CDI Foundations Flashcards

1
Q

7 D’s of CDI

A

Definition, Diagnosis, Documentation (providers); Deciphering, Delineation, Deployment (CDI/coders); Defense (everyone, w/audits)

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2
Q

Principal Diagnosis

A

The condition found after study to be chiefly responsible for occasioning the inpatient admission. *based on circumstances of admission, dx approach, treatment rendered

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3
Q

What is heart failure?

A

Inability of heart to effectively pump blood out with each beat.

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4
Q

What are the clinical indicators of heart failure?

A

Increased BNP, sx of pulmonary or systemic congestion (wheezing/resp distress, edema, weight gain, HTN, arrhythmias, JVD, decreased SpO2)

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5
Q

How do you know if a diagnosis is present or a “history of”?

A

History of = no longer present, no longer treated. Present = may be treated (meds, therapies, etc), but can be more or less asymptomatic d/t being well controlled. Ie breast cancer pt on tamoxifen, still “has” breast cancer d/t ongoing tx.

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6
Q

What is HF with reduced EF or preserved EF?

A

Reduced EF (systolic HF) = < 50% EF; Preserved EF (diastolic HF) = > 50% EF

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7
Q

What defines heart failure being “decompensated”?

A

Requiring urgent, unplanned/unscheduled care to manage. IE IV diuretics, increased monitoring, etc.

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8
Q

What are the different types of altered mental status?

A

Delirium: acute change in arousal and content
Depression: chronic change in arousal
Dementia: chronic change in arousal and content
Coma: dysfunction of arousal and content
Psychosis: acute change

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9
Q

Can you explain underlying causes of different types of altered mental states?

A

Possibilities: primary intercranial disease(s), systemic diseases affecting the CNS, exogenous toxins (toxic encephalopathies), drug withdrawal.
Consider: hypoglycemia, hypoxia, infection/sepsis,

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10
Q

Toxic vs metabolic encephalopathy

A

Toxic: usually d/t (external) toxic exposure; worksite neurotoxins, drugs, alcohol, CO2, hydrogen sulfide, cyanide, heavy metals
Metabolic: usually d/t internal factors; hypoglycemia, ischemia, hypoxia, hypercapnia, vitamin deficiencies, d/t peripheral organ dysfunction (hepatic, uremic/dialysis)

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11
Q

PATIO

A

Present at the time of inpatient (admission) order

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12
Q

What is sepsis?

A

The body’s systemic inflammatory response TO infection (not an infection itself).

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13
Q

Sepsis-2

A

Systemic Inflammatory Response to Infection.
clinical indicators = pt “looks” septic
organ dysfunction not required

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14
Q

Sepsis-3

A

Dysregulated host response to infection resulting in organ dysfunction.
requires documentation of “severe sepsis” or “septic shock” to be coded as sepsis-3

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15
Q

Septic shock

A

Patients who require vasopressors to maintain MAP > 65 mmHg or greater and lactate > 2 mmol/L in the absence of hypovolemia.

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16
Q

Sepsis vs cytokine release syndrome

A

Sepsis: body’s response to infection, Interferon rarely elevated, IL-6 and ferritin levels often very high
CKS: not d/t infection

17
Q

Which conditions may present like sepsis but not be due to infection?

A

Anaphylaxis, aspiration pneumonitis, adrenal insufficiency, SBO, DKA, heat emergencies, hypovolemia, PE, pancreatitis, intestinal ischemia, thyroid disease (thyrotoxicosis/thyroid storm), toxicity (ODs) and withdrawals, malignant hyperthermia, neuroleptic malignant syndrome, vasculitis

18
Q

Recommended anti-infective tx for adults with CAP and no concerns for Psuedo or MRSA:

A

Combination therapy: beta-lactam (ampicillin, rocephin, cefotaxime) AND macrolide (azithromycin or clarithromycin).
Monotherapy: respiratory flouroquinolone (levofloxacin)

19
Q

Recommended anti-infective tx for adults with CAP and concerns for MRSA:

A

Vanco or Zyvox (linezolid)

20
Q

Recommended anti-infective tx for adults with CAP and concerns for Pseudomonas Aeruginosa:

A

Zosyn (piperacillin-tazobactam), Cefepime, ceftazidime, aztreonam, meropenem, imipenem

21
Q

Respiratory insufficiency

A

Shortness of breath

22
Q

Respiratory distress

A

labored, difficult breathing

23
Q

Respiratory failure

A

Failure to ventilate or oxygenate (hypoxia, hypercapnia) AEB elevated PCO2 or decreased O2.

24
Q

How to differentiate between acute and chronic hypercapnic respiratory failure?

A

Acute: pH < 7.33
Chronic: pH > 7.33 (compensated)

25
Q

What is shock?

A

life-threatening circulatory failure that leads to tissue hypoperfusion and tissue dysoxia (abnormal tissue oxygen utilization)

26
Q

What are the 4 classifications of shock?

A

Cardiogenic, Distributive (septic, vasodilatory), Hypovolemic, Obstructive (think large PE blocking pulm artery and left ventricle)

27
Q

Clinical indicators of shock

A

sx of poor peripheral perfusion - cold, clammy skin, delayed cap refill, mottling
tachycardia
urine output
abnormal BPs
altered level of consciousness

28
Q

Lab values supporting shock dx

A

lactate levels (#1 cause of elevated lactate = shock, but not the only cause)

29
Q

What makes a debridement excisional vs non-excisional?

A

Excisional debridements are done by scalpel only. Other sharp instruments (scissors, curette, etc) do not apply to excisional debridements.

30
Q

Essentials of a condition: M.U.S.I.C.

A

Manifestation
Underlying Cause
Severity/Specificity - organism?
Instigating cause - what precipitated?
Consequences?