Cardiovascular Flashcards

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1
Q

What is Apical pulse used to determine?

A

size of the heart

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2
Q

find areas of ischemia in the heart muscle (do not show blockage). >70% blockage shows ischemia

A

Nuclear studies

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3
Q

dye in arteries, shows blockages

A

Heart catheterization

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4
Q

substance produced by liver in response to inflammation. High= general marker of inflammation (including infections and rheumatoid; not specific for CV disease) >10mg/L is high. <10 normal → increase risk, control RF

A

CRP

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5
Q

amount of calcium in the arteries; useful if pt his high risk but has no symptoms

A

Coronary artery calcium score (CAC)

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6
Q

How to do in office Ankle-Brachial Index (ABI)

A

(divide highest BP at ankle by highest BP in either arm given as a ratio). Decrease of ABI with exercise is a significant indicator of PAD. Normal is 1- 1.1 (ankle about the same as arm). The lower the ABI the worse the PAD
<0.95 indicates possible PAD

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7
Q

What does an ABI of <0.95 indicate?

A

possible PAD

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8
Q

How to confirm Unstable angina/ NSTEMI?

A

cardiac enzymes

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9
Q

meds to manage Past STEMI?

A

Management= Antiplatelet (aspirin or plavix), beta blocker (olol drugs), statin, ACE or ARB, nitro

Past STEMI= Q-wave

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10
Q

do not show blockages. Can have up to 70% blocked before _____ study is affected. Shows blood flow only.

A

Nuclear studies

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11
Q

Heaviness, tightness, burning, crushing, squeezing, discomfort often in the chest area (also in back, jaw, shoulder, neck, epigastric). Usually resolves with rest.
Women present with atypical symptoms (don’t feel right, lightheaded, SOB, fatigability)

NOT ____: sharp, jabbing
Usual age: 50s-70s

A

Angina

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12
Q

how to tx angina?

A

nitroglycerin (vasodilates, decreases afterload) beta blockers (decrease contractility and HR= decreased demand) apply oxygen

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13
Q

Predictable; occurs with exertion. Resolves with rest and/or SL Nitro

A

Stable angina

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14
Q

new onset, worsening, or unpredictable. Occurs at rest, unrelieved with rest and SL NTG.
This is a form of ACS, could lead to MI → ED

A

unstable angina

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15
Q

d/t vasospasm. Occurs spontaneously at rest, often at night. F>M; smokers
May have ST depression; clean coronaries on heart cath
Tx: CCBs (relax walls of arteries) and Nitrates

A

Prinzmetal/Vasospastic Angina

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16
Q

d/t microvascular dysfunction or spasm. No epicardial obstruction. Occurs with exertion and rest. Diagnosed with PET or CMR

A

Microvascular Angina

17
Q

narrowing of the arterial lumen that can lead to cardiac ischemia through thrombus formation, coronary vasospasm, endothelial cell dysfunction. 70% blockage can cause ischemia

A

Coronary Artery Disease/ASCVD

18
Q

gold standard diagnostic for Coronary Artery Disease/ASCVD

A

coronary angiography (heart cath) GOLD STANDARD;

stress test (less effective, less invasive), CRP (high CRP → increased inflammation) → not CVD indicator, coronary artery calcium

19
Q

What to do if CAD suspected ?

A

refer to cardiology!

20
Q

diagnosed with Cardiac enzymes

No EKG changes (same with unstable angina)

A

NSTEMI

21
Q

(most specific) detectable within hours, peak at 10-24 hours, can be detected for 10-14 days. Usually not ordered outpatient

A

Troponin

22
Q

claudication (heaviness, tightness, tired, sore, necrotic toe). Pain worse with activity, relieved by rest and if leg is in dependent position, which maximizes blood flow. Pain worse with elevation

A

Peripheral arterial disease

23
Q

Findings: diminished pulses, hair loss, pallor, rubor, gangrene, ulcers
dx: In office ABI/ Arterial ultrasound

A

Peripheral arterial disease

24
Q

Tx for PAD?

A

depends on severity of sx. Lifestyle modifications. Want them to walk to the point of pain daily.
Rx: antiplatelets (low dose ASA), statins
Angioplasty or bypass surgery if severe.

25
Q
increased pressure (d/t obesity, pregnancy, standing all day) damages valves and disrupts one way flow back to heart
PE: swelling, worse at end of the day (dependent edema); chronic skin changes (ruddy discoloration), potentially ulcerations. Discomfort worse when standing, relieved with elevation. Warm extremities with normal pulses
  • clinical diagnosis
    Tx: compression stockings and periodic leg elevation
A

Peripheral Venous insufficiency

26
Q

pathologic distension and proliferation of superficial veins

A

varicose veins

27
Q

familial (cannot have a hx of DVT for this classification) usually occur during pregnancy

A

primary varicose veins

28
Q

result from previous DVT. Incompetent valves after recannulation therapy.

A

Secondary varicose veins

29
Q

result from increased pressure in superficial veins

A

Telangiectasia

30
Q

how to dx varicose veins?

A

clinical, based on inspection. See veins fill while pt standing. Venous duplex to r/o DVT.

31
Q

(usu affects 2-5th costochondral junction)

Sharp pain, worse with movement or deep breathing
Recent physical exercise, illness with cough
Pain often one-sided; palpation elicits tenderness
Tx: heat, ice, NSAIDs

A

costochondritis

32
Q

involves single cartilage (usu. 2nd rib)
Sharp pain, worse with movement or deep breathing
Recent physical exercise, illness with cough
Pain often one-sided; palpation elicits tenderness
Tx: heat, ice, NSAIDs

A

Tietze syndrome

33
Q

Heartburn, cough, sore throat, atypical chest pain

Tx: PPI (omeprazole, pantoprazole)ss

A

GERD

34
Q

Acute onset. Sharp burning pain with inspiration
Increases with inspiration or reclining
lessens when leaning forward
PE: pericardial friction rub. EKG diffuse ST elevation and PR depression
Cause: virus (RSV, coxsackie, flu, mumps, adenovirus, CMV, EBV)
DDx: PE, Pna

A

Pleuritic CP