CDI Adult MDC 6 - Digestive Flashcards

1
Q

Esophageal Varices: Definition, Clinical Indicators, Treatment, Coding Guielines

A

Definition: Abnormally dilated submucosal veins in lower third esophagus associated with chronic liver disease (cirrhosis and portal hypertension)

CI: Hematemesis, Syncope, Melena, Hypovolemic Shock

Treatment: Usually requires surgical intervention (Ie: ligation, sclerotherapy)

Coding Guidelines: Require sequencing of cirrhosis as Primary Diagnosis with the Varices as Secondary Diagnosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Laennec’s Cirrhosis with Esophageal Varices
Coding Clinic - Nov/DEC 1985, p14

Question: If a patient is admitted for Bleeding Esophageal Varices and also has Laennec’s Cirrhosis, what is the proper sequencing of the codes?

A

Answer: The Cirrhosis of the liver is sequenced first with the bleeding esophageal varices sequenced second.
*Esophageal Varices are a complication of various types of cirrhosis of the liver.

It is coded:
571.2 Alcoholic cirrhosis of the liver
456.20 Bleeding Esophageal Varices in Diseases Classified elsewhere
303.9 Alcoholism, with fifth-digit designation as to type

If the physician establishes a link between the varices and a cirrhosis diagnosis, the underlying causes (cirrhosis) is the primary diagnosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Clarification Practice: A 64 year old male has been admitted with “bleeding esophageal varices”. He has a 20+ year history of ETOH abuse. The physical exam reveals an enlarged liver with elevated LFTs. An EGD with esophageal banding has been performed this afternoon.

A
  • Bleeding Esophageal Varcies d/t cirrhosis requiring treatment with banding
  • Other; with explanation of the clinical findings
  • Unable to determine (No explanation of clinical findings)

The Medical Record reflects the following clinical evidence:
** Clinical Indicator: **Enlarged liver, Elevated LFT, Esopahgeal Varices
**
Risk Factors: **20+ year history of ETOH Abuse
Treatment: Banding of Esopahgeal Varices

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Esophageal Varices

Secondary Diagnoses

A
  1. Peritonitis: Inflammation of Peritoneal Cavity.
    * Clinical Indicator: Abdominal Pain, Tenderness, Vomiting, Fever, Peristalsis is absent.
    * Treatment: Identify and treat underlying cause; Antibiotics, NGT, O2, Fluids, Electrolyte Replacement.
    * Complications: AKI, ARDS, Liver Failure DIC
  2. Hemorrhagic Shock: A condition of reduced tissue perfusion resulting in inadequate delivery of O2 and nutrients.
    * Clinical Indicator: Hypotension, Tachycardia, Decreased UOP, AMS, Pale/Diaphoretic Skin
    * Treatment: Identify and control source of bleeding, fluid/blood replacement
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Esophageal Banding
-Defintion
-Root Operation

A

Defintion: A banding procedure that uses an extraluminal device to occlude the esophageal varices; used to control the bleeding from the ruptured varix. Is a Valid OR Procedure.

Root Operation = Occlusion ; Completly closing orifice/lumen

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Control of Bleeding: Bleeding Esophageal Varices

A patient with Alcoholic Cirrhosis and Bleeding Esophageal Varices secondary to Portal Hypertension underwent Transorifice Endoscopic Ligation of Esophageal Varices using Bands.

Question: What is the appropriate Root Operation? “Control” or “Occlusion”?

A

The Esophageal Banding (Ligtation) was the definitive procedure and the varices were being treated to prevent further bleeding.

06L38CZ Occlusion of Esophageal Vein with Extraluminal Device, via Natural or Artificial Opening Endoscopic

Transorifice endoscopic banding of esophgeal varices involves complete occlusion of blood flow and meets the defintion of the root operation “Occlusion”. The lumen of the esophageal vein is being banded, not the esophagus.

Explanation:In this Coding Clinic question, the HIM professional seeks advice for the proper assignment of transorifice endoscopic ligation of esophageal varices. The procedure is considered an occlusion using a band to occlude the esophageal vein to prevent future bleeding. Every clinical situation is different. Always review the body of the procedure note to ensure appropriate procedure assignment.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Injection of Sclerosing Agent into Esophageal Varix

What is the ICD-10-PCS Code Assignment for injection of a sclerosing agent into an Esophageal Varix of the Lower Esophagus?

Coding Clinic 1Q 2013 p27

A

Assign Code 3E0G8TZ Introduction of Destructive Agent into Upper GI, via Natural or Artificial Opening Endoscopic for the Injection of a sclerosing agent into the esophageal varix.

This is the correct code assignment since the application of a sclerosing agent via injection into a varix prevents bleeding rather than destroying tissue.

Root Operations:
Destruction = Physical eradication of all or a portion of body part by use of energy, force, or destructive agent

Introduction = Putting in or on therapeutic, diagnostic, nutritional, physiological, or prophylactic substance except blood or blood products

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Clostridium Difficile Enterocolitis
-Definition
-Clinincal Indicators
-Treatment

A

Definition: A bacterium that causes symptoms ranging from diarrhea to life-threatening inflammation of the colon.

Clinical Indicators: Usually develop within 5-10 days after starting antibiotics, but may occur as soon as first day or up to 2 months.
The most common symptoms of mild to moderate C Diff Infection are:
-Watery Diarrhea three or more times a day for two or more days
-Mild abdominal cramping and tenderness

Indicators of severe infection Include:
-Watery diarrhea 10 to 15 times a day
-Abdominal cramping and pain, which may be severe/abdominal distention
-Tachycardia
-Fever
-Blood or pus in stool
-Nausea/Dehydration
-Loss of appetitie/Weight loss
-Acute Renal Failure
-Elevated WBC

Treatment: The first step is to discontinue the causative antibiotic. The standard treatment for C. difficile is another antibiotic. These antibiotics keep C. difficile from growing, which in turn treats diarrhea and other complications. For mild to moderate infection, PO Flagyl. For severe cases, vancomycin or Dificid.

Of note: Up to 20% of patients will experience a recurrence of the condition. In some instances, a fecal transplant may be required. Review your patient’s medical record closely.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Extraction Procedures

Fecal transplantation also known as fecal bacteriotherapy, or human probiotic infusion, is a medical treatment for patients with Clostridium difficile (C. difficile) enteritis or ulcerative colitis. C. difficile infection occurs in patients who have been administered antibiotics for a long period of time. The antibiotics destroy important disease-fighting bacterial flora in the intestine. Fecal transplants are believed to restore the bacteria back to normal and the patient can recover. The fecal transplant works by repopulating friendly flora in the infected intestines. The donated feces is screened for disease and then mixed with a saline solution to the consistency of a “milkshake.” The fecal material is administered by enema, nasogastric tube or endoscopy.

Coding Clinic 4Q2017, p41

A

Explanation:

Some patients with a C. diff infection or ulcerative colitis will undergo a fecal transplant to restore normal bacterial flora into the infected intestine. The donor feces is screened for disease, then mixed into a milkshake consistency with normal saline. The transplant is then administered by enema, NG tube, or endoscopy.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Acute Appendicits with Peritonitis

A

There are two MSDRG options for the assignment of appendicitis.
The first is for “simple” (acute) appendicitis, an uncomplicated diagnosis
The second, a **complicated ** diagnosis involves appendicitis with a complicating condition, like a malignancy or peritonitis (considered a major digestive disorder).

Review the medical record for these clinical clues to support a potential diagnosis of peritonitis:
― Leakage of bowel contents documented in the OP note
― Pathology report + for E. coli in the peritoneal fluid
― Pathology report documentation of a “perforation” noted on inspection of the appendix

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Appendicitis with Sepsis

A
  • If sepsis is confirmed by the physician, assign the sepsis as the PDx from MDC 18, the infectious disease MDC.
  • If sepsis is not present on admission, assign sepsis as a secondary diagnosis.

Review your patient’s medical record closely. If there is clinical evidence of sepsis at the time of admission, clarify the diagnosis with the physician.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Appendicitis with Sepsis Clinical Example

Scenario: This 69 yo female patient presented with lethargy, a temp of 102.5, RR 26, and an elevated WBC with a differential consisting of 34% bands. She is being treated with IVF resuscitation and a combination of Vancomycin and Levaquin. An emergent appendectomy has been performed for a ruptured appendix.

