Cardiology Flashcards

1
Q

What is the origin of the normal cardiac electrical impulse and describe the pathway of impulse transmission (AKA conduction) through the heart.

A
  • Generated in the SA node
  • Conducted rapidly through atria (atrial systole) > conducted through AV node slowly > rapidly conducted through the bundle of HIS > then right and left bundle branches comprised of Purkinje fibers > depolarize myocytes > ventricular systole > ventricular repolarization
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2
Q

What limb locations and charges do the three standard leads represent?

A
  • White on the front right limb
  • Black on the front left limb
  • Red on the left hind limb

Lead I = RF- to LF+
Lead II = RF- to LH+
Lead III = LF- to LH+

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3
Q

How do you calculate heart rate based on two in-class methods?

A
  • Find paper speed, count QRS complexes in 30 large boxes
  • For 25 mm/sec multiply by 10
  • For 50 mm/sec multiply by 20

OR

  • As long as the rhythm is regular, count the number of small boxes in 1 RR interval
  • For 25 mm/sec divide 1500 by # of small boxes
  • For 50 mm/sec divide 3000 by # of small boxes
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4
Q

What does each component of the PQRST represent? Know how to measure them on an ECG.

A
  • P-wave = atrial depolarization
  • QRS = ventricular depolarization
  • T = ventricular reploarization
  • PR interval = atrial depolarization and AV conduction
  • QT interval = ventricular depolarization and repolarization
  • RR interval = time between ventricular depolariazations
  • ST segment = time between end of ventricular depolarizarion and the begining of repolarization
  • PR segment = time between atrial depolarization and begining of ventricular depolarization
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5
Q

What is the normal duration of a canine and feline QRS complex?

A

Canines: less than 60ms
Felines: less than 40ms

*narrow is normal, anything higher than that is considered wide

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6
Q

What are the six steps in ECG interpretation?

A
  1. General assessment
  2. Calculate HR
  3. Measure RR intervals (regular vs irregular)
  4. Examine each PQRST
  5. Identify ectopic complexes
  6. Identify pauses
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7
Q

What are the ECG criteria for a sinus arrhythmia?

A

Greater than 10% variation in RR intervals (regularly irregular)

  • HR accelerates during inspiration
  • HR decelerates during expiration
  • Normal HR is often low normal

*Be able to identify a sinus arrhythmia

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8
Q

Which species is a sinus arrhythmia normal and common in?

A

Respiratory sinus arrhythmia common in resting dogs, but abnormal in awake cats

*Exaggerated in some patients with certain respiratory diseases

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9
Q

What are the three mechanisms of arrhythmia formation? And what are the names of the arrhythmias that belong to each mechanism?

A
  1. Disturbances of impulse formation (excitability)
    - Sinus (SB, ST)
    - Supraventricular (SVPCs, SVT, A fib, A flutter)
    - Ventricular (VPCs, VT, V fib, AIVR)
  2. Disorders of impulse transmission (conduction)
    - Atrioventricular block (first degree, second degree, third degree)
    - BBBs
    - Atrial standstill
  3. Complex (disorders involving both impulse formation and conduction)
    - SSS
    - Electrolyte derangements
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10
Q

What is the breed disposition for A-fib in canines?

A

Giant and large breed dogs

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11
Q

What is the breed disposition for sick sinus syndrome in canines?

A

Miniature schnauzers
West highland white terrier
Dachshunds
Cocker spaniels

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12
Q

What are the three differentials for a wide QRS complex?

A
  1. VPCs
  2. BBBs
  3. Electrolyte derrangements
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13
Q

What types of electrolyte derangements cause arrhythmias?

A
  • Hypokalemia causes VPCs and APCs
  • Hyperkalemia causes prolongation of normal waves and in severe cases V-fib and pulseless electrical activity
  • Hypomagnesemia causes VPCs, VT, AIVR
  • Hypocalcemia causes lower AP threshold and prolongs QT interval
  • Hypercalcemia raiser AP threshold causing bradycardia, shortened QT interval and VPCs
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14
Q

What are the ECG abnormalities that are associated with hyperkalemia and hypokalemia?

A
  • Hypokalemia causes VPCs and APCs
    (prolongs repolarization)
  • Hyperkalemia causes prolongation of normal waves and in severe cases V-fib and pulseless electrical activity and electromechanical dissociation
    (in mild, shortens repolarization, tented T waves)
    (more moderate, prolongs transmission velocity, wide QRS complexes)
    (severe, prolongs PR interval or absent P waves seen)
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15
Q

What factors do hemodynamic effects depend on?

A
  • Ventricular rate (if HR is too high ventricles don’t have time to fill so SV decreases if HR is too low ventricles are not ejecting enough blood)
  • Duration of the abnormal rhythm
  • Inherant myocardial and valvular function
  • Temporal relationship between atria and ventricles
  • Extra-cardiac influences
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