Cardiology Flashcards
Where is the most common location of the ectopic foci that cause Atrial Fibrillation?
Pulmonary Veins
An accessory atrioventricular bypass tract is seen in what cardiac syndrome?
Wolff-Parkinson-White Syndrome
Delta waves are seen on EKG.
What is the most common cause of atrial flutter?
A re-entrant circuit around the tricuspid annulus.
Interventricular Septum Rupture
Seen 3-5 days post-MI involving the LAD.
Presents with acute cardiogenic shock, JVD, and hepatomegaly.
Harsh, holocystolic murmur with palpable thrill at the LSB.
Diagnosis: pulmonary artery catheterization with step up in oxygenation from the RA to RV or via TTE
Papillary Muscle Rupture
3-5 days post-MI of the RCA.
Presents with severe, acute mitral regurgitation, sudden-onset hypotension, dyspnea, and pulmonary edema.
Soft, holocystolic murmur without palpable thrill.
Tennessee
S4 heart sound
Indicates stiff LV and LV hypertrophy from long-standing hypertension or restrictive cardiomyopathy.
In an adenosine or dypiramidole stress-test that is positive for ischemia, what is the mechanism responsible for the changes seen.
Augmentation of blood flow in non-obstructed vessels. This is because adenosine and dypiramidole both cause dilation of the vessels that is most prominent in non-obstructed vessels.
What is the mechanism responsible for the changes seen in a dobutamine stress test?
Increase myocardial oxygen demand.
Dobutamine increases HR and myocardial contractility.
Kentucky
S3 heart sound
Heard in left-sided heart failure.
May be a normal finding in young adults and well-trained athletes without other symptoms.
Complete AV dissociation
Complete heart block
For A-Fib with RVR in a patient with WPW, what drug do you use to convert stable patients?
Procainamide (Ibutilide as well)
Electro-cardioversion for unstable patients
Acute Mitral Regurgitation
Can occur due to papillary muscle displacement secondary to acute MI.
Presents with acute pulmonary edema.
Increase left atrial and ventricular filling pressures.
Symptomatic Sinus Bradycardia
Initial - IV atropine
No response with atropine - IV epi or dopamine
No response with epi or dopamine - trancutaneous pacing
Screening for AAA
Men aged 65-75 who have smoked should receive a one-time screening ultrasound.
ST segment elevations in II, III, and aVF
Inferior wall MI
Reciprocal changes in I and aVL
Involves the RCA or the Left Circumflex (LCX), most commonly the RCA
ST segment depressions in V1 and V2
Posterior wall MI
RCA if depressions in I and aVL
LCX if elevations in I and aVL
ST segment depression in V1 and V2 and elevations in II, III, aVF
Occlusion of the right coronary artery (RCA)
Anterior MI
Occlusion of the LAD
Changes in some or all of V1 - V6
Lateral MI
Involvement of the LCX or diagonal
ST elevations in I, aVL, V5, and V6
ST depression in II, III, and aVF
Right Ventricle MI
Occurs in 1/2 of all inferior MI
Involves the RCA
ST elevations in V4-V6R
Occlusion of the Left Main Coronary Artery
Usually causes sudden death as this supplies the LAD and LCX
Elevations in anterior and lateral leads: I, aVL, V1-V6
Vasospastic Angina Treatment
CCBs for prevention
Sublingual Nitroglycerin for abortive therapy
Hyponatremia in Heart Failure
Poor prognostic factor
New conduction abnormalities in a patient with infective endocarditis:
Perivalvular abscess
Tricuspid Regurgitation
Holosystolic murmur
Left Ventricular Aneurysm
Etiology: scar tissue following transmural MI
Presentation: several months post-MI, heart failure and angina, Ventricular arrhythmia (V-tach), systemic embolization (stroke)
EKG: persistent ST elevation, deep Q waves
Echo: thin, dyskinetic myocardial wall - impaired EF
Cardiac Index =
Cardiac Output / Body Surface Area
Reduced in HF
Costochondritis
Chest wall tenderness
Most accurate test is physical exam
Pericarditis
Pain worse with lying flat and better sitting up in a young patient <40 years old
Most accurate test is EKG with ST elevations in all leads and PR depression
Aortic Dissection
Radiation to the back, unequal blood pressure between arms
Most accurate test is a CXR with widened mediastinum, chest CT, MRI, or TEE to confirm diagnosis
Duodenal Ulcer Disease
Epigastric discomfort, pain relieved with eating
Most accurate test is endoscopy
GERD
Bad taste in mouth, cough, hoarseness
Most accurate test is response to PPIs, aluminum hydroxide, magnesium hydroxide, viscous lidocaine
Pneumonia
Cough, sputum, hemoptysis, chest pain
Most accurate test is CXR
Pulmonary Embolism
Sudden-onset SOB, tachycardia, hypoxia, chest pain
Most accurate test is spiral CT, V/Q scan
Pneumothorax
Sharp, pleuritic chest pain, tracheal deviation
Most accurate test is CXR
Exercise Tolerance Test
Used to determine the presence of ischemia
Shows ST segment depression if ischemia is detected
Exercise thallium
Used if there is an inability to read the EKG or there are baseline ST segment abnormalities
Shows decreased uptake of nuclear isotope if ischemia is detected
Exercise Echo
Used if there is an inability to read the EKG or there are baseline ST segment abnormalities
Shows wall motion abnormalities if there is ischemia
Dipyridamole Thallium Test
Used if there is an inability to exercise to target heart rate
Showed decreased uptake of nuclear isotope if there is ischemia
Dobutamine Echo Test
Used if there is an inability to exercise to target heart rate
Shows wall motion abnormalities if there is ischemia
What is the time to becoming abnormal in the setting of an MI for an EKG? What is the duration of the abnormality?