A

Sample Clarification:
* Sepsis, POA in the setting of ruptured appendix requiring aggressive treatment with IVFs and Vancomycin
* Other; with explanation of the clinical findings
* Unable to determine (no explanation for the clinical findings)

The medical record reflects the following clinical evidence:
* Clinical Indicators: lethargy, RR 26, temp 102.5, 34% bandemia
* Risk Factor: Ruptured Appendix
* Treatment: Appendectomy, IVFs, IV Vancomycin

Explanation:
In the clinical presentation of this patient admitted with a ruptured appendix, is acute appendicitis the best diagnosis for this patient? Probably not based on the available information. Note the clarification has been formatted specifically for sepsis, present on admission.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Root Operation: Appendectomy

A

An appendectomy is generally performed to cut out or off, without replacement, all the appendix. This is the definition of the root operation RESECTION, the root operation used for appendectomy.

Important Note: Like other surgical procedures, the intent of the intervention can change once the surgeon enters the patient’s abdomen. Review the operative note closely. If the appendix is very friable, the surgeon may elect to resect a portion of the cecum. This additional surgical work will impact the DRG assignment, as will a colostomy that might be required for concomitant diagnoses of sepsis, peritonitis, or infarcted bowel.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Appendicitis Requiring Resection of the Cecum

Question: A 55-year-old woman presents to the hospital with a five-day history of abdominal pain, fever and chills. An exploratory laparotomy was performed which revealed a perforated appendix. The entire appendix was gangrenous down to the base, which was extremely thin-walled and friable. Therefore, the surgeon performed an ileocecectomy, which included the entire cecum, the terminal ileum and the appendix.

Is it appropriate to assign separate codes for resections of the appendix and the cecum along with excision of the terminal ileum?

Coding Clinic 3Q 2014, p6

A

Answer:An ileocecectomy is a surgery performed to remove the cecum (first part of the large intestine) and the ileum (end part of the small intestine). A complete resection of the cecum always includes part of the terminal ileum (ileocecal valve); the appendix is removed as well. No separate or distinct surgery is carried out to remove the ileum and appendix. Do not separately code the excision of adjacent structures that are an inherent part of the procedure to resect an entire body part. Assign the following ICD-10-PCS code for the ileocecectomy.

0DTH0ZZ Resection of cecum, open approach

Explanation:This Coding Clinic provides the advice for the appropriate assignment of a resection of the cecum in an appendectomy patient. A 55-year-old female was admitted with a five-day history of abdominal pain accompanied by chills and fever. An exploratory lap revealed a gangrenous appendix. The appendix was removed along with the cecum, the starting point of the large intestine. You’ll assign only the resection of the cecum since the appendix is considered an appendage of the cecum. The resection is considered a major bowel procedure.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Colon Cancer
-Definition
-Clinical Indicators
-Treatment
-3 Acute Complications

A

**Defintion: **Colon cancer is a malignant tumor arising from the inner wall of the large intestine.
The most common metastatic sites are: Liver, which can lead to jaundaice and biliary obstruction, lungs, brain, abdominal cavity (ovaries for females)

Clinical Indicators: Patients in the early stage of colon cancer may have no symptoms, but the most common symptoms do involve gastrointestinal bleeding and an unexplained fever.

Risk Factors: Family history of colon cancer, colon polyps, ulcerative colitis

Treatment: Depends on size, location, metastatic sites

Complications: GIB, Bowel Obstruction, Bowel Perforation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Bowel Obstruction
-Obstruction caused by colon cancer is mainly manifested as ___________ obstruction
-The main physiological and pathological changes in patients with obstruction due to colon cancer include:
-Clinical Indicators:
-Clinical diagnosis:
-Treatment:

A

-The obstruction caused by colon cancer is mainly manifested as mechanical obstruction.
-Physiological and pathological changes include: Dilated colon proximal to the obstruction site, fluid and electrolyte loss, and infection. The severity of these changes depends on the site of the obstructed site, the duration of obstruction, and presence (or absence) of any blood supply dysfunction on the bowel wall.

-Clinical Indicators: Patients with a bowel obstruction from colon cancer will present with dull, persistent abdominal pain. Vomiting, abdominal distention, dehydration and electrolyte abnormalities are considered late manifestations.

-Clinical diagnosis: The diagnosis is usually made based on the patient’s clinical presentation. Obstruction will be further confirmed by radiographic studies.

-Treatment: For cancer patients, treatment is focused on correcting dehydration and GI decompression. Surgical intervention may be used to resect the offending tumor, but if resection is not possible, the surgeon may elect to bypass the obstruction with a colostomy.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Coding Clinic Advice - Intestinal Obstruction d/t Peritoneal Carcinomato

How is bowel obstruction due to peritoneal carcinomatosis coded?

Coding Clinic 2Q2017, p10

A

Answer: Assign only code C78.6, Secondary malignant neoplasm of retroperitoneum and peritoneum, as instructed by the Excludes 1 notes found under codes K56.60, Unspecified intestinal obstruction, and K56.69, Other intestinal obstruction, which state: “intestinal obstruction due to specified condition-code to condition.”

Explanation: This Coding Clinic from 2017 provides guidance for the appropriate capture and coding of an obstruction due to peritoneal carcinomatosis. In this situation, the malignancy is assigned as the principal diagnosis based on the instructional notes for obstruction due to a specified condition.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Colectomy
-Root Operation
-Types of coloectomy operations

A

If a portion of the bowel was removed (partial colectomy), index the procedure with the root operation EXCISION.

If the entire portion of bowel (ex: transverse colon) is removed, index the procedure with the root operation RESECTION.

  • Total colectomy involves removing the entire colon.
  • Partial colectomy involves removing part of the colon and may also be called subtotal colectomy.
  • Hemicolectomy involves removing the right or left portion of the colon.
  • Proctocolectomy involves removing both the colon and rectum.

  • Excision: takes out some of a body part
  • Resection: takes out all of a body part
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Colostomy
-Definition
-Root Operation

A

Definition: A colostomy reroutes the contents of the bowel to an opening in the abdominal wall.

You will use the root operation BYPASS to assign the creation of a colostomy.

In this bypass procedure, the body part represents the “from” and the qualifier the “to.” For example, the transverse colon is being rerouted to a cutaneous opening.

Bypass: altering the route of passage of the contents of a tubular body part

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Chemotherapy Administration
* Is Chemotherapy a Valid or Non-valid OR Procedure?
* What is the Root Operation for chemotherapy administration?

A
  • Chemotherapy administration is a non-valid OR procedure
  • During chemo administration, a therapeutic product is being introduced into the patient’s body. Use the root operation INTRODUCTION to index the administration of chemotherapy.

Here’s the graphic for chemo: Using the root operation INTRODUCTION, assign the body system or region where the procedure occurs—not the intended target for the chemo. Next is the approach—either percutaneous or open. Assign the substance and the qualifier.

Introduction: putting in or on a therapeutic substance except blood or blood product

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Testing Your Knowledge

Clinical Scenario: A 41 yo female with an inoperable stage 4 colon tumor presents with bowel obstruction. The surgeon has opted to perform a colostomy for symptomatic relief.

Use this root operation for the procedure:
1. Excision
2. Resection
3. Bypass

A

Answer: 3) Bypass.

Remember the Root Operation for a colostomy is Bypass. In the Bypass procedure, the body part represents the “from” and the qualifier the “to”.
For example, the transvere colon is being rerouted to a cutaneous opening.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

GIB Secondary to Gastric Ulcer - Coding Clinic 3Q2017, p27

Question: A patient presents due to acute gastrointestinal bleed (GIB). An esophagogastroduodenoscopy (EGD) was performed, which showed gastric ulcers as well as portal hypertension. The physician does not link the bleeding to the ulcer nor is it documented that these conditions are unrelated. Under the revised “With” guideline, it appears that we may assume a relationship between the gastrointestinal bleed and the ulcer.

How should we report gastric ulcer in a patient with gastrointestinal bleeding?

A

Answer: It would be appropriate to assign code K25.4, Chronic or unspecified gastric ulcer with hemorrhage. As stated in the ICD-10-CM Official Guidelines for Coding and Reporting, (I.A.15) the classification presumes a causal relationship between the two conditions linked by these terms in the Alphabetic Index or Tabular List. Unless the provider documents a different cause of the bleeding or states that the conditions are unrelated, it is appropriate to assign the combination code for these conditions.

Explanation: In this Coding Clinic from 2017, a coding professional seeks direction concerning the assumption of a relationship between a gastric ulcer identified by EGD and GI bleeding. According to Coding Clinic, it would be appropriate to assume a cause-and-effect relationship between the two conditions since they are linked by the term “with” in the alphabetic index, unless the physician documents a different cause for the bleeding.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Gastrointestinal Hemorrhage
* Definition
* GIB is often divided into — GIB or — GIB
* In most cases of GIB, what will be assigned as the PDX?

