Ch. 26: Common Respiratory Disorders Flashcards

1
Q

Acute Respiratory Failure (ARF)

A

Hypoxemic (Type 1) - Low PaO2, normal PaCO2

Hypercapnic (Type 2) - Low PaO2, High PaCO2

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2
Q

Causes of ARF

A
Obstruction
Bronchial Disease
Parenchymal disease
CVD
Extrapulmonary disorders
Muscular/neuromuscular junction disorders
Peripheral nerve/spinal cord disorders
CNS disorders
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3
Q

Hallmark signs of ARF?

A

Hypoxemia and Hypoventilation:

Ventilation/perfusion mismatch and/or intrapulmonary shunting

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4
Q

Intrapulmonary shunting?

A

Uncommon, but is when blood is shunted away from alveoli in pulmonary system. Main point is that high O2 delivery does NOT improve SaO2 by that much, very little improvement.

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5
Q

V/Q mismatch

A

Most common cause of ARF. There are areas of the lung in which V/Q is high and V/Q is low. O2 delivery DOES increase SaO2 because everywhere gets high O2 concentrations in lungs. Except in a pulmonary embolism case, then the other areas of the lung compensate for the lost area where the PE is blocking.

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6
Q

Initial Assessment of ARF?

A
ABCs, drowsiness, confusion, silent chest. If any of these are abnormal or present, do:
Consult ICU
Start short-acting beta agonist
O2
Prepare for intubation
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7
Q

Mild-moderate signs of ARF

A
Talks in phrases
Sitting
Non agitated
Increased RR
Tachycardic
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8
Q

Severe signs of ARF

A
Talks in words
Sits hunched forward
Agitated
Tachypneic >30
Tachycardic >120
O2 <90%
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9
Q

Medical Management for ARF?

A
Bronchodialators
Mucolytics
Sedatives (vent only)
Paralytics (vent only)
Bicarb therapy for acidosis if vent is not correcting fast enough
Nutritional support
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10
Q

Nutritional support for ARF, what is included?

A

Normal balanced diet, but in ARF r/t ARDS, high carb solutions are avoided to prevent excess CO2 production

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11
Q

Nursing Management for ARF?

A
Continual respiratory assessment
Vent management
Positioning
Prevent Desatting
Promoting secretion clearance
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12
Q

The triad, and components, of risk for pulmonary emboli

A

Virchow’s Triad:
Hypercoagulability
Vascular endothelium damage
Venous stasis

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13
Q

Other risk factors for PE

A
Trauma
Cancer
Pregnancy
Hormone therapy
Recent surgery
Venous stasis
Inherited thrombophilias
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14
Q

Assessment for PE

A
Chest pain
Dyspnea
Sustained hypotension w/o other explanations
Tachycardia
Hypoxia
Vague S/S
Hemodynamic changes
V/Q scan
CTPA (Computed pulmonary angiography)
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15
Q

Treatment for PE

A
Low molecular weight heparin (lovenox)
Unfractionated IV heparin
Oral anticoagulation
Thrombolytic therapy
Inferior Vena Cava Filter
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