Cardiovascular Flashcards
What is the heart sound S1? (2)
- Caused by the closure of AV valves
- Marks the end of diastole, the beginning of systole
What is the heart sound S2? (2)
- Caused by closure of the semilunar valves
- Marks the end of systole, beginning of diastole.
What is S2 splitting on inspiration caused by?
Right bundle branch block
What makes S2 louder?
Pulmonary Embolism
What is S3? (2)
- Caused by a rapid of rush of blood into a dilated ventricle
- Associated with heart failure; may occur before crackles.
What is S3 Caused by? (2)
- Pulmonary hypertension and cor pulmonale.
- Mitral, aortic, or tricuspid insufficiency
What are the 5 auscultatory points?
Aortic, pulmonic, erb’s point, tricuspid, mitral (apical)
What is S4?
- Caused by atrial contraction of blood into a noncompliant ventricle
What is S4 associated with? (6)
Myocardial ischemia, infarction, hypertension, ventricular hypertrophy and aortic stenosis
What is pulse pressure?
Systolic- Diastolic = Pulse pressure
Systolic blood pressure is an indirect measurement of what?
Cardiac output and stroke volume
What is narrowing pulse pressure usually seen with?
Severe hypovolemia or severe drop in cardiac output
Diastolic blood pressure is an indirect measurement of what?
Systemic vascular resistance
A widened pulse pressure may indicate what?
Vasodilation, a drop in systemic vascular resistance - sepsis or septic shock
Coronary arteries are perfused during what?
diastole
Causes of valvular heart disease
- CAD/ Ischemia, MI
- Cardiomyopathy
- Bicuspid aortic valve
- Fever/ infection
- Connective tissue diseases
Murmurs of insufficiency occur when the valve is what?
Closed
Murmurs of stenosis occur when the valve is what?
Open
Murmurs of stenosis are acute or chronic?
Chronic
The semilunar (pulmonic, aortic) are what during systole?
OPEN
The AV valves (tricuspid, mitral) valves are what during systole?
CLOSED
The semilunar valves are what during diastole?
CLOSED
The AV valves are what during diastole?
OPEN
Anticoagulant for acute chest pain 
Heparin or lovenox
And I platelet agents for acute chest pain 
- Plavix
- Reopro
- Integrilin
- Aggrastat
Cardioselective beta blocker
Metropolol
Non-cardioselective beta blocker
Propranolol
Contraindications to beta blockers in acute chest pain 
Sildenafil (viagra)
Changes in II, III aVF
Right coronary artery, inferior LV
Changed in V1, V2, V3, V4
Left anterior descending, anterior LV
Changed in V5,V6, I, aVL
Circumflex, Lateral LV
Changes in V5,V6
Low lateral LV
Changes in I, aVL
High lateral LV
Changed in VI, V2
Right coronary artery, posterior LV
Changes in V3R, V4R
Right coronary artery, right ventricular infarct
Inferior MI- ST elevation
II,III, aVF
Inferior wall- reciprocal changes
Lateral wall (I, aVL)
What is an inferior MI associated with
AV conduction disturbances ( 2° block, 3° block, sick sinus syndrome and sinus Brady)
What can an Inferior MI develop into
Systolic murmur, mitral valve regurg
Right ventricular infarction
Right coronary artery
Signs and symptoms of right ventricular infract
JVD, high cvp, hypotension, clear lungs, bradyarrythmias
Right ventricular infarct ST ST elevation
V3R, V4R
Avoid in right ventricular infarct 
Preload reducers (nitrates or diuretics) beta blockers
ST elevations/ depressions in anterior MI
V1-V4 / inferior wall (I,II,aVF)
Anterior my my develop
2° type II block, or RVBB
Lateral MI ST elevation
V5, V6 (low lateral) I, aVL (high lateral)
Signs of retroperitoneal bleed
Sudden, hypotension, severe low back pain
Evidence of reperfusion
Chest pain, relief, resolution of ST segment, deviations, marked elevation of troponin, reperfusion arrhythmias 
Signs of coronary artery re-occlusion
Chest pain, ST elevation
Hypertensive, emergency or crisis 
Elevated blood pressure where evidence of an organ damage that can be related to acute hypertension 
Hypertension urgency 
Elevated blood pressure without evidence of acute and organ damage 
Nitroprusside in HTN
Preload and after load reducer, assess for cyanide toxicity
Signs and symptoms of peripheral arterial disease
Pain, pallor, pulse absent, parenthesia, paralysis, poikilothermia
Normal ankle- brachial index
> 0.90
WPW syndrome
Genetic conduction abnormality, in which abnormal conduction pathway exist, that allows re-and Trent tachycardia pathway to bypass a normal AV node conduction pathway, resulting in superventricular tachycardia, 
EKG changes with WPW
Short PR interval, presence of delta wave
Do not give what medications with WPW
Adenine, digoxin, or calcium channel blockers- enhance conduction through abnormal pathway by increasing refractory. And AV node resulting in ventricular fibrillation.