A

Definition:Gastrointestinal (GI) bleeding refers to any bleeding that starts in the gastrointestinal tract.

Bleeding may come from any site along the GI tract, but is often divided into:
* Upper GI bleeding: esophagus, stomach, duodenum
* Lower GI bleeding: remainder of the small intestine (ileum, jejunum), large intestine, rectum, anus

  • In most cases of GI bleed, the lesion identified as the cause of bleeding will be assigned as the principal diagnosis.

Your documentation management will include ensuring the appropriate lesion or underlying cause has been identified. Do not make any assumption between a GIB and a potential underlying cause.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

GI Bleed
* The diagnosis of GIB is based on?
* Often a GIB is a clinical manifestation of …?
* If a link is established between a GIB and a potential underlying cause, what becomes the principal diagnosis?

A
  • The diagnosis of GI bleed is often a clinical diagnosis based on simple diagnostic assessment (guaiac) rather than observed frank hemorrhage.
  • Often a “GIB” is a clinical manifestation of an underlying condition. Review the clinical evidence closely and seek clarification of an alternative principal diagnosis if clinically appropriate. Do not make any assumption between a GIB and a potential underlying cause.
  • If a link is established, assign one of the following underlying causes as the principal diagnosis:
    Esophageal ulcer
    Ischemic colitis
    Mallory Weiss tear
    Scleroderma
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

GIB due to Multiple Possible Sources - Coding Clinic 3Q 2018, p16

Question: A patient admitted with hematochezia underwent colonoscopy. The provider’s diagnostic impression included non-thrombosed and non-bleeding internal hemorrhoids, sigmoid diverticulosis, colonic angiodysplasia, and adenomatous cecum polyp. Coding professionals understand that active bleeding does not have to be demonstrated during the hospital stay for the physician to clinically diagnose bleeding, and that the classification makes a linkage between bleeding and angiodysplasia, and diverticulosis with bleeding.

Is it appropriate to assign codes for multiple bleeding sites when more than one finding/possible cause is linked, because of indexing in the classification?

A

Answer: Assign code K57.31, Diverticulosis of large intestine without perforation or abscess with bleeding, and code K55.21, Angiodysplasia of colon with hemorrhage, for the diverticulosis and colonic angiodysplasia with GI bleeding. Either condition may be sequenced as the principal diagnosis. Assign also codes D12.0, Benign neoplasm of cecum, and K64.8, Other hemorrhoids, for the polyp and internal hemorrhoids.

The fact that bleeding is not seen during colonoscopy does not preclude the assignment of a code describing hemorrhage. ICD-10-CM makes a linkage between gastrointestinal hemorrhage and diverticulosis and angiodysplasia; therefore, the provider does not have to link the conditions in the documentation.

Explanation: In this Coding Clinic from 2018, a patient is admitted with hematochezia. A colonoscopy reveals multiple possible sources of the bleeding, including hemorrhoids, diverticulosis, angiodysplasia and a polyp.

The question: “is it appropriate to assign codes for multiple bleeding sites when more than one finding or possible cause is linked by indexing in the diagnostic classification?” The short answer is yes, and any of the conditions may be assigned as principal diagnosis.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

GIB d/t Esophageal Ulcer
* Where do esophageal ulcers typically occur in the esophagus? What clinical information in the documentation should the CDI be looking for?
* Clinical Indicators
* Treatment
* Complications

A
  • Esophageal ulcers occur in the lower end of the esophagus. If the patient has been admitted with what’s been described as an upper GI bleed, consider an esophageal ulcer as a potential underlying cause, particularly if the patient has a history of GERD, is a chronic user of NSAIDs, smokes or has bulimia.
  • Clinical Indicators: difficulty swallowing, painful swallowing, hoarse voice, chest pain with eating, decreased appetite, cough.
  • Treatment: Treatment is focused on the underlying cause, e.g., management of GERD, cessation of smoking, discontinuation of NSAIDs. Esophageal ulcers are prone to recurrence.
  • Complications of esophageal ulcers include upper GI bleeding and perforation.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

GIB d/t Ischemic Colitis
* Definition
* Risk Factors
* Clinical Indicators
* Diagnostics
* Treatment
* Complications

A
  • Ischemic colitis is a medical condition in which inflammation and injury of the large intestine result from inadequate blood supply. Ischemic colitis occurs when blood flow to part of the colon is reduced, usually due to narrowed or blocked arteries.
  • Risk Factors: age 60+, clotting abnormalities, high cholesterol, CHF, previous abdominal surgery, extreme amounts of exercise that decrease blood flow to the colon (marathon running)
  • Clinical Indicators: acute onset of left-sided abdominal pain, tenderness or abdominal cramping, blood in the stool, bowel urgency, nausea, diarrhea. Symptoms often diminish in 2-3 days.
  • Diagnostics: abdominal US/CT, MRI of the abdomen, colonoscopy
  • Treatment: prophylactic antibiotics, IVFs, treatment of the underlying condition, surgical intervention to remove necrotic tissue, repair a perforation, bypass a blocked intestinal artery, or excision of the affected portion of the colon
  • Complications: bowel obstruction (ischemic stricture), gangrene of the intestine, perforation of the intestine, peritonitis

The condition may be misdiagnosed because it can easily be confused with other digestive problems, such as nonspecific abdominal pain or colitis.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

GIB d/t Mallory Weiss Tear
* Definition
* Cause
* Clinical Indicators
* Treatment

A
  • Mallory-Weiss syndrome (MWS) is a condition marked by a tear in the mucous membrane of the esophagus. Most tears heal within 7 to 10 days without treatment, but Mallory-Weiss tears can cause significant bleeding.
  • Cause: The most common cause of MWS is severe or prolonged vomiting. While this type of vomiting occurs with illness, it also frequently occurs due to chronic alcohol abuse or bulimia.
  • Clinical Indicators: MWS doesn’t always produce symptoms. This is more common in mild cases when tears of the esophagus produce only a small amount of bleeding and heal quickly without treatment. In most cases, clinical indicators include:
    abdominal pain
    severe vomiting
    hematemesis
    involuntary retching
    bloody or black stools
  • Treatment: The bleeding that results from tears in the esophagus will stop on its own in about 80 to 90 percent of MWS cases. This typically occurs in a few days and doesn’t require treatment. Other treatment options include:
    surgery (sclerotherapy/coagulation therapy)
    blood replacement
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

GIB d/t Scleroderma
* Definition
* Risk Factors
* Clinical Indicators
* Treatment

A
  • Scleroderma is a group of rare diseases that involve the hardening and tightening of the skin and connective tissues. It is caused by an overproduction and accumulation of collagen in body tissues. In many individuals, the disease process also affects the circulatory system and internal organs, such as the kidneys, lungs, and digestive tract.
  • Scleroderma affects women more often than men and most commonly occurs between the ages of 30 and 50. There is no cure for scleroderma.
  • Clinical indicators of scleroderma’s digestive impact include GERD, gastroparesis, GI bleed, difficulties with nutrient absorption.
  • Treatment: a variety of medications can help control scleroderma symptoms or help prevent complications
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

GIB Secondary Diagnoses: ABLA
* Is there a specific lab paramteter indicating ABLA? What clinical clue may indicate the Physician is thinking and treating but not documenting ABLA?
* Can you assume a diagnosis of acute blood loss anemia from a diagnosis of “blood loss anemia”?

A
  • There is **no specific lab parameter **indicating “acute blood loss anemia”; however, serial monitoring of the H&H or blood transfusion may be a clinical clue of what the physician may be thinking and treating but not documenting.
  • Remember you are not allowed to assume a diagnosis of acute blood loss anemia from a diagnosis of “blood loss anemia.” The physician must provide explicit documentation of the condition.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Acute Blood Loss Anemia Clinical Example - Sample Clarification

Scenario: A 55 yo male presented to the ED complaining of frank blood in his stool; he tested guaiac positive. His H&H on admission was 10 and 24 – he was transfused with two units of PRBCs and taken emergently to the endoscopy suite where a bleeding diverticulum was found. He had been diagnosed with lower GI bleed d/t ruptured diverticulum and anemia. His inpatient orders include H&H q4 hours with an order to infuse another unit of PRBCs.

A

Sample Clarification:
* Acute blood loss anemia in the setting of lower GIB requiring treatment with blood replacement
* Other; with explanation of the clinical findings
* Unable to determine (no explanation for the clinical findings)

The medical record reflects the following clinical evidence:
* Clinical Indicators: blood in stool, guaiac positive, H&H 10 and 24
* Risk Factor: Ruptured Diverticulum
* Treatment: Serial labs and blood replacement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

Internal Guidelines for ABLA - Coding Clinic 1Q2014, p15

Question:We are considering developing internal coding guidelines and obtaining medical staff approval to code acute blood loss anemia. The guidelines would specify lab values pre- and post-surgery, as well as some clinical signs to allow coders to code acute blood loss anemia without the need to have physician documentation.

