Chapter 11: Cardiovascular Anatomy and Physiology Flashcards
Which structure is the primary or natural pacemaker of the heart?
a. Ventricular tissue
b. Atrioventricular node
c. Sinoatrial node
d. Purkinje fibers
ANS: C
With an intrinsic rate of 60 to 100 beats/min, the sinoatrial node is the primary pacemaker in a healthy heart. The atrioventricular node beats 40 to 60 beats/min. Ventricular tissue must have an electrical impulse to contract. Purkinje fibers beat 15 to 40 beats/min.
The atrioventricular (AV) node delays the conduction impulse from the atria (0.8 to 1.2 seconds) for what reasons?
a. To limit the amount of blood that fills the ventricle from the atria
b. To provide time for the ventricles to fill during diastole
c. To limit the number of signals the ventricles receive in some rhythms
d. To allow the atria to rest between signals
ANS: B
The atrioventricular (AV) node delays the conduction impulse from the atria (0.8 to 1.2 seconds) to provide time for the ventricles to fill during diastole.
A patient reports feeling dizzy after standing quickly. Which finding could provide a clue regarding the cause?
a. Hemoglobin level of 14.0 g/dL and hematocrit level of 42.3%
b. Poor skin turgor with extended tenting
c. Supine blood pressure of 146/93 mm Hg
d. Resting heart rate of 96 beats/min
ANS: B
Poor skin turgor could suggest dehydration. Dehydration can cause orthostatic hypotension because of low capacitance reserves from hypovolemia. Supine blood pressure of 146/93mmHg would be considered hypertensive, and the patient would most likely experience a headache rather than dizziness. A resting heart rate of 96 beats/min is still considered a normal value
Why do many patients with heart rates greater than 120 frequently have chest pain and shortness of breath?
a. Patients with heart disease frequently have an anxiety disorder as well.
b. The rapid pounding of the heart in the chest wall causes the physical pain.
c. The heart muscle gets tired from the increased work.
d. The decreased diastolic time decreases oxygen delivery to the myocardium.
ANS: D
The coronary arteries are perfused during diastole. When the heart rate increases, the diastolic time decreases as each contraction has less time to be completed. This decreases the time the coronary arteries have to deliver oxygenated blood to the myocardium. The symptoms described are caused by a lack of oxygen in the myocardium.
A patient presents with atrial fibrillation, a heart rate of 156 beats/min, and a blood pressure of 124/76 mm Hg. The practitioner orders diltiazem, a calcium channel blocker, to be given slowly by intravenous push. Why did the practitioner choose this medication to treat this patient’s atrial tachyarrhythmia?
a. Diltiazem decreases the calcium influx into the atrioventricular (AV) nodal tissue and decreases the speed of impulse conduction.
b. Diltiazem increases the calcium influx into the AV nodal tissue and decreases the speed of impulse conduction.
c. Diltiazem decreases the calcium influx into the myocardial tissue and decreases the strength of heart contraction.
d. Diltiazem increases the calcium influx into the myocardial tissue and decreases the strength of heart contraction.
ANS: A
Calcium channel-blocking drugs, such as verapamil and diltiazem, inhibit the inward Ca++ current into pacemaker tissue, especially the atrioventricular (AV) node. For this reason, they are used therapeutically to slow the rate of atrial tachydysrhythmias and protect the ventricle from excessive atrial impulses.
What is one hemodynamic effect of a pericardial effusion?
a. Increased ventricular ejection
b. Decreased ventricular filling
c. Myocardial ischemia
d. Increased afterload
ANS: B
If the fluid collection in the sac (pericardial effusion) impinges on ventricular filling, ventricular ejection, or coronary artery perfusion, a clinical emergency may exist that necessitates removal of the excess pericardial fluid to restore normal cardiac function. Myocardial ischemia is damage of the myocardium muscle as the result of a heart attack.
What percentage of volume does atrial kick contribute to ventricular filling?
a. 10%
b. 20%
c. 5%
d. 45%
ANS: B
Atrial contraction, also known as “atrial kick,” contributes approximately 20% of blood flow to ventricular filling; the other 80% occurs passively during diastole.
What is the function of the atrioventricular (AV) valves?
a. Prevent backflow of blood into the atria during ventricular contraction
b. Prevent blood regurgitation back into the ventricles
c. Assist with blood flow to the lungs and aorta
d. Contribute to ventricular filling by atrial kick
ANS: A
The atrioventricular (AV) valves are open during ventricular diastole (filling) and prevent backflow of blood into the atria during ventricular systole (contraction). Semilunar valves prevent the backflow of pulmonic and aortic blood back into the ventricles.
Which step of impulse conduction is most conducive to atrial kick?
a. The firing of the sinoatrial node, which results in atrial depolarization
b. The conduction delay at the atrioventricular (AV) node, allowing time for filling
c. Conduction through the bundle of His, enhancing ventricular depolarization
d. Conduction to the Purkinje fibers, allowing for ventricular contraction
ANS: B
The conduction delay at the atrioventricular (AV) node allows adequate time for ventricular filling from atrial contraction.
Which is an example of a physiologic shunt?
a. A ventricular septal defect
b. Blood returning from the inferior vena cava to the right atrium
c. A septal infarct
d. The thebesian vessels returning deoxygenated blood to the left ventricle
ANS: D
The thebesian vessels return blood to the left ventricle. The mixing of unoxygenated blood with freshly oxygenated blood is called a physiologic shunt. A ventricular septal defect (VSD) allows mixing of blood from both ventricles. The clinical impact depends on the size of the intracardiac shunt. A VSD is a congenital opening between the ventricles; a ventricular septal rupture can occur as a complication of a large anterior wall myocardial infarction.
What is the name of outermost layer of an artery?
a. Tunica
b. Intima
c. Adventitia
d. Media
ANS: C
The adventitia is the outermost layer of the artery that helps strengthen and shape the vessel. The media is the middle layer that is made up of smooth muscle and elastic tissue. The intima is the innermost layer consists of a thin lining of endothelium and a small amount of elastic tissue.
Which of the following is most descriptive of the capillary?
a. Large diameter, low pressure
b. Small diameter, high pressure
c. Large diameter, high pressure
d. Small diameter, low pressure
ANS: D
The diameter of a capillary is less than that of an arteriole, but the pressure is relatively low as a result of the large cross-sectional area of the branching capillary bed.
Depolarization of one myocardial cell will likely result in what physiologic response?
a. Completion of the action potential in that cell before a new cell can accept an impulse
b. Quick depolarization and spread to all of the heart
c. Depolarization of only cells superior to the initial depolarization
d. Quick depolarization of only cells inferior to the initial depolarization
ANS: B
The cardiac muscle is a functional syncytium in which depolarization started in any cardiac cell is quickly spread to all of the heart.
What is the normal resting membrane potential of a myocardial cell?
a. 10 to 20 mV
b. 30 to 40 mV
c. –20 to –30 mV
d. –80 to –90 mV
ANS: D
In a myocardial cell, the normal resting membrane potential is –80 to –90 mV.
Which phase is the final repolarization phase of the action potential?
a. Phase 1
b. Phase 2
c. Phase 3
d. Phase 4
ANS: C
The final repolarization phase is phase 3 of the action potential. Phases 1 and 2 (partial repolarization) occur as the AP slope returns toward zero. The plateau that follows is described as phase 2. In phase 4, the AP returns to an RMP of –80 to –90 mV