Chapter 19 Flashcards

0
Q

Depending on the degree of severity, where may fluid progressively move into?

A

The alveoli, bronchioles, and bronchi

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1
Q

Where does the fluid from the pulmonary vascular system first seep into?

A

Perivascular and peribronchial interstitial spaces

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2
Q

What does alveolar swelling and increased alveolar surface tension cause within the lung?

A

Alveolar shrinkage and atelectasis

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3
Q

What kind of pulmonary disorder is pulmonary edema?

A

Restrictive

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4
Q

What are the major pathological or structural changes of the lungs associated with pulmonary edema?

A

Interstitial edema, alveolar flooding, increased surface tension of alveolar fluids, alveolar shrinkage and atelectasis, frothy white (or pink) secretions

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5
Q

What are the two major categories of pulmonary edema?

A

Cardiogenic and non-cardiogenic

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6
Q

What is the most common cause of cardiac pulmonary edema?

A

CHF (congestive heart failure)

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7
Q

What demographic is heart failure most common?

A

People over the age of 65; and African Americans

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8
Q

How does cardiac pulmonary edema occur?

A

Blood cannot be pumped because of left ventricular failure, which increases the hydrostatic pressure inside the pulmonary veins and capillaries

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9
Q

What is normal hydrostatic pressure?

A

10-15 mm Hg

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10
Q

What does normal hydrostatic pressure tend to do with the pulmonary capillaries?

A

It tends to move fluid out of the pulmonary capillaries into the interstitial space

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11
Q

What offsets normal hydrostatic forces?

A

Oncotic pressure

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12
Q

What are normal oncotic pressures?

A

25-30 mm Hg

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13
Q

What happens when hydrostatic pressure within the pulmonary vascular system rises to more than 25 to 30 mm Hg?

A

Oncotic forces lose its holding force and capillary fluid spills into the interstitial and air spaces of the lungs

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14
Q

What are some clinical signs for the patient with left ventricular failure?

A

Anxiety, delirium, cough, fatigue, and adventitious breath sounds, cool skin, diaphoresis, cyanosis of the digits and peripheral pallor

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15
Q

What are some non-cardiac causes of Cardiogenic Pulmonary Edema?

A

Systemic Hypertension, excessive fluid administration, pulmonary embolus, renal failure

16
Q

In non cardiogenic pulmonary edema, what can happen even in the absence of the back pressure caused by an abnormal heart?

A

Fluid easily flows from the pulmonary capillaries into the alveoli.

17
Q

What are four common causes of noncardiogenic pulmonary edema?

A

Increased capillary permeability, lymphatic insufficiency, decreased intrapleural pressure, decreased oncotic pressure

18
Q

What are some causes of increased capillary permeability?

A

Alveolar hypoxia, ARDS, inhalation of toxic agents, pulmonary infections (ie pneumonia), acute head injury (aka cephalogenic pulmonary edema)

19
Q

What may cause a decreased oncotic pressure?

A

Overtransfusion and/or rapid transfusion of intravenous fluids, uremia, hypoproteinemia, acute nephritis, and polyarteritis nodosa

20
Q

What are some common therapeutic interventions for managing pulmonary edema?

A

Antidysrhythemic agents, positive inotropic agents, cardiac workload reduction, sodium and fluid retention therapy, albumin and mannitol

21
Q

What is the most frequently prescribed inotropic agent for heart failure?

A

Digitalis

22
Q

What is the most effective way to decrease the cardiac workload?

A

Reduce the cardiac afterload (afterload reduction)

23
Q

What are some examples of direct acting vasodilators?

A

Nitroglycerin, nitroprusside, and isosorbide

24
Q

What are some indirect-acting vasodilators?

A

Alpha -adrenergic receptor blocking agent such as Prazosin or trimazosin

25
Q

What do angiotension converting enzyme (ACE) inhibitors do?

A

Vasodilation and afterload reduction

26
Q

What does bed rest in the supine position do?

A

Enhances natural diuresis by the kidneys, reducing sodium and fluid retention

27
Q

What will a restriction of sodium and water intake, and a high-dose diuretic therapy do for a patient with pulmonary edema?

A

Reduce sodium and fluid retention

28
Q

Why is albumin and mannitol sometimes administered for patients with pulmonary edema?

A

To increase the patient’s oncotic pressure in an effort to offset the increased hydrostatic forces of cardiogenic pulmonary edema

29
Q

What are some of the physical manifestations for pulmonary edema?

A

Tachypnea, tachycardia, increased blood pressure, Cheyne-Stokes Respiration, cyanosis, cough and sputum,

30
Q

What are some common breath sounds found in patients with pulmonary edema?

A

Crackles, rhonchi, and wheezing

31
Q

What kind of patients have results of hypokalemia, hyponatremia, and hypocholemia?

A

Patients with left-sided heart failure

32
Q

What are common radiologic findings in patients with pulmonary edema?

A

Bilateral fluffy opacities, diluted pulmonary arteries, cardiomegaly, Kerley A and B lines, bats wing or butterfly pattern, pleural effusion

33
Q

Pulmonary venous congestion, Kerley A and B lines and cardiomegaly are examples of what sort of pulmonary edema?

A

Cardiogenic

34
Q

Fluffy densities, without an enlarged cardiac silhouette are common with which kind of pulmonary edema?

A

Noncardiogenic