Would this be acceptable?

A

Answer: No, it is not acceptable.
The Official Coding Guideline Section III.B., states: “Abnormal findings (laboratory, x-ray, pathologic, and other diagnostic results) are not coded and reported unless the physician indicates their clinical significance. If the findings are outside the normal range and the physician has ordered other tests to evaluate the condition or prescribed treatment, it is appropriate to ask the physician whether the diagnosis should be added.” Therefore, internal guidelines should not replace physician documentation.

Facilities can work together with their medical staff to develop facility specific coding guidelines, which promote complete documentation needed for consistent code assignment. Additionally, these guidelines can guide the coding professionals as to when they should query physicians for clarification of their documentation. Any guidelines developed must be applied consistently to all records coded. An internal facility guideline should not interpret abnormal findings to replace physician documentation or physician query. The guideline may provide assistance in determining when a physician query is appropriate, but it may not interpret abnormal test results.

These facility guidelines must not conflict with the “Official ICD-b0-CM Guidelines for Coding and Reporting “ developed by the Cooperating Parties and, additionally, they should not be developed to replace the physician documentation needed to support code assignment.

Explanation:
Here’s an important Coding Clinic from 2014 addressing the issue of a facility developing its own internal coding guidelines that would negate the need for a coding professional or CDS to query the physician for a diagnosis of acute blood loss anemia.

According to Coding Clinic, it would be inappropriate to assign a diagnosis based on lab values without the physician indicating the clinical significance. However, you may certainly work with your medical staff to develop guidelines that help guide the coding professional or CDS when to query or clarify abnormal lab values with the physician. The guideline must be applied consistently across all records, and it may not be used to interpret abnormal lab results for diagnostic purposes. Finally, the guidelines must not conflict with the Official Guidelines.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

GIB Secondary Diagnoses: Hemorrhagic Shock
* Why are patients with GIB at risk for developing hemorrhagic or hypovolemic shock?
* Definition
* Clinical Indicators
* Treatment

A
  • Patients with GI bleed are also at risk for developing hemorrhagic or hypovolemic shock from the loss of circulating blood volume.
  • Hemorrhagic shock is a condition of reduced tissue perfusion, resulting in the inadequate delivery of oxygen and nutrients that are necessary for cellular function.
  • Clinical Indicators: The hallmark clinical indicators of shock have generally been the presence of abnormal vital signs, such as hypotension, tachycardia, decreased urine output, and altered mental status. These findings represent secondary effects of circulatory failure, not the primary etiologic event. The patient may also present with pale, diaphoretic skin, and/or confusion/agitation.
  • Treatment: The primary treatment of hemorrhagic/hypovolemic shock is to control the source of bleeding/fluid loss as soon as possible and to replace that loss. Crystalloid is the first fluid of choice for resuscitation. PRBCs should be transfused if the patient remains unstable after 2000 mL of crystalloid resuscitation.

Whenever cellular oxygen demand outweighs supply, both the cell and the organism are in a state of shock. Hypovolemic shock, the most common type, results from a loss of circulating blood volume from clinical etiologies, such as penetrating and blunt trauma, gastrointestinal bleeding, and obstetrical bleeding. Hypovolemia may also result from overwhelming body fluid loss such as that with extensive or full-thickness burns.

33
Q

GIB Secondary Diagnoses: Acute Kidney Injury
* Is AKI typically seen more with UGIB or LGIB?
* Clinical Indicators of AKI
* Treatment for AKI

A
  • Duration of hospital stay and mortality increases in patients with UGIB complicated by acute kidney injury (AKI).
  • Clinical Indicators: AKI is based clinically on RIFLE, AKIN or KDIGO criteria, including serum creatinine, GFR and/or urinary output. Other indicators may include, anorexia, nausea, vomiting, weakness, confusion.
  • Treatment: Fluid resuscitation, correction of electrolyte imbalances, diuretics or fluid restriction for management of volume overload, discontinuation of any nephrotoxins, hemodialysis.

Gastrointestinal bleeding, especially upper gastrointestinal bleeding (UGIB), is a frequently encountered condition that has a high morbidity, leading to increased treatment costs. Duration of hospital stay and mortality increases in patients with UGIB complicated by acute kidney injury (AKI). It has been reported that AKI develops in 1–11.4% of patients with acute UGB.

34
Q

A 52 yo female with a history of alcohol abuse was admitted to ICU for coffee-ground emesis and “upper GI bleed.” She denied melena or hematochezia and her last bowel movement was several days before admission. Physical exam revealed jaundice and distended abdomen with tenderness on palpation. The patient was noted to have a temp of 100.2 with WBC 15.2 and 83% segs. CT of the abdomen was positive for widespread inflammation, diffuse enteritis, ascites and liver cirrhosis. The patient was made NPO, CIWA protocol initiated, NG tube to suction placed and IV Vancomycin initiated. An endoscopy was performed demonstrating no active source of bleed.

1) What is the patient’s principal diagnosis?
A. (lower) GI Bleed
B. (upper) GI Bleed
C. Ascites
D. Liver Cirrhosis

2)The endoscopy was negative for an active bleed. Does this change the principal diagnosis?
A. Yes
B. No

3) Is there clinical evidence of a potential secondary diagnosis based on the CT findings?
A. Yes
B. No

4) What is the potential secondary diagnosis?
A. Jaundice
B. Distended Abdomen
C. Tenderness to palpation
D. Peritonitis

5) Are you allowed to assume a diagnosis from lab or radiological findings?
A. Yes
B. No

A

Answers:
1) B. Upper GI Bleed
2) B. No
3) A. Yes
4) D. Peritonitis
5) B. No

**Explanation: **
This patient’s principal diagnosis is the upper GI bleed, even though the endoscopy was negative for an active bleeding site. The CT findings provide clinical evidence of a potential important secondary diagnosis—peritonitis. We’ll need to ask a question because we are not allowed to make assumptions of diagnoses from a lab or radiological finding.

If you need remediation, review the GI hemorrhage content.

35
Q

Important Note: Ulcers
* Where can an ulceration occur?
* In the MS-DRG System, most bleeding ulcers will assign to what DRG?
* The Ulcers excluded from the GIB Triplet include?
* What makes an ulcer complicated?
* What makes an ulcer uncomplicated?

A

An ulceration can occur almost anywhere along the GI tract including the esophagus, stomach and duodenum.

**In the MS-DRG system, most bleeding ulcers will assign to a GI bleed DRG: **
* Bleeding Gastric Ulcer
* Bleeding Duodenal Ulcer
* Bleeding Peptic Ulcer
* Bleeding Gastrojejunal Ulcer

The ulcers that are excluded from the GI bleed triplet include:
* Esophageal (with/without bleeding)
* Complicated (ex: specified as perforated)
* Uncomplicated

A complicated ulcer is perforated. If there is bleeding associated with the perforation, the ulcer will be assigned as a GIB.

An uncomplicated ulcer is just that; it’s NOT bleeding or perforated.

It is not likely that a patient with an uncomplicated ulcer would require an inpatient admission. If the documentation does not provide information concerning the inpatient’s ulcer, look for opportunities to further refine the diagnosis, such as

? Bleeding
? Perforated
? Esophageal
Capturing this additional information will help support inpatient medical necessity.

36
Q

Gastric Ulcers
* Definition
* Clinical Indicators
* Risk Factors
* Treatment
* Complications

A
  • A gastric ulcer occurs when the thick layer of mucus that protects the stomach from digestive juices is reduced. This allows the digestive acids to eat away at the tissues that line the stomach, causing an ulcer.
  • Clinical Indicators: Abdominal pain that improves with eating, nausea, vomiting, acid reflux, symptoms of anemia, tarry stools, coffee ground emesis.
  • Risk Factors: Two common causes of gastric ulcers are the H. pylori organism and long-term use of NSAIDs. These cause the thick layer of mucus lining the stomach to become significantly reduced causing the digestive juices to eat away at the lining of the stomach.
  • Treatment: If the underlying cause is H. pylori, expect the patient to be placed on antibiotics. Proton pump inhibitors and H2 receptor blockers are also commonly prescribed; probiotics.
  • Complications: Complications include bleeding, perforation, bowel obstruction, peritonitis, and sepsis.
37
Q

GI Bleed Secondary to Gastric Ulcer - Coding Clinic 3Q2017, p27

**Question: ** A patient presents due to acute gastrointestinal bleed (GI). An esophagogastroduodenoscopy (EGD) was performed, which showed gastric ulcers as well as portal hypertension. The physician does not link the bleeding to the ulcer nor is it documented that these conditions are unrelated. Under the revised “With” guideline, it appears that we may assume a relationship between the gastrointestinal bleed and the ulcer.

