Chapter 19 Flashcards

0
Q

Depending on the degree of severity, where may fluid progressively move into?

A

The alveoli, bronchioles, and bronchi

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1
Q

Where does the fluid from the pulmonary vascular system first seep into?

A

Perivascular and peribronchial interstitial spaces

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2
Q

What does alveolar swelling and increased alveolar surface tension cause within the lung?

A

Alveolar shrinkage and atelectasis

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3
Q

What kind of pulmonary disorder is pulmonary edema?

A

Restrictive

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4
Q

What are the major pathological or structural changes of the lungs associated with pulmonary edema?

A

Interstitial edema, alveolar flooding, increased surface tension of alveolar fluids, alveolar shrinkage and atelectasis, frothy white (or pink) secretions

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5
Q

What are the two major categories of pulmonary edema?

A

Cardiogenic and non-cardiogenic

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6
Q

What is the most common cause of cardiac pulmonary edema?

A

CHF (congestive heart failure)

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7
Q

What demographic is heart failure most common?

A

People over the age of 65; and African Americans

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8
Q

How does cardiac pulmonary edema occur?

A

Blood cannot be pumped because of left ventricular failure, which increases the hydrostatic pressure inside the pulmonary veins and capillaries

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9
Q

What is normal hydrostatic pressure?

A

10-15 mm Hg

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10
Q

What does normal hydrostatic pressure tend to do with the pulmonary capillaries?

A

It tends to move fluid out of the pulmonary capillaries into the interstitial space

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11
Q

What offsets normal hydrostatic forces?

A

Oncotic pressure

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12
Q

What are normal oncotic pressures?

A

25-30 mm Hg

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13
Q

What happens when hydrostatic pressure within the pulmonary vascular system rises to more than 25 to 30 mm Hg?

A

Oncotic forces lose its holding force and capillary fluid spills into the interstitial and air spaces of the lungs

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14
Q

What are some clinical signs for the patient with left ventricular failure?

A

Anxiety, delirium, cough, fatigue, and adventitious breath sounds, cool skin, diaphoresis, cyanosis of the digits and peripheral pallor

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15
Q

What are some non-cardiac causes of Cardiogenic Pulmonary Edema?

A

Systemic Hypertension, excessive fluid administration, pulmonary embolus, renal failure

16
Q

In non cardiogenic pulmonary edema, what can happen even in the absence of the back pressure caused by an abnormal heart?

A

Fluid easily flows from the pulmonary capillaries into the alveoli.

17
Q

What are four common causes of noncardiogenic pulmonary edema?

A

Increased capillary permeability, lymphatic insufficiency, decreased intrapleural pressure, decreased oncotic pressure

18
Q

What are some causes of increased capillary permeability?

A

Alveolar hypoxia, ARDS, inhalation of toxic agents, pulmonary infections (ie pneumonia), acute head injury (aka cephalogenic pulmonary edema)

19
Q

What may cause a decreased oncotic pressure?

A

Overtransfusion and/or rapid transfusion of intravenous fluids, uremia, hypoproteinemia, acute nephritis, and polyarteritis nodosa

20
Q

What are some common therapeutic interventions for managing pulmonary edema?

A

Antidysrhythemic agents, positive inotropic agents, cardiac workload reduction, sodium and fluid retention therapy, albumin and mannitol

21
Q

What is the most frequently prescribed inotropic agent for heart failure?

22
Q

What is the most effective way to decrease the cardiac workload?

A

Reduce the cardiac afterload (afterload reduction)

23
Q

What are some examples of direct acting vasodilators?

A

Nitroglycerin, nitroprusside, and isosorbide

24
What are some indirect-acting vasodilators?
Alpha -adrenergic receptor blocking agent such as Prazosin or trimazosin
25
What do angiotension converting enzyme (ACE) inhibitors do?
Vasodilation and afterload reduction
26
What does bed rest in the supine position do?
Enhances natural diuresis by the kidneys, reducing sodium and fluid retention
27
What will a restriction of sodium and water intake, and a high-dose diuretic therapy do for a patient with pulmonary edema?
Reduce sodium and fluid retention
28
Why is albumin and mannitol sometimes administered for patients with pulmonary edema?
To increase the patient's oncotic pressure in an effort to offset the increased hydrostatic forces of cardiogenic pulmonary edema
29
What are some of the physical manifestations for pulmonary edema?
Tachypnea, tachycardia, increased blood pressure, Cheyne-Stokes Respiration, cyanosis, cough and sputum,
30
What are some common breath sounds found in patients with pulmonary edema?
Crackles, rhonchi, and wheezing
31
What kind of patients have results of hypokalemia, hyponatremia, and hypocholemia?
Patients with left-sided heart failure
32
What are common radiologic findings in patients with pulmonary edema?
Bilateral fluffy opacities, diluted pulmonary arteries, cardiomegaly, Kerley A and B lines, bats wing or butterfly pattern, pleural effusion
33
Pulmonary venous congestion, Kerley A and B lines and cardiomegaly are examples of what sort of pulmonary edema?
Cardiogenic
34
Fluffy densities, without an enlarged cardiac silhouette are common with which kind of pulmonary edema?
Noncardiogenic