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MOD1 > chap 6 symp > Flashcards

chap 6 symp Flashcards

1
Q

Raynaud phenomenon

A

Scleroderma: Episodic vasoconstriction of the a’s of the extremities.

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2
Q

Synovitis

A

Sjogren syndrome

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3
Q

Obliterative phlebitis

A

IgG4 Related Disease: inflamm results in pemanent closure of vessel

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4
Q

increased incidence of tumors

A

Ataxia-telangiectasia synd

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5
Q

women aged 50-60

A

Sjogren syndrome, Scleroderma

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6
Q

Coronary a. dz

A

SLE: can lead to angina or MI.

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7
Q

adverse side effects of HAART

A
  • Lipoatrophy (loss of facial fat)
  • Lipoaccumulation (excess fat deposition centrally)
  • Elevated lipids
  • Insulin resistance
  • Peripheral neuropathy
  • Premature cardiovascular kidney and liver disease
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8
Q

Dryness of the nasal mucosa and epistaxis.

A

Sjogren syndrome

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9
Q

Peripheral neuropathy

A

Sjogren syndrome

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10
Q

edema, erythema, scaliness, follicular plugging, and skin atrophy surrounded by an elevated erythematous border; scarring

A

SLE: Chronic discoid lupus erythematosus

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11
Q

Wide spread damage to small blood vessels

A

Scleroderma

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12
Q

increased PTT

A

SLE

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13
Q

Dysphagia

A

Scleroderma: from esophageal fibrosis that leads to hypomotility

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14
Q

prone to developing AI dis’s

A

Brutons Agammaglobinemia, Isolated IgA deficeincy

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15
Q

Progressive interstitial and perivascular fibrosis in the skin(early) and multiple organs like the GI tract, kidneys, heart, muscles, and lungs later

A

Scleroderma

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16
Q

Renal:
• Mild proteinuria
• Malignant hypertension

A

Scleroderma

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17
Q

middle-aged and older men

A

IgG4 Related Disease

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18
Q

Neuropsychiatric symptoms

A

SLE Due to non-inflammatory occlusion of small vessels

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19
Q

increased serum IgG4

A

IgG4 Related Disease

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20
Q

Diffuse pulmonary fibrosis

A

Sjogren syndrome

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21
Q

Keratoconjunctivitis

A

Sjogren syndrome: blurring of vision, burning, and itching.

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22
Q

defect in ability of helper T cells to deliver activating signals to B cells and macrophages.

A

Hyper IgM synd

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23
Q

susceptable to recurrent pyogenic infections. and to Pneumocystis jiroveci pneumonia.

A

Hyper IgM synd

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24
Q

recurrent infection that leads to early death.

A

Wiskott-Aldrich synd

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25
Q

Respiratory difficulty

A

Scleroderma

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26
Q

make IgM antibodies can’t produce IgG, IgA, and IgE antibodies.

A

Hyper IgM synd

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27
Q

Inflammation of the serosal membranes

A

SLE

  • acute: fibrinous exudate
  • later: thickened
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28
Q

eczema

A

Wiskott-Aldrich synd

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29
Q

loss of T cell imunity

A

DiGeorge Synd

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30
Q

skin rash widespread, superficial, and non-scarring

A

SLE: Subacute cutaneous lupus erythematosus

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31
Q

Splenomegaly, capsular thickening, and follicular hyperplasia.

A

SLE

32
Q

congenital defects of <3 and great valves

A

DiGeorge Synd

33
Q

Valvular (libman-sacks) endocarditis

A

SLE

  • Uncommon now due to the use of steroids.
  • Nonbacterial verrucous endocarditis.
34
Q

immunodeficiency

A

Ataxia-telangiectasia synd

35
Q

telangiectasia , , and.

A

Ataxia-telangiectasia synd (vascular malformations)

36
Q
  • Calcinosis
  • Raynaud phenomenon
  • Esophageal dysmotility
  • Sclerodactyly
  • Telangiectasia
A

CREST Synd

37
Q

butterfly malar rash

A

SLE: on cheeks and forehead; may have on ext’s and trunk; also on uninvolved skin

38
Q

false + for syphilis

A

SLE: d/t antiphospholipids abs

39
Q

Recurrent bronchitis and pneumonitis

A

Sjogren syndrome

40
Q

hypocalcemia and tetany

A

DiGeorge Synd

41
Q

Increased number of infections

A

SLE: d/t immune system dysfunction or tx with immunosuppressive drugs.