How should we report gastric ulcer in a patient with gastrointestinal bleeding?

A

Answer: It would be appropriate to assign code K25.4, Chronic or unspecified gastric ulcer with hemorrhage. As stated in the ICD-10-CM Official Guidelines for Coding and Reporting, (I.A.15) the classification presumes a causal relationship between the two conditions linked by these terms in the Alphabetic Index or Tabular List. Unless the provider documents a different cause of the bleeding or states that the conditions are unrelated, it is appropriate to assign the combination code for these conditions.

Explanation: This Coding Clinic from 2017 provides guidance for the appropriate assignment of a GI bleed and gastric ulcers found on EGD. But the physician does not link the two in the documentation. The HIM professional seeks advice about assuming a relationship between the two conditions based on the “with” official guideline. According to Coding Clinic, it would be appropriate to assume this causal relationship linked by the term “with” in the alphabetic index. You would not assume the relationship if the physician documented a different cause of the bleeding.

38
Q

Bleeding Duodenal Ulcer - Coding Clinic 4Q 2016, p99

**Question: ** A patient presents with bleeding duodenal ulcer and an esophagogastroduodenoscopy was carried out. Multiple clips were applied to the vessels to control the multiple hemorrhaging ulcers.

Should “control” be assigned for the root operation? What is the appropriate ICD-10-PCS procedure code?

A

Answer: The definition of the root operation “control” has been revised and is now applicable to correct postprocedural or other acute bleeding. In this case, the bleeding is of the duodenum. Most of the body’s organs and tissues are vascular, and they bleed when cut or eroded. Control of bleeding of a cut or eroded body part is coded to the body part where the bleeding was controlled, rather than to a vascular system body part. In this case, the bleeding from the duodenal ulcer was controlled via an endoscopic approach, with clips placed on vessels eroded by the ulcers.

Assign the following ICD-10-PCS code:
0W3P8ZZ Control bleeding in gastrointestinal tract, via natural or artificial opening endoscopic

Explanation:This clinic and the next provides advice for the capture of procedures performed to control GI bleeding due to ulcers; this first for the endoscopic application of clips to control the bleeding. Use the root operation “control” to assign this valid OR procedure.

39
Q

Control Gastrointestinal Bleeding - Coding Clinic 4Q 2017, p82

Question: The patient underwent endoscopy due to upper gastrointestinal bleeding. A large duodenal ulcer with adherent clot was found and injected with epinephrine for hemostasis and 3 clips were placed.

Is “Control” of bleeding appropriate for this procedure when there is no documentation of active bleeding?

A

Answer: When a patient is seen for bleeding and a procedure is performed to control the hemorrhage and prevent a recurrent bleed, assign a code for control of bleeding. Active bleeding does not need to be demonstrated during the encounter to diagnose and treat an acute hemorrhage.

Assign the following ICD-10-PCS code:
0W3P8ZZ Control bleeding in gastrointestinal tract, via natural or artificial opening endoscopic

Explanation: In this clinic, an injection of epinephrine was used along with clips on an ulcer that was not actively bleeding. So, is it appropriate to index the procedure with the root operation “control”? Coding Clinic advises, “Active bleeding does not need to be demonstrated during the encounter to diagnose and treat an acute hemorrhage.” So, yes, control is the appropriate root operation for the procedure.

40
Q

Control of a Bleeding Ulcer
* Why is a bleeding ulcer a surgical emergency?
* What is the intent of surgical intervention?
* What is the root operation to index the procedure?

A
  • A bleeding ulcer of any kind is a surgical emergency given the subsequent complications (acute blood loss anemia, acute renal failure, hypovolemic shock).
  • The intent of surgical intervention is to control the frank bleeding or oozing from the ulcer.
  • Use the root operation CONTROL to index the procedure.

Control: restoring body part to its normal structure

41
Q

Inflammatory Bowel Disease

A
  • Crohn’s and ulcerative colitis are two distinct disease processes, although each causes an inflammatory reaction in the GI tract.
  • Do not confuse the terms “inflammatory bowel disease” and “irritable bowel syndrome.”
  • Crohn’s disease can occur anywhere along the patient’s GI tract, while ulcerative colitis is limited to the colon.
  • Ulcerative colitis only affects the innermost lining of the colon; Crohn’s disease can occur in all the layers of the bowel walls.
42
Q

Crohn’s Disease

A
  • Affects the entire GI Tract
  • Most common ite: Terminal end of the small intestine
  • Patchy appearance
  • May extend through the entire thickness of the bowel
  • Symptoms: Frequent diarrhea, rectal bleeding, unintended weight loss, fever, abdominal pain, fatigue, poor appetite
  • Treatment: Antibiotics, Aminoglycosides, corticosteroids, immunomodulators to suppress the immune response

Surgical options: excision/resection of the affected portion of the bowel

43
Q

Ulcerative Colitis

A
  • Limited to the large intestine
  • Most common site: portion or entire colon including the rectum
  • Continuous pattern of inflammation
  • Inflammatory process occurs only in the lining of the colon
  • Symptoms: Abdominal pain, blood or pus in stool, fever, unintended weight loss, frequent diarrhea, fatigue, poor apetite, tenesmus
  • Treatment: Antibiotics, Aminoglycosides, corticosteroids, immunomodulators to suppress the immune response

Surgical Options: Resection of the colon, rectum and anus with the creation of an Ileostomy; Resection of the colon and rectum with creation of an ileal pouch to anus anastomosis

Types of Ulerative Colitis Include:
Ulcerative Proctitis
Proctosigmoiditis
Left-sided Colitis
Pancolitis
Acute severe ulcerative colitis

Complications: GIB, Perforated bowel, Severe Dehydration, Toxic Megacolon

44
Q

Ulcerative proctitis

A

Inflammation is confined to the area closest to the anus (rectum), and rectal bleeding may be the only sign of the disease. This form of ulcerative colitis tends to be the mildest.

45
Q

Proctosigmoiditis

A

Inflammation involves the rectum and sigmoid colon (lower end of the colon). Signs and symptoms include bloody diarrhea, abdominal cramps and pain, and an inability to move the bowels in spite of the urge to do so (tenesmus).

46
Q

Left-sided colitis

A

Inflammation extends from the rectum up through the sigmoid and descending colon. Signs and symptoms include bloody diarrhea, abdominal cramping and pain on the left side, and unintended weight loss.

47
Q

Pancolitis

A

Pancolitis often affects the entire colon and causes bouts of bloody diarrhea that may be severe, abdominal cramps and pain, fatigue, and significant weight loss.

48
Q

Acute severe ulcerative colitis

A

This rare form of colitis affects the entire colon and causes severe pain, profuse diarrhea, bleeding, fever and inability to eat.

49
Q

Ulcerative Colitis and Toxic Megacolon
* Definition
* Clinical Indicators
* Treatment
* Complications

A
  • Toxic megacolon occurs when inflammatory bowel diseases cause the colon to expand, dilate, and distend. It is considered a life-threatening condition.
  • Clinical Indicators: The patient will present with abdominal pain and distention. Other indications include fever, tachycardia, and symptoms of shock. The patient may also experience profuse bloody diarrhea.
  • Treatment: Surgical intervention including partial and total colectomy. The surgical procedure of choice for toxic megacolon is total colectomy with preservation of rectum and diverting ileostomy.
  • Complications: The complications associated with toxic megacolon are just as life-threatening: bowel infarction, perforation, peritonitis, and sepsis.

Important Note: The 30-day mortality rate is 57%, with an in-hospital mortality rate of 49%. You’ll want to ensure you’ve captured all evidence of SOI and ROM.

50
Q

Crohn’s Disease with Bowel Obstruction or Rectal Abscess
* What are 2 common complications of Crohn’s Disease?
* Is there an assumed link between Crohn’s Disease and Bowel Obstructions?
* What is the Principal Diagnosis if a patient is admitted with an obstruction due to Crohn’s Disease?
* What is the Principal Diagnosis when an Obstruction is secondary to Crohn’s Disease?
* If there are multiple potential causes of the bowel obstruction, what must the CDI do?

