chapter 1 Flashcards
After Action Potential
Neurotransmitter released then re-uptake, enzyme destruction, binding to autoreceptors
SSRIs
Neurotransmitters are blown up by enzyme destruction
MAOI
all neurotranmitters are made up of a simple protein called (monalamy - simple protein)
Venom
interrupts job of neurotransmitters (acetocholine controls movement and if stopped you are paralyzed)
Glutamate
primary excilatory (makes neurons fire more), too much causes seizures (random neurons firing for no reason)
GABA
primary inhibitory - stops seizures (#1 transmitter to tell other neurons to slow da fk down)
Benzohioaphine
increases the effectiveness of GABA (alcohol is example)
Acetycholine
important to cause movement, for memory, implicated in Alzheimer’s (part of our memory system uses acetycholine and breakdown of memory system leads to Alzheimer’s)
Nor-epinephrine
wakefulness, precursor to epinephrine and adrenaline
Serotonin
hunger, sleep, arousal, aggression (makes you feel content and when this and nor-epinephrine are low causes mood disorders)
SSNRIs
drugs that work on serotonin
Dopamine
motivation, pleasure, arousal (attention), motor control and implicated in Parkinson’s (cocaine and ampheteimene makes you feel good for activity you are doing)
Endorphine
make you feel less pain, gives you “runner’s high” (drug is so powerful that it kicks out heroin and used in heroin overdose)
Agonists
release mimic, block re-uptake (THC similar to molecule in our brain, nicotine mimics acetycholine-2 different types)
Antagonist
block action of neurotranmitters like “jamming the lock” (neurotransmitter is key)