Chapt 23- Promoting Aspesis/Preventing Disease Flashcards

1
Q

Portal of Entry

A

site where pathogen enters and causes infection

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2
Q

Where do pathogens enter and leave the body

A

Through the same location they came

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3
Q

Why is skin important

A

1 protection from Pathogens

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4
Q

cilia

A

in nose and resp tract and hold onto them and prevent infection

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5
Q

vector

A

caused by parasites- typical mosquitoes, flies, ticks, lice

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6
Q

susceptible host

A

The human person

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7
Q

What can help us prevent being a host

A

staying healthy, eating right

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8
Q

Who are susceptible host

A

cancer pts, auto immune d/o

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9
Q

Classifying infection points

A

Site, Source, Starting Point, How long

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10
Q

Two def of Site

A

Localized- certain loctation

Systemic- all over body

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11
Q

Starting point

A

Primary Vs Secondary

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12
Q

Primary Infection

A

first time exposed to infection, less affected SECOND time infected due to antibodies

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13
Q

What happens in primary

A

No antibodies and no defenses

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14
Q

Secondary Infection

A

Infection during or after treatment of some OTHER infection

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15
Q

Examples of Secondary Infection

A
  1. vagina yeast infection post antibiotics

2.

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16
Q

Endo vs Exogenous infection

A

Person’s on body vs infected from outside source of body

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17
Q

Acute Infection

A

rapin onset and is resolved in several days

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18
Q

Chronic Infections causes

A

viruses

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19
Q

Examples of Chronic

A

Hep, herpes, and epstein barr virus

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20
Q

Latent Infection

A

dormant infections

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21
Q

Examples of Latent

A

Herpes, Mono, Shingles

22
Q

Incubation period

A

generally in acute disease, initial entry into host(pt unaware pathogen is developing

23
Q

Incubation virus example

A

Strep Throat- prior exposed and onset later of virus

24
Q

Infectious Stages

A

Incubation, Prodromal, Illness, Decline, Convalescence

25
Q

Prodromal Period

A

After incubation, pathogens multiple and symptoms begin

26
Q

Illness Stage

A

Full blown illness and symptoms

27
Q

Decline Stage

A

Pathogens are declining but pt at risk for SECONDARY infection here

28
Q

Convalescence Stage

A

Pt’s body goes back to normal, some LONG TERM affects may remain ( ex- hearing lose)

29
Q

Epidemic

A

affect large number of people in ONE are

30
Q

Pandemic

A

Epidemic that has spread all over

31
Q

Drug Resistance bacteria

A

MRSA(staph aureus)
VRE-enterocci
CDIFF

32
Q

MSRA

A
-lives on skin and nose
dormant in healthy person
-treat differently then normal meds
-spreads by skin to skin, clothing, environment
-Aggressive hand washing to get off
33
Q

VRE-enterococci

A
  • Lives in intestine and female genital tract
  • Spread when in the hospital and failure to control
  • LEADING cause of healthcare acquired bacteria, wounds and UTI
  • Risks- weakened immune, surgical procedure, catheters
34
Q

CDIFF

A
  • New virulent strain which can lead to death
  • thrives in hospital environments and wrong prescriptions being prescribed
  • CONTACT PRECAUTION
  • Very strong Oder
  • WASH with SOAP and WATER to kill SPORES
  • USE bleach- cleaning disinfectant
35
Q

Bodies defenses against infection

A

Mucus Membranes- Skin,Respiratory, Eyes, Mouth, GI and GU tract

36
Q

Skin

A
  • Biggest organ of defense
  • tough, resilient and layers
  • Normal Flora inhibits pathogens from entering
37
Q

Resp Tract

A

Nares, trachea and bronchi traps pathogens

-Nares- upper pathway protection via cilia and cough clears them

38
Q

Eyes

A

lac gland produces tears

39
Q

Mouth

A

saliva has lysosomes that wash microbes

-normal flora helps too

40
Q

GI Tract

A
  • acidic environment and bile kills pathogens
  • probiotics help with increasing normal flora
  • Diarrhea and vomiting are F.L of defense to help secrete those pathogens
41
Q

Gu Tract

A

-protective mucus membrane
-urine has high acidity and lysozymes
mucus mem in vagina, and anus help keep pathogens away

42
Q

Secondary Defenses

A

Phagocytosis, complement cascade, inflammation and fever

43
Q

Phagocytosis

A

enfulf pathogens and kill

44
Q

Complement Cascade

A
  • Proteins called complements trigger chemicals and cause rupture of pathogens
  • Complements also signal basophils to release histamine and promote INFLAMMATION
45
Q

Inflammation

A
  • blood vessel dilate and become more permeable for basophils
  • warmth
  • fluid leading= edema
46
Q

Fever

A
  • low grade are necessary for natural defense

- DON’T treat until 102F

47
Q

Active Immunity

A

Occurs when OWN body makes antibodies

48
Q

Passive Immunity

A

Given antibiotics rather than producing their own antibodies

49
Q

Specific Immunity

A

immune cells in body recognize and destroy pathogen

50
Q

Lymphocytes (WBC) types

A

B cells and T cells

-Lymphs are stores in spleen

51
Q

Antigens

A

Molecules that trigger specific immunity response

52
Q

Factors that influence patient’s infection

A

Age, immuno- compromised