A
  • Bowel obstructions are one common complication of Crohn’s disease; as such, there is an assumed link between the two conditions.
  • If the patient is admitted with an obstruction due to Crohn’s disease, assign Crohn’s as the principal diagnosis.
  • When the obstruction is secondary to Crohn’s disease, the principal diagnosis is Crohn’s disease with obstruction.
  • If there are potential multiple causes of the bowel obstruction, it may be necessary to seek clarification.
  • Another common complication for the Crohn’s patient is a rectal abscess. The rectal abscess qualifies as a potential severity diagnosis.

Crohn’s Disease with Rectal Abscess: This condition may not be addressed in the medical record documentation but might be mentioned on a colonoscopy procedural note. Always review any procedural note carefully to ensure clinically supported diagnoses are captured in the medical record documentation.

51
Q

Crohn’s Disease and Rectal Abscess - Coding Clinic 4Q2012, p104

Question: In ICD-10-CM codes K50.014, K50.114, K50.814 and K50.914 are used to identify Crohn’s disease with intestinal abscess.

When a patient presents with Crohn’s disease of the small intestine with a rectal abscess, would it be appropriate to assign an additional code for the rectal abscess?

A

Yes, it is appropriate to assign code K50.014, Crohn’s disease of small intestine with abscess, along with code K61.1, Rectal abscess, since the additional code provides information regarding the specific site of the abscess. Codes in category K50 describe intestinal abscess only.

Explanation: This Coding Clinic from 2012 is a reminder to review the medical record closely for the Crohn’s patient. If a rectal abscess is documented and meets the guideline for reporting of a secondary diagnosis, capture the rectal abscess. Assign two separate codes—K50.014 for Crohn’s with abscess, and the second code, K61.1, to identify the type of abscess.

52
Q

A 30 yo male with Crohn’s disease has been admitted with a “flare.” During your review of the documentation, you find mention of a rectal abscess with orders for surgical consult and PO medications for pain.

Does the rectal abscess meet the definition for capture as a secondary diagnosis?
1. Yes
2. No
3. Only if it is surgically drained

A

Answer: A. Yes.

Explanation:
The correct answer is Yes. In this situation the rectal abscess does meet the definition of a secondary diagnosis since it requires clinical evaluation, therapeutic treatment and increased nursing care.

53
Q

Bowel Obstruction
* The main Physiological and Pathological changes in patients with obstruction include?
* The severity of the Physiological and Pathological changes depend on?
* Clinical diagnosis
* Clinical Indicators
* Treatment

A
  • The main physiological and pathological changes in patients with obstruction include a dilated bowel proximal to the obstruction site, fluid and electrolyte loss, and infection.
  • The severity of these changes depends on the site of the obstructed site, the duration of obstruction, and presence (or absence) of any blood supply dysfunction on the bowel wall.
  • Clinical diagnosis: The diagnosis is usually made based on the patient’s clinical presentation. Obstruction will be further confirmed by radiographic studies.
  • Clinical Indicators: abdominal pain (persistent and dull), vomiting (late manifestation), abdominal distention (late manifestation), dehydration, hypokalemia.
  • Treatment: correct dehydration and electrolytes, GI decompression (NG tube), treat the underlying cause.
54
Q

Causes of Small Bowel Obstruction

A

Adhesions from previous abdominal surgery (most common cause)
Hernias containing bowel
Crohn’s* disease causing adhesions or inflammatory strictures
Neoplasms, benign or malignant
Volvulus

55
Q

Causes of Large Bowel Obstruction

A

Neoplasms, benign or malignant
Hernias containing bowel
Inflammatory bowel disease
Adhesions
Fecal impaction
Diverticular disease
Endometriosis

56
Q

Documentation Validation

Clinical Scenario: A 17 yo male presented to the ED with complaints of severe abdominal pain and vomiting. An NG tube was inserted for decompression and he was admitted with a diagnosis of “bowel obstruction.” He has a significant history of Crohn’s disease as well as surgical adhesiolysis, hernia repair and appendectomy. Other components of the treatment regimen include NPO status and IVFs.

What is the physician thinking and treating but not documenting?

Review this scenario for an unfortunate 17-year-old male with a bowel obstruction. Determine if there is a question that should be presented to the physician and what evidence you’ll use to support that question.

A

Clarification Example
* Bowel obstruction d/t Crohn’s disease requiring NGT decompression
* Other; with explanation of the clinical findings
* Unable to determine (no explanation for the clinical findings)

The medical record reflects the following clinical evidence:
* Clinical Indicators: bowel obstruction
* Risk Factors: Crohn’s disease, previous multiple bowel surgeries
* Treatment:NGT, NPO status, IVFs

57
Q

Abdominal Pain d/t SLE Enteritis
* Definition
* Cardinal Signs
* Gold Standard for diagnosis with 3 classic patterns
* Clinical Indicators
* Treatment
*

A
  • Lupus enteritis is defined as either vasculitis or inflammation of the small bowel.
  • Acute abdominal pain, often nonspecific with diarrhea and vomiting, are typically the cardinal signs for lupus enteritis.
  • CT of Abdomen is the golden standard for diagnosis with 3 classic patterns suggestive of lupus enteritis:
    1. Bowel wall thickening grater than 3mm, also referred to as Target Sign
    2. Engorgement of the Mesenteric Vessels, also referred to as Comb Sign
    3. Increased attenuation of mesenteric fat
  • Clinical indicators: Focal or diffuse abdominal pain is the cardinal manifestation with associated symptoms of nausea, vomiting, ascites, diarrhea, with or without fever, and possible rebound tenderness with guarding
  • Treatment: Corticosteroids is typically the first-line therapy along with complete bowel rest. Cyclophosphamide or mycophenolate may be recommended for patients with severe or steroid-resistant lupus enteritis.
  • Sequenced as the principal diagnosis, the SLE will assign to MDC 8 which more accurately reflects the severity of illness and risk of mortality associated with the systemic disease.

Patients with SLE can often have gastrointestinal tract involvement.

58
Q

SLE Clinical Example

Scenario: A 28 yo female with a history of SLE presented to the ED complaining of abdominal pain, nausea, and vomiting x2 days. A CT of her abdomen in the ED revealed a combination of ascites, Comb sign and increased attenuation of mesenteric fat. She was admitted with a diagnosis of “abdominal pain,” and her treatment regimen includes IV steroids and bowel rest. Today’s progress notes still reflect a diagnosis of “abdominal pain.“

A

Sample Clarification:
* Abdominal pain d/t SLE enteritis requiring treatment with IV steroids
* Other; with explanation of the clinical findings
* Unable to determine (no explanation for the clinical findings)

The medical record reflects the following clinical evidence:
* Clinical Indicators: abdominal pain with N/V, CT + for ascites, Comb sign, and increased attenuation of mesenteric fat
* Risk Factor: SLE
* Treatment: IV steroids, bowel rest

59
Q

Abdominal Pain d/t Postcholecystectomy Syndrome
* Definition
* Risk Factor
* Clinical Indicators
* Treatment

A
  • Patients with this condition continue to experience biliary symptoms associated with gallbladder disease, such as bloating, burping, and intolerance to fatty foods even post-surgery after the removal of the gallbladder.
  • Risk Factor: s/p cholecystectomy
  • Clinical indicators include:
    persistent pain in the upper right abdomen
    nausea
    vomiting
    intolerance to fatty foods
    referred pain to right shoulder
    belching
    bloating
    dyspepsia
    biliary colic
  • Treatment: treatment of symptoms, GI workup, ERCP

Review the clinical presentation of the patient carefully. If the patient has a history of cholecystectomy, consider seeking validation of postcholecystectomy syndrome from the physician to more accurately describe the patient’s severity of illness. There is no definitive treatment for postcholecystectomy syndrome, but the symptoms do require maintenance. Symptoms occur in 5-40% of patients who undergo cholecystectomy.

60
Q

Postcholecystectomy Syndrome - Coding Clinic 1Q 1988, p10

Question:
In I10: K91.5 Postcholecystectomy Syndrome
What is a postcholecystectomy syndrome, 576.0?

A

Answer: Postcholecystectomy syndrome, 576.0, classifies those cases in which symptoms suggestive of biliary tract disease either persist or develop following cholecystectomy but for which no demonstrable cause or abnormality is found on workup.

Explanation: In 1988, Coding Clinic addressed the diagnosis of postcholecystectomy syndrome, describing it as those cases in which symptoms suggestive of biliary tract disease either persist or develop following cholecystectomy but for which no demonstrable cause or abnormality is found on workup. Postcholecystectomy syndrome is an MDC 7 or hepatobiliary diagnosis.

