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Foundation of Nursing > ch. 22 promoting asepsis & preventing infections > Flashcards

ch. 22 promoting asepsis & preventing infections Flashcards

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1
Q

Healthcare-Acquired Infections (HAI’s)

A

infections associated with healthcare giving setting

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2
Q

Nosocomial infections

A

these are more specific to hospital setting

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3
Q

Colonization

A

occurs when a microorganism invades the host but does not cause infection

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4
Q

Communicable disease

A

infectious process transmitted from one person to another

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5
Q

chain of infection

A 
infectious agent
reservoir
portal of exit
mode of transmission 
 portal of entry 
susceptible host
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6
Q

normal flora

A

general term that is used for flora that is beneficial or even essential for human health and well-being.

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7
Q

Normal (resident) flora

A

microorganisms that are always present on or in a person and usually do not cause any disease.

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8
Q

Transient flora

A

episodic microorganisms found on or in a person. An example of this is the Norovirus, which can cause the stomach flu.

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9
Q

pathogens

A

causes disease

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10
Q

virulence

A

power of the organism

to cause disease

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11
Q

reservoir

A

living or nonliving material in or on which an infectious agent multiplies and/or develops and is dependent for its survival in nature.

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12
Q

reservoir for infections

A

People, animals, plants, water supplies

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13
Q

reservoir

A 
Nutrients
Moisture
Temperature
Oxygen
pH & Electrolytes
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14
Q

portal of exit

A

site from where micro-organisms leave the host to enter another host and cause disease/infection.

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15
Q

modes of transmission

A

DIRECT CONTACT
Touch
Kiss
Sex

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16
Q

modes of transmission

A

INDIRECT CONTACT
Computer keyboard
pen
Stethoscope

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17
Q

portal of entry

A

site through which micro-organisms enter the susceptible host and cause disease/infection. Infectious agents enter the body through various portals, including the mucous membranes, the skin, the respiratory and the gastrointestinal tracts.
site through which micro-organisms enter the susceptible host and cause disease/infection. Infectious agents enter the body through various portals, including the mucous membranes, the skin, the respiratory and the gastrointestinal tracts.

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18
Q

pathogen

A

often enter the body of the host through the same route they exited the reservoir

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19
Q

Vector-borne diseases

A

human illnesses caused by parasites, viruses and bacteria that are transmitted by mosquitoes, sandflies, triatomine bugs, blackflies, ticks, tsetse flies, mites, snails and lice.

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20
Q

susceptible host

A

someone who is at the risk of infection

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21
Q

localized infection

A

infection stays local at the infected site

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22
Q

systemic infection

A

an infection that travels and spread through out the body. E.G. the blood stream

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23
Q

primary infection

A

the first time you are exposed to and infected by a pathogen.

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24
Q

primary infection

A

when does your body have no innate defenses against the organism, such as antibodies

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25
Q

secondary infection

A

infection that occurs during or after treatment for another infection

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26
Q

secondary infection

A

may be caused by the first treatment or by changes in the immune system.

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27
Q

secondary infection

A

A vaginal yeast infection after taking antibiotics to treat an infection caused by bacteria

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28
Q

pathogens

A

micro-organisms that are capable of causing diseases or infections.

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29
Q

endogenous infection

A

micro-organisms from a person’s own body cause an infection

30
Q

exogenous infection

A

a micro-organism derived from sources outside a person’s own body causes an infection

31
Q

acute viral infection

A

characterized by rapid onset of disease, a relatively brief period of symptoms, and resolution within days. It is usually accompanied by early production of infectious virions and elimination of infection by the host immune system

32
Q

Chronic infections

A

most commonly caused by viruses – this is the case with diseases like hepatitis, herpes, and the Epstein-Barr virus, however, they can be caused by a range of different pathogens.

33
Q

latent infection

A

infection that is hidden, inactive, or dormant. As opposed to active infections, where a virus or bacterium is actively replicating and potentially causing symptoms, are essentially static

34
Q

incubation period

A

occurs in an acute disease after the initial entry of the pathogen into the host (patient).

35
Q

incubation period

A

It is during this time the pathogen begins multiplying in the host. However, there are insufficient numbers of pathogen particles (cells or viruses) present to cause signs and symptoms of disease.

36
Q

incubation period

A

During this period, the patient is unaware that a disease is beginning to develop.

37
Q

prodromal period

A

occurs after the incubation period. During this phase, the pathogen continues to multiply and the host begins to experience general signs and symptoms of illness, which typically result from activation of the immune system, such as fever, pain, soreness, swelling, or inflammation.

38
Q

period of illness

A

during which the signs and symptoms of disease are most obvious and severe.
Full blown s/s of disease are most obvious

39
Q

period of decline

A

the number of pathogen particles begins to decrease, and the signs and symptoms of illness begin to decline. However,
patients may become susceptible to developing secondary infections because their immune systems have been weakened by the primary infection.

40
Q

period of convalescence

A

patient generally returns to normal functions, although some diseases may inflict permanent damage that the body cannot fully repair.