Immediately at the onset of pain
Will show ST elevations that progress to Q waves over several days to a week
What is the time to becoming abnormal in the setting of an MI for Myoglobin? What is the duration of the abnormality?
1-4 hours
Resolves in 1-2 days
What is the time to becoming abnormal in the setting of an MI for CK-MB? What is the duration of the abnormality?
4-6 hours
Resolves in 1-2 days
What is the time to becoming abnormal in the setting of an MI for Troponin? What is the duration of the abnormality?
4-6 hours
Resolves in 10-14 days
Treatment indications in the setting of MI: ASPIRIN
Everyone, the best initial therapy
Treatment indications in the setting of MI:
CLOPIDOGREL or PRASUGREL or TICAGRELOR
Those undergoing angioplasty or stenting, second antiplatelet drug with aspirin
Treatment indications in the setting of MI:
BETA BLOCKERS
Everyone, effect is not dependent on time so can be started at any time during the admission
Treatment indications in the setting of MI:
ACEI/ARB
Everyone, benefit best with an EF <40%
Treatment indications in the setting of MI:
STATINS
Everyone, goal LDL <70 mg/dL
Treatment indications in the setting of MI:
NITRATES
Everyone, no clear mortality benefit
Treatment indications in the setting of MI:
HEPARIN
After thrombolytics/PCI to prevent restenosis, initial therapy with ST depression and other non-ST elevation events like unstable angina
Treatment indications in the setting of MI:
CCB’s
If a patient can’t use B-blockers, in cocaine-induced pain, in Prinzmetal or vasospastic variant angina
What are the absolute contraindications to thrombolytics?
Major bleeding (bowel or brain) Recent surgery (<2 weeks) Severe HTN (>180/110) Nonhemorrhagic stroke (<6 months)
What medications should every patient be started on after an MI?
Aspirin
Beta Blocker
Statin
ACEI/ARB
What is the most likely diagnosis in the setting of post-MI?
Bradycardia, cannon A waves
Third-degree AV block
What is the most likely diagnosis in the setting of post-MI?
Bradycardia without cannon A waves
Sinus Bradycardia
What is the most likely diagnosis in the setting of post-MI?
Sudden loss of pulse, jugulovenous distention
Tamponade/Wall Rupture
What is the most likely diagnosis in the setting of post-MI?
IWMI in history, clear lungs, tachycardia, hypotension with nitroglycerin
RV infarction
What is the most likely diagnosis in the setting of post-MI?
New murmur, rales/congestion
Valve rupture
What is the most likely diagnosis in the setting of post-MI?
New murmur, increase is oxygen saturation on entering the right ventricle (step-up)
Septal Rupture
What is the most likely diagnosis in the setting of post-MI?
Loss of pulse, need EKG in order to answer this question
Ventricular fibrillation
What is the most likely diagnosis in the setting of Dyspnea?
Sudden onset dyspnea with clear lungs on exam
PE
What is the most likely diagnosis in the setting of Dyspnea?
Sudden onset, wheezing, increased expiratory phase
Asthma
What is the most likely diagnosis in the setting of Dyspnea?
Slow onset dyspnea, fever, sputum, unilateral rales/rhonchi
Pneumonia
What is the most likely diagnosis in the setting of Dyspnea?
Decreased breath sounds unilaterally, tracheal deviation
PTX
What is the most likely diagnosis in the setting of Dyspnea?
Circumoral numbness, caffeine use, hx of anxiety
Panic attack
What is the most likely diagnosis in the setting of Dyspnea?
Pallor, gradual over days to weeks
Anemia
What is the most likely diagnosis in the setting of Dyspnea?
Pulsus paradoxus, decreased heart sounds, JVD
Tamponade
What is the most likely diagnosis in the setting of Dyspnea?
Palpitations, syncope
Arrhythmia of any kind
What is the most likely diagnosis in the setting of Dyspnea?
Dullness to percussion at the bases
Pleural effusion
What is the most likely diagnosis in the setting of Dyspnea?
Long smoking history, barrel chest
COPD
What is the most likely diagnosis in the setting of Dyspnea?
Recent anesthetic use, brown blood, not improved with oxygen, clear lungs, cyanosis
Methemoglobinemia
What is the most likely diagnosis in the setting of Dyspnea?
In a burning building or car, wood-burning stove in the winter, suicide attempt
Carbon monoxide poisoning
Effects of Maneuvers on:
MS and AS
Squatting = increase Standing/Valsalva = decrease
Effects of Maneuvers on:
MR and AR
Squatting = increase Standing/Valsalva = decrease
Effects of Maneuvers on:
MVP
Squatting = decrease Standing/Valsalva = increase
Effects of Maneuvers on:
HOCM
Squatting = decrease Standing/Valsalva = increase