61
Q

Yes or No? The clinical indicators of postcholecystectomy syndrome include:
* nausea
* localized pain at McBurney’s point
* watery diarrhea

A

Answer: No

Explanation: The correct answer is No. The clinical indicators include persistent pain in the upper right abdomen, nausea, vomiting, intolerance to fatty foods, referred pain to right shoulder, belching, bloating, dyspepsia, biliary colic but not localized pain at McBurney’s point which is often associated with appendicitis.

62
Q

Nausea and Vomiting with Aspiration

A

If your patient has nausea and vomiting, review the clinical presentation and treatment regimen closely. It would seem odd to find a patient admitted for vomiting with a treatment regimen including aspiration precautions and IV Clindamycin. If the clinical evidence suggests a diagnosis of aspiration pneumonia or bronchitis, ask a question. The physician may be thinking and treating the condition but not adequately documenting the diagnosis.

63
Q

Esophagitis d/t Candida
* Definition of Esophagitis
* Causes of Esophagitis
* How is Esophagitis diagnosed?

A
  • Esophagitis is an inflammation of the lining of the esophagus.
  • Esophagitis can be caused by several underlying conditions such as reflux, irritation from medications, or infections.
  • It is usually diagnosed by endoscopic exam.

Esophagitis is one of the diagnoses that is considered a miscellaneous d

Investigate the record carefully, especially if the patient undergoes an EGD. If the EGD reveals the characteristic white patches of candida or the pathology returns positive for candida, seek clarification for candidal esophagitis, a major esophageal disorder. This specified type of esophagitis more accurately reflects the SOI and ROM associated with the organism.

Be sure to review any cultures obtained as clinical support for your clarification, but remember, a lone lab value does not adequately support an alternative diagnosis.

64
Q

Eosinophilic Esophagitis - Coding Clinic 4Q2020, p8

Question: A patient was seen due to dysphagia with chest and abdominal discomfort. The provider performed an esophagogastroduodenoscopy with biopsies. Heavy eosinophilic infiltration and furrows were seen on the esophageal mucosa. Eosinophilic esophagitis was the final diagnosis.

How is eosinophilic esophagitis coded?

A

Answer: Assign code K20.0 Eosinophilic esophagitis, for eosinophilic esophagitis. It would not be appropriate to assign code D72.18, Eosinophilia in diseases classified elsewhere, as eosinophilic esophagitis is specifically indexed to code K20.0 and the code already identifies both the esophagitis and the eosinophilia.

Explanation:In some instances, the EGD doesn’t return positive for candida but for eosinophils. This Coding Clinic advises the assignment of code K20.0, eosinophilic esophagitis rather than D72.18 because the condition can be specifically indexed.

Patients at risk include those living in a cold/dry climate, during the spring/fall season of the year, male gender, family history, history of food or environmental allergies, and chronic respiratory disease. The symptoms include dysphagia, food impaction, substernal chest pain, and regurgitation of undigested food. The patient will require treatment with dietary management, proton pump inhibitors, topical steroids, and in many cases, dilation of the esophagus.

65
Q

Extraction Procedures - Coding Clinic 4Q 2017, p41

Question 1: A patient underwent an endoscopic brush biopsy of the stomach. What is the procedure code assignment for this biopsy?

Question 2:How should a debridement of the right lower leg including removal of necrotic muscle using jet lavage be coded?

A

Answer 1:Assign the following ICD-10-PCS code:
0DD68ZX Extraction of stomach, via natural or artificial opening endoscopic, diagnostic, for the endoscopic brush biopsy of the stomach

Answer 2:The jet lavage debridement is considered nonsurgical mechanical debridement. It does not involve cutting away or excising devitalized tissue. Assign the following ICD-10-PCS code:
0KDS0ZZ   Extraction of right lower leg muscle, open approach

When multiple layers of the same site are debrided, assign only a code for the deepest layer of debridement. In this instance, only debridement of the muscle as the deepest layer is assigned.

The root operation value “D” Extraction, has been added to the following

The change will allow the capture of additional detail for the root operation “Extraction,” including percutaneous aspiration biopsies and brush biopsies for the respiratory, gastrointestinal, and lymphatic body systems, and nonexcisional debridement for the muscles, tendons and bones body systems.

Explanation:Brush biopsies (including GI sites) are assigned to root operation Extraction, therefore the previous advice provided per Coding Clinic 1Q 2016 regarding the assigning of Excision for a brush biopsy of the esophagus is no longer valid.

66
Q

Gastroenteritis or Toxic Gastroenteritis?

A
  • Chemotherapy can cause inflammation anywhere along the GI tract resulting in the symptoms generally associated with Gastroenteritis “GE”
  • Gastroenteritis caused by chemotherapy (or exposure to other toxic substances) is referred to as “toxic gastroenteritis.”
  • Important Note: The inclusion terms for toxic gastroenteritis include drug-induced gastroenteritis, drug-induced colitis, and drug-induced diarrhea.

Do not assume a link between the two conditions

Colitis and gastroenteritis as miscellaneous GI disorders do not capture the appropriate SOI when the condition is caused by exposure to radiation or a toxic agent, such as chemotherapy. The patient with toxic gastroenteritis or radiation colitis will present with symptoms mimicking colitis or gastroenteritis. Consider this documentation strategy for any cancer patient admitted with a non-infectious form of gastroenteritis.

67
Q

Toxic Gastroenteritis Clinical Example - Seek Clarification

A 56 yo female has been admitted with “non-infectious GE” (N/V) after her second round of chemotherapy; she is currently being treated for stage 2 pancreatic cancer. C. diff and other differential diagnoses have been ruled out. Her treatment regimen includes NPO status, antibiotics and IV fluids.

A

Sample Clarification:
* Toxic gastroenteritis d/t chemotherapy requiring treatment with IV antibiotics and IV fluids
* Other; with explanation of the clinical findings
* Unable to determine (no explanation for the clinical findings)

The medical record reflects the following clinical evidence:
* Clinical Indicators: “non-infectious GE”
* Risk Factor: chemotherapy
* Treatment: IV fluids, antibiotics, NPO status

68
Q

Gastroparesis
* Definition
* What is the Most Common Cause of Gastroparesis?
* Risk Factors
* Clinical Indicators
* Treatment

A
  • Gastroparesis, or gastric stasis, is a disorder of delayed gastric emptying in the absence of mechanical obstruction. It occurs when the nerves to the stomach are damaged and don’t work properly.
  • Diabetes is the most common cause.
  • Risk factors: diabetes mellitus, prior gastric surgery with or without vagotomy, a preceding infectious illness, pseudo-obstruction, collagen vascular disorders, and anorexia nervosa
  • The clinical indicators are a set of largely nonspecific symptoms such as early satiety, bloating, nausea, anorexia, vomiting, abdominal pain, and weight loss. Among these, vomiting and postprandial fullness are the most common.
  • Treatment:
    Drugs used to relieve nausea and vomiting in gastroparesis include promotility drugs such as Reglan and erythromycin, anti-nausea medications, and serotonin antagonists (Zofran).
    **Drugs used to relieve abdominal pain in gastroparesis include nonsteroidal anti-inflammatory drugs, low-dose tricyclic antidepressants, and drugs that block nerves that sense pain such as Neurontin.

*Erythromycin is an antibiotic. At doses lower than those used to treat infections, erythromycin stimulates contractions of the muscles of the stomach and small intestine and is useful for treating gastroparesis.

Of note: Evidence suggests that after 10-20 years of clinically apparent diabetes, 30-60% of diabetics develop overt signs of visceral autonomic neuropathy, of which gastroparesis is one form. Diabetic gastroparesis is detected with equal frequency in type 1 and type 2 diabetics. If the clinical evidence suggests a diagnosis of diabetic-related gastroparesis, seek clarification from the physician. Diabetic-related gastroparesis is an MDC 1 diagnosis.

69
Q

An 84 yo female has been admitted with “nausea and vomiting.” Her treatment regimen includes aspiration precautions, bronchodilators, and Tessalon Perles. Her chest x-ray is negative.

The CDS should seek clarification for:
* Aspiration Pneumonia
* Aspiration Bronchitis
* Aspiration Gastroenteritis

A

Answer: Aspiration Bronchitis

69
Q

Clinical Scenario: A 60 yo female has been admitted with “esophagitis”; she is currently undergoing Methotrexate therapy for severe rheumatoid arthritis. Her EGD this AM was positive for white patches in the esophagus, and she has been started on IV fluconazole.

