Ch 14: Fluid and Electrolytes Quiz Flashcards
Osmosis
movement of water across semi-permeable membrane from low solute concentration to high solute concentration.
Osmolality
of particles of solute in a unit of fluid based on weight (blood, urine)
Osmolarity
of particles of solute in a unit of fluid based on volume. (Spec. gravity)
Tonicity
ability of solutes to cause an osmotic driving force and promote water movement from one compartment to another.
Adrenal gland
releases aldosterone in response to decreased Na+, or increased K+ or renin.
Parathyroid
regulates calcium and phosphate. Secretes PTH which causes bone resorption, calcium absorption from intestines, calcium reabsorption from renal tubules.
Renin-Angiotensin-Aldosterone System
- Renin = enzyme that converts angiotensinogen into angiotensin I. (liver)
- Renin is released by the juxtaglomerular cells of kidneys in response to decreased renal perfusion
- ACE converts angiotensin I to angiotensin II
- Vasoconstriction increases arterial perfusion pressure and stimulates thirst.
- Sympathetic nervous system stimulates release of aldosterone in response to increase renin
- Aldosterone regulates volume
Atrial Natriuretic Peptide (ANP)
opposite of renin-angiotensin-aldosterone system
Gerontologic considerations for increased risk of F&E disorders
- decreased renal and pulmonary function
- altered ratio of body fluid to muscle mass
- Altered response to F&E changes
* Atypical
* Rapid onset - Changes in acid-base balance
Gerontologic Assessment Considerations
- Thirst mechanism
- NPO risk
- Test preps
- Cardiac, kidney, lung, adrenal function
- Attention to intake/output and DAILY WEIGHTS
- Medication effects
Fluid gains/losses
Gain: PO, IV, SQ, Enteral Loss: Kidneys: 1500mL/day - urine **normal u/o = 1 ml/kg/day Insensible loss: *Skin 600 mL/day *Lungs 400 mL/day * GI tract 100-200 mL/day
S/S of hypovolemia
- weight loss
- thirst, dry mucous membranes
- poor skin turgor
- Decreased LOC
- HR, orthostatic hypotension
- Hemoconcentration, increased urine spec. grav., decreased u/o.
- flat jugular veins, time for veins to fill
Causes of hypovolemia
- decreased intake
- blood loss/ hemorrhage
- GI: V/D/ GI suction
- Renal: diuretics, Addison’s, diabetes insipidus, osmotic diuresis
- 3rd space shift: decreased oncotic pressure
- Ascites
The nurse would best assess the adequacy of fluid volume replacement in a patient with hypovolemia by monitoring:
Vital signs and daily weights
S/S of hypervolemia
- rapid weight gain
- Ascites, decreased serum proteins
- Decreased serum and ua osmolality
- decreased urine Na+
- Dyspnea
- HTN, edema, JVD, time for veins to empty
Causes of hypervolemia
- excess fluid intake
- excess Na+ intake
- increased retention of sodium & water
- renal failure
- SIADH (increased ADH) - heart failure
- liver failure
- decreased serum proteins
- liver failure
- malnutrition
- burns
- nephrotic syndrome
Normal Sodium levels
135-145 mEq/L
Functions of Sodium
- # 1 ECF cation
- Major determinant of ECF osmolality
- Muscle contraction/nerve impulse transmission
- Controls water distribution
Hyponatremia
Sodium less than 135
Severe = less than 120
Causes of hyponatremia
- Na+ deficit
- net gain of water (w/o salt)
- *Causes of Na+ deficit
- decreased intake
- increased loss
- diuretics
- GI suction
- Excess sweating
- decreased aldosterone
S/S of hyponatremia
s/s dependent on:
rate of fall
duration of low Na+ levels
ECF volume
- GI: anorexia, N/V, cramping
- Neuro: h/a, lethargy, confusion, seizures caused by H2O moving into brain cell
- Low serum and urine osmolality
Hypernatremia
sodium level greater than 145
Causes of hypernatremia
- decreased fluid intake
- hypertonic tube feeding without adequate water
- increased water losses
- increased insensible losses
- diabetes insipidus (decreased ADH)
- Increased sodium
- increased aldosterone
- corticosteroids
- excess sodium bicarb. or sodium chloride