41
Q

epidemic

A

diseases are those that affect large number of people in one area simultaneously

42
Q

pandemic

A

disease that has spread to more than one area

43
Q

MRSA: Staphylococcus aureus

A

Lives on the skin and in the nose usually without causing any problems

44
Q

“Staph” infections

A

usually treated with methicillin, but those infections that are resistant to the drug must be given something else to treat it.

45
Q

“Staph” infections

A

The organism is spread by skin to skin contact and by living in crowded conditions.

46
Q

“Staph” infections

A

It can survive on hands, clothing, environmental surfaces and equipment, it is easily spread to vulnerable patients

47
Q

VRE: Enterococci

A

Normally resides in the intestine and the female genital tract, also occurs in the environment.

48
Q

EColi

A

occur in the hospital and spread is attributed to the failure of infection control measures

49
Q

EColi

A

leading cause of healthcare acquired bacteremia, surgical wound infections, and UTI’s.

50
Q

EColi

A

Risk factors include previous long term use of abx therapy, a weakened immune system, surgical procedures, long term devices (catheters) or colonization

51
Q

C-diff: Clostridium difficile

A

3% of healthy adults carry a harmless version of

52
Q

C.diff

A

It can become harmful when abx destroy healthy bacteria that would normally protect against the disesase.

53
Q

C. diff

A

A newer strain has emerged that is resistant to nearly all abcx and virulent enough to cause sepsis, lasting intestinal damage, the need for colon removal and even death.

54
Q

C.diff

A

It thrives in hospital environments and the federal government is pressuring physicians and hospitals to better enforce sanitation standards and to prevent the overuse of prescription abx.

55
Q

Preventing the spread:

A

Apply contact precautions for patients with diarrhea

56
Q

Preventing the spread:

A

Accurately identify patients with GI issues or infection

57
Q

Preventing the spread:

A

rigorous with cleaning patients rooms and equipment

58
Q

Preventing the spread:

A

Consistent hand hygiene, using soap and water rather than alcohol-based hand rubs for mechanical removal of spores (spores can live on surfaces for days and alcohol based scrubs are not effective on C.diff. spores)

59
Q

Preventing the spread:

A

Use a bleach-containing disinfectant for environmental disinfection

60
Q

primary defenses: Respiratory Tree

A

Nares, trachea and bronchi are all covered with mucous membranes that trap pathogens, which are then expelled.
The nose hair filters the upper airway
The nasal passages, sinuses, trachea, and larger bronchi are lined with cilia that sweep microorganisms upward from the lower airways.
Coughing and sneezing forcefully expel organisms

61
Q

Primary Defenses

A 
skin
mucous membranes
urinary tract 
tears
gastrointestinal tract
62
Q

primary defenses: eyes

A

The lacrimal glands produce tears that contain lysozyme which is an antimicrobial enzyme.
Tears help the body wash infective organisms away

63
Q

primary defenses: mouth

A

Saliva contains lysozyme and helps continually wash microbes from the teeth and gums
Normal flora in the mouth compete for nutrition with invading organisms, thereby limiting the number of pathogens

64
Q

primary defenses: GI tract

A

Many organisms that reach the stomach are destroyed in its acidic environment. Those that successfully enter the small intestine face an antimicrobial action of the bile.
Normal flora in the intestine secrete antibacterial substances.
Diarrhea and vomiting are actually some of the first line defense mechanisms for pathogens that invade

65
Q

Primary Defenses: G Urinary tract

A

is protected with mucous membranes.
The epitheisal cells lining the urethra and the anus secrete mucus, which adheres to pathogens to promote their excretion through urine and stool.
Urine is typically highly acidic and contains lysozymes as well.
Mucous membranes in the vagina also help keep pathogens from est. colonies there.
The vagina also has a high acidity environment and the normal flora keep pathogens in check.

66
Q

SECONDARY DEFENSES: phagocytosis

A

special WBC that engulf and destroy pathogens directly.

67
Q

SECONDARY DEFENSES: complement cascade

A

Proteins called complements, trigger the release of chemicals causing them to rupture.
Complements also signals basophils (another special WBC) to release histamine. Histamine prompts inflammation.

68
Q

SECONDARY DEFENSES: inflammation

A

When histamine and other chemicals are released either from damaged cells, or from basophils being activated by the complement produces inflammation
Blood vessels dilate and become more permeable. This increases the flow of the phagocytes, antimicrobial chemicals, oxygen, and nutrients to the affected area.

69
Q

SECONDARY DEFENSES: Fever

A

A rise in core body temp that increases metabolism, inhibits multiplication of pathogens, and triggers specific immune responses.
Believing that low grade fevers are a necessary natural defense mechanism, many clinicians do not treat a fever unless it is greater than 102F/38.9C

70
Q

The classic s/s of inflammation

A

The classic s/s of inflammation is: localized warmth, and erythema because of increased blood flow. Also, fluid leaking from the more permeable blood vessels accumulates in the surrounding tissue, causing edema, pain and pressure on the nerves.

71
Q

TERTIARY DEFENSES

A 
active immunity
passive immunity 
specific immunity 
lymphocytes
B cells 
T cells
72
Q

Active Immunity

A

Occurs when the body makes it’s own antibodies or T cells to protect the body against pathogens.

Foundation of Nursing (1 decks)

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