicated by recurrent infection, change in vision, numbness.
Hyperosmolar Hyperglycemic Nonketotic Syndrome
Occurs when blood glucose levels are not controlled with DM 2.
Symptoms: Hyperglycemia, AMS, dehydration, visual or sensory deficits, partial paralysis, muscle weakness, seizures.
Hyperosmolarity (diabetes)
Concentrated blood from dehydration.