Questions
1) What is this patient’s principal diagnosis?
A. Esophagitis
B. Rheumatoid Arthritis
C. Thrush

2) What is a potential alternative principal diagnosis for her?
A. Rheumatoid Arthritis
B. Strep Throat
C. Esophagitis d/t Candida

3. What is the appropriate root operation for the brush biopsy performed during the EGD?
A. Repair
B. Control
C. Excision
D. Extraction

A

Answers:
1) A. Esophagitis
2) C. Esophagitis d/t Candida
3) D. Extraction

70
Q

Gastroparesis d/t Diabetes

A
  • Diabetes is a common cause of gastroparesis
  • There is an assumed link between DM and gastroparesis; as such, assign the appropriate E10 or E11 code assignment.
  • However, if the condition has not been diagnosed as gastroparesis, you may be required to seek clarification from the physician.
71
Q

Hernia
* Definition
* Root Operation

A
  • Hernia: a protrusion of a part or structure through the tissue that normally contains it
  • During the patient’s hernia repair, the surgeon returns the bowel back to its normal anatomic position. In some instances, the muscle wall will need to be reinforced with mesh to prevent a recurrence of the hernia. When your patient undergoes a herniorrhaphy, one of two actions are being performed—a “REPAIR” or “SUPPLEMENT.” These are the root operations that you’ll use to index a hernia repair, including a procedure for failed mesh graft.

Repair without mesh uses “repair.” If mesh is used, use “supplement”.

Repair: restoring, to the extent possible, a body part to its normal anatomic structure and function
Supplement: putting in or on biological or synthetic material that physically reinforces and/or augments the function of a portion or body part

If the patient undergoes a hernia repair without mesh, use the root operation “repair.” If mesh is used, use “supplement” instead. This detail is available in the body of the operative report.

72
Q

A “Re-Do” Hernia Repair
* What if this ‘redo’ hernia repair is because of a failed mesh graft from a previous hernia repair?

A
  • In some instances, previously placed grafting material may fail, causing a reoccurrence (recurrent) hernia. If a ‘redo’ hernia repair is because of a failed mesh graft from a previous hernia repair, then the hernia is NOT the principal diagnosis, it’s the failed mesh graft, a complication of a medical device.
  • This complication of treatment is not an MDC 6 diagnosis but an MDC 21 complication diagnosis and, as an MDC 21 PDx, more accurately describes the patient’s severity of illness.

Under these circumstances, it is important to have the physician validate in the documentation the failed graft as the underlying cause of the recurrent hernia.

73
Q

Hernia Repair with Additional Surgical Work: Adhesiolysis

A
  • During a hernia repair, an adhesiolysis may be required. Dense significant adhesions requiring tedious dissection adds OR time and additional work to an otherwise routine hernia repair.
  • If the documentation suggests significant adhesiolysis but it is not well documented, seek clarification from the physician regarding the clinical significance of the adhesions.
  • Of note, the adhesiolysis takes precedence in the surgical hierarchy, not the hernia repair.

A patient who is admitted for a hernia repair may require ‘additional surgical work.’ As you review the hernia repair operative note, look for indicators of additional surgical work.

74
Q

Adhesiolysis

A

Your patient may require an adhesiolysis for abdominal adhesions. You will use the root operation RELEASE since the intent of the procedure is to release an anatomical structure from a band of adhesions

  • Release: freeing a body part from an abnormal physical constraint by cutting or by the use of force

*When a release is performed, none of the constrained organ is removed. Appropriate assignment requires the capture of each organ released during the adhesiolysis; laterality may be required.

75
Q

Lysis of Adhesions - Coding Clinic 1Q 2014, p3

Question:A 65-year-old man was admitted and underwent emergent surgery secondary to incarcerated ventral hernia due to adhesions. The surgeon carried out laparoscopic ventral hernia repair with mesh. According to the operative report, at surgery the patient was found to have significant adhesions to the anterior abdominal wall consisting mostly of the greater omentum, which were carefully and slowly taken down exposing the entire defect. The hernia was then repaired with mesh.

In this case, should the adhesiolysis be coded?

Coders should carefully review the entire operative report to determine the clinical significance of the adhesions and the complexity of the lysis of adhesions. Coders should not code adhesions and lysis thereof, based solely on mention of adhesions or lysis in an operative report. As is customary with other surgeries, it is irrelevant whether the adhesions or lysis of adhesions are included in the title of the operation. Determination as to whether the adhesions and the lysis are significant enough to code and report must be made by the surgeon

A

Answer: In this instance, the lysis of adhesions is separately coded (root operation Release), because it was more than simply procedural steps necessary to reach the operative site. According to the ICD-10-PCS Official Coding Guidelines, B3.13, “In the root operation Release, the body part value coded is the body part being freed and not the tissue being manipulated or cut to free the body part.”

Therefore, the body part value for the lysis of adhesions is the greater omentum.

Explanation: Many patients, especially those with a history of inflammatory bowel disease or multiple GI surgeries, are at risk for the development of adhesions, strong bands of connective tissue that develop in response to inflammation. Adhesions can cause pain, obstruction, and incarceration of surrounding tissues and organs. Lysis of adhesions is often considered inherent to procedures involving the abdomen and pelvis and would not require the assignment of an additional procedure code. However, if the adhesions are dense, significant, require additional work, increase the duration of the surgery or impede the progression of the procedure until they are removed, the lysis should be captured. When in doubt, question the surgeon.

76
Q

Hernia Repair with Additional Surgical Work: Bowel Manipulation

A

*During a hernia repair, a bowel manipulation may be required to address a portion of compromised bowel (bowel that might be twisted or kinked).
*An incarcerated or strangulated hernia requiring the surgeon to manipulate the bowel back into position represents additional surgical work as well as increased SOI and ROM.
*Do not confuse bowel manipulation with a standard running of the bowel conducted prior to the closure of the surgical wound.
* Bowel manipulation takes precedence in the surgical hierarchy. The bowel manipulation procedure would be considered the principal procedure—not the hernia repair.

77
Q

Bowel Manipulation
* Definition
* Root Operation

A
  • During a bowel manipulation, the surgeon moves the bowel to a more suitable location. Bowel manipulation is the manual untwisting or un-kinking of the bowel.
  • Use the root operation REPOSITION to index the procedure.

Reposition: moving, to normal or other suitable location

Remember, do not confuse bowel manipulation with the standard running of the bowel which is inherent to all bowel procedures as the surgeon inspects the bowel prior to closing.

78
Q

Sample Operative Note

PRE-OP DIAGNOSIS: Incarcerated ventral hernia.
POST-OP DIAGNOSIS: Incarcerated ventral hernia

PROCEDURE PERFORMED: Incisional hernia repair

DESCRIPTION OF PROCEDURE: “…A skin incision was made along the right costal margin, overlying the palpable bulge and the dissection carried into the subcutaneous tissue. All bleeding points were controlled with Bovie electrocautery. The hernia sac was identified and using sharp dissection was mobilized from the surrounding soft tissue of the abdominal wall, down to the level of the anterior fascia. The hernia appeared to involve the majority of her previous right subcostal incision. There were a couple of areas where it looked like the bowel had twisted on itself. It appeared dusky so we untwisted the entirety of the bowel, we ran the entire bowel, and determined it to be completely viable. The fascial edges were then cleaned circumferentially for a distance of at least 3-4 cm. The hernia sac was opened and any adherent omentum was mobilized sharply and returned back to the abdominal cavity. The midline fascia was palpated superiorly and inferiorly to ensure that there were no other defects. Finding none, a retromuscular extraperitoneal plane was developed including mobilization of the posterior rectus sheath. This dissection extended to at least 4 cm beyond the margins of the fascial opening. The excess hernia sac was excised and the posterior sheath and peritoneum closed with running 2-0 PDS suture…”

A

You’ll note the only procedure that is listed as being performed is an incisional hernia repair. However, as we review the body of the report, we find the following statement:* “There were a couple of areas where it looked like the bowel had twisted on itself. It appeared dusky so we untwisted the entirety of the bowel, we ran the entire bowel, and determined it to be completely viable.”*

We will need to clarify with the physician the bowel manipulation based on the evidence found in the report.
* Hernia repair is considered a minor procedure.
* Manipulation of the bowel is considered a major digestive procedure that will influence DRG assignment.

79
Q

Concept Challenge: Hernia Repair

Scenario: A 33 yo female has been admitted for a “recurrent ventral hernia” repair; her previous repair was 7 years ago. The op report indicates removal of the previous “compromised” grafting material and replacement with a new graft. She is recovering well, and discharge is anticipated within 24 hours.

Yes or No? The CDS should seek clarification of recurrent incisional hernia.

A

Answer: No

Explanation: The correct answer is NO. The CDS should seek clarification of a cause-and-effect relationship between the recurrent hernia and the failed mesh graft. If you need to remediate this strategy, review the hernia repair content.