42
Q

Parotid gland enlargement

A

Sjogren syndrome

43
Q

normal levels B cells, but low Ig’s

A

CVID

44
Q

esp involves skin, joints, kidney, and serosal membranes.

A

SLE

45
Q

have inherited dificicnces of early compliment components

A

SLE –> impaired removal fo immune complexes

46
Q

low levels of both serum and secretory IgA.

A

Isolated IgA deficiency

47
Q

Chronic interstitial fibrosis and secondary pulmonary HTN.

A

SLE

48
Q

mixture of the features of SLE, systemic sclerosis, and polymyositis.

A

Mixed Connective Tissue Disease

49
Q

B cells are absent or markedly decreased in the circulation

A

Brutons Agammaglobinemia

50
Q

susceptable to recurent infections:
• Bacterial infections of the respiratory tract: Haemophilus influenzae, Streptococcus pneumoniae, or Staphylococcus aureus.
• Viruses in the bloodstream or mucosal secretions: Enteroviruses, such as echovirus, poliovirus, and coxsackievirus.
• Giardia lamblia (intestinal protozoan).

A

Brutons Agammaglobinemia

51
Q

skin thickening

A

Scleroderma

52
Q

Thrombocytopenia

A

Wiskott-Aldrich synd

53
Q

assoc w autoimmune hemolytic anemia, thrombocytopenia, and neutropenia.

A

Hyper IgM synd

54
Q

storiform fibrosis

A

IgG4 Related Disease (whirled like storage pattern)

55
Q

Affects Hispanic and African American women more than white men

A

SLE

56
Q

Infants: Extensive diaper rash.

A

SCID

57
Q

Infants: Failure to thrive.

A

SCID

58
Q

Renal: Hematuria, red cell casts, proteinuria, and sometimes present as classic nephrotic syndrome.

A

SLE

59
Q

Xerostomia

A

Sjogren syndrome: difficultly in swallowing solid foods, decrease in taste, cracks and fissures in the mouth, dryness of the buccal mucosa.

60
Q

Myocarditis

A

SLE:(inflammation of the myocardium) can lead to tachycardia and EKG abnormalities.

61
Q

Abnormal gait

A

Ataxia-telangiectasia synd

62
Q

T cell–mediated reactions are normal

A

Brutons Agammaglobinemia

63
Q

has anas and antiphospholipids abs

A

SLE

64
Q

Myocardial fibrosis

A

Scleroderma: leading to arrhythmias or cardiac failure

65
Q

susceptible to recurrent, severe infections:

•Candida albicans, Pneumocystis jiroveci, Pseudomonas, CMV, varicella, and many bac

A

SCID

66
Q

Pleuritic and pleural effusions

A

SLE

67
Q

recurrent sinopulmonary infections and diarrhea

A

Isolated IgA deficiency

68
Q

neurologic deficits

A

Ataxia-telangiectasia synd

69
Q

Infants have oral candidiasis

A

SCID: (prominant thrush)

70
Q

abn mouth, ears, face

A

DiGeorge Synd

71
Q

acute necrotizing vasculitis

A

SLE

72
Q

Plasma cells are absent throughout the body

A

Brutons Agammaglobinemia, CVID

73
Q

Renal: renal tubular acidosis, uricosuria, phosphaturia

A

Sjogren syndrome

74
Q

Non-erosive synovitis w lttle deformity

A

SLE

75
Q

immune destruction of the lacrimal and salivary glands by lymphocytes.

A

Sjogren syndrome

76
Q

Valve prbs

A

SLE(mostly to mitral and aortic) can lead to stenosis and/or regurgitation.

77
Q

Serum levels of all classes of immunoglobulins are depressed.

A

Brutons Agammaglobinemia, CVID

MOD1 (6 decks)

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