ch 27. psychopathy Flashcards

1
Q

history psychopathy term

A

early 19th century; Pinel; patients who behaved violently and inadequately
1940s; Milton Cleckley; combi socially abnormal behavior and personality traits

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2
Q

clinical picture & diagnostics psychopathy

A

Lombroso 1867: facial features like wide jaws and deep-set eyes
1991: PCL = degree of psychopathy measured in prisoners and forensic patients
PPI= normal population personality check

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3
Q

psychopathy characteristics

A
  • insensitivity
  • coldness
  • lack of empathy
  • pathological lying
  • manipulation
    precursor = child conduct disorder
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4
Q

prevalence and risk factors

A

1-4% normal pupulation, 15-30% international patients
men score higher on characteristics bc of masculine description

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5
Q

4 models/hypothesis about aetiology neuropathology psychopathy

A
  • Low Fear model (LFM) = abnormal reduced anxiety responses + coordinate behavior less as result of negative feedback
    –> looks specifically at insensitivity to punishment + sensation seeking
    criticism: would hinder moral socialization, studies dont show lowered fear response
  • Response Modulation Hypothesis (RMH) Harpur &Hare (1990) = peripheral stimuli that dont receive immidiate attention are not properly processed. They dont make attention shift so that they can calculate their behavioral consequences and adapt their behavior.
    criticism; they can make this shift
  • Violence Inhibition Model (VIM) Blair et al (2005) = psychopaths have disorder in Basal Threat System which is activates by grief&anxiety, consequently their violence inhibition mechanism isnt developed.
    criticism; doesnt expalin emotional and attentional disturbances
  • Integrated Emotion System (IES) model Blair et al 2005 = five systems; 1. transfer sensory representations, 2. make quick decisions based on reward/punishment (valence repfresentation) –> disturbed 3. motor responses 4. response selection system based on largest possible reward 5. response-gating system activates when reward/punishment pattern is violated
    Amygdala plays large role! explains why aversive conditioning not possible in psychopaths
    = more broadly applicable
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6
Q

why neuroimaging data difficult to compare

A
  • definition psychopathy differs
    -collection & processing data differs
  • there is almost always comorbidity
  • no consensus about perfect control group
  • no consensus perfect cutoff score PCL-R
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7
Q

neuroimaging findings psychopathy

A
  • structural deviations prefrontal-temporo-limbic structures; smaller PFC volume gray matter, amygdala, posterior hippocampal regions and superior temporal gyrus + corpus callosum larger
  • lower metabolism + reduced perfusion in both temporal and frontal lobes
  • smaller error related negativities (ERNs) which detect errors
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8
Q

behavior & cognition psychopaths

A
  • higher intelligence =/= inhibitory factor problem behavior as in normal population
  • attention (deviations in shifting to non-dominant stimuli)
  • learning (from rewards rather than punishment, early error detection system is weak & low error awareness)
  • decisionmaking (riskier)
  • social cognition (preference negative stimuli, reduced apathy abilities (respons less to others distress), cant distinguish between harming behavior or behavior that disrupts social order moral vs conventional transgression)
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9
Q

why aggression in psychopaths & how to fix this

A
  • lower serotonin level causes uninhibited aggressive behavior
    –> SSRIs + tryptohpan increase these levels
    –> antipsychotics block dopamine receptors
  • strong positive relation testosterone and violent/antisocial behavior; psychopaths higher testosterone level
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